Search results for "Amy"

showing 10 items of 1486 documents

Muscle follistatin gene delivery increases muscle protein synthesis independent of periodical physical inactivity and fasting

2020

Blocking of myostatin and activins effectively counteracts muscle atrophy. However, the potential interaction with physical inactivity and fasting in the regulation of muscle protein synthesis is poorly understood. We used blockade of myostatin and activins by recombinant adeno-associated virus (rAAV)-mediated follistatin (FS288) overexpression in mouse tibialis anterior muscle. To investigate the effects on muscle protein synthesis, muscles were collected 7 days after rAAV-injection in the nighttime or in the daytime representing high and low levels of activity and feeding, respectively, or after overnight fasting, refeeding, or ad libitum feeding. Muscle protein synthesis was increased by…

Male0301 basic medicineFollistatinMuscle Proteinsphysical activitylihaksetMyostatinBiochemistryMice0302 clinical medicineTibialis anterior musclemedia_common2. Zero hungerbiologyChemistryactivinsFastingDependovirusMuscle atrophyCircadian RhythmMuscular Atrophymyostatinmedicine.symptomfyysinen aktiivisuusBiotechnologymedicine.medical_specialtyfastingmedia_common.quotation_subjectMechanistic Target of Rapamycin Complex 1Gene delivery03 medical and health sciencesPhysical Conditioning AnimalInternal medicineGeneticsmedicineAnimalsMolecular BiologypaastoPI3K/AKT/mTOR pathwaysolufysiologiaSarcolemmaJNK Mitogen-Activated Protein Kinasesmechanistic target of rapamycin proteinAppetiteGenetic TherapyMice Inbred C57BL030104 developmental biologyEndocrinologybiology.protein1182 Biochemistry cell and molecular biology3111 BiomedicineproteiinitEnergy Metabolismlihassurkastumasairaudet030217 neurology & neurosurgeryFollistatin
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Exercise and probiotics attenuate the development of Alzheimer's disease in transgenic mice: Role of microbiome

2018

Abstract It has been suggested that exercise training and probiotic supplementation could decelerate the progress of functional and biochemical deterioration in APP/PS1 transgenic mice (APP/PS1TG). APP/PS1TG mice were subjected to exercise training and probiotic treatments and functional, biochemical and microbiome markers were analyzed. Under these conditions the mice significantly outperformed controls on The Morris Maze Test, and the number of beta-amyloid plaques decreased in the hippocampus. B. thetaiotaomicron levels correlated highly with the results of the Morris Maze Test (p

Male0301 basic medicineGenetically modified mouseAgingalzheimer; exercise; microbiome; probioticsalzheimermicrobiomeMice TransgenicPlaque AmyloidDiseaseHippocampusBiochemistrylaw.inventionAmyloid beta-Protein PrecursorMice03 medical and health sciencesProbiotic0302 clinical medicineEndocrinologyAlzheimer DiseaselawPhysical Conditioning Animalmental disordersPresenilin-1GeneticsAnimalsHippocampus (mythology)MedicineMicrobiomeMolecular BiologyAmyloid beta-Peptidesexercisebusiness.industryMicrobiotaCell BiologyDisease Models Animal030104 developmental biologyprobioticsImmunologybusiness030217 neurology & neurosurgeryExperimental Gerontology
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Impact of Resilience on the Association Between Amyloid-β and Longitudinal Cognitive Decline in Cognitively Healthy Older Adults

2019

The present study aims at investigating if the association between amyloid-β and longitudinal cognitive decline in cognitively healthy elderly is modulated by resilience capacity. Resilience capacity was quantified by education, which is a common proxy of resilience and has been shown to be related to a wide range of behaviors promoting resilience. Analyses were conducted with longitudinal cognitive data from the Alzheimer's Disease Neuroimaging Initiative (ADNI). 276 cognitively healthy older individuals (≥56 years) were included in the study. Baseline amyloid pathology was quantified using CSF amyloid-β 1-42 measurements. Longitudinal cognitive decline was assessed using ADAS13, Clinical …

