Search results for "Cerebellum"

showing 10 items of 202 documents

Organotypic rat cerebellar slice culture as a model to analyze the molecular pharmacology of GABAA receptors

2002

The preservation of the neuronal circuitry in rat cerebellar slice cultures provides an advantage in monitoring the development and characterizing the pharmacology of GABA(A) receptor subtypes. Sprague-Dawley rats, 8-11 days of age, were decapitated, their cerebella were cut into 400-microm slices and transferred into culture dishes. Cell viability and organotypic cerebellar organization of the culture remained well preserved up to 3 weeks. Autoradiographic procedures were introduced in these advanced culture technique and employed [(3)H]Ro 15-4513 in the absence and presence of 10 microM diazepam to visualize all benzodiazepine (BZD) and diazepam-insensitive (DIS) binding sites, respective…

MaleAgonistAzidesCerebellumCell Survivalmedicine.drug_classProtein subunitBiologyPharmacologyRats Sprague-DawleyBenzodiazepinesCerebellumCulture TechniquesmedicineAnimalsPharmacology (medical)Viability assayReceptorCells CulturedBiological PsychiatryPharmacologyBenzodiazepineBinding SitesGABAA receptorAffinity LabelsReceptors GABA-ARatsPsychiatry and Mental healthmedicine.anatomical_structureAnimals NewbornNeurologyOrgan SpecificityNeurology (clinical)NeuroscienceDiazepammedicine.drugEuropean Neuropsychopharmacology
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Characterization of γ-aminobutyrate type A receptors with atypical coupling between agonist and convulsant binding sites in discrete brain regions

2001

Abstract γ-Aminobutyric acid type A (GABA A ) receptor ionophore ligand t -[ 35 S]butylbicyclophosphorothionate ([ 35 S]TBPS) was used in an autoradiographic assay on brain cryostat sections to visualize and characterize atypical GABA-insensitive [ 35 S]TBPS binding previously described in certain recombinant GABA A receptors and the cerebellar granule cell layer. Picrotoxinin-sensitive but 1-mM GABA-insensitive [ 35 S]TBPS binding was present in the rat cerebellar granule cell layer, many thalamic nuclei, subiculum and the internal rim of the cerebral cortex, amounting in these regions up to 6% of the basal binding determined in the absence of exogenous GABA. Similar binding properties wer…

MaleAgonistAzidesmedicine.medical_specialtyCerebellumSesterterpenesmedicine.drug_classLoreclezoleConvulsantsBiologySulfur RadioisotopesTritiumBinding CompetitiveBenzodiazepinesRadioligand AssayCellular and Molecular Neurosciencechemistry.chemical_compoundThalamusCerebellumInternal medicinemedicineAnimalsHumansPicrotoxinRats WistarBinding siteReceptorGABA AgonistsMolecular Biologygamma-Aminobutyric AcidMuscimolGABAA receptorAffinity LabelsBridged Bicyclo Compounds HeterocyclicReceptors GABA-AGranule cellRatsEndocrinologymedicine.anatomical_structurenervous systemMuscimolchemistryBiophysicsChickensmedicine.drugMolecular Brain Research
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Continuous theta burst stimulation (cTBS) on left cerebellar hemisphere affects mental rotation tasks during music listening.

2013

Converging evidence suggests an association between spatial and music domains. A cerebellar role in music-related information processing as well as in spatial-temporal tasks has been documented. Here, we investigated the cerebellar role in the association between spatial and musical domains, by testing performances in embodied (EMR) or abstract (AMR) mental rotation tasks of subjects listening Mozart Sonata K.448, which is reported to improve spatial-temporal reasoning, in the presence or in the absence of continuous theta burst stimulation (cTBS) of the left cerebellar hemisphere. In the absence of cerebellar cTBS, music listening did not influence either MR task, thus not revealing a “Moz…

