Search results for "Alternative complement pathway"

showing 10 items of 35 documents

Inflammatory Response of the Ascidian Ciona intestinalis

2016

Abstract The Ciona intestinalis inflammatory response to several irritants appears to be composed of a complex reaction set. The cellular reactions in the tunic involve hemocyte infiltration, hemocyte and epidermis activities, vacuolization, and cell disruption, while cell products can contribute to form capsule components and/or cause a tunic wound. In this response, the involvement of the pharynx, as the main immune-competent organ, has been disclosed by a lipopolysaccharide challenge that upregulates innate immunity genes and transcription activation genes. The pharynx responds through hemocyte recruitment into the pharynx vessels, enhancement of galectin-like lectins in the serum hemoly…

InflammationProphenoloxidaseInnate immune systemAscidianCiona intestinaliSettore BIO/05 - ZoologiaMedicine (miscellaneous)HemocyteLipopolysaccharideInflammationProphenoloxidaseBiologybiology.organism_classificationCell biologyImmune systemImmunologymedicineAlternative complement pathwayPharynxCiona intestinalismedicine.symptomCytokineLectinGeneGalectin
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The lipopolysaccharide O side chain of Vibrio vulnificus serogroup E is a virulence determinant for eels

1997

Vibrio vulnificus is a gram-negative bacterium capable of producing septicemic infections in eels and immunocompromised humans. Two biotypes are classically recognized, with the virulence for eels being specific to strains belonging to biotype 2, which constitutes a homogeneous lipopolysaccharide (LPS)-based O serogroup (which we have designated serogroup E). In the present study we demonstrated that the O side chain of this LPS determines the selective virulence of biotype 2 for eels: (i) biotype 1 strains (which do not belong to serogroup E) are destroyed by the bactericidal action of nonimmune eel serum (NIS) through activation of the alternative pathway of complement, (ii) biotype 2 str…

Lipopolysaccharidesendocrine systemanimal structuresLipopolysaccharideComplement Pathway AlternativeImmunologyMutantVirulenceVibrio vulnificusBiologyMicrobiologyMicrobiologychemistry.chemical_compoundPhagocytosisVibrionaceaeAnimalsVibrioEelsVirulenceO Antigensbiology.organism_classificationVirologyVibrioInfectious DiseaseschemistryAlternative complement pathwayImmunizationParasitologyBacteriaResearch Article
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Involvement of complement pathways in patients with bacterial septicemia.

2007

The complement system is a major humoral portion of the innate immune system, playing a significant role in host defence against microorganisms. The biological importance of this system is underlined by the fact that at least three different pathways for its activation exist, the classical, the MBL and the alternative pathway. To elucidate the involvement of the classical and/or the MBL pathway during bacterial septicemia, 32 patients with gram-positive and 30 patients with gram-negative bacterial infections were investigated. In patients with gram-positive bacteria, a significant consumption of C1q (p=0.005) but not of mannose-binding lectin (MBL) (p=0.2) was found during the acute phase o…

MESH: Complement Pathway Mannose-Binding LectinLipopolysaccharidesSalmonellaMESH: Complement C1qLipopolysaccharideImmunologychemical and pharmacologic phenomenaBacteremiamedicine.disease_causeGram-Positive BacteriaMannose-Binding LectinMicrobiologyMESH: Gram-Positive Bacteria03 medical and health scienceschemistry.chemical_compoundClassical complement pathway0302 clinical medicinemedicineHumans[SDV.BBM]Life Sciences [q-bio]/Biochemistry Molecular BiologyComplement Pathway ClassicalMESH: BacteremiaMolecular Biology030304 developmental biology0303 health sciencesInnate immune systemMESH: HumansbiologyComplement C1qLectinSalmonella entericaComplement Pathway Mannose-Binding LectinMESH: Complement Pathway Classicalbiology.organism_classificationbacterial infections and mycoses3. Good healthComplement systemMESH: Mannose-Binding LectinchemistryMESH: Salmonella entericaImmunologyAlternative complement pathwaybiology.proteinMESH: LipopolysaccharidesBacteria030215 immunologyMolecular immunology
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Effect of in vivo stimulation of mice on the secretion of factor B of the alternative complement pathway by peritoneal macrophages