Male0301 basic medicineGerontologyAmyloid pathologyAmyloid βClinical Dementia RatingDiseaseNeuropsychological Tests03 medical and health sciences0302 clinical medicineNeuroimagingAlzheimer Diseasemental disordersHumansMedicineCognitive DysfunctionLongitudinal StudiesCognitive declineAgedAged 80 and overAmyloid beta-Peptidesbusiness.industryGeneral NeuroscienceCognitionGeneral MedicineMiddle AgedResilience PsychologicalPeptide FragmentsPsychiatry and Mental healthClinical Psychology030104 developmental biologyMixed effectsFemaleGeriatrics and GerontologybusinessBiomarkers030217 neurology & neurosurgeryFollow-Up StudiesJournal of Alzheimer's Disease
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CSF neurofilament proteins as diagnostic and prognostic biomarkers for amyotrophic lateral sclerosis

2017

Elevated cerebrospinal fluid (CSF), Neurofilament Light (NF-L) and phosphorylated Heavy (pNF-H) chain levels have been found in Amyotrophic Lateral Sclerosis (ALS), with studies reporting a correlation of both neurofilaments (NFs) with the disease progression. Here, we measured NF-L and pNF-H concentrations in the CSF of ALS patients from a single tertiary Center and investigated their relationship with disease-related variables. A total of 190 ALS patients (Bulbar, 29.9%; Spinal, 70.1%; M/F = 1.53) and 130 controls with mixed neurological diseases were recruited. Demographic and clinical variables were recorded, and Delta FS was used to rate the disease progression. Controls were divided i…

Male0301 basic medicineNeurologypNF-HNeurofilamentKaplan-Meier EstimateGastroenterology0302 clinical medicineCerebrospinal fluidNeurofilament ProteinsMedicinePhosphorylationAmyotrophic lateral sclerosisNeuroradiologyNeurofilament ProteinMiddle AgedPrognosisNeurologyArea Under CurveCohortDisease ProgressionFemaleHumanmedicine.medical_specialtyPrognostic variableNeurofilamentPrognosiCSFEnzyme-Linked Immunosorbent AssayFollow-Up StudieDiagnosis Differential03 medical and health sciencesOligoclonal BandInternal medicineHumansAgedInflammationbusiness.industryAmyotrophic Lateral SclerosisOligoclonal BandsBiomarkermedicine.disease030104 developmental biologyROC CurveNF-LNeurology (clinical)ALSDifferential diagnosisbusinessBiomarkers030217 neurology & neurosurgeryAmyotrophic Lateral SclerosiFollow-Up StudiesJournal of Neurology
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Molecular properties underlying regional vulnerability to Alzheimer’s disease pathology

2018

Amyloid deposition and neurofibrillary degeneration in Alzheimer's disease specifically affect discrete neuronal systems, but the underlying mechanisms that render some brain regions more vulnerable to Alzheimer's disease pathology than others remain largely unknown. Here we studied molecular properties underlying these distinct regional vulnerabilities by analysing Alzheimer's disease-typical neuroimaging patterns of amyloid deposition and neurodegeneration in relation to regional gene expression profiles of the human brain. Graded patterns of brain-wide vulnerability to amyloid deposition and neurodegeneration in Alzheimer's disease were estimated by contrasting multimodal amyloid-sensiti…

Male0301 basic medicinePathologyphysiology [Amyloidogenic Proteins]genetics [Transcriptome]genetics [Alzheimer Disease]Cohort StudiesTranscriptomepathology [Alzheimer Disease]metabolism [Amyloidogenic Proteins]methods [Magnetic Resonance Imaging]0302 clinical medicinepathology [Brain]Gene expressiongenetics [Genetic Predisposition to Disease]Aged 80 and overmethods [Positron-Emission Tomography]NeurodegenerationBrainNeurofibrillary TanglesHuman brainMagnetic Resonance Imagingmetabolism [Neurofibrillary Tangles]medicine.anatomical_structureFemalemethods [Neuroimaging]Alzheimer's Disease Neuroimaging InitiativeAmyloidmedicine.medical_specialtyAmyloidmetabolism [Amyloid beta-Peptides]Amyloidogenic ProteinsNeuroimagingtau ProteinsBiology03 medical and health sciencesAlzheimer DiseasemedicineHumansDementiaGenetic Predisposition to Diseaseddc:610metabolism [Amyloid]AgedAmyloid beta-PeptidesCyclin-dependent kinase 5medicine.diseasemetabolism [tau Proteins]030104 developmental biologyPositron-Emission Tomographypathology [Neurofibrillary Tangles]Neurology (clinical)Transcriptome030217 neurology & neurosurgeryBrain
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The landscape of epilepsy-related GATOR1 variants

