Search results for "B1"

showing 10 items of 1109 documents

Detachment of Chain-Forming Neuroblasts by Fyn-Mediated Control of cell–cell Adhesion in the Postnatal Brain

2018

In the rodent olfactory system, neuroblasts produced in the ventricular-subventricular zone of the postnatal brain migrate tangentially in chain-like cell aggregates toward the olfactory bulb (OB) through the rostral migratory stream (RMS). After reaching the OB, the chains are dissociated and the neuroblasts migrate individually and radially toward their final destination. The cellular and molecular mechanisms controlling cell–cell adhesion during this detachment remain unclear. Here we report that Fyn, a nonreceptor tyrosine kinase, regulates the detachment of neuroblasts from chains in the male and female mouse OB. By performing chemical screening andin vivoloss-of-function and gain-of-f…

Male0301 basic medicineanimal structuresRostral migratory streamNerve Tissue ProteinsProto-Oncogene Proteins c-fynAdherens junctionMice03 medical and health sciencesFYNNeural Stem CellsNeuroblastCell MovementCell AdhesionmedicineAnimalsCell adhesionResearch ArticlesChemistryGeneral NeurosciencefungiBrainCateninsCadherinsDAB1Granule cellOlfactory BulbOlfactory bulbCell biology030104 developmental biologymedicine.anatomical_structurenervous systemGene Knockdown Techniquesembryonic structuresFemaleThe Journal of Neuroscience
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Patients experiencing statin-induced myalgia exhibit a unique program of skeletal muscle gene expression following statin re-challenge

2017

Statins, the 3-hydroxy-3-methyl-glutaryl (HMG)-CoA reductase inhibitors, are widely prescribed for treatment of hypercholesterolemia. Although statins are generally well tolerated, up to ten percent of statin-treated patients experience myalgia symptoms, defined as muscle pain without elevated creatinine phosphokinase (CPK) levels. Myalgia is the most frequent reason for discontinuation of statin therapy. The mechanisms underlying statin myalgia are not clearly understood. To elucidate changes in gene expression associated with statin myalgia, we compared profiles of gene expression in skeletal muscle biopsies from patients with statin myalgia who were undergoing statin re-challenge (cases)…

Male0301 basic medicinemyalgiaGene Expressionlcsh:MedicineApoptosis030204 cardiovascular system & hematologyPathology and Laboratory MedicineBioinformaticsBiochemistry0302 clinical medicineMedicine and Health SciencesGene Regulatory Networkslcsh:ScienceMusculoskeletal SystemEnergy-Producing OrganellesMyositisRegulation of gene expressionMultidisciplinaryCell DeathbiologyMusclesDrugsMiddle AgedMitochondriaCell ProcessesHMG-CoA reductaseFemalelipids (amino acids peptides and proteins)AnatomyCellular Structures and Organellesmedicine.symptomResearch ArticleSenescencemedicine.medical_specialtyStatinmedicine.drug_classPainBioenergeticsPolymorphism Single Nucleotide03 medical and health sciencesSigns and SymptomsDiagnostic MedicineInternal medicineGeneticsmedicineHumansGene Regulationcardiovascular diseasesMuscle SkeletalAgedPharmacologybusiness.industrylcsh:RStatinsBiology and Life SciencesComputational Biologynutritional and metabolic diseasesMyalgiaCell Biologymedicine.disease030104 developmental biologyEndocrinologyGene Expression RegulationSkeletal MusclesLeukocytes Mononuclearbiology.proteinProtein prenylationlcsh:QHydroxymethylglutaryl-CoA Reductase InhibitorsSLCO1B1businessPLOS ONE
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Total Sugar Intake and Macro and Micronutrients in Children Aged 6-8 Years: The Aniva Study

2020

The objective of this study was to study the association between total sugar intake (TSI) levels of children aged 6-8 years old, nutrient intake and anthropometry. Food and beverage intakes were collected by a prospective three-day recall questionnaire. The 2237 children were distributed into three groups according to TSI percentiles. Mean TSI was 93.77 &plusmn

Male0301 basic medicinesugar intakeWaistDietary Sugarsmedicine.medical_treatmentNutritional Statuschemistry.chemical_elementlcsh:TX341-641030209 endocrinology & metabolismEating patternsIodineBody compositionArticleChildhood obesity03 medical and health sciences0302 clinical medicineAnimal scienceNutrientchildrenSurveys and QuestionnairesSugar intakeVitamin D and neurologyHumansMedicineMicronutrientsProspective StudiesVitamin B12ChildChildrenPublic health nutritionbody composition030109 nutrition & dieteticsNutrition and Dieteticsbusiness.industryVitamin EFeeding BehaviorAnthropometryNutrition Surveysmedicine.diseasechemistrypublic health nutritionMental RecallFemaleeating patternsbusinesslcsh:Nutrition. Foods and food supplyFood Science
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Effect of some indole derivatives on xenobiotic metabolism and xenobiotic-induced toxicity in cultured rat liver slices.

