Search results for "CQ"
showing 10 items of 1801 documents
"Table 1" of "First measurement of the production of a W boson in association with a single charm quark in p anti-p collisions at s**(1/2) = 1.96-TeV"
2009
The measured cross section for leptonic decay of the W.
"Table 4" of "Measurement of $\gamma$ + b + X and $\gamma$ + c + X production cross sections in $p \bar{p}$ collisions at $\sqrt{s}$=1.96 TeV"
2009
Differential cross section for (GAMMA CJET X) production in the region YRAP(GAMMA)*YRAP(JET) < 0.
"Table 3" of "Measurement of $\gamma$ + b + X and $\gamma$ + c + X production cross sections in $p \bar{p}$ collisions at $\sqrt{s}$=1.96 TeV"
2009
Differential cross section for (GAMMA CJET X) production in the region YRAP(GAMMA)*YRAP(JET) > 0.
"Table 2" of "The Forward - backward asymmetry for charm quarks at the Z pole"
1995
No description provided.
"Table 1" of "A measurement of alpha(s) from the scaling violation in e+ e- annihilation."
1996
SIG(Q=BQ, Q=CQ, Q=UDS) corresponds to BQ, CQ, and U,D,S quarks fragmentation into charged hadron.
Epidemiology of intra-abdominal infection and sepsis in critically ill patients: "AbSeS", a multinational observational cohort study and ESICM Trials…
2019
Pardo-Oviedo, Juan Mauricio/0000-0003-0084-3449; Lopez-Delgado, Juan Carlos/0000-0003-3324-1129; Corradi, Francesco/0000-0002-5588-2608; De Backer, Daniel/0000-0001-9841-5762; POTA, VINCENZO/0000-0001-9999-3388; Tomescu, Dana/0000-0001-9673-5754; Sabetian, Golnar/0000-0001-8764-2150; Girardis, Massimo/0000-0002-2453-0829; Brazzi, Luca/0000-0001-7059-0622; Leone, Marc/0000-0002-3097-758X; Zabolotskikh, Igor Borisovich/0000-0002-3623-2546; De Lange, Dylan/0000-0002-0191-7270; ALMEKHLAFI, GHALEB A./0000-0002-0323-7025; Elke, Gunnar/0000-0002-4948-1605; Grigoras, Ioana/0000-0001-9412-9574; Czuczwar, Miroslaw/0000-0002-9025-6717; Nora, David/0000-0002-1133-7368; Masjedi, Mansoor/0000-0001-6175-9…
Exploring a regulatory role for mast cells: 'MCregs'?
2010
Regulatory cells can mould the fate of the immune response by direct suppression of specific subsets of effector cells, or by redirecting effectors against invading pathogens and infected or neoplastic cells. These functions have been classically, although not exclusively, ascribed to different subsets of T cells. Recently, mast cells have been shown to regulate physiological and pathological immune responses, and thus to act at the interface between innate and adaptive immunity assuming different functions and behaviors at discrete stages of the immune response. Here, we focus on these poorly defined, and sometimes apparently conflicting, functions of mast cells.
Complement and atherosclerosis—united to the point of no return?
2012
Atherosclerosis is widely regarded as a chronic inflammatory disease that develops as a consequence of entrapment of oxidized low-density lipoprotein (LDL) in the arterial intima and its interaction with components of both innate and adaptive immunity. This article reviews the role of the complement system in the context of a different concept on atherogenesis. Arguments are forwarded in support of the contention that enzymatic and not oxidative modification of LDL is the prerequisite for transforming the lipoprotein into a moiety that is recognized by the innate immune system. In a departure from general wisdom, it is proposed that these processes are initially not pathological. To the con…
miR-155: On the Crosstalk Between Inflammation and Cancer
2009
MicroRNAs are short non-coding RNAs that posttranscriptionally modulate the expression of multiple target genes and are thus implicated in a wide array of cellular and developmental processes. miR-155 is processed from BIC, a non-coding transcript highly expressed in both activated B and T cells and in monocytes/macrophages. miR-155 levels change dynamically during both hematopoietic lineage differentiation and the course of the immune response. Different mouse models developed recently indicate that miR-155 plays a critical role during hematopoiesis and regulates lymphocyte homeostasis and tolerance. A moderate increase of miR-155 levels is observed in many types of malignancies of B cell …
Analysis and modulation of the inflammatory response through lung agression related to bacterial infection and mechanical ventilation
2015
Despite major advances since decades in the management of ventilated patients, ventilator-associated pneumonia (VAP) continues to complicate the course of approximately 28% of the patients receiving mechanical ventilation (MV). Among patients hospitalized in intensive care units, the risk of pneumonia is 3- to 10- fold increased in MV patients. However, MV is often the only way to care for critically ill patients with respiratory failure. It has now been clearly demonstrated that MV, in particular adverse ventilatory strategies could activate lung cells, thus leading to a proinflammatory response, even in the absence of pathogen. This is the biotrauma paradigm, which accounts, at least in p…