Search results for "Cell Fusion"

showing 10 items of 30 documents

Anti-HIV-1 activity of inorganic polyphosphates.

1997

Human blood plasma, serum, peripheral blood mononuclear cells, and erythrocytes contain significant amounts of inorganic polyphosphates (ranging from 53 to 116 microM, in terms of phosphate residues). Here we demonstrate that at higher concentrations linear polyphosphates display cytoprotective and antiviral activity. Sodium tetrapolyphosphate and the longer polymers, with average chain lengths of 15, 34, and 91 phosphate residues, significantly inhibited human immunodeficiency virus type 1 (HIV-1) infection of cells in vitro at concentrations > or = 33.3 microg/ml (> or = 283-324 microM phosphate residues), whereas sodium tripolyphosphate was ineffective. In the tested concentration range,…

ErythrocytesCell SurvivalSodiumT-LymphocytesImmunologychemistry.chemical_elementBiologyPeripheral blood mononuclear cellGiant CellsCell LineCell Fusionchemistry.chemical_compoundDrug StabilityPolyphosphatesVirologyImmunology and AllergyHumansHost cell surfaceSyncytiumCell fusionDose-Response Relationship DrugPolyphosphateBiological activityPhosphateBiochemistrychemistryHIV-1Leukocytes MononuclearCell DivisionJournal of acquired immune deficiency syndromes and human retrovirology : official publication of the International Retrovirology Association
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Curcumin and trans-resveratrol exert cell cycle-dependent radioprotective or radiosensitizing effects as elucidated by the PCC and G2-assay

2013

Curcumin and trans-resveratrol are well-known antioxidant polyphenols with radiomodulatory properties, radioprotecting non-cancerous cells while radiosensitizing tumor cells. This dual action may be the result of their radical scavenging properties and their effects on cell-cycle checkpoints that are activated in response to radiation-induced chromosomal damage. It could be also caused by their effect on regulatory pathways with impact on detoxification enzymes, the up-regulation of endogenous protective systems, and cell-cycle-dependent processes of DNA damage. This work aims to elucidate the mechanisms underlying the dual action of these polyphenols and investigates under which conditions…

G2 PhaseRadiation-Sensitizing AgentsRadiosensitizerCurcuminAntioxidantDNA damageHealth Toxicology and Mutagenesismedicine.medical_treatmentRadioprotectorCellRadiation-Protective AgentsCHO CellsBiologyRadiation ToleranceCell Fusionchemistry.chemical_compoundCricetulusCricetinaeStilbenesGeneticsmedicineAnimalsHumansRadiosensitivityMolecular BiologyCells CulturedMutagenicity TestsCell CycleCell cycleChromatin Assembly and DisassemblyRadiosensitizermedicine.anatomical_structureG2-assayBiochemistrychemistryResveratrolPeripheral blood lymphocyteCancer researchCurcumintrans-ResveratrolPremature chromosome condensationMutation Research/Fundamental and Molecular Mechanisms of Mutagenesis
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Cyclosporin A resistance of herpes simplex virus-induced "fusion from within" as a phenotypical marker of mutations in the Syn 3 locus of the glycopr…

1994

We here report research in which nine strains of Herpes simplex virus (HSV) with fusing activity were investigated in order to establish precise phenotypical markers of mutations in the carboxy terminus of glycoprotein B (gB). The gene region encoding the carboxy terminus of gB was isolated, then cloned, and finally sequenced. Our investigation showed that seven strains have different mutations in the syn 3 locus. We observed no base difference in the gB gene region encoding the carboxy terminus of gB of two other strains. Strains with a mutation in the carboxy terminus of gB induced fusion from within (FFWI) in the presence of Cyclosporin A (CyA) at a concentration up to 150 µM. There are …

Genetic MarkersGenes ViralLocus (genetics)Biologymedicine.disease_causeVirusCell LineCell FusionViral Envelope ProteinsVirologyCyclosporin aGeneticsmedicineAnimalsHumansSimplexvirusMolecular BiologyGenechemistry.chemical_classificationGeneticsCell fusionDrug Resistance MicrobialGeneral MedicinePhenotypeMolecular biologyHerpes simplex virusPhenotypechemistryMutationCyclosporineGlycoproteinViral Fusion ProteinsVirus genes
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Jellyfish Stings Trigger Gill Disorders and Increased Mortality in Farmed Sparus aurata (Linnaeus, 1758) in the Mediterranean Sea