1999

In this study the effect of some indole derivatives on xenobiotic metabolizing enzymes and xenobiotic-induced toxicity has been examined in cultured precision-cut liver slices from male Sprague-Dawley rats. While treatment of rat liver slices for 72 hours with 2-200 microM of either indole-3-carbinol (I3C) or indole-3-acetonitrile (3-ICN) had little effect on cytochrome P-450 (CYP)-dependent enzyme activities, enzyme induction was observed after in vivo administration of I3C. The treatment of rat liver slices with 50 microM 3,3'-diindolylmethane (DIM; a dimer derived from I3C under acidic conditions) for 72 hours resulted in a marked induction of CYP-dependent enzyme activities. DIM appears…

Male33'-DiindolylmethaneAflatoxin B1IndolesCarcinogenicity TestsDiindolylmethaneIn Vitro TechniquesToxicologyXenobioticsRats Sprague-Dawleychemistry.chemical_compoundCytochrome P-450 Enzyme SystemAnimalsAnticarcinogenic AgentsDrug InteractionsEnzyme inducerMonocrotalinebiologyCytochrome P450General MedicineGlutathioneRatschemistryBiochemistryLiverToxicitybiology.proteinCarcinogensXenobioticDrug metabolismFood ScienceFood and chemical toxicology : an international journal published for the British Industrial Biological Research Association
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HLA association is different in children and adults with severe acquired aplastic anemia

2007

Background Severe aplastic anemia (SAA) is defined as pancytopenia caused by bone marrow failure. The pathogenesis of SAA is thought to involve autoimmune processes. Increased susceptibility to autoimmunity has been shown to be associated with several different HLA alleles. In SAA, few large studies based on data mainly from adults describe a positive HLA correlation with HLA-DR2 (DRB1*15) and HLA-B14. Procedure This study explored the HLA constitution of 181 children with SAA who were enrolled in the prospective multi-center study SAA94 between January 1994 and January 2002. The control group consisted of 303 healthy individuals of comparable demographic background. Allelic frequencies bet…

MaleAdolescentHuman leukocyte antigenmedicine.disease_causeAutoimmunityPathogenesisHLA-B14 AntigenHLA Antigenshemic and lymphatic diseasesmedicineHumansHLA-DR2 AntigenProspective StudiesChildAllelesbusiness.industryBone marrow failureAnemia AplasticInfantHematologymedicine.diseasePancytopeniaPathophysiologyExact testOncologyHLA-B AntigensChild PreschoolPediatrics Perinatology and Child HealthImmunologyCohortFemalebusinessPediatric Blood & Cancer
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Rat liver endothelial and Kupffer cell-mediated mutagenicity of polycyclic aromatic hydrocarbons and aflatoxin B1.

1990

The ability of isolated rat liver endothelial and Kupffer cells to activate benzo(a)pyrene (BP), trans-7,8-dihydroxy-7,8-dihydrobenzo(a)pyrene (DDBP), trans-1,2-dihydroxy-1,2-dihydrochrysene (DDCH), and aflatoxin B1 (AFB1) to mutagenic metabolites was assessed by means of a cell-mediated bacterial mutagenicity assay and compared with the ability of parenchymal cells to activate these compounds. Endothelial and Kupffer cells from untreated rats were able to activate AFB1 and DDBP; DDBP was activated even in the absence of an NADPH-generating system. Pretreating the animals with Aroclor 1254 strongly enhanced the mutagenicity of the dihydrodiol, whereas the mutagenicity of AFB1 showed a sligh…

MaleAflatoxin B1EndotheliumKupffer CellsLiver cytologyHealth Toxicology and MutagenesisIn Vitro TechniquesBiologychemistry.chemical_compoundAflatoxinsmedicineOrganoidAnimalsPolycyclic CompoundsTestosteroneBiotransformationCarcinogenKupffer cellPublic Health Environmental and Occupational Healthfood and beveragesRats Inbred StrainsRatsmedicine.anatomical_structureLiverBiochemistrychemistryBenzopyreneToxicityMicrosomeEndothelium VascularResearch ArticleMutagensEnvironmental Health Perspectives
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Interspecies differences in cancer susceptibility and toxicity.

1999

One of the most complex challenges to the toxicologist represents extrapolation from laboratory animals to humans. In this article, we review interspecies differences in metabolism and toxicity of heterocyclic amines, aflatoxin B1, 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD), and related compounds, endocrine disrupters, polycyclic aromatic hydrocarbons, tamoxifen, and digitoxin. As far as possible, extrapolations to human toxicity and carcinogenicity are performed. Humans may be more susceptible to the carcinogenic effect of heterocyclic amines than monkeys, rats, and mice. Especially, individuals with high CYP1A2 and 3A4 activities and the rapid acetylator phenotype may be expected to have …

