Search results for "Antidepressant"

showing 10 items of 161 documents

Piracetam counteracts the effects of amitriptyline on inhibitory avoidance in CD1 mice.

2005

The purpose of the present work was to study the effects of amitriptyline on animal cognition in relation to some characteristics of its therapeutic effects. The modulation of acute and chronic effects of amitriptyline on inhibitory avoidance in male and female mice by piracetam was investigated. In Experiment 1, mice were subjected to the training phase of inhibitory avoidance conditioning 60 min after acute piracetam (100 mg/kg) or physiological saline administration. Immediately after the behavioural task, they received a single injection of the tricyclic antidepressant amitriptyline (30 mg/kg) or physiological saline. Twenty-four hours later, subjects were tested for avoidance. In Exper…

MaleElevated plus mazemedicine.medical_specialtymedicine.drug_classAmitriptylineTricyclic antidepressantPharmacologyAntidepressive Agents TricyclicInhibitory postsynaptic potentialDrug Administration ScheduleStatistics NonparametricBehavioral NeuroscienceMiceSex FactorsMemorymedicineAvoidance LearningAnimalsAmitriptylineDrug InteractionsPsychiatryNootropic AgentsAnalysis of VarianceReactive inhibitionTherapeutic effectPiracetamReactive InhibitionPiracetamFemaleAnalysis of variancePsychologymedicine.drugBehavioural brain research
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Anticonvulsant and antidepressant activity of the selected terpene GABA derivatives in experimental tests in mice

2006

The present study was designed to investigate the central nervous system activity of terpene GABA (and piracetam) derivatives designated as BF-1, BF-2, BF-3, BF-4, BF-5, BF-6. We assessed their anticonvulsant activity in the two main mouse models of seizures (MES-test, PTZ-test), an antidepressant-like effect in the forced swim test (FST), as well as an influence on spontaneous locomotor activity. Our study demonstrated the strong anticonvulsant activity of (1S,3R,7R)-(-)-3,8,8-trimethyl-4-aza-bicyclo[5.1.0]acetate-5-one hydrochloride (compound BF-2) in the PTZ-test. Activity of BF-2 was equipotent to ethosuximide (380 mg/kg, po) in the PTZ-test, when used at a dose of 100 mg/kg, po. No neu…

MaleGABAantidepressant-like activitymiceReceptors GABA-BAnimalsAnticonvulsantsMotor Activityanticonvulsant-Antidepressive AgentsSwimminggamma-Aminobutyric AcidterpenesPharmacological Reports
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A Simple Microassay for the Determination of Hydrazine in Biological Samples. Effect of Hydrazine and Isoniazid on Liver and Brain Glutathione

1987

A simple microassay for the determination of hydrazine in laboratory samples is presented. The colored product of the reaction of p-dimethylaminobenzaldehyde with hydrazine was tested at 470 nm using double-beam mode in different samples. Internal standards and data on blood serum, liver, and brain of rats treated with hydrazine or isoniazid are presented. The tissue glutathione content of these rats was determined, and the possible implication of glutathione in the brain toxicity of hydrazine is discussed.

MaleHealth Toxicology and MutagenesisMetaboliteToxicologyAnalytical Chemistrychemistry.chemical_compoundBlood serumSpectrophotometryIsoniazidmedicineAnimalsEnvironmental ChemistryHydrazine (antidepressant)Brain ChemistryChemical Health and SafetyChromatographymedicine.diagnostic_testIsoniazidMetabolismGlutathioneGlutathioneRatsHydrazinesLiverchemistryBiochemistryToxicitymedicine.drugJournal of Analytical Toxicology
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Structural connectivity and subcellular changes after antidepressant doses of ketamine and Ro 25-6981 in the rat: an MRI and immuno-labeling study

2021

© The Author(s) 2021.

MaleHistologyDendritic spineInfralimbic cortexPrefrontal CortexNeuroimagingNeurofilamentRats Sprague-DawleyInfralimbic cortexWhite matterDorsal raphe nucleusPhenolsPiperidinesmedicineAnimalsPrefrontal cortexbiologyChemistryGeneral NeuroscienceDorsal raphe nucleusPsychotomimeticMagnetic Resonance ImagingAntidepressive AgentsRatsMyelin basic proteinMyelinizationmedicine.anatomical_structureFast-acting antidepressantbiology.proteinNMDA receptorOriginal ArticleKetamineAnatomyNeurosciencemedicine.drugBrain Structure and Function
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Molecular adaptations of the blood–brain barrier promote stress resilience vs. depression

