Search results for "CHEMICALS"

showing 10 items of 991 documents

Essential oil of Foeniculum vulgare subsp. piperitum fruits exerts an anti‑tumor effect in triple‑negative breast cancer cells

2022

At present, the growing spread of tumor cases worldwide renders the research of new promising and selective anticancer drugs urgent. The biological action of extracts of medicinal plants or their essential oils (EOs) is an emerging field of interest, since they could comprise a rich source of phytochemicals that can prove promising. In the present study, the biological activity and mechanism of action of the EO of Foeniculum vulgare subsp. piperitum fruits (FVPEO) were investigated using MTT assays, morphological analyses and western blotting in MDA‑MB231 cells, a triple‑negative breast cancer cell line. The findings revealed that FVPEO could exert strong anticancer effects, causing a dose‑…

Cancer Researchessential oil apoptotic cell deathOncologyphytochemicals antitumor effect breast cancerSettore BIO/10 - BiochimicaGeneticsMolecular MedicineSettore CHIM/06 - Chimica OrganicaMolecular BiologyBiochemistryMolecular Medicine Reports
researchProduct

Indicaxanthin inhibits NADPH oxidase (NOX)-1 activation and NF-κB-dependent release of inflammatory mediators and prevents the increase of epithelial…

2014

Dietary redox-active/antioxidant phytochemicals may help control or mitigate the inflammatory response in chronic inflammatory bowel disease (IBD). In the present study, the anti-inflammatory activity of indicaxanthin (Ind), a pigment from the edible fruit of cactus pear (Opuntia ficus-indica, L.), was shown in an IBD model consisting of a human intestinal epithelial cell line (Caco-2 cells) stimulated by IL-1β, a cytokine known to play a major role in the initiation and amplification of inflammatory activity in IBD. The exposure of Caco-2 cells to IL-1β brought about the activation of NADPH oxidase (NOX-1) and the generation of reactive oxygen species (ROS) to activate intracellular signal…

Cell Membrane PermeabilityPyridinesPyridinemedicine.medical_treatmentInterleukin-1betaMedicine (miscellaneous)Nitric Oxide Synthase Type IIIndicaxanthinNADPH OxidaseInflammatory bowel diseaseIntestinal absorptionAntioxidantschemistry.chemical_compoundSettore BIO/10 - BiochimicaInflammation MediatorCaco-2 CellNutrition and DieteticsNADPH oxidasebiologyNF-kappa BNADPH Oxidase 1OpuntiaCell biologyBetaxanthinsCytokineNADPH Oxidase 1EnterocyteAntioxidantmedicine.symptomInflammation MediatorsReactive Oxygen SpecieIndicaxanthinHumanRedox-active phytochemicalInflammationIn vitro modelmedicineHumansIndicaxanthin Betalain pigments Inflammatory bowel disease Redox-active phytochemicalsInterleukin 8Inflammationbusiness.industryInterleukin-6Interleukin-8NADPH OxidasesInflammatory Bowel DiseasesEnzyme ActivationEnterocyteschemistryIntestinal AbsorptionCaco-2Cyclooxygenase 2BetaxanthinFruitImmunologybiology.proteinCaco-2 CellsbusinessReactive Oxygen SpeciesThe British journal of nutrition
researchProduct

Inhibitory effects oftrans-resveratrol analogs molecules on the proliferation and the cell cycle progression of human colon tumoral cells

2008

International audience; Resveratrol may function as a cancer chemopreventive agent. However, few data are available on the antitumoral activities of its dimer, epsilon-viniferin, also present in human diet. So, the effects of resveratrol, epsilon-viniferin, of their acetylated forms (resveratrol triacetate, epsilon-viniferin pentaacetate) and of vineatrol (a wine grape extract) were compared on human adenocarcinoma colon cells. Resveratrol and resveratrol triacetate inhibit cell proliferation and arrest cell cycle. epsilon-Viniferin and epsilon-viniferin pentaacetate slightly reduce cell proliferation. Vineatrol inhibits cell proliferation and favors an accumulation in the S phase of the ce…

