Search results for "Cardiomyopathy"

showing 10 items of 242 documents

Branching enzyme deficiency/glycogenosis storage disease type IV presenting as a severe congenital hypotonia: muscle biopsy and autopsy findings, bio…

2010

The fatal infantile neuromuscular presentation of branching enzyme deficiency (glycogen storage disease type IV) due to mutations in the gene encoding the glycogen branching enzyme, is a rare but probably underdiagnosed cause of congenital hypotonia. We report an infant girl with severe generalized hypotonia, born at 33 weeks gestation who required ventilatory assistance since birth. She had bilateral ptosis, mild knee and foot contractures and echocardiographic evidence of cardiomyopathy. A muscle biopsy at 1 month of age showed typical polyglucosan storage. The autopsy at 3.5 months of age showed frontal cortex polymicrogyria and polyglucosan bodies in neurons of basal ganglia, thalamus, …

Pathologymedicine.medical_specialtyMuscle HypotoniaCardiomyopathyAutopsyGlycogen Storage Disease Type IVFatal Outcome14-alpha-Glucan Branching EnzymemedicineGlycogen branching enzymePolymicrogyriaHumansGlycogen storage disease type IVMuscle SkeletalGenetics (clinical)Muscle biopsymedicine.diagnostic_testbiologyInfant NewbornBrainInfantmedicine.diseaseNeurologyPediatrics Perinatology and Child Healthbiology.proteinMuscle HypotoniaFemaleNeurology (clinical)Differential diagnosisInfant PrematureNeuromuscular disorders : NMD
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Primary desminopathies.

2007

•  Introduction •  Desmin is an essential component of the extrasarcomeric cytoskeleton in striated muscle cells •  Distal myopathy,cardiac arrhythmias,cardiomyopathy:classical criteria of primary desminopathies •  Sub-sarcolemmal and cytoplasmic desmin-positive protein aggregates:the morphological hallmark of primary and secondary desminopathies •  The spectrum of pathogenic desmin gene mutations •  The molecular pathogenesis of primary desminopathies: some answers gained,but even more questions raised •  Diagnostic work-up to distinguish primary from secondary desminopathies •  Treatment and clinical management of primary desminopathy patients Abstract Mutations of the human desmin gene o…

Pathologymedicine.medical_specialtyintermediate filamentsCardiomyopathyReviewsgranulofilamentous materialdesmininclusion bodiesmacromolecular substancesBiologymyofibrillar myopathyprotein aggregationdesmin-related myopathySarcolemmaMuscular DiseasesmedicineMyocyteAnimalsHumansIntermediate filamentMyopathyMuscle SkeletalCytoskeletonGenetic heterogeneityCardiac muscleCell Biologymedicine.diseasemusculoskeletal systemmutationsmedicine.anatomical_structuredesminopathyMutationMolecular MedicineDesminmedicine.symptomMyofibrilJournal of cellular and molecular medicine
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Broken heart, tako-tsubo or stress cardiomyopathy? Metaphors, meanings and their medical impact

2016

The cardiac impact of psychological stress historically and socially understood as boundary experiences of human life has long since become an icon. From the aching heart to the sudden death provoked by awe, horror, grief, anger, and humiliation on one side and extreme enchantment, enthusiasm, and excitement on the other, the broken heart has become a globally recognized and powerful metaphor present from folklore to popular culture to high literature and back to everyday communication. In medicine, the "broken heart syndrome" is described as a relatively new nosological entity that has been used synonymously with the term tako-tsubo or stress cardiomyopathy. Among those three terms, howeve…

Pathologymedicine.medical_specialtymedia_common.quotation_subjectEmotionsCardiomyopathyBroken heart030204 cardiovascular system & hematologyGlobal HealthRisk AssessmentSudden deathBroken heart syndromeElectrocardiography03 medical and health sciences0302 clinical medicineRisk FactorsTakotsubo CardiomyopathymedicineHumans030212 general & internal medicinePsychiatrymedia_commonbusiness.industryIncidenceMortality rateHumiliationmedicine.diseaseSurvival RateSpouseMetaphorGriefCardiology and Cardiovascular MedicinebusinessStress PsychologicalInternational Journal of Cardiology
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0331 : Pathophysiology of the ubiquitine ligase E3, PDZRN3, in the development of dilated cardiomyopathies