1977

After in vivo treatment of mice with thioglycollate medium, the amount of native factor B which could be detected in vitro in culture supernatants of peritoneal macrophages was much lower than that found in supernatants of macrophages taken from untreated mice. However, when the macrophages from thioglycollate medium-treated mice were cultured on a plastic surface covered with glutardialdehyde-linked bovine serum albumin, the culture supernatants contained larger quantities of native factor B than culture supernatants of macrophages from untreated mice under the same conditions. Thus, the effect of in vivo thioglycollate medium treatment on the in vitro secretion of factor B by peritoneal m…

MacrophagesGuinea PigsImmunologyCell CountSerum Albumin BovineStimulationComplement System ProteinsBiologyComplement factor BIn vitroMicrobiologyMiceGlutaralIn vivobiology.proteinAlternative complement pathwayAnimalsImmunology and AllergySecretionFactor DBovine serum albuminPlasticsCells CulturedEuropean Journal of Immunology
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Two‐dimensional analysis of myocardial protein expression following myocardial ischemia and reperfusion in rabbits

2002

Myocardial ischemia and reperfusion injury (MI/R) can be related to leukocyte activation with subsequent release of cytokines and oxygen derived free radicals. Activation of the complement system has been implicated in the pathogenesis of myocardial ischemia and reperfusion injury. Inflammatory injury will subsequently result in cellular activation and protein synthesis. In the present study we analyzed the myocardial protein expression and its pattern following myocardial ischemia and reperfusion, with and without complement inhibition with the synthetic serine protease inhibitor Futhan/nafamstat mesilate (FUT-175) known to inhibit classical and alternative complement pathway in a rabbit m…

MaleSerine Proteinase InhibitorsNecrosisProteomeNeutrophilsMyocardial IschemiaIschemiaMyocardial Reperfusion InjuryPharmacologyGuanidinesBiochemistrySuperoxide dismutaseNecrosisRandom AllocationComplement inhibitormedicineAnimalsElectrophoresis Gel Two-DimensionalCreatine KinaseMolecular BiologybiologySuperoxide DismutaseChemistryGene Expression ProfilingMyocardiumHemodynamicsProteinsalpha-Crystallin B Chainmedicine.diseaseBenzamidinesComplement systemBiochemistrybiology.proteinAlternative complement pathwayCreatine kinaseRabbitsmedicine.symptomReperfusion injuryPROTEOMICS
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Prognostic Value of Complement Properdin in Cancer

2021

© 2021 Mangogna, Varghese, Agostinis, Alrokayan, Khan, Stover, Belmonte, Martorana, Ricci, Bulla and Kishore. The complement system is readily triggered by the presence of damage-associated molecular patterns on the surface of tumour cells. The complement alternative pathway provides rapid amplification of the molecular stress signal, leading to complement cascade actvation to deal with pathogens or malignant cells. Properdin is the only known positive regulator of the alternative pathway. In addition, properdin promotes the phagocytic uptake of apoptotic T cells by macrophages and dendritic cells without activating the complement system, thus, establishing its ability to recognize “altered…

Malelcsh:Immunologic diseases. Allergybioinformatics analysisNeutrophilsComplement Pathway AlternativeImmunologyDatasets as TopicInflammationchemical and pharmacologic phenomenaBiologyLymphocytes Tumor-InfiltratingImmune systemNeoplasmsTumor Microenvironmentmedicinebioinformatics; cancer; complement; innate immunity; prognosis; properdinData MiningHumansImmunology and AllergyCytotoxic T cellcancercomplementRNA MessengerRNA Neoplasminnate immunityOriginal ResearchTumor microenvironmentInnate immune systembioinformaticMacrophagesDendritic CellsbioinformaticsLymphocyte SubsetsComplement systemproperdinAlternative complement pathwayCancer researchProperdinFemaleprognosismedicine.symptomTranscriptomelcsh:RC581-607prognostic markerprognosi
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Biosynthesis and transformation of 20α 21-dihydroxycholesterol by rat adrenal preparations