Cell Membrane Permeabilityendocrine system diseasesvineatrolMESH: Cell CycleMESH: DNA ReplicationMESH: Flow CytometryresveratrolResveratrolMESH : Antineoplastic Agents PhytogenicWine grapechemistry.chemical_compoundMESH: Structure-Activity RelationshipMESH: StilbenesStilbenesMESH : Structure-Activity RelationshipMESH: Cell Membrane Permeabilityskin and connective tissue diseasesfood and beveragesDNA NeoplasmMESH : Cell DivisionCell cycleFlow CytometryMESH : Colonic Neoplasmscolon cancerBiochemistryColonic NeoplasmsMESH: Cell Divisioncell cycleMESH : DNA NeoplasmCell Divisionhormones hormone substitutes and hormone antagonistsMESH : DNA ReplicationBiotechnologyDNA ReplicationMESH: XenobioticsMESH: Cell Line TumorMESH : Flow CytometryMESH: Antineoplastic Agents PhytogenicMESH: DNA NeoplasmMESH : XenobioticsBiologyXenobioticsMESH : StilbenesStructure-Activity RelationshipCell Line TumorMESH : Cell Cycle[SDV.BBM] Life Sciences [q-bio]/Biochemistry Molecular BiologyHumansStructure–activity relationship[SDV.BBM]Life Sciences [q-bio]/Biochemistry Molecular Biology[ SDV.BBM ] Life Sciences [q-bio]/Biochemistry Molecular BiologypolyphenolsS phaseMESH: Colonic NeoplasmsMESH: HumansMESH : Cell Line TumorCell growthorganic chemicalsMESH : HumansAntineoplastic Agents PhytogenicchemistryMESH : Cell Membrane PermeabilityAcetylationCell cultureCancer researchFood ScienceMolecular Nutrition & Food Research
researchProduct

All-trans retinoic acid restores gap junctional intercellular communication between oral cancer cells with upregulation of Cx32 and Cx43 expressions …

2012

Objective: All-trans retinoic acid (ATRA) has been demonstrated to inhibit tumor growth by restoration of gap junctional intercellular communication (GJIC) via upregulation of connexin (Cx) expression in some solid tumors. However, the relationship between ATRA and GJIC remains unclear in oral squamous cell carcinoma (OSCC). The aim of this study was to investigate the effect of ATRA on the GJIC function of OSCC. Study design: We measured the effects of ATRA on the viability and cell cycle distribution of SCC9 and Tca8113 OSCC cells. The GJIC function was observed using the scrape-loading dye transfer technique, and the mRNA and protein levels of Cx32 and Cx43 were detected by qRT-PCR, West…

Cell cycle checkpointRetinoic acidConnexinAntineoplastic AgentsTretinoinOdontologíaCell CommunicationConnexinschemistry.chemical_compoundDownregulation and upregulationTretinoinmedicineTumor Cells CulturedHumansRNA MessengerGeneral DentistryneoplasmsMouth neoplasmOral Medicine and Pathologyorganic chemicalsGap JunctionsCell cycle:CIENCIAS MÉDICAS [UNESCO]Ciencias de la saludbiological factorsUp-RegulationGene Expression Regulation Neoplasticstomatognathic diseasesOtorhinolaryngologychemistryConnexin 43ImmunologyCancer cellUNESCO::CIENCIAS MÉDICASCancer researchCarcinoma Squamous CellSurgeryMouth NeoplasmsResearch-Articlemedicine.drug
researchProduct

Comparison between tumors in plants and human beings: Mechanisms of tumor development and therapy with secondary plant metabolites

2019

Abstract Background Human tumors are still a major threat to human health and plant tumors negatively affect agricultural yields. Both areas of research are developing largely independent of each other. Treatment of both plant and human tumors remains unsatisfactory and novel therapy options are urgently needed. Hypothesis The concept of this paper is to compare cellular and molecular mechanisms of tumor development in plants and human beings and to explore possibilities to develop novel treatment strategies based on bioactive secondary plant metabolites. The interdisciplinary discourse may unravel commonalities and differences in the biology of plant and human tumors as basis for rational …

Cellular immunityPhytochemicalsPlant TumorsPhysical CarcinogenesisSecondary MetabolismPharmaceutical ScienceBiologymedicine.disease_cause03 medical and health sciences0302 clinical medicineImmune systemCancer stem cellNeoplasmsDrug DiscoveryBiological CarcinogenesisPlant defense against herbivorymedicineAnimalsHumansPlant ImmunityPlant Physiological PhenomenaPlant Diseases030304 developmental biologyPharmacology0303 health sciencesAntibiotics Antineoplasticfungifood and beveragesPlantsAntineoplastic Agents PhytogenicComplementary and alternative medicineAgrobacterium tumefaciensDrug Resistance Neoplasm030220 oncology & carcinogenesisCancer researchMolecular MedicineCarcinogenesisPhytomedicine
researchProduct

Gray Matter NG2 Cells Display Multiple Ca2+-Signaling Pathways and Highly Motile Processes

