Search results for "BIOTRANSFORMATION"

showing 10 items of 183 documents

Inactivation of electrophilic metabolites by glutathione S-transferases and limitation of the system due to subcellular localization

1977

Benzo(a)pyrene was activated to metabolites mutagenic for Salmonella typhimurium TA 98 by liver microsomes from control and phenobarbital treated mice. Under these conditions benzo(a)pyrene 4,5-oxide accounts for most of the mutagenicity. We have therefore investigated (1) the conjugation of benzo(a)pyrene 4,5-oxide with glutathione and (2) the effect of glutathione on the mutagenicity of benzo(a)pyrene.

MaleSalmonella typhimuriumendocrine systemHealth Toxicology and MutagenesisMutagenToxicologymedicine.disease_causeMicechemistry.chemical_compoundCytosolBiotransformationpolycyclic compoundsmedicineAnimalsBenzopyrenesBiotransformationGlutathione Transferasebiologyfungifood and beveragesGeneral MedicineGlutathioneSubcellular localizationGlutathioneCytosolGlutathione S-transferaseBenzo(a)pyrenechemistryBiochemistryMicrosomes Liverbiology.proteinPyreneMutagensArchives of Toxicology
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The effect of dietary imbalances on the activation of benzo[a]pyrene by the metabolizing enzymes from rat liver.

1987

Abstract Male Sprague-Dawley rats (70–80 g) were fed ad libitum a standard control diet (22% casein, 5% lard), or a high lipid diet (30% lard) or a low protein diet (6% casein) or a standard diet containing 50 ppm phenoclor DP6. After 6 weeks on these diets, the cytochrome P-450 microsomal content, the benzo[ a ]pyrene monooxygenase (BaP-MO) and the epoxide hydrolase (EH) were assayed. The formation of mutagenic B(a)P metabolites which covalently bind with DNA was compared. The activity of BaP-MO and of EH were increased by the high lipid diet (+27% and 106% respectively) and by the phenoclor DP6 treatment (+63% and 400% respectively), compared to the standard diet. In animals fed a low pro…

MaleSalmonella typhimuriummedicine.medical_treatmentchemistry.chemical_compoundLow-protein dietCaseinmedicineBenzo(a)pyreneAnimalsFood scienceEpoxide hydrolaseBenzopyrene HydroxylaseCarcinogenBiotransformationEpoxide HydrolasesCocarcinogenesisChemistryMutagenicity TestsRats Inbred StrainsGeneral MedicineMonooxygenaseDietary FatsPolychlorinated BiphenylsRatsBiochemistryBenzo(a)pyreneMicrosomeMicrosomes LiverPyreneAryl Hydrocarbon HydroxylasesDietary ProteinsDNA DamageMutation research
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Bisdihydrodiols, rather than dihydrodiol oxides, are the principal microsomal metabolites of tumorigenic trans-3,4-dihydroxy-3,4-dihydrodibenz[a,h]an…

1994

Several studies on metabolism and biological activity of tumorigenic dibenz[a,h]anthracene (DBA) and its derivatives have led to the conclusion that the M-region dihydrodiol, trans-3,4-dihydroxy-3,4-dihydro-DBA (DBA-3,4-dihydrodiol), is the precursor of the ultimate mutagenic and tumorigenic metabolite of DBA with the presumed structure of a bay-region dihydrodiol oxide. Incubations of DBA-3,4-dihydrodiol (50 microM) with the microsomal hepatic fraction of Sprague-Dawley rats pretreated with Aroclor 1254 yielded more than 13 metabolites upon separation by HPLC. anti-3,4-Dihydroxy-1,2-epoxy-1,2,3,4-tetrahydro-DBA [0.27 nmol/(nmol of P450.15 min)] could be identified for the first time by UV …

MaleStereochemistryMetaboliteToxicologyHigh-performance liquid chromatographyRats Sprague-Dawleychemistry.chemical_compoundStructure-Activity Relationshippolycyclic compoundsBenz(a)AnthracenesAnimalsEpoxide hydrolaseCarcinogenBiotransformationChromatography High Pressure LiquidMutagenicity TestsDiastereomerBiological activityRats Inbred StrainsGeneral MedicineMetabolismRatsModels StructuralchemistryMicrosomeMicrosomes LiverChemical research in toxicology
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Intestinal transport of cefuroxime axetil in rats: absorption and hydrolysis processes.

