Search results for "BN"

showing 10 items of 1136 documents

Cytogenetic analysis of epithelial renal-cell tumors: Relationship with a new histopathological classification

1993

Renal-cell carcinomas (RCC) are clinically, histologically and cytogenetically very heterogeneous. The present histological WHO classification shows no clear correlation between histologic subtypes and specific chromosomal abnormalities. In 1986, a new classification was proposed by Thoenes and Storkel based on the cell type from which the tumor arises. They distinguish S cell types: clear-cell, chromophilic, chromophobic, ductus Bellini and oncocytic. Results of 105 primary tumors show that, in this new classification, there is a correlation between different subtypes of renal-cell tumor and specific chromosomal abnormalities at a microscopic and/or molecular level. The clear-cell compact …

Cancer ResearchPathologymedicine.medical_specialtyMonosomyCell typeCARCINOMAChromosome DisordersHistogenesisBiologyPolysomy 7Loss of heterozygositymedicineHumansCarcinoma Renal CellChromosome AberrationsChromosome 7 (human)PolysomyPloidiesABNORMALITIESCytogeneticsDNA Neoplasmmedicine.diseaseKidney NeoplasmsONCOCYTOMASOncologyTISSUEKaryotypingInternational Journal of Cancer
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No evidence of EMAST in whole genome sequencing data from 248 colorectal cancers.

2021

Microsatellite instability (MSI) is caused by defective DNA mismatch repair (MMR), and manifests as accumulation of small insertions and deletions (indels) in short tandem repeats of the genome. Another form of repeat instability, elevated microsatellite alterations at selected tetranucleotide repeats (EMAST), has been suggested to occur in 50% to 60% of colorectal cancer (CRC), of which approximately one quarter are accounted for by MSI. Unlike for MSI, the criteria for defining EMAST is not consensual. EMAST CRCs have been suggested to form a distinct subset of CRCs that has been linked to a higher tumor stage, chronic inflammation, and poor prognosis. EMAST CRCs not exhibiting MSI have b…

Cancer Researchcongenital hereditary and neonatal diseases and abnormalities3122 Cancerscolorectal cancersuolistosyövätBiologymikrosatelliititmedicine.disease_causeGenomeDNA sequencingEMAST03 medical and health sciences0302 clinical medicineINDEL MutationGeneticsmedicineHumansGenetic TestingIndelneoplasmsGeneticsWhole genome sequencingnext generation sequencingMutationDNA-analyysiWhole Genome Sequencing1184 Genetics developmental biology physiologyMicrosatellite instabilitymedicine.diseasedigestive system diseases3. Good health030220 oncology & carcinogenesisgenome sequencing dataMicrosatellitesyöpätauditDNA mismatch repaircolorectal cancersColorectal NeoplasmsMicrosatellite RepeatsGenes, chromosomescancerREFERENCES
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Involvement of the chromosomal region 11q13 in renal oncocytoma: case report and literature review.

1997

Renal oncocytomas comprise a cytogenetically heterogeneous group of tumors consisting potentially of cytogenetic distinguishable subgroups. Review of the literature revealed loss of chromosome 1 and Y as a possible anomaly for at least one subset of oncocytomas. The frequent finding of rearrangements involving chromosome 11 band q13 characterizes another subset of oncocytomas. We report the cytogenetic and pathological features of a renal oncocytoma diagnosed in a 72-year-old woman and found a t(9;11)(p23;q13) as a consistent abnormality. This supports the idea that translocations involving 11q13 define a further subset of oncocytoma. (C) Elsevier Science Inc., 1997.

Cancer Researchmedicine.medical_specialtyPathologyChromosomes Human Pair 21Chromosomes Human Pair 20Chromosomal translocationChromosome DisordersBiologyurologic and male genital diseasesTranslocation GeneticGeneticsmedicineAdenoma OxyphilicHumansOncocytomaRenal oncocytomaCYTOGENETIC ABNORMALITIESMolecular BiologyAgedGeneticsChromosome AberrationsChromosomes Human Pair 11CytogeneticsChromosomeLOCALIZATIONKaryotypemedicine.diseaseTUMORSGENEKidney NeoplasmsChromosome BandingChromosomal regionFemaleAbnormalityChromosomes Human Pair 9Cancer genetics and cytogenetics
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Influence of segmental chromosome abnormalities on survival in children over the age of 12 months with unresectable localised peripheral neuroblastic…

2014

Background: The prognostic impact of segmental chromosome alterations (SCAs) in children older than 1 year, diagnosed with localised unresectable neuroblastoma (NB) without MYCN amplification enrolled in the European Unresectable Neuroblastoma (EUNB) protocol is still to be clarified, while, for other group of patients, the presence of SCAs is associated with poor prognosis. Methods: To understand the role of SCAs we performed multilocus/pangenomic analysis of 98 tumour samples from patients enrolled in the EUNB protocol. Results: Age at diagnosis was categorised into two groups using 18 months as the age cutoff. Significant difference in the presence of SCAs was seen in tumours of patients…

