Search results for "CHON"

showing 10 items of 1866 documents

Neuroactive compounds produced by bacteria from the marine sponge Halichondria panicea: activation of the neuronal NMDA receptor

1998

Abstract Previous studies revealed that the marine sponge Halichondria panicea habors symbiotic- and commensalic bacteria ( Althoff et al., 1998 . Marine Biol. 130, 529–536). In the present study the hypothesis was tested whether some of those bacteria synthesize neuroactive compounds. For the first time the effect of bacterial bioactive compounds on the neuronal ionotropic glutamate receptors [iGluR], subtype N -methyl- d -aspartate (NMDA) receptor, was checked. In cortical neurons from rats as cell system the supernatant of two bacterial cultures isolated from H. panicea proved to agonize the NMDA receptor. The response of the NMDA receptor to the bioactive compounds was determined by mea…

PharmacologybiologyHealth Toxicology and MutagenesisGlutamate receptorMemantineGeneral MedicineToxicologybiology.organism_classificationHalichondria paniceaBiochemistrymedicineIonotropic glutamate receptorNMDA receptorReceptorBacteriaIonotropic effectmedicine.drugEnvironmental Toxicology and Pharmacology
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Swelling properties of the mitochondria of unfertilized and newly fertilized sea urchin eggs

1957

Mitocondri isolati da uova vergini e appena fecondate diParacentrotus lividus sono stati trattati con soluzioni di saccarosio a concentrazioni decrescenti, a partire da 0,5M, e con Duponol. Si e trovato che nelle soluzioni ipotoniche il rigonfiamento dei mitocondri e notevolmente maggiore per quelli preparati da uova vergini che per quelli preparati da uova fecondate. Anche al trattamento con Duponol sono piu sensibili i mitocondri delle uova vergini.

PharmacologybiologyZygoteCell BiologyAnatomyMitochondrionMolecular biologyMitochondriaCellular and Molecular NeuroscienceSea Urchinsbiology.animalAnimalsMolecular MedicineMolecular BiologySea urchinExperientia
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Mitochondrial basis of the anti-arrhythmic action of lidocaine and modulation by the n-6 to n-3 PUFA ratio of cardiac phospholipids

2012

The aim of this study was to evaluate the involvement of mitochondria in the mechanism of the anti-arrhythmic lidocaine. Rats were fed with a diet containing either n-6 polyunsaturated fatty acids (PUFAs, SSO group) or an equimolecular mixture of n-3 and n-6 PUFAs (FO group) for 8 weeks. The hearts were perfused according to the working mode using a medium with or without lidocaine 5 μM. They were then subjected to local ischemia (20 min) and reperfusion (30 min). Dietary n-3 PUFAs triggered the expected decrease in the n-6/n-3 PUFA ratio of cardiac phospholipids. Reperfusing the ischemic area favored the incidence of severe arrhythmias. Lidocaine treatment abolished almost completely reper…

Pharmacologychemistry.chemical_classification0303 health sciencesLidocaineLocal anestheticmedicine.drug_classmedicine.medical_treatmentIschemiachemistry.chemical_element030204 cardiovascular system & hematologyCalciumMitochondrionAntiarrhythmic agentPharmacologymedicine.disease03 medical and health sciences0302 clinical medicineAnticonvulsantchemistryAnesthesiamedicinePharmacology (medical)030304 developmental biologyPolyunsaturated fatty acidmedicine.drugFundamental & Clinical Pharmacology
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Zur Wirkung von Butazolidin auf Glutamins�ure-dehydrogenase und ?-Ketoglutarat-oxydase

1957

Glutamic dehydrogenase of rat liver mitochondria is inhibited by phenylbutazone at a final concentration of 2,5 mg-% (0,81 · 10−4 M). However, glutamic dehydrogenase in heart muscle homogenate and the cristalline enzyme from liver are not inhibited by phenylbutazone up to 10 mg-% (3,24 · 10−4 M), but they are inhibited by a metabolite arising during incubation of liver mitochondria with phenylbutazone.

Pharmacologychemistry.chemical_classificationRat liver mitochondriaMetabolitePharmacology toxicologyGeneral MedicineMitochondrionchemistry.chemical_compoundEnzymeBiochemistrychemistryPhenylbutazonemedicineGlutamic dehydrogenaseIncubationmedicine.drugNaunyn-Schmiedebergs Archiv f�r Experimentelle Pathologie und Pharmakologie
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Perspectives and Potential Applications of Mitochondria-Targeted Antioxidants in Cardiometabolic Diseases and Type 2 Diabetes

2013

There is abundant evidence to suggest that mitochondrial dysfunction is a main cause of insulin resistance and related cardiometabolic comorbidities. On the other hand, insulin resistance is one of the main characteristics of type 2 diabetes, obesity, and metabolic syndrome. Lipid and glucose metabolism require mitochondria to generate energy, and when O2 consumption is low due to inefficient nutrient oxidation, there is an increase in reactive oxygen species, which can impair different types of molecules, including DNA, lipids, proteins, and carbohydrates, thereby inducing proinflammatory processes. Factors which contribute to mitochondrial dysfunction, such as mitochondrial biogenesis and…

Pharmacologymedicine.medical_specialtyContext (language use)Type 2 diabetesBiologyMitochondrionBioinformaticsmedicine.diseasemedicine.disease_causeEndocrinologyInsulin resistanceMitochondrial biogenesisDiabetes mellitusInternal medicineDrug DiscoverymedicineMolecular MedicineMetabolic syndromeOxidative stressMedicinal Research Reviews
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Phylogeny and classification of poison frogs (Amphibia: dendrobatidae), based on mitochondrial 16S and 12S ribosomal RNA gene sequences.

