Search results for "C3"

showing 10 items of 1295 documents

"Table 6" of "Freeze-out radii extracted from three-pion cumulants in pp, p-Pb and Pb-Pb collisions at the LHC"

2014

Mixed charge C3 and c3 in p Pb collisions projected against Q3.

P PB --> PI- XInclusive5020.0C3P PB --> PI+ X
researchProduct

"Table 3" of "Freeze-out radii extracted from three-pion cumulants in pp, p-Pb and Pb-Pb collisions at the LHC"

2014

Same charge C3 and c3 in p Pb collisions projected against Q3.

P PB --> PI- XInclusive5020.0C3P PB --> PI+ X
researchProduct

MCC1019, a selective inhibitor of the Polo-box domain of Polo-like kinase 1 as novel, potent anticancer candidate

2019

Polo-like kinase (PLK1) has been identified as a potential target for cancer treatment. Although a number of small molecules have been investigated as PLK1 inhibitors, many of which showed limited selectivity. PLK1 harbors a regulatory domain, the Polo box domain (PBD), which has a key regulatory function for kinase activity and substrate recognition. We report on 3-bromomethyl-benzofuran-2-carboxylic acid ethyl ester (designated: MCC1019) as selective PLK1 inhibitor targeting PLK1 PBD. Cytotoxicity and fluorescence polarization-based screening were applied to a library of 1162 drug-like compounds to identify potential inhibitors of PLK1 PBD. The activity of compound MC1019 against the PLK1…

PBD Polo box domainMTD maximal tolerance doseCDC25 cell division cycle 25HIF-1α hypoxia-inducible factor 1 αMST microscale thermophoresisIC50 50% inhibition concentrationMFP M phase promoting factorPARP-1 poly(ADP-ribose) polymerase-10302 clinical medicineFOXO forkhead box ONec-1 necrostatin 1CDC2 cell division cycle protein 2 homologGeneral Pharmacology Toxicology and PharmaceuticsMitotic catastropheCDK cyclin-dependent kinase0303 health sciencesChemistryPolo-like kinaseMono-targeted therapyCell cycleBUBR1 budding uninhibited by benzimidazole-related 1Polo box domain030220 oncology & carcinogenesisPLK1 Polo-like kinaseNecroptosisSpindle damagePLK1IHC immunohistochemistryOriginal articleNecroptosisCell cyclePLK1APC/C anaphase-promoting complex/cyclosomePLK3ABC avidin-biotin complexPI propidium iodide03 medical and health sciencesFBS fetal bovine serumPDB Protein Data BankKd the dissociation constantKinase activity030304 developmental biologyAkt/PKB signaling pathwayCell growthlcsh:RM1-950LC3 light chain 3lcsh:Therapeutics. PharmacologyCancer researchDAPKs death-associated protein kinase3-MA 3-methyladenineDAPI 4′6-diamidino-2-phenylindoleSAC spindle assembly checkpointActa Pharmaceutica Sinica B
researchProduct

Through Predictive Personalized Medicine.

2020

Neuroblastoma (NBM) is a deadly form of solid tumor mostly observed in the pediatric age. Although survival rates largely differ depending on host factors and tumor-related features, treatment for clinically aggressive forms of NBM remains challenging. Scientific advances are paving the way to improved and safer therapeutic protocols, and immunotherapy is quickly rising as a promising treatment that is potentially safer and complementary to traditionally adopted surgical procedures, chemotherapy and radiotherapy. Improving therapeutic outcomes requires new approaches to be explored and validated. In-silico predictive models based on analysis of a plethora of data have been proposed by Lomba…

PD-L1medicine.medical_treatmentcomputational modellingHost factorsBioinformaticsSettore BIO/09 - Fisiologialcsh:RC321-57103 medical and health sciencesneuroblastoma0302 clinical medicineIntracellular signaling pathwaysSAFERMedicineSolid tumorlcsh:Neurosciences. Biological psychiatry. Neuropsychiatry030304 developmental biology0303 health sciencesbusiness.industryGeneral NeurosciencePediatric ageImmunotherapySurgical proceduresEditorial030220 oncology & carcinogenesisPersonalized medicineimmunotherapybusinessBrain sciences
researchProduct

Frequency-specific network activity predicts bradykinesia severity in Parkinson’s disease

2021

Highlights • Parallel subnetworks are affected in bradykinesia. • The primary motor and the premotor cortex are common nodes with task-specificity. • Beta activity decreases, gamma activity increases with improvement of bradykinesia. • Subthalamic stimulation reduces beta, increases gamma power in ipsilateral cortex. • Subnetworks act with frequency-specific oscillations.