Cancer Researchmedicine.medical_specialtyPathologyMYCN AmplificationKaplan-Meier EstimateunresectableGastroenterologyDisease-Free Survivalsegmental chromosome alterationsNeuroblastomaneuroblastomaDDX1FISHaCGHOlder patientsPeripheral Nervous System NeoplasmsInternal medicineNeuroblastomaMYCNmedicineHumansMultiplex ligation-dependent probe amplificationGainChromosome AberrationsOncogene ProteinsComparative Genomic HybridizationN-Myc Proto-Oncogene Proteinbusiness.industrySignificant differenceGene AmplificationSegmental Chromosome abnormalitiesInfantNuclear ProteinsChromosomePrognosislocalisedmedicine.diseaseDoenças GenéticasMLPA3. Good healthPeripheralOncologyMycn amplificationClinical StudyHistopathologybusinessBritish Journal of Cancer
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Genotype-first in a cohort of 95 fetuses with multiple congenital abnormalities: when exome sequencing reveals unexpected fetal phenotype-genotype co…

2020

PurposeMolecular diagnosis based on singleton exome sequencing (sES) is particularly challenging in fetuses with multiple congenital abnormalities (MCA). Indeed, some studies reveal a diagnostic yield of about 20%, far lower than in live birth individuals showing developmental abnormalities (30%), suggesting that standard analyses, based on the correlation between clinical hallmarks described in postnatal syndromic presentations and genotype, may underestimate the impact of the genetic variants identified in fetal analyses.MethodsWe performed sES in 95 fetuses with MCA. Blind to phenotype, we applied a genotype-first approach consisting of combined analyses based on variants annotation and …

Candidate genemedicine.medical_specialtyGenotype[SDV]Life Sciences [q-bio]BiologyCongenital AbnormalitiesCohort Studiescomplex traits03 medical and health sciencesFetusMolecular geneticsGenotypemedicineHumansAbnormalities MultipleExomeClinical significancegeneticsGeneGenetic Association StudiesGenetics (clinical)Exome sequencing030304 developmental biologyGenetics0303 health sciencesFetus030305 genetics & hereditySequence Analysis DNAPhenotype[SDV] Life Sciences [q-bio]molecular geneticsreproductive medicine
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Long-term cardiac pro-B-type natriuretic peptide gene delivery prevents the development of hypertensive heart disease in spontaneously hypertensive r…

2011

Background— Diastolic dysfunction associated with high blood pressure (BP) leads to cardiac remodeling and fibrosis and progression to congestive heart failure. B-type natriuretic peptide (BNP) has BP-lowering, antifibrotic, and antihypertrophic properties, which makes BNP an attractive agent for attenuating the adverse cardiac remodeling associated with hypertension. In the current study, we tested the effects of sustained cardiac proBNP gene delivery on BP, cardiac function, and remodeling in spontaneously hypertensive rats (SHR). Methods and Results— We used the myocardium-tropic adeno-associated virus serotype 9 (AAV9) vector to achieve continuously enhanced cardiac rat proBNP expressi…

Cardiac function curvemedicine.medical_specialtymedicine.drug_classBNP; Natyriuretic peptides; hypertensive heart diseaseDiastolePhysiology (medical)Internal medicinemedicineNatriuretic peptideNatyriuretic peptidescardiovascular diseasesVentricular remodelingEjection fractionbusiness.industryheart failure natriuretic peptide hypertension.medicine.diseaseSettore MED/11 - Malattie Dell'Apparato CardiovascolareHypertensive heart diseaseBlood pressureEndocrinologyHeart failurecardiovascular systemCardiologyCardiology and Cardiovascular Medicinebusinesshypertensive heart diseasehormones hormone substitutes and hormone antagonistsBNP
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THE CAUCHY DUAL AND 2-ISOMETRIC LIFTINGS OF CONCAVE OPERATORS

2018

We present some 2-isometric lifting and extension results for Hilbert space concave operators. For a special class of concave operators we study their Cauchy dual operators and discuss conditions under which these operators are subnormal. In particular, the quasinormality of compressions of such operators is studied.

