Search results for "Autoimmune"

showing 10 items of 648 documents

Expression profiling of autoimmune regulator AIRE mRNA in a comprehensive set of human normal and neoplastic tissues.

2006

Defects in the autoimmune regulator (AIRE) gene cause the monogenic autoimmune disease autoimmune polyendocrinopathy syndrome type 1 (APS-1), which is characterized by a loss of self-tolerance to multiple organs. In concordance with its role in immune tolerance, AIRE is strongly expressed in medullary thymic epithelial cells (mTECs). Data on mechanisms controlling AIRE activation and the expression of this gene in other tissues are fragmentary and controversial. We report here AIRE mRNA expression profiling of a large set of normal human tissues and cells, tumor specimen and methylation deficient cell lines. On this broad data basis we found that AIRE mRNA expression is confined to mTECs in…

Immune Tolerance/geneticsThymus Gland/immunologyTranscription GeneticImmunologyTranscription Genetic/immunologyThymus GlandBiologyLymph Nodes/immunologymedicine.disease_causeAutoimmunityImmune toleranceCell Line TumorNeoplasmsmedicineTranscriptional regulationImmune ToleranceImmunology and AllergyHumansRNA MessengerPolyendocrinopathies AutoimmuneGeneTranscription Factors/biosynthesisAutoimmune diseaseReverse Transcriptase Polymerase Chain ReactionGene Expression ProfilingRNA Messenger/biosynthesisDNA Methylationmedicine.diseaseAutoimmune regulatorNeoplasms/geneticsPolyendocrinopathies Autoimmune/geneticsGene expression profilingGene Expression Regulation NeoplasticDNA methylationImmunologyCancer researchLymph NodesGene Expression Profiling/methodsTranscription FactorsImmunology letters
researchProduct

Increased Goodpasture antigen-binding protein expression induces type IV collagen disorganization and deposit of immunoglobulin A in glomerular basem…

2007

Increased expression of Goodpasture antigen-binding protein (GPBP), a protein that binds and phosphorylates basement membrane collagen, has been associated with immune complex-mediated pathogenesis. However, recent reports have questioned this biological function and proposed that GPBP serves as a cytosolic ceramide transporter (CERTL). Thus, the role of GPBP in vivo remains unknown. New Zealand White (NZW) mice are considered healthy animals although they convey a genetic predisposition for immune complex-mediated glomerulonephritis. Here we show that NZW mice developed age-dependent lupus-prone autoimmune response and immune complex-mediated glomerulonephritis characterized by elevated GP…

Immunoglobulin ACollagen Type IVAgingMice Inbred StrainsMice TransgenicAntigen-Antibody ComplexProtein Serine-Threonine Kinasesurologic and male genital diseasesPathology and Forensic MedicinePathogenesisType IV collagenMiceGlomerulonephritisSpecies SpecificityGlomerular Basement MembranemedicineGoodpasture syndromeAnimalsHumansLupus Erythematosus SystemicAutoantibodiesAutoimmune diseaseBasement membranebiologyGlomerular basement membraneGlomerulonephritismedicine.diseaseImmunoglobulin Amedicine.anatomical_structureImmunologyCancer researchbiology.proteinRegular Articles
researchProduct

Nucleic acid recognizing Toll-like receptors and autoimmunity

2007

The understanding of autoimmune diseases experienced an impressive boost since the Toll-like receptors (TLRs) have been identified as possible key players in autoimmune pathophysiology. Although these receptors recognize a variety of structures derived from viruses, bacteria, and fungi leading to subsequent initiation of the relevant immune responses, recent data support the idea that TLRs are crucial in the induction and perpetuation of certain autoimmune diseases, especially the systemic lupus erythematosus (SLE). In this review, we will summarize recent data on involvement of TLRs in the development of autoimmune diseases. We will focus on TLRs 7, 8, and 9 that were originally identified…

ImmunologyGene ExpressionReceptors Antigen B-CellAutoimmunityContext (language use)Biologymedicine.disease_causeAutoimmune DiseasesAutoimmunityImmune systemAntigenGene expressionmedicineAnimalsHumansImmunology and AllergyReceptorToll-Like ReceptorsRNADNADendritic CellsToll-Like Receptor 7Toll-Like Receptor 8Toll-Like Receptor 9ImmunologyRNASignal transductionSignal TransductionCurrent Opinion in Immunology
researchProduct