MaleAnatomy and PhysiologyTime FactorsCTBSlcsh:MedicineAudiologySocial and Behavioral SciencesMental rotationMUSICBehavioral NeuroscienceMental ProcessesCerebellar hemisphereTask Performance and AnalysisPsychologyTheta Rhythmlcsh:ScienceMultidisciplinaryMusic psychologyCognitive NeurologyBRAIN STIMULATIONExperimental PsychologyhumanitiesMental HealthNeurologyAuditory PerceptionMedicineSensory PerceptionFemalepsychological phenomena and processesResearch ArticleAdultmedicine.medical_specialtyAdolescentCognitive NeuroscienceNeurophysiologyBiologybehavioral disciplines and activitiesLateralization of brain functionNeurological SystemNOYoung AdultMotor imageryNeuropsychologymedicineReaction TimeHumansActive listeningMozart effectBiologyCEREBELLUMSettore M-PSI/02 - Psicobiologia E Psicologia Fisiologicalcsh:RCognitive PsychologyNeuroanatomyAcoustic Stimulationlcsh:QNeuroscience
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Aspartoacylase-lacZ knockin mice: an engineered model of Canavan disease.

2011

Canavan Disease (CD) is a recessive leukodystrophy caused by loss of function mutations in the gene encoding aspartoacylase (ASPA), an oligodendrocyte-enriched enzyme that hydrolyses N-acetylaspartate (NAA) to acetate and aspartate. The neurological phenotypes of different rodent models of CD vary considerably. Here we report on a novel targeted aspa mouse mutant expressing the bacterial β-Galactosidase (lacZ) gene under the control of the aspa regulatory elements. X-Gal staining in known ASPA expression domains confirms the integrity of the modified locus in heterozygous aspa lacZ-knockin (aspa(lacZ/+)) mice. In addition, abundant ASPA expression was detected in Schwann cells. Homozygous (…

MaleCentral Nervous SystemCerebellumPathologyAnatomy and PhysiologyCanavan DiseaseMouseMutantlcsh:MedicineNeural HomeostasisBiochemistryMiceNeurobiology of Disease and Regenerationlcsh:ScienceSex CharacteristicsMultidisciplinaryNeuromodulationNeurochemistryGenomicsAnimal ModelsFunctional Genomicsmedicine.anatomical_structureLac OperonNeurologyHomeostatic MechanismsMedicineFemaleNeurochemicalsGenetic EngineeringResearch ArticleNervous System PhysiologyBiotechnologymedicine.medical_specialtyTransgeneCentral nervous systemNeurophysiologyMice TransgenicNeuroimagingBiologyNeurological SystemAmidohydrolasesWhite matterModel OrganismsGeneticsmedicineAnimalsBiologyNeuropeptidesLeukodystrophylcsh:RComputational Biologymedicine.diseaseMolecular biologyCanavan diseaseAspartoacylaseDisease Models AnimalMetabolismnervous systemSmall MoleculesCellular NeuroscienceMetabolic DisordersMutationGenetics of DiseaseNervous System Componentslcsh:QGene FunctionMolecular NeuroscienceAnimal GeneticsNeurosciencePLoS ONE
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Vestibular compensation in cerebellar stroke patients.

2014

Background and purpose There is little evidence about the site where compensatory vestibular mechanisms in patients with cerebellar strokes take place. Methods To determine whether the location of a cerebellar lesion might be a crucial variable in vestibular compensation a sample of 22 patients with cerebellar stroke were tested for graviceptive function in the acute and chronic stage. Results Our statistical anatomical lesion analysis indicated that mainly lesions of the cerebellar hemispheres (lobule V, VI, VIIa) hinder vestibular compensation and might lead to an overcompensation. Conclusions Overcompensation-induced dysfunction can be explained by the absence of cerebellar inhibitory si…