MaleAflatoxinAflatoxin B1Cardiotonic AgentsPolychlorinated DibenzodioxinsAntineoplastic Agents HormonalHamsterEndocrine SystemPharmacologyToxicologychemistry.chemical_compoundMiceDigitoxinSpecies SpecificityHeterocyclic CompoundsCricetinaeNeoplasmsBenzo(a)pyreneAnimalsHumansPharmacology (medical)General Pharmacology Toxicology and PharmaceuticsCarcinogenCYP1A2EstrogensGlutathioneAntiestrogenRatsTamoxifenBenzo(a)pyrenechemistryToxicityMicrosomes LiverFemaleDisease SusceptibilityRabbitsDrug metabolism reviews
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Mechanisms involved in the chemoprevention of flavonoids

2001

Flavonoids, widespread in edible plants, have been studied extensively for their anticarcinogenic properties. However, only few studies have been done with these constituents being administered by the dietary route. In our research, the effects of feeding rats with flavone, flavanone, tangeretin, and quercetin were investigated on two steps of aflatoxin B1 (AFB1)-induced hepatocarcinogenesis (initiation and promotion). Nonpolar flavonoids such as flavone, flavanone and tangeretin administered through the initiation period, decreased the number of -gamma-glutamyl transpeptidase-preneoplastic foci. In the same conditions of administration, quercetin, a polyhydroxylated flavonoid, showed no pr…

MaleAflatoxinAflatoxin B1[SDV]Life Sciences [q-bio]Clinical BiochemistryFlavonoidChemopreventionBiochemistryFlavones03 medical and health scienceschemistry.chemical_compoundTangeretinCytosolLiver Neoplasms Experimental0302 clinical medicineAnimalsAnticarcinogenic Agentsheterocyclic compoundsRats WistarComputingMilieux_MISCELLANEOUS030304 developmental biologyFlavonoidschemistry.chemical_classification0303 health sciencesDNAGeneral MedicineGlutathioneFlavonesGlutathioneCANCERDietRats3. Good health[SDV] Life Sciences [q-bio]LiverchemistryBiochemistryPhenobarbital030220 oncology & carcinogenesisFlavanonesCarcinogensChemoprotectiveMolecular MedicineQuercetinQuercetinFlavanoneBioFactors
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DNA binding, adduct characterisation and metabolic activation of aflatoxin B1 catalysed by isolated rat liver parenchymal, Kupffer and endothelial ce…

1991

In vitro studies with rat liver parenchymal, Kupffer and endothelial cells isolated from male Sprague-Dawley rats were undertaken to investigate cell-specific bioactivation of aflatoxin B1, DNA binding and adduct formation. In the mutagenicity studies, using homogenates of all three separated liver cell populations (co-incubated with NADP+ and glucose-6-phosphate as cofactors for the cytochrome P-450 monooxygenase system) parenchymal, Kupffer and endothelial cells were able to activate aflatoxin B1 to a metabolite mutagenic to Salmonella typhimurium TA 98. In the case of nonparenchymal cells (i.e. Kupffer and endothelial cells) 10-fold higher concentrations of aflatoxin B1 had to be used to…

MaleAflatoxinAroclorsAflatoxin B1Kupffer CellsHealth Toxicology and MutagenesisMetaboliteBiologyIn Vitro TechniquesToxicologychemistry.chemical_compoundmedicineAnimalsTestosteroneEndotheliumBiotransformationMutagenicity TestsLiver cellKupffer cellfood and beveragesRats Inbred StrainsGeneral MedicineDNAMonooxygenaseChlorodiphenyl (54% Chlorine)In vitroRatsEndothelial stem cellmedicine.anatomical_structurechemistryBiochemistryLiverMicrosomeArchives of toxicology
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Age differences in the role of the cannabinoid type 1 receptor on glutamatergic neurons in habituation and spatial memory acquisition

2015

Abstract Aims Aging is typically linked with a decline in memory performance and alterations in neural integrity. In pathological aging such as Alzheimer's disease, these effects are aggravated. Studies using cannabinoid CB1 receptor-deficient mice have shown a role of the endocannabinoid system in memory processing and neuroprotection. As the CB1 receptor is expressed in various neuronal populations, in this study, we aimed at investigating the consequences of CB1 receptor gene inactivation in cortical glutamatergic neurons in mice (Glu-CB1-KO) in regard to age-related alterations in spatial memory performance. Main methods Juvenile (5.5–7.5 weeks), adult (5.5–7 months), and old (11.5–14 m…

MaleAgingCannabinoid receptormedicine.medical_treatmentMorris water navigation taskBiologyGeneral Biochemistry Genetics and Molecular BiologyMiceGlutamatergicGlutamatesReceptor Cannabinoid CB1medicineAnimalsMemory impairmentGeneral Pharmacology Toxicology and PharmaceuticsHabituationHabituation PsychophysiologicMaze LearningSpatial MemoryMice KnockoutNeuronsThigmotaxisLearning DisabilitiesGeneral MedicineEndocannabinoid systemMice Inbred C57BLnervous systemlipids (amino acids peptides and proteins)CannabinoidNeurosciencePsychomotor Performancepsychological phenomena and processesLife Sciences
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