Immunosuppression Drugs Seize the Overacting Immune System by Preventing the Cytokine Storm In COVID-19 Symptoms

2021

The COVID-19 over-active other diseases within the human body especially respiratory system in which cytokine storm abnormally developed that results in the excessive killing of cells within the autoimmune system. To resolve this major issue, a systematic review paper is developed whose major aim is to propose such immunosuppression drugs that can easily seize the overacted immune system by blocking the cytokine storm action within human body, by systematically overview the previous scholars' articles, case reports and authentic websites based valid data. To justify this aim, different latest and authentic medicine's journals and papers are collected by posting four major keywords like "Cyt…

Immunosuppression Drugsautoimmune systemCOVID-19Cytokine StormSystematic Reviews in Pharmacy
researchProduct

Immunosuppressant therapy of thyroid eye disease.

1988

Thyroid eye disease is attributed to an autoimmune process where both cellular and humoral immunity play a role. In this report, after a short introduction dealing with immunopathogenesis of the disease, immunosuppressant therapy is discussed. Treatment with glucocorticoids (as the standard substance), nonsteroid immunosuppressants (azathioprine, cyclophosphamide, lately cyclosporin) and with the immunomodulatory substance ciamexone is reviewed. Retroorbital irradiation as a "local" immunosuppressive method and plasmapheresis are also discussed. While systemic glucocorticoids and to a lesser extent orbital radiotherapy are routinely administered for severe Graves' ophthalmopathy, nonsteroid…

Immunosuppression TherapyCyclophosphamidebusiness.industrymedicine.medical_treatmentEye diseaseThyroidAzathioprineGeneral MedicineDiseasemedicine.diseaseeye diseasesGraves DiseaseRadiation therapymedicine.anatomical_structureAutoimmune ProcessDrug DiscoveryImmunologymedicineMolecular MedicineHumansPlasmapheresisbusinessGenetics (clinical)Immunosuppressive Agentsmedicine.drugKlinische Wochenschrift
researchProduct

Hsp10: Anatomic distribution, functions, and involvement in human disease

2013

There is growing evidence that molecular chaperones/heat shock proteins are involved in the pathogenesis of a number of human diseases, known as chaperonopathies. A better molecular understanding of the pathogenetic mechanisms is essential for addressing new strategies in diagnostics, therapeutics and clinical management of chaperonopathies, including those in which Hsp10 is involved. This chaperonin has been studied for a long time as a member of the mitochondrial protein-folding machine. However, although in normal cells Hsp10 is mainly localized in the mitochondrial matrix, it has also been found during and after stress in other subcellular compartments, such as cytosol, vesicles and sec…

InflammationAgingGeneral Immunology and MicrobiologySettore BIO/16 - Anatomia UmanaVesicleBiologyGeneral Biochemistry Genetics and Molecular BiologyChaperoninCell biologyAutoimmune DiseasesPathogenesisSettore MED/18 - Chirurgia GeneraleCytosolSettore MED/38 - Pediatria Generale E SpecialisticaBiochemistryMitochondrial matrixHeat shock proteinNeoplasmsCancer cellExtracellularChaperonin 10HumansHsp10chaperonopathies molecular chaperones human diseases cellular localization mitochondria
researchProduct

Hsp60 chaperonopathies and chaperonotherapy: targets and agents

2014

Hsp60 (Cpn60) assembles into a tetradecamer that interacts with the co-chaperonin Hsp10 (Cpn10) to assist client polypeptides to fold, but it also has other roles, including participation in pathogenic mechanisms.Hsp60 chaperonopathies are pathological conditions, inherited or acquired, in which the chaperone plays a determinant etiologic-pathogenic role. These diseases justify selection of Hsp60 as a target for developing agents that interfere with its pathogenic effects. We provide information on how to proceed.The information available encourages the development of ways to improve Hsp60 activity (positive chaperonotherapy) when deficient or to block it (negative chaperonotherapy) when pa…

InflammationPharmacologyanimal structuresChaperonin 60biologyProtein ConformationfungiClinical BiochemistryChaperonin 60BioinformaticsAutoimmune Diseasesautoimmunity cancer carboranylphenoxyacetanilide chaperonopathies chaperonotherapy chemical compounds Cpn60 electrophilic compounds epolactaene functional domain GroEL Hsp60 inflammation mizoribine structural domainNeoplasmsChaperone (protein)Expert opinionDrug DiscoveryImmunologybiology.proteinAnimalsHumansMolecular MedicineHSP60Cytokine formationA determinantExpert Opinion on Therapeutic Targets
researchProduct