2000

An analysis of partial sequences of the 16S ribosomal rRNA gene (582 bp) of 20 poison frog species (Dendrobatidae) confirmed their phylogenetic relationships to bufonid and leptodactylid frogs. Representatives of the ranoid families and subfamilies Raninae, Mantellinae, Petropedetinae, Cacosterninae, Arthroleptidae, Astylosternidae, and Microhylidae did not cluster as sister group of the Dendrobatidae. Similar results were obtained in an analysis using a partial sequence of the 12S gene (350 bp) in a reduced set of taxa and in a combined analysis. Within the Dendrobatidae, our data supported monophyly of the genus Phyllobates but indicated paraphyly of Epipedobates and Colostethus. Minyobat…

PhyllobatesArthroleptidaebiologyColostethusMicrohylidaeDendrobatesMolecular Sequence DataZoologyDNASequence Analysis DNAbiology.organism_classificationMitochondriaEpipedobatesAmphibiansMantellinaeRNA RibosomalRNA Ribosomal 16SGeneticsAnimalsAllobatesMolecular BiologyEcology Evolution Behavior and SystematicsPhylogenyMolecular phylogenetics and evolution
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Chemical abundances determined from meteor spectra II. Evidence for enlarged sodium abundances in meteoroids

2004

9 pages, 5 figures.-- Printed version published Mar 2004.

PhysicsAstrochemistryMeteoroidSodiumComet dustchemistry.chemical_elementTechniques: spectroscopicgeneral [Comets]Astronomy and AstrophysicsAstrobiologyspectroscopic [Techniques]Comets: generalInterplanetary dust cloudchemistryMeteoriteSpace and Planetary ScienceChondriteAbundance (ecology)Meteors meteoroidsAstrochemistry
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DIRECT EVIDENCE FOR CONDENSATION IN THE EARLY SOLAR SYSTEM AND IMPLICATIONS FOR NEBULAR COOLING RATES

2009

We have identified in an acid resistant residue of the carbonaceous chondrite Murchison a large number (458) of highly refractory metal nuggets (RMNs) that once were most likely hosted by Ca,Al-rich inclusions (CAIs). While osmium isotopic ratios of two randomly selected particles rule out a presolar origin, the bulk chemistry of 88 particles with sizes in the submicron range determined by energy dispersive X-ray (EDX) spectroscopy shows striking agreement with predictions of single-phase equilibrium condensation calculations. Both chemical composition and morphology strongly favor a condensation origin. Particularly important is the presence of structurally incompatible elements in particl…

PhysicsMurchison meteoriteRange (particle radiation)AstrochemistrySpace and Planetary ScienceChemical physicsChondriteCarbonaceous chondriteCondensationAstronomy and AstrophysicsAstrophysicsFormation and evolution of the Solar SystemChemical compositionThe Astrophysical Journal
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The MAVS Immune Recognition Pathway in Viral Infection and Sepsis.

2021

Significance: It is estimated that close to 50 million cases of sepsis result in over 11 million annual fatalities worldwide. The pathognomonic feature of sepsis is a dysregulated inflammatory response arising from viral, bacterial, or fungal infections. Immune recognition of pathogen-associated molecular patterns is a hallmark of the host immune defense to combat microbes and to prevent the progression to sepsis. Mitochondrial antiviral signaling protein (MAVS) is a ubiquitous adaptor protein located at the outer mitochondrial membrane, which is activated by the cytosolic pattern recognition receptors, retinoic acid-inducible gene I (RIG-I) and melanoma differentiation associated gene 5 (M…

PhysiologyClinical BiochemistryBiologyBiochemistrySepsisImmune systemInterferonSepsismedicineAnimalsHumansMolecular BiologyGeneral Environmental ScienceMitochondrial antiviral-signaling proteinAdaptor Proteins Signal TransducingImmune EvasionLGP2Pattern recognition receptorSignal transducing adaptor proteinMDA5Cell Biologymedicine.diseaseForum Review ArticlesVirus DiseasesImmunologyHost-Pathogen InteractionsGeneral Earth and Planetary Sciencesmedicine.drugSignal TransductionAntioxidantsredox signaling
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Molecular Mechanisms of the Crosstalk Between Mitochondria and NADPH Oxidase Through Reactive Oxygen Species—Studies in White Blood Cells and in Anim…

2014

Aims: Oxidative stress is involved in the development of cardiovascular disease. There is a growing body of evidence for a crosstalk between different enzymatic sources of oxidative stress. With the present study, we sought to determine the underlying crosstalk mechanisms, the role of the mitochondrial permeability transition pore (mPTP), and its link to endothelial dysfunction. Results: NADPH oxidase (Nox) activation (oxidative burst and translocation of cytosolic Nox subunits) was observed in response to mitochondrial reactive oxygen species (mtROS) formation in human leukocytes. In vitro, mtROS-induced Nox activation was prevented by inhibitors of the mPTP, protein kinase C, tyrosine kin…

PhysiologyNeutrophilsClinical BiochemistryBiologyMitochondrionmedicine.disease_causeBiochemistryModels BiologicalSuperoxide dismutaseCyclophilinsMiceForum Original Research CommunicationsMitochondria (A. Daiber Ed.)medicineLeukocytesAnimalsHumansMolecular BiologyGeneral Environmental ScienceRespiratory Burstchemistry.chemical_classificationMice KnockoutReactive oxygen speciesNADPH oxidaseSuperoxide DismutaseAngiotensin IINADPH OxidasesBiological TransportCell BiologyRespiratory burstMitochondriaPeroxidesEnzyme ActivationCrosstalk (biology)Oxidative StressMitochondrial permeability transition poreBiochemistrychemistrybiology.proteincardiovascular systemGeneral Earth and Planetary SciencesReactive Oxygen SpeciesOxidation-ReductionOxidative stressCyclophilin D
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