2002

Studies were performed using three cefuroxime axetil solutions (11.8, 118 and 200 microM) in three selected intestinal segments and one cefuroxime axetil solution (118 microM) in colon of anaesthetized rats. First-order absorption rate pseudoconstants, k(ap) and effective permeability coefficients, P(eff), were calculated in each set. Absorption of cefuroxime axetil can apparently be described as a carrier-mediated transport, which obeys Michaelis-Menten and first order kinetics in the proximal segment of the small intestine and a passive diffusion mechanism in the mean and distal segments. The absorption kinetic parameters for cefuroxime axetil were obtained: Vm=0.613 (0.440) microM min-1;…

MaleStereochemistryPharmaceutical ScienceModels BiologicalIntestinal absorptionPharmacokineticsmedicineAnimalsProdrugsIntestinal MucosaRats WistarBiotransformationAntibacterial agentCefuroximeIntestinal permeabilityChromatographyChemistryHydrolysisBiological TransportProdrugmedicine.diseaseSmall intestineCephalosporinsRatsPerfusionmedicine.anatomical_structureIntestinal AbsorptionCefadroxilCefuroximeAlgorithmsmedicine.drugInternational journal of pharmaceutics
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Activation of propane 2-nitronate to a genotoxicant in V79-derived cell lines engineered for the expression of rat hepatic sulfotransferases

1999

2-Nitropropane (2-NP) is a genotoxic hepatocarcinogen in rats. The genotoxicity of the compound has been attributed to a sulfotransferase-mediated formation of DNA-reactive species from the anionic form of 2-NP, propane 2-nitronate (P2N). Several observations have suggested that sulfotransferases (SULTs) 1A1 and/or 1C1 may be important in the activation of P2N to a genotoxicant in rat liver, but a definite proof is lacking. In order to identify the sulfotransferase(s) of rat liver that are capable of activating P2N, we have investigated the genotoxicity of P2N in various V79-derived cell lines engineered for expression of individual forms of rat hepatic sulfotransferases. Genotoxicity was a…

MaleSulfotransferaseDNA RepairDNA repairHealth Toxicology and MutagenesisHamstermedicine.disease_causeCell LineNitroparaffinsPropanechemistry.chemical_compoundCricetulusCricetinaeGeneticsmedicineAnimalsRats WistarBiotransformationchemistry.chemical_classificationRatsEnzymeLiverBiochemistrychemistryCell culture2-NitropropaneCarcinogensHydroxysteroidSulfotransferasesGenotoxicityMutagensMutation Research/Genetic Toxicology and Environmental Mutagenesis
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Sulfotransferase-mediated chlorination of 1-hydroxymethylpyrene to a mutagen capable of penetrating indicator cells.

1990

Methylated polycyclic aromatic hydrocarbons are common in the human environment. Many of them are stronger carcinogens than their purely aromatic congeners. They may be metabolized to benzylic alcohols. We report here on biochemical and toxicological characteristics of 1-hydroxymethylpyrene (HMP), a typical representative of this class of compounds. Rat liver cytosol, fortified with 3'-phosphoadenosine-5'-phosphosulfate, converted HMP into its sulfate ester (HMPS), HMPS bound covalently to isolated DNA. In physiological buffer at 37 degrees C, HMPS had a half-life of 2 min, the major decomposition product being HMP. Thus, cyclic activation is possible. When Cl- anions were present at physio…

MaleSulfotransferaseHealth Toxicology and MutagenesisMutagenIn Vitro Techniquesmedicine.disease_causeAdductchemistry.chemical_compoundBiotransformationChloridesmedicineAnimalsCarcinogenBiotransformationchemistry.chemical_classificationPyrenesMutagenicity TestsCell MembranePublic Health Environmental and Occupational HealthRats Inbred StrainsRatsEnzymechemistryBiochemistryLiverPyreneSulfotransferasesDNAResearch ArticleMutagensEnvironmental Health Perspectives
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Kinetics of tienilic acid bioactivation and functional generation of drug–protein adducts in intact rat hepatocytes

2005

13 pages; Drug-induced autoimmune hepatitis is among the most severe hepatic idiosyncratic adverse drug reactions. Considered multifactorial, the disease combines immunological and metabolic aspects, the latter being to date much better known. As for many other model drugs, studies on tienilic acid (TA)-induced hepatitis have evidenced the existence of bioactivation during the hepatic oxidation of the drug, allowing the identification of the neoantigen of anti-LKM2 autoantibodies and the pathway responsible for its formation. However, most of these results are based on the use of microsomal fractions whose relevance to the liver in vivo still needs to be established. In the more complex int…