Induction of regulatory Tr1 cells and inhibition of TH17 cells by IL-27

2011

Accumulating evidence indicates that IL-27, a member of the IL-12 family of cytokines, alleviates the severity of autoimmune diseases in both mice and men. The IL-27-induced activation of signal transducer and activator of transcription (Stat)1 and Stat3 promotes the generation of IL-10- producing type 1 regulatory T (Tr1) cells that inhibit effector T cells. In addition, IL-27 also suppresses the development of pathogenic IL-17-producing CD4(+) T cells (T(H)17) cells suggesting that pharmacological manipulations of IL-27 signaling pathway could be exploited therapeutically in regulating tissue inflammation. Here, we review how IL-27 controls inflammation through the regulation of Tr1 and T…

Interleukins/immunologyImmunologyInterleukin-10/biosynthesis/immunologyT-Lymphocytes Regulatory/immunologyddc:616.07T-Lymphocytes RegulatoryArticleAutoimmune Diseases03 medical and health sciences0302 clinical medicineImmunology and AllergyCytotoxic T cellTh17 Cells/immunologyAnimalsHumansIL-2 receptorSTAT3STAT4030304 developmental biology0303 health sciencesbiologyZAP70InterleukinsFOXP33. Good healthCell biologyddc:616.8Interleukin-10Interleukin 10Autoimmune Diseases/immunologyImmunologyInterleukin 12biology.proteinTh17 Cells030215 immunology
researchProduct

Gastric cancer in autoimmune gastritis: A case-control study from the German centers of the staR project on gastric cancer research

2020

Objectives Patients with autoimmune gastritis (AIG) are reported to have an increased risk of developing gastric cancer (GC). In this study, we assess the characteristics and outcomes of GC patients with AIG in a multicenter case-control study. Methods Between April 2013 and May 2017, patients with GC, including cancers of the esophagogastric junction (EGJ) Siewert type II and III, were recruited. Patients with histological characteristics of AIG were identified and matched in a 1:2 fashion for age and gender to GC patients with no AIG. Presenting symptoms were documented using a self-administered questionnaire. Results Histological assessment of gastric mucosa was available for 572/759 GC …

Intrinsic FactorMalemedicine.medical_specialtyAutoimmune Gastritisautoimmune gastritisRisk AssessmentGastroenterologysurvivalAutoimmune DiseasesGerman03 medical and health sciences0302 clinical medicineParietal Cells GastricRisk FactorsStomach NeoplasmsInternal medicineAnemia PerniciousmedicineHumansEndoscopy Digestive SystemAgedAutoantibodiesbiologyHelicobacter pyloribusiness.industryGastroenterologyCase-control studyCancerOriginal ArticlesMiddle AgedHelicobacter pyloribiology.organism_classificationmedicine.diseaselanguage.human_languageIncreased riskOncologyGastric MucosaCase-Control StudiesGastritis030220 oncology & carcinogenesislanguagesymptomsFemale030211 gastroenterology & hepatologybusinessGastric cancerUnited European Gastroenterology Journal
researchProduct

Molecular mimicry may explain multi-organ damage in COVID-19

2020

International audience

Kawasaki vasculitiVascular damagemedicine.disease_causeEpitopes0302 clinical medicineOR7D4PandemicSevere acute respiratory syndrome coronavirus 2Immunology and AllergyComputingMilieux_MISCELLANEOUS0303 health sciencesLeukopenia[SDV.BIBS]Life Sciences [q-bio]/Quantitative Methods [q-bio.QM]Molecular mimicryPARP9Cross ReactionEpitopemedicine.symptomCoronavirus InfectionsHuman2019-20 coronavirus outbreakCoronavirus disease 2019 (COVID-19)AnosmiaPneumonia ViralImmunologyAnosmiaCross ReactionsBiologyAutoimmune DiseaseArticleAutoimmune DiseasesBetacoronavirus03 medical and health sciencesKawasaki vasculitismedicineHumansPandemics030304 developmental biologyBetacoronaviruPandemicSARS-CoV-2Coronavirus InfectionModels ImmunologicalCOVID-19LeukopeniaMulti organbiology.organism_classificationVirologySLC12A6Molecular mimicry030217 neurology & neurosurgeryBetacoronavirusAutoimmunity Reviews
researchProduct