1979

Abstract The biosynthesis of [ 3 H]-20α, 21 dihydroxycholestderol from [ 3 H]-20α-hydroxycholesterol and its transformation to [ 3 H]-21-hydroxypregnenolone by rat adrenal preparations has been demonstrated. 20α-Hydroxycholesterol was transformed to 20α, 21-dihydroxycholesterol by microsomal preparations in the presence of NADPH and 20α-21-dihydroxycholesterol was metabolized to 21-hydroxypregnenolone by mitochondrial preparations in the presence of a NADPH-generating-system. Comparison of the Michaelis-Menten-Kinetics of the steps “20α, 21-dihydroxycholesterol → 21-hydroxycholesterol” and “20α-hydroxycholesterol → pregnenolone” revealed that both compounds behaved as analogue substrates of…

Malemedicine.drug_classStereochemistryBiology17-alpha-HydroxypregnenoloneBiochemistrychemistry.chemical_compoundEndocrinologyBiosynthesisMicrosomesAdrenal Glandspolycyclic compoundsmedicineAnimalsCholesterol Side-Chain Cleavage EnzymeCholesterol side-chain cleavage enzymeHydroxycholesterolsMitochondriaRatsTransformation (genetics)BiochemistrychemistryAlternative complement pathwayPregnenoloneMicrosomeCorticosteroidlipids (amino acids peptides and proteins)NADPmedicine.drugJournal of Steroid Biochemistry
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Activation of the lectin pathway in murine lupus nephritis.

2004

In systemic lupus erythematosus (SLE), hypocomplementaemia and complement deposition have been described both in man and in experimental models. A major involvement of the classical pathway of complement activation has been demonstrated in this disease, however relatively little is known about the involvement of the lectin pathway. Therefore in the present study we have analyzed the activity of all three pathways of complement activation in murine models of SLE. In the mouse, MBL is expressed in two forms, namely MBL-A and MBL-C. In the present study young and old MRL-lpr and control MRL+/+ mice were compared for the levels of complement activity with specific attention for the lectin pathw…

Mice Inbred MRL lprImmunologychemical and pharmacologic phenomenaurologic and male genital diseasesmedicine.disease_causeAutoimmunityClassical complement pathwayMiceImmune systemimmune system diseasesMurine lupusLectinsmedicineAnimalsskin and connective tissue diseasesMolecular BiologyAutoantibodiesChemistrybacterial infections and mycosesmedicine.diseaseLupus NephritisComplement systemLectin pathwayImmunologyAlternative complement pathwayNephritisMolecular immunology
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Immunological and pathological status of gilthead sea bream (Sparus aurata L.) under different long-term feeding regimes

2003

The possible influence of the feeding regime (FR) on the immune system and pathological status of gilthead sea bream was studied. Two growth trials were performed starting at different seasons (trial 1 = March; trial 2 = June) under controlled experimental conditions. In both trials, FR-1 groups received a restricted amount of food, whereas FR-2 groups were fed to visual satiety. The pathology study included parasitological and bacteriological examination, and the immunological traits analysed were respiratory burst activity of head kidney leucocytes, serum lysozyme and alternative pathway complement activity (ACH50). The immunological status of gilthead sea bream not only was not impaired …

Respiratory burstBacteriaSparidaebiologyEcologyVibrio harveyiFish farmingLysozymeComplementPhysiologyParasitismAquatic Sciencebiology.organism_classificationRespiratory burstchemistry.chemical_compoundImmune systemchemistryGilthead sea breamAlternative complement pathwayParasitesLysozymeRation sizeAquaculture
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Activation of complement by the alternative pathway as a factor in the pathogenesis of periodontal disease.

1976

Dental plaque and a bacterium, Actinomyces viscosus, isolated from plaque that can reproduce periodontal disease in germ-free rats, are activators of complement by the alternative pathway. It is suggested that this process is involved in the pathogenesis of chronic inflammatory periodontal disease.

T-LymphocytesGuinea PigsDental PlaqueAntigen-Antibody ComplexDental plaquePathogenesisstomatognathic systemPeriodontal diseasemedicineActinomycesAnimalsHumansActinomyces viscosusBone ResorptionPeriodontitisGlycoproteinsB-LymphocytesEnzyme Precursorsbusiness.industryMacrophagesGlobulinsGeneral MedicineComplement C3Complement System Proteinsmedicine.diseaseCathepsinsComplement (complexity)RatsEndotoxinsstomatognathic diseasesMicrobial CollagenaseImmunologyAlternative complement pathwaybusinessLancet (London, England)
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