2019

Purpose:\ud \ud To define the phenotypic and mutational spectrum of epilepsies related to DEPDC5, NPRL2 and NPRL3 genes encoding the GATOR1 complex, a negative regulator of the mTORC1 pathway.\ud \ud Methods:\ud \ud We analyzed clinical and genetic data of 73 novel probands (familial and sporadic) with epilepsy-related variants in GATOR1-encoding genes and proposed new guidelines for clinical interpretation of GATOR1 variants.\ud \ud Results:\ud \ud The GATOR1 seizure phenotype consisted mostly in focal seizures (e.g., hypermotor or frontal lobe seizures in 50%), with a mean age at onset of 4.4 years, often sleep-related and drug-resistant (54%), and associated with focal cortical dysplasia…

Male0301 basic medicineProbandDEPDC5SUDEP030105 genetics & heredityBioinformaticsLoss of Function Mutation/geneticsEpilepsyINDEL MutationLoss of Function MutationmTORC1 pathwayGenetics(clinical)ChildGenetics (clinical)Multiprotein Complexes/geneticsBrugada SyndromeDNA Copy Number VariationBrugada syndromeINDEL Mutation/geneticsGTPase-Activating ProteinsNPRL3SeizureDEPDC5PhenotypePedigree3. Good healthBrugada Syndrome/geneticsChild PreschoolFemaleHumanSignal TransductionDNA Copy Number VariationsAdolescentSeizures/complicationsMechanistic Target of Rapamycin Complex 1/geneticsDNA Copy Number Variations/geneticsMechanistic Target of Rapamycin Complex 1Tumor Suppressor Proteins/geneticsArticleFocal cortical dysplasia03 medical and health sciencesSeizuresGTPase-Activating Proteins/geneticsmedicineHumansGenetic Predisposition to DiseaseDEPDC5; Focal cortical dysplasia; Genetic focal epilepsy; mTORC1 pathway; SUDEPGenetic focal epilepsyEpilepsy/complicationsRepressor Proteins/geneticsEpilepsybusiness.industryGTPase-Activating ProteinTumor Suppressor ProteinsInfant NewbornCorrectionInfantRepressor ProteinCortical dysplasiamedicine.diseaseddc:616.8Repressor Proteins030104 developmental biologyFrontal lobe seizures[SDV.GEN.GH]Life Sciences [q-bio]/Genetics/Human geneticsMultiprotein ComplexesMultiprotein ComplexeSignal Transduction/geneticsHuman medicinebusiness
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Genetic investigation of amyotrophic lateral sclerosis patients in south Italy: a two-decade analysis

2020

Amyotrophic lateral sclerosis (ALS) is a multifactorial disease characterized by the interplay of genetic and environmental factors. In the majority of cases, ALS is sporadic, whereas familial forms occur in less than 10% of patients. Herein, we present the results of molecular analyses performed in a large cohort of Italian ALS patients, focusing on novel and already described variations in ALS-linked genes. Our analysis revealed that more than 10% of tested patients carried a mutation in one of the major ALS genes, with C9orf72 hexanucleotide expansion being the most common mutation. In addition, our study confirmed a significant association between ALS patients carrying the ATNX-1 interm…

Male0301 basic medicineSanger sequencingAgingTime FactorsDiseaseCohort Studies03 medical and health sciencessymbols.namesake0302 clinical medicineRisk FactorsC9orf72HumansMedicineAmyotrophic lateral sclerosisRisk factorGenePathologicalAtaxin-1Genetic Association StudiesAmyotrophic lateral sclerosiSanger sequencingGeneticsDNA Repeat ExpansionC9orf72 ProteinMolecular analysibusiness.industryMolecular analysisGeneral NeuroscienceGenetic VariationAmyotrophic lateral sclerosismedicine.disease030104 developmental biologyItalyMutation (genetic algorithm)symbolsFemaleNeurology (clinical)Geriatrics and Gerontologybusiness030217 neurology & neurosurgeryDevelopmental BiologyNeurobiology of Aging
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Female exogamy and gene pool diversification at the transition from the final neolithic to the early Bronze Age in central Europe