PPC posterior parietal cortexBradykinesiaParkinson's diseaseDeep brain stimulationCognitive Neurosciencemedicine.medical_treatmentComputer applications to medicine. Medical informaticsR858-859.7FT finger tappingHypokinesiaElectromyographyElectroencephalographyPS pronation-supinationGamma oscillationPremotor cortexCER cerebellumSubthalamic NucleusDeep brain stimulationmedicineHumansRadiology Nuclear Medicine and imagingRC346-429SMA supplementary motor cortexM1 primary motor cortexResting state fMRImedicine.diagnostic_testbusiness.industryRegular ArticleBeta oscillationmedicine.diseasehumanitiesnervous system diseasesParkinson diseaseHG hand graspingSubthalamic nucleusCross-Sectional Studiesmedicine.anatomical_structurePMC premotor cortexNeurologyDLPFC dorsolateral prefrontal cortexFinger tappingStrEM structural equation modellingNeurology. Diseases of the nervous systemNeurology (clinical)businessNeuroscienceSTN subthalamic nucleusNeuroImage: Clinical
researchProduct

Vascular Senescence: A Potential Bridge Between Physiological Aging and Neurogenic Decline

2021

The adult mammalian brain contains distinct neurogenic niches harboring populations of neural stem cells (NSCs) with the capacity to sustain the generation of specific subtypes of neurons during the lifetime. However, their ability to produce new progeny declines with age. The microenvironment of these specialized niches provides multiple cellular and molecular signals that condition NSC behavior and potential. Among the different niche components, vasculature has gained increasing interest over the years due to its undeniable role in NSC regulation and its therapeutic potential for neurogenesis enhancement. NSCs are uniquely positioned to receive both locally secreted factors and adhesion-…

ParabiosisGeneral NeuroscienceNicheNeurogenesisneurogenic nicheNeurosciences. Biological psychiatry. NeuropsychiatryReviewBiologyadult neural stem cellNeural stem cellPhysiological AgingBridge (graph theory)senescence-associated secretory phenotypeAging brainparabiosisHeterochronyNeuroscienceNeuroscienceendothelial cell senescenceRC321-571Frontiers in Neuroscience
researchProduct

G2019S Variation in LRRK2: An Ideal Model for the Study of Parkinson's Disease?

2019

Parkinson’s disease (PD) is the second most common neurodegenerative disorder and has plagued humans for more than 200 years. The etiology and detailed pathogenesis of PD is unclear, but is currently believed to be the result of the interaction between genetic and environmental factors. Studies have found that PD patients with the LRRK2:G2019S variation have the typical clinical manifestations of PD, which may be familial or sporadic, and have age-dependent pathogenic characteristics. Therefore, the LRRK2:G2019S variation may be an ideal model to study the interaction of multiple factors such as genetic, environmental and natural aging factors in PD in the future. This article reviewed the …

Parkinson's diseaseNatural agingDiseaseReviewBioinformatics050105 experimental psychologylcsh:RC321-57103 medical and health sciencesBehavioral Neuroscience0302 clinical medicineG2019s mutationG2019S mutationmedicine0501 psychology and cognitive scienceslcsh:Neurosciences. Biological psychiatry. NeuropsychiatryBiological Psychiatrybusiness.industrydisease modelpathogenesis05 social sciencesLRRK2medicine.diseaseLRRK2nervous system diseasesPsychiatry and Mental healthNeuropsychology and Physiological PsychologyMultiple factorsVariation (linguistics)NeurologyEtiologyParkinson’s diseasebusiness030217 neurology & neurosurgeryNeuroscienceFrontiers in human neuroscience
researchProduct

Imaging of Substantia Nigra in Parkinson's Disease: A Narrative Review.