Cauchy dual operatorsubnormal operatorPure mathematics[MATH.MATH-FA]Mathematics [math]/Functional Analysis [math.FA]01 natural sciencessymbols.namesakeFOS: Mathematics0101 mathematicsconcave operatorMathematics47A05 47A15 47A20 47A63Mathematics::Functional AnalysisMathematics::Operator AlgebrasApplied Mathematics010102 general mathematicsHilbert spaceCauchy distributionExtension (predicate logic)Special class2-isometric liftingsA-contractionFunctional Analysis (math.FA)Dual (category theory)Mathematics - Functional Analysis010101 applied mathematicssymbolsAnalysis
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A GFP-tagged Muscleblind C protein isoform reporter construct

2010

Drosophila muscleblind (mbl), the ortholog of human Muscleblind-like 1 (MBNL1) gene involved in Myotonic Dystrophy (DM), gives raise to protein isoforms MblA to G. The specific functions and subcellular distribution of isoforms are still largely unknown. To overcome the lack of isoform-specific antibodies we generated transgenic flies that express a GFP:MblC fusion protein under the control of the Gal4/UAS system. The reporter fusion protein was able to functionally complement mbl loss of function mutations, demonstrating activity, and accumulated predominantly in adult muscle nuclei. The fluorescent nature of the reporter makes it appropriate for live imaging detection of MblC protein isof…

Cell NucleusProtein isoformGene isoformMusclesRecombinant Fusion ProteinsTransgeneGreen Fluorescent ProteinsNuclear ProteinsBiologyMolecular biologyFusion proteinGreen fluorescent proteinAnimals Genetically Modifiedchemistry.chemical_compoundchemistryGenes ReporterLive cell imagingInsect ScienceAnimalsDrosophila ProteinsMBNL1DrosophilaGenetic EngineeringGeneFly
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TGF-β superfamily signaling is essential for tooth and hair morphogenesis and differentiation

2007

Members of the transforming growth factor beta (TGF-beta) superfamily of signaling molecules are involved in the regulation of many developmental processes that involve the interaction between mesenchymal and epithelial tissues. Smad7 is a potent inhibitor of many members of the TGF-beta family, notably TGF-beta and activin. In this study, we show that embryonic overexpression of Smad7 in stratified epithelia using a keratin 5 promoter, results in severe morphogenetic defects in skin and teeth and leads to embryonic and perinatal lethality. To further analyze the functions of Smad7 in epithelial tissues of adult mice, we used an expression system that allowed a controlled overexpression of …

Cell signalingmedicine.medical_specialtyHistologyMorphogenesisEmbryonic DevelopmentMice TransgenicNerve Tissue ProteinsBiologySmad7 ProteinPathology and Forensic MedicineNestinMice03 medical and health sciences0302 clinical medicineIntermediate Filament ProteinsGenes ReporterTransforming Growth Factor betaInternal medicineMorphogenesismedicineAnimalsHumansTransgenes030304 developmental biology0303 health sciencesR-SMADIntegrasesintegumentary systemTooth Abnormalities[SDV.BBM.BM]Life Sciences [q-bio]/Biochemistry Molecular Biology/Molecular biologyCell DifferentiationCell BiologyGeneral MedicineHair follicleSurvival AnalysisCell biologyKeratin 5Endocrinologymedicine.anatomical_structureGene Expression RegulationLac OperonTransforming growth factor beta 3030220 oncology & carcinogenesisRabbitsAmeloblastToothHairSignal TransductionTransforming growth factorEuropean Journal of Cell Biology
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Craniomaxillofacial morphology alterations in children, adolescents and adults with neurofibromatosis 1 : a cone beam computed tomography analysis of…

2017

Background Oral manifestations are common in neurofibromatosis 1 (NF1), and include jaws and teeth alterations. Our aim was to investigate the craniomaxillofacial morphology of Brazilian children, adolescents and adults with NF1 using cone beam computed tomography. Material and Methods This study was conducted with 36 Brazilian individuals with NF1 with ages ranging from 4 to 75. The participants were submitted to anamnesis, extra and intraoral exam and cephalometric analysis using cone beam computed tomography. Height of the NF1 individuals was compared to the length of jaws and skull base. The results of the cephalometric measurements of the NF1 group were compared with a control group pa…

Cephalometric analysisAdultMaleCone beam computed tomographycongenital hereditary and neonatal diseases and abnormalitiesNeurofibromatosis 1AdolescentCephalometry03 medical and health sciencesYoung Adult0302 clinical medicinestomatognathic systemmedicineHumansProspective StudiesNeurofibromatosisChildGeneral DentistryneoplasmsAgedAnamnesisOrthodonticsSkull BaseOral Medicine and Pathologybusiness.industryResearchMandible030206 dentistryCone-Beam Computed TomographyMiddle Aged:CIENCIAS MÉDICAS [UNESCO]medicine.diseasenervous system diseasesSkullstomatognathic diseasesmedicine.anatomical_structureOtorhinolaryngologyJawMaxillaCase-Control StudiesChild PreschoolUNESCO::CIENCIAS MÉDICASEarly adolescentsSurgeryFemalebusiness030217 neurology & neurosurgeryBrazil
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