2011

NG2 cells, the fourth type of glia in the mammalian CNS, receive synaptic input from neurons. The function of this innervation is unknown yet. Postsynaptic changes in intracellular Ca(2+)-concentration ([Ca(2+)](i)) might be a possible consequence. We employed transgenic mice with fluorescently labeled NG2 cells to address this issue. To identify Ca(2+)-signaling pathways we combined patch-clamp recordings, Ca(2+)-imaging, mRNA-transcript analysis and focal pressure-application of various substances to identified NG2-cells in acute hippocampal slices. We show that activation of voltage-gated Ca(2+)-channels, Ca(2+)-permeable AMPA-receptors, and group I metabotropic glutamate-receptors provo…

Central Nervous SystemAnatomy and PhysiologyVesicular glutamate transporter 1Glycobiologylcsh:MedicineHippocampal formationBiochemistryIon ChannelsTransmembrane Transport ProteinsMice0302 clinical medicinePostsynaptic potentialBiomacromolecule-Ligand Interactionslcsh:ScienceCells CulturedMembrane potential0303 health sciencesMultidisciplinarybiologyReverse Transcriptase Polymerase Chain ReactionDepolarizationNeurochemistryNeurotransmittersCell biologyElectrophysiologymedicine.anatomical_structureNeurologyNeurogliaMedicineProteoglycansNeurochemicalsGlutamateNeurogliaResearch ArticleNervous System PhysiologySignal TransductionCell PhysiologyMotilityNeuroimagingMice TransgenicNeurological System03 medical and health sciencesNeuropharmacologymedicineAnimalsHumansddc:610Biology030304 developmental biologyEndoplasmic reticulumlcsh:RProteinsGamma-Aminobutyric AcidTransmembrane ProteinsLuminescent ProteinsMicroscopy Electronnervous systemMicroscopy FluorescenceSynapsesVesicular Glutamate Transport Protein 1biology.proteinNervous System Componentslcsh:QCalciumPhysiological Processes030217 neurology & neurosurgeryNeurosciencePLoS ONE
researchProduct

AAV vector-mediated overexpression of CB1 cannabinoid receptor in pyramidal neurons of the hippocampus protects against seizure-induced excitoxicity.

2010

The CB1 cannabinoid receptor is the most abundant G-protein coupled receptor in the brain and a key regulator of neuronal excitability. There is strong evidence that CB1 receptor on glutamatergic hippocampal neurons is beneficial to alleviate epileptiform seizures in mouse and man. Therefore, we hypothesized that experimentally increased CB1 gene dosage in principal neurons would have therapeutic effects in kainic acid (KA)-induced hippocampal pathogenesis. Here, we show that virus-mediated conditional overexpression of CB1 receptor in pyramidal and mossy cells of the hippocampus is neuroprotective and moderates convulsions in the acute KA seizure model in mice. We introduce a recombinant a…

Central Nervous SystemCannabinoid receptormedicine.medical_treatmentHippocampuslcsh:MedicineHippocampal formationHippocampuschemistry.chemical_compoundMiceReceptor Cannabinoid CB1Neurobiology of Disease and RegenerationTransgeneslcsh:ScienceNeuronsRecombination GeneticMultidisciplinaryBehavior AnimalNeuromodulationmusculoskeletal neural and ocular physiologyfood and beveragesNeurochemistryGenomicsGene TherapyDependovirusEndocannabinoid systemCell biologyFunctional GenomicsNeurologyHomeostatic MechanismsMedicinelipids (amino acids peptides and proteins)Viral VectorsNeurochemicalsGenetic EngineeringResearch ArticleBiotechnologyKainic acidGenetic VectorsGreen Fluorescent ProteinsNeurophysiologyBiologyMicrobiologyVector BiologyGlutamatergicGenomic MedicineSeizuresmedicineGeneticsAnimalsBiologyEpilepsyIntegrasesDentate gyruslcsh:RMolecular biologyMice Inbred C57BLchemistryGene Expression Regulationnervous systemGenetics of DiseaseSynapseslcsh:QCannabinoidGene FunctionMolecular NeuroscienceAnimal GeneticsTransgenicsNeuroscienceEndocannabinoidsPLoS ONE
researchProduct

Hypothalamic S-Nitrosylation Contributes to the Counter-Regulatory Response Impairment following Recurrent Hypoglycemia