2021

Parkinson’s disease (PD) is a progressive neurodegenerative disorder, characterized by motor and non-motor symptoms due to the degeneration of the pars compacta of the substantia nigra (SNc) with dopaminergic denervation of the striatum. Although the diagnosis of PD is principally based on a clinical assessment, great efforts have been expended over the past two decades to evaluate reliable biomarkers for PD. Among these biomarkers, magnetic resonance imaging (MRI)-based biomarkers may play a key role. Conventional MRI sequences are considered by many in the field to have low sensitivity, while advanced pulse sequences and ultra-high-field MRI techniques have brought many advantages, partic…

Parkinson's diseaseSettore MED/50 - Scienze Tecniche Mediche ApplicateSubstantia nigraNeurosciences. Biological psychiatry. NeuropsychiatryDiseaseStriatumReview030218 nuclear medicine & medical imaging03 medical and health sciences0302 clinical medicineironneurodegenerative diseasemedicinemagnetic resonance imagingneurodegenerative diseasesmedicine.diagnostic_testPars compactabusiness.industryGeneral NeuroscienceradiomicSettore MED/37 - NeuroradiologiabiomarkersMagnetic resonance imagingmedicine.diseaseparkinsonian disordersnigrosome-1radiomicsParkinson’s diseasebiomarkerSettore MED/26 - NeurologiaDifferential diagnosisneuromelaninbusinessSettore MED/36 - Diagnostica Per Immagini E RadioterapiaNeuroscience030217 neurology & neurosurgeryDiffusion MRIRC321-571Brain sciences
researchProduct

Drosophila Models of Parkinson's Disease: Discovering Relevant Pathways and Novel Therapeutic Strategies

2010

Parkinson's disease (PD) is the second most common neurodegenerative disorder and is mainly characterized by the selective and progressive loss of dopaminergic neurons, accompanied by locomotor defects. Although most PD cases are sporadic, several genes are associated with rare familial forms of the disease. Analyses of their function have provided important insights into the disease process, demonstrating that three types of cellular defects are mainly involved in the formation and/or progression of PD: abnormal protein aggregation, oxidative damage, and mitochondrial dysfunction. These studies have been mainly performed in PD models created in mice, fruit flies, and worms. Among them, Dro…

Parkinson's diseasebiologyved/biologyved/biology.organism_classification_rank.speciesDopaminergicNeuroscience (miscellaneous)Review ArticleDiseasebiology.organism_classificationmedicine.diseaseBioinformaticslcsh:RC346-429PathogenesisPsychiatry and Mental healthmedicineNeurology (clinical)Model organismDrosophilaGenelcsh:Neurology. Diseases of the nervous systemFunction (biology)Parkinson's Disease
researchProduct

Longitudinal 18F-FDG PET and MRI Reveal Evolving Imaging Pathology That Corresponds to Disease Progression in a Patient With ALS-FTD

2019

Single time point positron emission tomography (PET) studies of patients with amyotrophic lateral sclerosis and frontotemporal dementia (ALS-FTD), have demonstrated hypometabolism or hypermetabolism in certain brain regions. To determine whether longitudinal (at baseline and 20.4 months later) PET and magnetic resonance imaging (MRI) reveal evolving brain imaging pathology corresponding to clinical progression in a patient with ALS-FTD, cerebral glucose metabolic rate, cortical thickness (CT) and cortical area (CA) were obtained and symmetric percent change (SPC) for each calculated. The patient had worsening symptoms and signs of bulbar-onset upper motor neuron-predominant ALS as well as l…

Pathologymedicine.medical_specialtyCase ReportLateralization of brain functionlcsh:RC346-429030218 nuclear medicine & medical imagingPrimary progressive aphasia03 medical and health sciences0302 clinical medicineNeuroimagingAphasiaALS-FTDMedicineAmyotrophic lateral sclerosislcsh:Neurology. Diseases of the nervous systemcortical areamedicine.diagnostic_testbusiness.industryMagnetic resonance imagingcortical thicknessmedicine.diseaseaphasiaPETNeurologyNeurology (clinical)medicine.symptombusinessNeurocognitive030217 neurology & neurosurgeryFrontotemporal dementiaMRIFrontiers in Neurology
researchProduct