2020

Significance Thirty to fifty percent of depressed individuals are unresponsive to commonly prescribed antidepressant treatments, suggesting that biological mechanisms, such as stress-induced inflammation and blood vessel dysfunction, remain untreated. The blood–brain barrier is the ultimate frontier between the brain and harmful toxins or inflammatory signals circulating in the blood. Depression and vulnerability to chronic social stress are associated with loss of this barrier integrity; however, the mechanisms involved remain poorly understood. Identification of adaptations leading to resilience under stressful conditions could help develop novel treatments. Here we combined behavioral, p…

MaleHistone Deacetylase 1InflammationFOXO1Blood–brain barrierNucleus AccumbensEpigenesis GeneticProinflammatory cytokineMice03 medical and health sciences0302 clinical medicinevascularmedicineAnimalsHumansClaudin-5030304 developmental biologyInflammationSocial stressDepressive Disorder Major0303 health sciencesantidepressantMultidisciplinaryDepressionbusiness.industrySystems BiologyBiological Sciencesmedicine.diseasemood disordersAntidepressive Agents3. Good healthMice Inbred C57BLDisease Models Animalmedicine.anatomical_structureMood disordersBlood-Brain BarrierMajor depressive disorderAntidepressantmedicine.symptombusinessNeuroscienceStress Psychologicalepigenetic030217 neurology & neurosurgerySignal TransductionProceedings of the National Academy of Sciences
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Attenuation of sucrose consumption in mice by chronic mild stress and its restoration by imipramine

1995

Chronic exposure to mild unpredictable stressors (CMS) has previously been found to reduce the consumption of palatable, sweet solutions in rats. In the present study, the utility of this procedure was assessed in mice. Male AP mice subjected to CMS showed reduced consumption of a 2% or 4% sucrose solution. This effect was reversed by chronic (3 weeks) treatment with the tricyclic antidepressant imipramine (20 mg/kg per day). These results extend previous reports of a generalized decrease in sensitivity to reward (anhedonia) in rats caused by CMS and the efficacy of antidepressant treatment in this paradigm. Chronic unpredictable mild stress in mice appears to provide a realistic animal mod…

MaleImipramineSucrosemedicine.medical_specialtySucroseRatónmedicine.drug_classmedicine.medical_treatmentTricyclic antidepressantImipramineEatingMicechemistry.chemical_compoundInternal medicineAnimalsMedicinePsychiatryDepression (differential diagnoses)PharmacologyAnalysis of VarianceDepressive DisorderChemotherapyBehavior Animalbusiness.industryAnhedoniaDisease Models AnimalEndocrinologychemistryAntidepressantmedicine.symptombusinessStress Psychologicalmedicine.drugPsychopharmacology
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Role of the amygdala in antidepressant effects on hippocampal cell proliferation and survival and on depression-like behavior in the rat

2021

The stimulation of adult hippocampal neurogenesis by antidepressants has been associated with multiple molecular pathways, but the potential influence exerted by other brain areas has received much less attention. The basolateral complex of the amygdala (BLA), a region involved in anxiety and a site of action of antidepressants, has been implicated in both basal and stress-induced changes in neural plasticity in the dentate gyrus. We investigated here whether the BLA modulates the effects of the SSRI antidepressant fluoxetine on hippocampal cell proliferation and survival in relation to a behavioral index of depression-like behavior (forced swim test). We used a lesion approach targeting th…

MaleLong-Term Potentiationlcsh:MedicineHippocampal formationElement-Binding ProteinAmygdala/*drug effects/physiopathologyHippocampusMemory FormationRats Sprague-Dawleyddc:616.890302 clinical medicineMedial Prefrontal CortexElevated Plus-MazeSerotonin Uptake Inhibitors/*pharmacologylcsh:ScienceBasolateral Amygdala0303 health sciencesMultidisciplinaryNeuroscience/Behavioral NeuroscienceDepressionNeurogenesisBLAAmygdalaImmunohistochemistryChronic FluoxetineAdult-RatNeuroscience/Psychologymedicine.anatomical_structureFluoxetine/*pharmacologyDepression/*pathologyAntidepressantAntidepressive Agents Second-GenerationSelective Serotonin Reuptake InhibitorsResearch ArticleEstrèsElevated plus mazemedicine.medical_specialtyAnimal-ModelAntidepressive Agents Second-Generation/*pharmacologyCell SurvivalAmygdala03 medical and health sciencesFluoxetineNeuroplasticityHippocampus/cytology/*drug effectsmedicineAnimalsPsychiatryMaze Learning030304 developmental biologyCell Proliferationbusiness.industryDentate gyrusMental Health/Mood Disorderslcsh:RBasolateral complex of the amygdaleRatsCell Proliferation/*drug effectsDentate Gyruslcsh:QCell Survival/*drug effectsbusinessNeuroscience030217 neurology & neurosurgeryBasolateral amygdala
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Altered brain concentrations of citalopram and escitalopram in P-glycoprotein deficient mice after acute and chronic treatment

