Search results for "Benzylamine"

showing 10 items of 29 documents

ChemInform Abstract: Reaction of 2,2′-Bis(N-methylindolyl) with Dimethyl Acetylenedicarboxylate and Thermally and Photochemically Induced Cyclization…

2010

2,2′-Bis(N-methylindolyl) 1 reacts with dimethyl acetylenedicarboxylate to furnish the 3-dimethyl maleoyl-substituted 2,2′-bisindolyl 2. Compound 2 cyclizes under aluminum trichloride catalysis according to a polar process to give a cyclopenta[2,1-b:3,4-b′]diindole derivative 4. Reaction of compound 4 with benzyl-amine yields the spiro derivative 5. Photochemically-induced 1,6-electrocyclization of compound 2 gives rise to the indolo[2,3-a]carbazole 6 directly, which is readily transformed to the pyrrolo-annelated carbazole 7 by treatment with benzylamine.

Dimethyl acetylenedicarboxylatechemistry.chemical_compoundBenzylaminechemistryCarbazoleAluminum trichlorideGeneral MedicineMedicinal chemistryDerivative (chemistry)CatalysisChemInform
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Reaction of 2,2′-bis(N-methylindolyl) with dimethyl acetylenedicarboxylate and thermally- and photochemically-induced cyclizations of the product

1995

2,2′-Bis(N-methylindolyl) 1 reacts with dimethyl acetylenedicarboxylate to furnish the 3-dimethyl maleoyl-substituted 2,2′-bisindolyl 2. Compound 2 cyclizes under aluminum trichloride catalysis according to a polar process to give a cyclopenta[2,1-b:3,4-b′]diindole derivative 4. Reaction of compound 4 with benzyl-amine yields the spiro derivative 5. Photochemically-induced 1,6-electrocyclization of compound 2 gives rise to the indolo[2,3-a]carbazole 6 directly, which is readily transformed to the pyrrolo-annelated carbazole 7 by treatment with benzylamine.

Dimethyl acetylenedicarboxylatechemistry.chemical_compoundBenzylaminechemistryCarbazoleAluminum trichlorideOrganic ChemistryOrganic chemistryMedicinal chemistryDerivative (chemistry)CatalysisJournal of Heterocyclic Chemistry
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[11C]-DASB microPET imaging in the aged rat: Frontal and meso-thalamic increases in serotonin transporter binding

2011

Whereas molecular imaging studies in the aging human brain have predominantly demonstrated reductions in serotonin transporter (5-HTT) availability, the majority of the rodent studies, using autoradiographic methods, report increases in neural 5-HTT levels with age. To our knowledge, however, no previous rodent studies have assessed this topic in vivo, and therefore it remains unclear whether this discrepancy arises from methodological or inter-species differences. We performed an [(11)C]-DASB microPET study to evaluate the effects of aging on 5-HTT availability in the rat brain. To generate binding potential estimates, quantitative tracer kinetic modeling was applied using the simplified r…

MaleBenzylaminesAgingThalamusDASBSerotonergicBiochemistrychemistry.chemical_compoundEndocrinologyThalamusGeneticsmedicineAnimalsCarbon RadioisotopesMolecular BiologySerotonin transporterSerotonin Plasma Membrane Transport ProteinsbiologyMembrane Transport ProteinsBinding potentialCell BiologyHuman brainAnatomyFrontal LobeRatsmedicine.anatomical_structureFrontal lobechemistryPositron-Emission TomographySerotonin Plasma Membrane Transport Proteinsbiology.proteinNeuroscienceExperimental Gerontology
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iNOS-derived nitric oxide mediates the increase in TFF2 expression associated with gastric damage: role of HIF-1.

2009

Trefoil (TFF) peptides are involved in gastrointestinal mucosal restitution. An hypoxia inducible factor 1 (HIF-1)-dependent induction of TFF genes has been reported in gastric epithelial cells. Nitric oxide (NO) is associated with mucosal damage and modulates HIF-1 activity. The aim of the present study was to analyze the role of iNOS-derived NO in HIF-1alpha stabilization and TFF gene expression in damaged gastric mucosa. Aspirin caused gastric injury that peaked 6 h after dosing and returned to normality at 24 h. iNOS mRNA expression occurs in the corpus in parallel with damage. Blockade of iNOS activity did not modify gastric lesions induced by aspirin but delayed mucosal healing. Aspir…