MaleCerebellumInhibitory postsynaptic potentialCerebellar Diseasesphysiopathology [Vestibule Labyrinth]otorhinolaryngologic diseasesMedicineCerebellar strokeHumansIn patientddc:610StrokeAgedVestibular systemChronic stagephysiopathology [Stroke]pathology [Cerebellar Diseases]business.industryMiddle AgedVestibular Function Testsmedicine.diseaseCerebellar lesionMagnetic Resonance Imagingphysiopathology [Cerebellar Diseases]Strokemedicine.anatomical_structurenervous systemNeurologypathology [Stroke]FemaleNeurology (clinical)Vestibule LabyrinthbusinessNeuroscienceEuropean journal of neurology
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A Multiomics Study To Unravel the Effects of Developmental Exposure to Endosulfan in Rats: Molecular Explanation for Sex-Dependent Effects

2019

Exposure to low levels of environmental contaminants, including pesticides, induces neurodevelopmental toxicity. Environmental and food contaminants can reach the brain of the fetus, affecting brain development and leading to neurological dysfunction. The pesticide endosulfan is a persistent pollutant, and significant levels still remain detectable in the environment although its use is banned in some countries. In rats, endosulfan exposure during brain development alters motor activity, coordination, learning, and memory, even several months after uptake, and does so in a sex-dependent way. However, the molecular mechanisms driving these effects have not been studied in detail. In this wor…

MaleCerebellumInsecticidescerebellumPhysiologyCognitive NeuroscienceMetabolitePhysiologyBiologyMotor ActivityBiochemistry03 medical and health scienceschemistry.chemical_compoundPhosphatidylinositol 3-Kinases0302 clinical medicineImmune systemSex FactorsPregnancyneurotoxicitymedicineCyclic GMP-Dependent Protein KinasesAnimalsCyclic GMPdevelopmentEndosulfanpesticide030304 developmental biologyCalcium signaling0303 health sciencesFetusBehavior AnimalNeurotoxicityCell BiologyGeneral Medicinemedicine.diseasesignaling pathwaysRatsmedicine.anatomical_structurechemistryPrenatal Exposure Delayed EffectsToxicityFemaleTranscriptome030217 neurology & neurosurgeryEndosulfanmultiomicsSignal Transduction
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Abnormal synchrony and effective connectivity in patients with schizophrenia and auditory hallucinations

2014

Auditory hallucinations (AH) are the most frequent positive symptoms in patients with schizophrenia. Hallucinations have been related to emotional processing disturbances, altered functional connectivity and effective connectivity deficits. Previously, we observed that, compared to healthy controls, the limbic network responses of patients with auditory hallucinations differed when the subjects were listening to emotionally charged words. We aimed to compare the synchrony patterns and effective connectivity of task-related networks between schizophrenia patients with and without AH and healthy controls. Schizophrenia patients with AH (n = 27) and without AH (n = 14) were compared with healt…

MaleCerebellumMVAR multivariate autoregressionHallucinationsAH auditory hallucinationsAuditory hallucinationsBPRS Brief Psychiatric Rating ScaleAudiologylcsh:RC346-429BOLD blood oxygenation level dependentDevelopmental psychologyFunctional connectivityCerebellumNeural PathwaysEffective connectivityICA-TC ICA-time courseFunctional connectivityEmotional stimuliMiddle AgedTemporal LobeICA independent component analysisSynchronymedicine.anatomical_structureNeurologySchizophreniaMRI functional magnetic resonance imaginglcsh:R858-859.7PsychologyAdultmedicine.medical_specialtyCognitive NeuroscienceEmotional processinglcsh:Computer applications to medicine. Medical informaticsArticleYoung AdultmedicineHumansRadiology Nuclear Medicine and imagingIn patientPANSS Positive and Negative Syndrome ScaleCoI component of interestCCTC cortico-cerebellar–thalamic–corticallcsh:Neurology. Diseases of the nervous systemAuditory CortexSPM statistical parametric mapsmedicine.diseaseGCCA Granger causal connectivity analysisAcoustic StimulationFISICA APLICADASchizophreniaAuditory stimuliPSYRATS Psychotic Symptom Rating ScaleNeurology (clinical)NeuroImage: Clinical
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Expression of connexin36 in the adult and developing rat brain.