2015

Dilated cardiomyopathy is a major cause of heart failure with a poor prognostic. Molecular mechanisms underlying the transition toward the dilated phenotype are still not known. In heart, individual cardiomyocytes connect some with the others via their extremities by junctional platform (Intercalated Discs, ID) crucial for the mechanical coupling and the anisotropic conduction of the electric signal. In this project, we are interested in an Ubiquitine ligase E3 called PDZRN3, which is expressed and regulated in cardiomyocytes during their maturation. We have previously identified PDZRN3 involvement in the the Wnt Planar Cell Polarity (Wnt/PCP) signaling in vascular morphogenesis. In the hea…

Pathologymedicine.medical_specialtymedicine.diagnostic_testbiologybusiness.industryWnt signaling pathwayDilated cardiomyopathyProtein degradationmedicine.diseaseCell biologyWestern blotUbiquitinProteasomeFibrosismedicinebiology.proteinMyocytebusinessCardiology and Cardiovascular MedicineArchives of Cardiovascular Diseases Supplements
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Two cases of tako-tsubo cardiomyophaty in Caucasians

2005

Tako-tsubo cardiomyopathy is a recently described disease characterized by chest pain, transient left ventricular dysfunction and specific electrocardiographic changes. The disease takes its name from the typical left apical ballooning observed at left ventriculogram. Tako-tsubo cardiomyopathy was first described by Sato in 1990. Since then sporadic cases were reported by Japanese authors, and only a few European publications are available. We describe 2 cases of patients affected by this syndrome.

PatientTakotsubo Cardiomyopathyapical ballooning
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Global, regional, and national levels and causes of maternal mortality during 1990-2013: A systematic analysis for the Global Burden of Disease Study…

2014

BACKGROUND: The fifth Millennium Development Goal (MDG 5) established the goal of a 75% reduction in the maternal mortality ratio (MMR; number of maternal deaths per 100,000 livebirths) between 1990 and 2015. We aimed to measure levels and track trends in maternal mortality, the key causes contributing to maternal death, and timing of maternal death with respect to delivery.METHODS: We used robust statistical methods including the Cause of Death Ensemble model (CODEm) to analyse a database of data for 7065 site-years and estimate the number of maternal deaths from all causes in 188 countries between 1990 and 2013. We estimated the number of pregnancy-related deaths caused by HIV on the basi…

PediatricsTime FactorsNutrition and Diseasehiv-infectionimmunodeficiency virus-1 infectionperipartum cardiomyopathyPoison controlHIV InfectionsIMMUNODEFICIENCY VIRUS-1 INFECTIONSocioeconomic FactorGlobal Health0302 clinical medicinePERIPARTUM CARDIOMYOPATHYpregnancy-related mortalityRisk FactorsPregnancyVoeding en ZiekteCause of DeathGlobal healthHIV Infection030212 general & internal medicinePregnancy Complications Infectious10. No inequalityCause of death030219 obstetrics & reproductive medicinesouth-africaMedicine (all)1. No povertyWOMENPREGNANCY-RELATED MORTALITYhealth initiativesGeneral MedicineSOUTH-AFRICA3. Good healthMaternal MortalityWorld HealthCHILD SURVIVALFemaleMaternal deathwomenHumanmedicine.medical_specialtyTime FactorDEATHSchild survivalArticleadult mortality03 medical and health sciencesAge DistributionHEALTH INITIATIVESSDG 3 - Good Health and Well-beingHIV-INFECTIONADULT MORTALITYInjury preventionmedicineOrganizational ObjectivesOrganizational ObjectiveHumansVLAGPregnancyModels Statisticalbusiness.industryRisk Factormedicine.diseasedeathsStandardized mortality ratioSocioeconomic FactorsRelative riskPregnancy Complications InfectioubusinessDemography
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Clinical and Genetic Aspects of Juvenile Onset Pompe Disease

2021

AbstractLittle is known about clinical symptomatology and genetics of juvenile onset Pompe disease (JOPD). The aims of this study were to analyze how these children are diagnosed, what clinical problems they have, and how phenotype is related to genotype. To accomplish this, we analyzed retrospectively data of 34 patients diagnosed after their first and before completion of their 18th birthday. Median age at diagnosis was 3.9 (range 1.1–17) years. Eight patients (23.5%) developed initial symptoms in the first year, 12 (35%) between 1 and 7 years, and 6 (18%) thereafter. Eight (23.5%) had no clinical symptoms at the time of diagnosis. Indications for diagnostics were a positive family histor…

Pediatricsmedicine.medical_specialtyGeneralized muscle weaknessDisease03 medical and health sciences0302 clinical medicineGenotypeHumansMedicineFamily historyRetrospective Studies030304 developmental biology0303 health sciencesGlycogen Storage Disease Type IIbusiness.industryHypertrophic cardiomyopathyMuscle weaknessalpha-GlucosidasesGeneral Medicinemedicine.disease3. Good healthPhenotypeJuvenile onsetMutationPediatrics Perinatology and Child HealthFailure to thriveNeurology (clinical)medicine.symptombusiness030217 neurology & neurosurgeryNeuropediatrics
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Hypertrophic cardiomyopathy associated with polyarteritis nodosa: A case of sudden cardiac death