2017

Human mobility has been vigorously debated as a key factor for the spread of bronze technology and profound changes in burial practices as well as material culture in central Europe at the transition from the Neolithic to the Bronze Age. However, the relevance of individual residential changes and their importance among specific age and sex groups are still poorly understood. Here, we present ancient DNA analysis, stable isotope data of oxygen, and radiogenic isotope ratios of strontium for 84 radiocarbon-dated skeletons from seven archaeological sites of the Late Neolithic Bell Beaker Complex and the Early Bronze Age from the Lech River valley in southern Bavaria, Germany. Complete mitocho…

Male0301 basic medicineengineering.materialDiversification (marketing strategy)03 medical and health sciencesBronze AgeBeakerKinshipHumans0601 history and archaeologyBronzeAnthropology CulturalHistory AncientMultidisciplinary060102 archaeologyExogamyGene Pool06 humanities and the artsBiological SciencesArchaeologyEurope030104 developmental biologyAncient DNAGeographyGenome MitochondrialengineeringFemaleGene pool
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Elevated Testosterone Level and Urine Scent Marking in Male 5xFAD Alzheimer Model Mice

2019

Background:Function of the Amyloid Precursor Protein (AβPP) and its various cleavage products still is not unraveled down to the last detail. While its role as a source of the neurotoxic Amyloid beta (Aβ) peptides in Alzheimer’s Disease (AD) is undisputed and its property as a cell attachment protein is intriguing, while functions outside the neuronal context are scarcely investigated. This is particularly noteworthy because AβPP has a ubiquitous expression profile and its longer isoforms, AβPP750 and 770, are found in various tissues outside the brain and in non-neuronal cells.Objective:Here, we aimed at analyzing the 5xFAD Alzheimer’s disease mouse model in regard to male sexual function.…

Male0301 basic medicinemedicine.medical_specialtymiceAmyloid betaCentral nervous system610Mice Transgenicamyloid precursor proteinAmyloid beta-Protein PrecursorSexual Behavior Animal03 medical and health sciences0302 clinical medicinesexual behaviorAlzheimer DiseaseInternal medicineTestisPresenilin-1medicineAmyloid precursor proteinAnimalsSperm CountbiologyWild typeBrainOrgan SizeAlzheimer's diseaseSertoli cellSpermPhenotypeAndrogen receptorDisease Models Animal030104 developmental biologymedicine.anatomical_structureEndocrinologyNeurologytestosteronebiology.proteinNeurology (clinical)030217 neurology & neurosurgeryurine scent marking test
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Effect of High‐Caloric Nutrition on Survival in Amyotrophic Lateral Sclerosis

2019

International audience; Objective: Weight loss has been identified as a negative prognostic factor in amyotrophic lateral sclerosis, but there is no evidence regarding whether a high-caloric diet increases survival. Therefore, we sought to evaluate the efficacy of a high-caloric fatty diet (HCFD) for increasing survival.Methods: A 1:1 randomized, placebo-controlled, parallel-group, double-blinded trial (LIPCAL-ALS study) was conducted between February 2015 and September 2018. Patients were followed up at 3, 6, 9, 12, 15, and 18 months after randomization. The study was performed at 12 sites of the clinical and scientific network of German motor neuron disease centers (ALS/MND-NET). Eligible…

Male0301 basic medicinemortality [Amyotrophic Lateral Sclerosis]MESH: Combined Modality Therapy[SDV]Life Sciences [q-bio]law.invention0302 clinical medicineMESH: RiluzoleRandomized controlled triallawdiet therapy [Amyotrophic Lateral Sclerosis]Clinical endpointMedicineMESH: Double-Blind Methodtherapeutic use [Riluzole]MESH: Amyotrophic Lateral Sclerosismethods [Combined Modality Therapy]education.field_of_studyRiluzoleMESH: Middle AgedHazard ratioMESH: Neuroprotective AgentsMiddle Agedtherapeutic use [Neuroprotective Agents]Combined Modality Therapy3. Good healthRiluzole[SDV] Life Sciences [q-bio]Neuroprotective AgentsNeurologyMESH: Survival AnalysisFemalemedicine.drugmortality [Diet High-Fat]medicine.medical_specialtyPopulationDiet High-FatPlacebo03 medical and health sciencesDouble-Blind MethodInternal medicineHumansddc:610educationSurvival analysisMESH: Humansdrug therapy [Amyotrophic Lateral Sclerosis]business.industryAmyotrophic Lateral SclerosisSurvival AnalysisConfidence intervalMESH: MaleMESH: Diet High-Fat030104 developmental biologyNeurology (clinical)businessMESH: Female030217 neurology & neurosurgery
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