2016

11 pages, 4 figures

Gills0106 biological sciences0301 basic medicineGillFish mortalityFish DiseaseJellyfishScyphozoaRespiratory SystemMarine and Aquatic Scienceslcsh:MedicineAquaculture01 natural sciencesCell FusionBites and StingFish DiseasesAquacultureOceansMedicine and Health SciencesMarine FishBites and StingsAnimal Anatomylcsh:ScienceMultidisciplinarybiologyPhysicsFishesClassical MechanicsAgricultureSurvival RateVertebratesPhysical SciencesAnatomyAnimals Aquaculture Bites and Stings Fish Diseases Gills Mediterranean Sea Scyphozoa Sea Bream Survival Rate TunisiaResearch ArticleSettore BIO/07 - EcologiaCell PhysiologyTunisiaFish BiologyFish farmingMarine BiologyCnidaria03 medical and health sciencesFish physiologyBodies of waterbiology.animalFish PhysiologyMediterranean SeaAnimalsAnimal Physiology14. Life underwaterDamage MechanicsAnimalbusiness.industry010604 marine biology & hydrobiologylcsh:ROrganismsBiology and Life SciencesCell BiologyScyphozoabiology.organism_classificationPelagia noctilucaInvertebratesVertebrate PhysiologySea BreamFishery030104 developmental biologyAquatic Respiratory Anatomy13. Climate actionEarth SciencesGilllcsh:QJellyfishbusinessZoologyPLOS ONE
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Involvement of actin-containing microfilaments in HSV-induced cytopathology and the influence of inhibitors of glycosylation.

1986

Two and a half hours after infection with a high dose of different strains of HSV-1 which induce rounding of cells, breakdown of actin containing microfilaments can be observed. At the periphery of the cell, actin containing knob-like protuberances were visible. Later on, actin seems to be located exclusively on the surface of cells. Observations were done by immunofluorescence microscopy, scanning electron-microscopy and immunoperoxidase staining of ultrathin sections. The envelope of HSV appears to be stained by anti-actin. Strain IES produces rounding of cells at a high dose of infection before fusion proceeds at 37 degrees C. Similar alterations were not observed with the fusing strains…

GlycosylationGlycosylationCellBiologyDeoxyglucoseMicrofilamentVirusCell LineCell Fusionchemistry.chemical_compoundCytopathogenic Effect ViralFluorodeoxyglucose F18VirologymedicineAnimalsSimplexvirusActinCells CulturedCytoskeletonchemistry.chemical_classificationTunicamycinCell MembraneGeneral MedicineTunicamycinVirologyActinsActin CytoskeletonMicroscopy Electronmedicine.anatomical_structurechemistryCytopathologyGlycoproteinArchives of virology
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Enhancement of Gene Expression by Somatic Hybridization with Primary Cells: High-Level Synthesis of the Hepatitis B Surface Antigen in Monkey Vero Ce…

1990

Vero cells transfected with the S gene encoding the surface antigen (HBsAg) of the hepatitis B virus (HBV) synthesize HBsAg at low levels. We have obtained a large increase in S gene expression by somatic hybridization of Vero cells with primary hepatocytes, which are the natural target cells for HBV infection. Fusion with cells other than hepatocytes did not enhance expression of the S gene. The Vero/hepatocyte hybrid clones analyzed are stable and have maintained a high level of HBsAg synthesis over prolonged periods. Hybrid cell lines may be of general interest for the high-level synthesis of proteins using cloned genes.

HBsAgLiver cytologyvirusesGenetic VectorsBiomedical EngineeringGene ExpressionBioengineeringHybrid CellsBiologyKidneyTransfectionmedicine.disease_causeApplied Microbiology and BiotechnologyCell LineAntigenChlorocebus aethiopsmedicineAnimalsVero CellsHepatitis B virusHepatitis B Surface AntigensCell fusionvirus diseasesTransfectionVirologyMolecular biologydigestive system diseasesLiverCell cultureVero cellRNAMolecular MedicinePapioBiotechnologyNature Biotechnology
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Microtubules and intermediate filaments of herpes simplex virus infected cells.

1987

The fate of microtubules and of vimentin or keratin containing intermediate filaments during infection with fusion or rounding producing strains of herpes simplex virus (HSV) was investigated. Microtubules polymerize early after fusion of cells. However, they do not reconstitute 6–7 hours post infection (p.i.) after release of a colcemid block. Keratin and vimentin are maintained around the original nucleus still inside of recruited cells in the polykaryocyte. Cells of fibroblastic and epithelial origin fuse. Inside of polykaryocytes keratin or vimentin containing fibers seem to polymerize. Keratin is to be found in invaginations in the nuclei surrounded by the inner layer of the nuclear me…