MaleBenzylaminesAmidinesNitric Oxide Synthase Type IINitric OxideBiochemistryNitric oxideCell LineRats Sprague-Dawleychemistry.chemical_compoundMiceDownregulation and upregulationGene expressionGeneticsGastric mucosamedicineGene silencingAnimalsHumansRNA MessengerEnzyme InhibitorseducationMolecular BiologyDNA Primerseducation.field_of_studyWound HealingAspirinBase SequenceAnti-Inflammatory Agents Non-SteroidalTrefoil factor 2Macrophage ActivationHypoxia-Inducible Factor 1 alpha SubunitCoculture TechniquesRatsUp-RegulationMicroRNAsmedicine.anatomical_structurechemistryCell cultureGastric MucosaCancer researchTrefoil Factor-2Wound healingPeptidesBiotechnologyFASEB journal : official publication of the Federation of American Societies for Experimental Biology
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Inhibition of iNOS activity by 1400W decreases glutamate release and ameliorates stroke outcome after experimental ischemia

2005

Background and purpose. It has been shown that the reversed operation of glutamate transporters when ATP levels fall accounts for most glutamate release induced by severe cerebral ischemia. Nitric oxide (NO) is formed after ischemia and causes ATP depletion. Our purpose is to test if NO release from inducible NO synthase (iNOS) after stroke may cause a delayed glutamate release due to ATP depletion that might underlie progression of the ischemic infarct. We have studied the effect of the highly selective inhibitor of iNOS activity 1400W on brain ATP levels, extracellular glutamate, and stroke outcome after transient focal cerebral ischemia in rats. Methods. To induce focal ischemia, the mid…

MaleBenzylaminesAmino Acid Transport System X-AGIschemiaAmidinesInfarctionDown-RegulationGlutamic AcidNitric Oxide Synthase Type IIL-argininePharmacologyNeuroprotectionNitric oxidelcsh:RC321-571chemistry.chemical_compoundAdenosine TriphosphateWestern blotmedicine.arteryStroke outcomeMedicineAnimalscardiovascular diseasesEnzyme InhibitorsRats Wistarlcsh:Neurosciences. Biological psychiatry. Neuropsychiatrymedicine.diagnostic_testbusiness.industryGlutamate receptorBrainInfarction Middle Cerebral ArteryNitric oxideCerebral Infarctionmedicine.diseaseNeuroprotectionRatsATPStrokeDisease Models AnimalNeuroprotective AgentsTreatment OutcomeNeurologychemistryCytoprotectionIschemic Attack TransientAnesthesiaMiddle cerebral arteryNitric Oxide SynthaseGlutamatebusinessNeurobiology of Disease
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Synthesis and evaluation in rats of the dopamine D2/3 receptor agonist 18F-AMC20 as a potential radioligand for PET

2015

Dopamine D2/3 receptor (D2/3R) agonist PET tracers are better suited for the imaging of synaptic dopaminergic neurotransmission than D2/3R antagonists and may also offer the opportunity to study in vivo the high-affinity state of D2/3R (D2/3RHigh). With the aim to develop 18F-labeled D2/3R agonists suitable for widespread clinical application, we report here on the synthesis and in vitro and in vivo evaluation of a D2/3R agonist ligand from the aminomethyl chromane (AMC) class-(R)-2-[(4- 18Fluorobenzylamino)methyl]chroman-7-ol (18F-AMC20). Methods: In vitro affinities of AMC20 toward dopaminergic receptor subtypes were measured in membrane homogenates prepared from HEK293 cells expressing h…

MaleBenzylaminesChemistry Techniques SyntheticPharmacologyLigandsRats Sprague-DawleyCricetinaeBINDINGRadioligandIN-VIVORacloprideRadiochemistryChemistryDopaminergicBrainStereoisomerismLigand (biochemistry)SERIESADDICTIONDopamine receptorDopamine AgonistsHIGH-AFFINITY STATEmedicine.drugAgonistmedicine.drug_classDRUG-ABUSECHO CellsPOSITRON-EMISSION-TOMOGRAPHYCricetulusFIn vivoDopamine receptor D2medicineAnimalsHumansRadiology Nuclear Medicine and imagingBenzopyransChromansReceptors Dopamine D2F-18Receptors Dopamine D3Biological TransportAgonist tracerRatsKineticsHEK293 CellsPETDopamine receptorRaclopridePositron-Emission TomographyRADIOTRACERSYSTEMJournal of Nuclear Medicine
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The Anticonvulsant FCE 26743 is a Selective and Short-acting MAO-B Inhibitor Devoid of Inducing Properties towards Cytochrome P450-dependent Testoste…

1994

Abstract The effects of the potent anticonvulsant FCE 26743 ((S)-2-(4-(3-fluorobenzyloxy)benzylamino)propionamide) on monoamine oxidase (MAO) activity were measured in-vitro and ex-vivo using rat tissue homogenates. In-vitro, FCE 26743 showed potent and selective inhibitory properties towards liver MAO-B, with IC50 values about 10−7  m for MAO-B and higher than 10−5  m for MAO-A. When determined ex-vivo in brain, the ED50 value for the inhibition of MAO-B was 1·1 mg kg−1 (p.o.) 1 h post-dosing, whereas MAO-A remained virtually unaffected after administration of 60 mg kg−1. Similar effects were seen in liver. Following oral administration of 5 mg kg−1 FCE 26743 to rats, brain MAO-B inhibitio…