2000

The distribution of connexin36 (Cx36) in the adult rat brain and retina has been analysed at the protein (immunofluorescence) and mRNA (in situ hybridization) level. Cx36 immunoreactivity, consisting primarily of round or elongated puncta, is highly enriched in specific brain regions (inferior olive and the olfactory bulb), in the retina, in the anterior pituitary and in the pineal gland, in agreement with the high levels of Cx36 mRNA in the same regions. A lower density of immunoreactive puncta can be observed in several brain regions, where only scattered subpopulations of cells express Cx36 mRNA. By combining in situ hybridization for Cx36 mRNA with immunohistochemistry for a general neu…

MaleCerebellumPathologymedicine.medical_specialtygenetic structuresHippocampusIn situ hybridizationBiologyPineal GlandConnexinsmedicineAnimalsRNA MessengerEye ProteinsMolecular BiologyNeuronsBrain MappingGeneral NeuroscienceAge FactorsBrainGap JunctionsNuclear ProteinsImmunohistochemistryOlfactory bulbCell biologyRatsmedicine.anatomical_structureParvalbuminsnervous systemAnimals NewbornCerebral cortexCerebellar cortexPituitary Glandbiology.proteinsense organsNeurology (clinical)NeuronNeuNBiomarkersDevelopmental BiologyBrain research
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Hippocampo-cerebellar theta band phase synchrony in rabbits.

2009

Hippocampal functioning, in the form of theta band oscillation, has been shown to modulate and predict cerebellar learning of which rabbit eyeblink conditioning is perhaps the most well-known example. The contribution of hippocampal neural activity to cerebellar learning is only possible if there is a functional connection between the two structures. Here, in the context of trace eyeblink conditioning, we show (1) that, in addition to the hippocampus, prominent theta oscillation also occurs in the cerebellum, and (2) that cerebellar theta oscillation is synchronized with that in the hippocampus. Further, the degree of phase synchrony (PS) increased both as a response to the conditioning sti…

MaleCerebellumPeriodicityHippocampusContext (language use)Hippocampal formationHippocampus03 medical and health sciencesRandom Allocation0302 clinical medicineNeural ensembleCerebellummedicineOscillation (cell signaling)AnimalsLearningCortical SynchronizationTheta Rhythm030304 developmental biology0303 health sciencesAnalysis of VarianceGeneral NeuroscienceConditioning EyelidElectrodes Implantedmedicine.anatomical_structurenervous systemEyeblink conditioningPractice PsychologicalRabbitsPsychologyNeuroscience030217 neurology & neurosurgeryCortical SynchronizationNeuroscience
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Interaction of uridine with GABA binding sites in cerebellar membranes of the rat

1983

The effect of uridine, a postulated anticonvulsant agent, on GABA receptors has been investigated. Uridine inhibits [3H]GABA binding to rat cerebellar buffer-washed membranes. Pretreatment of the membranes with Triton X-100 increases the effect of uridine on GABA-binding. The Scatchard analysis reveals that both high and low affinities of GABA for its receptors are affected by 1 mM uridine, while the apparent number of binding sites remains unchanged. The ability of uridine to interact competitively with GABA binding sites, also examined by the Lineweaver-Burk analysis, suggests a possible mechanism of action of this anticonvulsant agent, so including it among those compounds characterized …

MaleCerebellumReceptors Cell SurfaceBiologyBinding CompetitiveBiochemistrygamma-Aminobutyric acidCellular and Molecular Neurosciencechemistry.chemical_compoundGABA receptorCerebellummedicineAnimalsBinding siteReceptorUridinegamma-Aminobutyric AcidGABAA receptorCell MembraneRats Inbred StrainsGeneral MedicineReceptors GABA-AUridineRatsmedicine.anatomical_structurenervous systemBiochemistryMechanism of actionchemistryAnticonvulsantsmedicine.symptommedicine.drugNeurochemical Research
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