2013

Background: According to both in vitro and in vivo data P-glycoprotein (P-gp) may restrict the uptake of several antidepressants into the brain, thus contributing to the poor success rate of current antidepressant therapies. The therapeutic activity of citalopram resides in the Senantiomer, whereas the R-enantiomer is practically devoid of serotonin reuptake potency. To date, no in vivo data are available that address whether the enantiomers of citalopram and its metabolites are substrates of P-gp. Methods: P-gp knockout (abcb1ab (-/-)) and wild-type (abcb1ab (+/+)) mice underwent acute (single-dose) and chronic (two daily doses for 10 days) treatment with citalopram (10 mg/kg) or escitalop…

MaleMedicin och hälsovetenskapescitalopramenantiomersCitaloprammice knockoutP-glycoproteinCitalopramPharmacologyMedical and Health Sciencesbehavioral disciplines and activitiesMiceIn vivomental disordersmedicineAnimalsEscitalopramPotencyPharmacology (medical)ATP Binding Cassette Transporter Subfamily B Member 1Biological PsychiatryP-glycoproteinMice KnockoutPharmacologybiologybusiness.industryBrainPsychiatry and Mental healthNeurologyKnockout mousebiology.proteinAntidepressive Agents Second-GenerationAntidepressantNeurology (clinical)Enantiomerbusinessmedicine.drugEuropean Neuropsychopharmacology
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Biomarkers for Antidepressant Efficacy of Electroconvulsive Therapy: An Exploratory Cerebrospinal Fluid Study

2018

<b><i>Background:</i></b> No candidate biomarkers based on cerebrospinal fluid (CSF) have been identified as prognostic factors in patients with major depression treated with electroconvulsive therapy (ECT), yet. <b><i>Method:</i></b> Following different underlying hypotheses, we analysed baseline CSF levels of markers of neurodegeneration (tau proteins, β-amyloids and neurogranin), elements of the innate immune system (interleukin [IL]-6, neopterin, soluble CD14, soluble CD163, migration inhibitory factor and monocyte chemotactic protein 1), endocannabinoids, sphingolipids and Klotho before ECT in patients with depression (<i>n</i&gt…

MaleOncologymedicine.medical_treatmentchemistry.chemical_compound0302 clinical medicineCerebrospinal fluidElectroconvulsive therapyNeurogranincerebrospinal fluid [Sphingolipids]Electroconvulsive TherapyKlothoGlucuronidaseAged 80 and overtherapy [Depressive Disorder Major]NeopterinInterleukinMiddle AgedPsychiatry and Mental healthTreatment OutcomeNeuropsychology and Physiological Psychologycerebrospinal fluid [Biomarkers]cerebrospinal fluid [Glucuronidase]Biomarker (medicine)AntidepressantFemaleAdultmedicine.medical_specialtyklotho proteinYoung Adult03 medical and health sciencesInternal medicinemental disordersmedicineHumansddc:610Klotho ProteinsBiological Psychiatrycerebrospinal fluid [Nerve Degeneration]AgedDepressive Disorder MajorSphingolipidsbusiness.industrycerebrospinal fluid [Depressive Disorder Major]Immunity Innate030227 psychiatrychemistryNerve Degenerationcerebrospinal fluid [Endocannabinoids]businessBiomarkers030217 neurology & neurosurgeryEndocannabinoidsNeuropsychobiology
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Quantitative analysis of the EEG effects produced by imipramine, desipramine, promazine, and monodesmethyl promazine in the isolated perfused rat bra…

1974

The effects of imipramine, desipramine, promazine and monodesmethyl promazine on the EEG of the isolated perfused rat brain were studied. The brain preparation was perfused for 30 min with simulated blood, containing of the drugs in a concentration of 10−5 M. Control experiments were performed without a drug added to the simplified blood. The EEG was recorded at various times on a magnetic tape and was evaluated visually and quantitatively (amplitude and interval histography). The EEG effects of imipramine and promazine as well as the effects of these drugs with their monodesmethyl metabolites were compared. The drugs produced clear EEG changes compared with the control EEG. An increase of …

MalePharmacologyAnalysis of VarianceImipraminemedicine.diagnostic_testChemistryDesipramineBrainElectroencephalographyIn Vitro TechniquesPharmacologyElectroencephalographyRat brainImipramineRatsPerfusionDesipraminemedicineAnimalsAntidepressantQuantitative analysis (chemistry)PromazinePromazinemedicine.drugDemethylationPsychopharmacologia
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