2018

This case concerns a rare sudden cardiac death characterized by macroscopic and microscopic postmortem findings of hypertrophic cardiomyopathy and polyarteritis nodosa. A complete autopsy was carried out, and histological and histochemical methods were employed. The cause of death was acute multifocal ischemic myocitolitic damage caused by both myocardial structural alteration attributable to hypertrophic cardiomyopathy (widespread interstitial fibrosis and multifocal myocyte disarray) and coronary arteritis attributable to polyarteritis nodosa. This is the first case in which the cause of death was attributed to both diseases.

Polyarteritis-NodosaSudden cardiac deathCase reportHypertrophic CardiomyopathyForensic pathology
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A Case of Cardiomyopathy Due to Premature Ductus Arteriosus Closure: The Flip Side of Paracetamol.

2017

Paracetamol (acetaminophen or N-acetyl-p-aminophenol) is considered a safe analgesic and antipyretic nonsteroidal antiinflammatory drug commonly used during pediatric ages and during pregnancy. We report on a term neonate with closed ductus arteriosus, severe cardiomyopathy, right ventricular dysfunction, and functional stenosis of pulmonary arteries at birth after maternal self-medication with paracetamol and consumption of polyphenol-rich foods in late pregnancy. This drug, especially when associated with other vasoconstrictors (such as polyphenols), interferes with prostaglandin metabolism, which seriously accentuates the intrauterine ductus arteriosus constriction and leads to pharmacol…

Polyphenolmedicine.medical_specialtyDuctus ArteriosuVentricular Dysfunction RightCardiomyopathyHeart MurmurPulmonary Valve StenosiSelf MedicationDiet Mediterranean03 medical and health sciences0302 clinical medicinePregnancy030225 pediatricsInternal medicineDuctus arteriosusmedicineHumansVasoconstrictor AgentsAdverse effectCardiomyopathieAcetaminophenPregnancy030219 obstetrics & reproductive medicineHeart Murmursbusiness.industryDuctus arteriosus closureInfant NewbornPolyphenolsDuctus ArteriosusAnalgesics Non-Narcoticmedicine.diseaseAcetaminophenPulmonary Valve Stenosismedicine.anatomical_structurePrenatal Exposure Delayed EffectsPediatrics Perinatology and Child HealthPulmonary valve stenosiscardiovascular systemCardiologyHeart murmurFemalemedicine.symptomVasoconstrictor AgentbusinessCardiomyopathiesmedicine.drugHumanPediatrics
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Clinical cardiac assessment in newborns with prenatally diagnosed intrathoracic masses

2018

Abstract Background Congenital space-occupying thoracic malformations and diaphragmatic hernia have in common pulmonary hypoplasia. Our study aims to assess cardiac involvement during post-natal adaptation. Methods A retrospective study was carried out on newborns with prenatally diagnosed intrathoracic mass. Gathering for respiratory distress syndrome (RDS), 35 neonates were compared for clinical course, cardiovascular enzymes, ECG, and ultrasound. Results The analysis revealed a high left heart defect rate in patients with severe RDS, without being influenced by the laterality. Ultrasound or laboratory assessment did not detect altered cardiac dimension or cardiomyopathy. Solely ECG signs…

Prenatal DiagnosiMaleCardiomyopathyCardiovascular abnormalitiesCongenital diaphragmatic herniaCohort StudiesElectrocardiography0302 clinical medicineRetrospective StudiePrenatal DiagnosisRespiratory system abnormalitiesDiaphragmatic hernia030219 obstetrics & reproductive medicineRespiratory distressCardiovascular abnormalitieUltrasoundClinical courselcsh:RJ1-570PrognosisSurvival RateRetrospective studyCardiologyFemaleHumanHeart Defects Congenitalmedicine.medical_specialtyPrognosiRisk AssessmentUltrasonography Prenatal03 medical and health sciencesPulmonary hypoplasia030225 pediatricsInternal medicinemedicineHumansAbnormalities MultipleRetrospective StudiesRespiratory Distress Syndrome Newbornbusiness.industryResearchInfant NewbornRespiratory system abnormalitieCongenital diaphragmatic herniaUltrasonography DopplerRetrospective cohort studylcsh:Pediatricsmedicine.diseaseCohort StudieHernias Diaphragmatic CongenitalbusinessItalian Journal of Pediatrics
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