2013

http://www.ncbi.nlm.nih.gov/pubmed/23894333; International audience; AIMS: Hypoglycemia is a severe side effect of intensive insulin therapy. Recurrent hypoglycemia (RH) impairs the counter-regulatory response (CRR) which restores euglycemia. During hypoglycemia, ventromedial hypothalamus (VMH) production of nitric oxide (NO) and activation of its receptor soluble guanylyl cyclase (sGC) are critical for the CRR. Hypoglycemia also increases brain reactive oxygen species (ROS) production. NO production in the presence of ROS causes protein S-nitrosylation. S-nitrosylation of sGC impairs its function and induces desensitization to NO. We hypothesized that during hypoglycemia, the interaction b…

Central Nervous SystemMaleespèce active de l'oxygènemedicine.medical_treatmentlcsh:Medicinechemistry.chemical_compoundEndocrinology0302 clinical medicineDesensitization (telecommunications)Insulinhypothalamuslcsh:ScienceNeurons0303 health sciencesMultidisciplinaryStatisticsNeurochemistryOrvostudományokAnimal Models[ SDV.MHEP.EM ] Life Sciences [q-bio]/Human health and pathology/Endocrinology and metabolism[SDV.MHEP.EM]Life Sciences [q-bio]/Human health and pathology/Endocrinology and metabolism3. Good healthEpinephrineHomeostatic MechanismsAlimentation et NutritionMedicineNeurochemicalshypoglycémieResearch Articlediabètemedicine.drugmedicine.medical_specialtyRecurrent hypoglycemiamonoxide d'azoteinsulino-thérapie intensiveNeurophysiologyBiostatisticsHypoglycemiaKlinikai orvostudományokNitric OxideGlucagonNitric oxide03 medical and health sciencesModel OrganismsInternal medicinemedicineFood and NutritionAnimalscontre-régulationBiologyNutrition030304 developmental biologyDiabetic EndocrinologyEndocrine Physiologybusiness.industryInsulinlcsh:Rneurone sensible au glucosenutritional and metabolic diseasesmonoxide d'azote;espèce active de l'oxygène;S-nitrosylation;hypoglycémie;neurone sensible au glucose;hypothalamus;contre-régulation;diabète;insulino-thérapie intensiveDiabetes Mellitus Type 1NeuroendocrinologyDiabetes Mellitus Type 2medicine.diseaseHypoglycemiaS-nitrosylationAcetylcysteineRatsGlucoseEndocrinologychemistryMetabolic DisordersRatlcsh:QReactive Oxygen SpeciesbusinessSoluble guanylyl cyclaseMathematics030217 neurology & neurosurgeryNeurosciencePLoS ONE
researchProduct

Dimethylsulfoxide as carrier in enzyme cytochemistry.

1971

Addition of dimethylsulfoxide (DMSO) to the incubation medium of succinate dehydrogenase in a concentration of 10% enhances the staining reaction in the hyphae of the fungus Cercosporella herpotrichoides after an incubation period of 15 min. Controls without DMSO remain unstained. DMSO causes a rapid penetration of the components of the medium through the mucilage that covers the hyphae.

CercosporellaHistologyintegumentary systembiologyHyphaStaining and LabelingHistocytochemistryorganic chemicalsSuccinate dehydrogenasefungiCell BiologyStainingIncubation periodMedical Laboratory TechnologyBiochemistryMucilagebiology.proteinCytochemistryDimethyl SulfoxideMitosporic FungiMolecular BiologyIncubationHistochemie. Histochemistry. Histochimie
researchProduct

The Effect of Taper and Apical Diameter on the Cyclic Fatigue Resistance of Rotary Endodontic Files Using an Experimental Electronic Device

2021

The aim of this study was to analyze the effect of the taper and apical diameter of nickel&ndash

Characteristic strengthinorganic chemicalsCyclic stressmedicine.medical_specialtyMaterials scienceapical diameterlcsh:TechnologyDynamic resistancelcsh:Chemistry03 medical and health sciences0302 clinical medicine0502 economics and businessEndodontic filesmedicineotorhinolaryngologic diseasesGeneral Materials SciencetaperInstrumentationlcsh:QH301-705.5endodontic rotary filesWeibull distributionFluid Flow and Transfer ProcessesOrthodonticsWeibull moduluslcsh:TProcess Chemistry and Technology05 social sciencesGeneral Engineering030206 dentistryEndodonticscyclic fatiguelcsh:QC1-999Computer Science Applicationsendodonticslcsh:Biology (General)lcsh:QD1-999Nickel titaniumlcsh:TA1-2040050211 marketingrotary movementlcsh:Engineering (General). Civil engineering (General)lcsh:PhysicsApplied Sciences
researchProduct