MaleBenzylaminesMonoamine Oxidase InhibitorsMonoamine oxidaseMetabolite3003 Pharmaceutical Science10050 Institute of Pharmacology and ToxicologyPharmaceutical Science610 Medicine & healthMice Inbred StrainsIn Vitro TechniquesPharmacologyHydroxylationRats Sprague-DawleyHydroxylationMicechemistry.chemical_compoundCytochrome P-450 Enzyme SystemOral administrationmedicineAnimalsTestosteroneED50PharmacologyAlanineDose-Response Relationship DrugbiologyChemistryBrainCytochrome P450Rats3004 PharmacologyLiverMechanism of actionbiology.protein570 Life sciences; biologyAnticonvulsantsMonoamine oxidase Bmedicine.symptomJournal of Pharmacy and Pharmacology
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Studies on the reliability of a bihyperbolic functional absorption model. II. Phenylalkylamines

1987

Evidence is given that demonstrates the reliability of the bihyperbolic equation, proposed by Pla-Delfina and Moreno, in fitting the correlation between absorption rate constants (ka) found in the small intestine and in the colon of the living anesthetized rat, and partition constants (1/R.F−1), for a series of phenylalkylamines, a group of compounds which differ largely from others which have been tested. Emphasis is laid on the nonexistence of an optimum of lipophilicity for intestinal absorption/partition correlation: This feature makes inapplicable the probabilistic approaches to the reported data.

MaleBenzylaminesPsychotropic DrugsAniline CompoundsPropylaminesSeries (mathematics)ChemistryStereochemistryThermodynamicsRats Inbred StrainsButylaminesModels BiologicalIntestinal absorptionRatsAbsorption rateIntestinal AbsorptionColonic absorptionPhenethylaminesLipophilicityAnimalsPartition (number theory)Pharmacology (medical)General Pharmacology Toxicology and PharmaceuticsAbsorption (chemistry)Reliability (statistics)Journal of Pharmacokinetics and Biopharmaceutics
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(2S,3S)-2-(N,N-dibenzylamino)butane-1,3-diol refined using a multipolar atom model.

2008

The crystal structure of the title compound, C18H23NO2, was determined using the experimental library multipolar atom model. The refinement showed a significant improvement of crystallographic statistical indices when compared with a conventional spherical neutral atom refinement.

Models MolecularBenzylaminesEnergetic neutral atomMolecular Structure010405 organic chemistryChemistryStatistical indexDiolButaneGeneral MedicineCrystal structure010402 general chemistryCrystallography X-Ray01 natural sciencesGeneral Biochemistry Genetics and Molecular Biology3. Good health0104 chemical sciencesCrystallographychemistry.chemical_compound[CHIM.CRIS]Chemical Sciences/Cristallography[CHIM]Chemical SciencesButylene GlycolsComputingMilieux_MISCELLANEOUSAtom modelActa crystallographica. Section C, Crystal structure communications
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Plerixafor is effective and safe for stem cell mobilization in heavily pretreated germ cell tumor patients.

2010

Up to 10% of germ cell tumor patients require salvage high-dose chemotherapy with stem cell support, achieving cure rates in the range of 10-60%. Stem cell mobilization may be difficult in these patients because of multiple lines of treatment known to seriously hamper stem cell recovery. Plerixafor significantly enhances the success of the CD34+ cell harvest, even in cases where prior mobilization attempts have failed. Six germ cell tumor patients provided informed consent and were included in the compassionate use program. All patients were heavily pretreated, with a median of 3.5 prior lines of therapy. All failed prior mobilization with G-CSF in combination with chemotherapy. Five patien…

OncologyAdultCompassionate Use TrialsMalemedicine.medical_specialtyBenzylaminesPlatelet Engraftmentmedicine.medical_treatmentCD34Hematopoietic stem cell transplantationCyclamsYoung AdultTesticular NeoplasmsHeterocyclic CompoundsInternal medicineAntineoplastic Combined Chemotherapy ProtocolsmedicineHumansUltrasonographyTransplantationChemotherapyMobilizationbusiness.industryPlerixaforHematopoietic Stem Cell TransplantationHematologyMiddle AgedNeoplasms Germ Cell and EmbryonalCombined Modality TherapyHematopoietic Stem Cell MobilizationSurgerySeminomamedicine.anatomical_structureStem cellbusinessGerm cellmedicine.drugBone marrow transplantation
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