MaleTicrynafen[SDV.BC]Life Sciences [q-bio]/Cellular BiologyAutoimmune hepatitisPlasma protein bindingHydroxylationBiochemistryRats Sprague-Dawley03 medical and health scienceschemistry.chemical_compound0302 clinical medicineIn vivoCYP[SDV.BBM] Life Sciences [q-bio]/Biochemistry Molecular BiologymedicineAnimalsPrimary cultured hepatocytesTienilic acid[SDV.BBM]Life Sciences [q-bio]/Biochemistry Molecular BiologyCytochrome P450 Family 2[SDV.BC] Life Sciences [q-bio]/Cellular BiologyBiotransformationCells Cultured030304 developmental biologyPharmacologyHepatitis0303 health sciencesDrug bioactivationChemistryGlutathionemedicine.diseaseGlutathioneIn vitroRats3. Good health[SDV.TOX] Life Sciences [q-bio]/Toxicologymedicine.anatomical_structureSteroid 16-alpha-HydroxylaseBiochemistryTienilic acid[SDV.TOX]Life Sciences [q-bio]/Toxicology030220 oncology & carcinogenesisHepatocyteHepatocytesAryl Hydrocarbon HydroxylasesDrug–protein adductsProtein Bindingmedicine.drugBiochemical Pharmacology
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Metabolism of metyrapone by a soluble enzyme system in rat liver.

1970

The formation of a metabolite of metyrapone by an oxygen sensitive soluble enzyme system in rat liver 105,000×g supernatant has been demonstrated. Enzyme activity requires the intact keto function of metyrapone.

MaleUltraviolet RaysMetabolitechemistry.chemical_elementOxygenchemistry.chemical_compoundEnzyme systemmedicineAnimalsBiotransformationPharmacologyMetyraponebiologyChemistryGeneral MedicineMetabolismMetyraponeEnzyme assayRatsBiochemistryLiverSpectrophotometryRat liverbiology.proteinMicrosomes LiverChromatography Thin LayerOxidoreductasesFunction (biology)NADPmedicine.drugNaunyn-Schmiedebergs Archiv fur Pharmakologie
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Long-term expression of differentiated functions in hepatocytes cultured in three-dimensional collagen matrix.

1998

Hepatocytes entrapped in collagen gel and cultured in serum-free conditions survived longer than cells cultured on plastic (5 days vs. 3 weeks), showed fewer signs of early cell senescence (no increase in c-fos oncoprotein expression), and maintained the expression of differentiated hepatic metabolic functions over a longer period of time. Cells cultured in collagen gels retained their ability to respond to hormones. The insulin-stimulated glycogen synthesis rate remained fairly constant during 18 days in culture (between 5.4 +/- 0.37 and 9 +/- 2.7 nmol glucose/h/microg DNA). Collagen-cultured hepatocytes recovered glycogen stores to levels similar to those found in liver, or in hepatocytes…

Malemedicine.medical_specialtyPhysiologyCellular differentiationClinical BiochemistryCell Culture TechniquesIsozymeCulture Media Serum-FreeRats Sprague-Dawleychemistry.chemical_compoundCytochrome P-450 Enzyme SystemInternal medicinemedicineAnimalsInsulinUreaRNA MessengerEnzyme inducerGlycogen synthaseBiotransformationCells CulturedbiologyGlycogenReverse Transcriptase Polymerase Chain ReactionGenes fosCell DifferentiationCell BiologyGlutathioneMolecular biologyExtracellular MatrixLiver GlycogenRatsIsoenzymesEndocrinologychemistryGene Expression RegulationLiverPharmaceutical PreparationsCell cultureEnzyme InductionMethylcholanthrenebiology.proteinMicrosomes LiverHepatocytesCollagenProto-Oncogene Proteins c-fosTranscription Factors
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Sympathetic Nerve Stimulation on the perfused rat heart. Affinities of N-methylatropine and pirenzepine at pre- and postsynaptic muscarine receptors.

1982

Rat isolated hearts with the sympathetic nerves attached were perfused with (-)-3H-noradrenaline in order to label the storage vesicles of the adrenergic nerves. Release was induced either by electrical stimulation of the nerves (3 Hz, 1 min) or by perfusion with high K+ solution (54 mM). The overflow of 3H-noradrenaline and its metabolites was determined by liquid scintillation counting after separation of the compounds by column chromatography. The experimental conditions ensured a minor contribution of 3H-metabolites to the evoked total tritium overflow. The release of 3H-noradrenaline evoked by nerve stimulation or high K+ solution was decreased in the presence of the muscarinic agonist…

Malemedicine.medical_specialtySympathetic Nervous SystemStimulationIn Vitro TechniquesPiperazineschemistry.chemical_compoundNorepinephrineColumn chromatographyPostsynaptic potentialInternal medicinemedicineAnimalsMethacholine CompoundsReceptors CholinergicAtropine DerivativesReceptorBiotransformationPharmacologyBenzodiazepinonesMuscarineMyocardiumHeartRats Inbred StrainsGeneral MedicinePirenzepinePirenzepineReceptors MuscarinicElectric StimulationRatsEndocrinologychemistryMethacholineFemalePerfusionmedicine.drugNaunyn-Schmiedeberg's archives of pharmacology
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