Intermediate FilamentsVimentinmacromolecular substancesmedicine.disease_causeMicrofilamentMicrotubulesEpitheliumCell LineCell Fusionchemistry.chemical_compoundCytopathogenic Effect ViralVirologyKeratinmedicineAnimalsSimplexvirusVimentinNuclear membraneIntermediate filamentCytoskeletonchemistry.chemical_classificationintegumentary systembiologyColcemidHerpes SimplexGeneral MedicineFibroblastsVirologyHerpes simplex virusmedicine.anatomical_structurechemistryCytoplasmbiology.proteinKeratinsArchives of virology
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Xenobiotic-metabolizing enzyme activities in hybrid cell lines established by fusion of primary rat liver parenchymal cells with hepatoma cells

1992

1. The activities of xenobiotic-metabolizing enzymes were determined in hybrid cell lines (hepatocytoma, HPCT) which have been established by fusion of liver parenchymal cells from adult rat (PC) with cells from a Reuber hepatoma cell line (FAO). 2. Cytochrome P450 was not measurable spectrophotometrically in FAO and HPCT. P450-dependent conversion of testosterone was below the detection limit in FAO and only marginally present in HPCT. 3. Microsomal and cytosolic epoxide hydrolase, glutathione S-transferase and phenol sulphotranserase were low or even below detection limit in FAO. These enzyme activities were significantly higher in HPCT and correspond to about 1-10% the activities measure…

Male1303 BiochemistryHealth Toxicology and Mutagenesis10050 Institute of Pharmacology and Toxicology610 Medicine & healthHybrid CellsToxicologyBiochemistryCell LineMixed Function OxygenasesXenobioticsCell FusionRats Sprague-Dawleychemistry.chemical_compoundLiver Neoplasms Experimental2307 Health Toxicology and MutagenesisTumor Cells CulturedAnimalsEnzyme inducerEpoxide hydrolasePharmacologychemistry.chemical_classificationbiologydigestive oral and skin physiologyCytochrome P4503005 ToxicologyGeneral MedicineGlutathioneRatsEnzyme3004 PharmacologychemistryBiochemistryLiverCell cultureEnzyme inhibitorbiology.proteinMicrosome570 Life sciences; biology
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Regulation of connexin gene expression during skeletal muscle regeneration in the adult rat

2009

In the adult skeletal muscle, various kinds of trauma promote proliferation of satellite cells that differentiate into myoblasts forming new myofibers or to repair the damaged one. The aim of present work was to perform a comparative spatial and temporal analysis of connexin (Cx) 37, Cx39, Cx40, Cx43, and Cx45 expression in the adult regenerating skeletal muscle in response to crush injury. Within 24 h from injury, Cx37 expression was upregulated in the endothelial cells of blood vessels, and, 5 days after injury, Cx37-expressing cells were found inside the area of lesion and formed clusters generating new blood vessels with endothelial cells expressing Cx37. Three days after injury, Cx39 m…

MalePathologymedicine.medical_specialtyTime FactorsPhysiologyMuscle Fibers SkeletalConnexinNeovascularization Physiologicconnexin 45BiologyConnexinsconnexin 43Cell Fusionconnexin 40Muscle regenerationGene expressionmedicineConnexin 30MyocyteAnimalsRegenerationRNA MessengerRats WistarMuscle SkeletalIn Situ HybridizationCell AggregationCell ProliferationMyogenic cellsconnexin 39Regeneration (biology)Skeletal muscleEndothelial CellsCell Biologyconnexin 37biology.organism_classificationConstrictionImmunohistochemistryCell biologyRatsMuscle regenerationmedicine.anatomical_structureGene Expression Regulationmyogenic cellSatellite (biology)Muscle regeneration; Connexins; Myogenic cells
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Cell fusion as a mechanism for the formation of giant cells (Langhans’ type)

1982

The formation of multinuclear giant cells of the Langhans' type in tubulo-interstitial auto-immune nephritis in the rat has been investigated by means of autoradiography. While in the majority of giant cells all nuclei were radiolabeled, in a few both labeled and unlabeled nuclei were present. This latter finding represents strong evidence in favour of the hypothesis that giant cells do not form by endomitotic processes but rather through fusion of certain precursor cells. According to previous studies this precursor cell population consists mainly of epitheloid cells, i.e. modified monocytes.

MalePathologymedicine.medical_specialtyanimal structuresCell fusionurogenital systemChemistryLanghans giant cellGeneral Medicineurologic and male genital diseasesmedicine.diseaseAutoimmune DiseasesRatsCell biologyCell FusionGiant cellRats Inbred BNcardiovascular systemmedicineAnimalsAutoradiographyNephritis InterstitialtissuesNephritisThymidineVirchows Archiv B Cell Pathology Including Molecular Pathology
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