Search results for "Colitis"

showing 10 items of 483 documents

Imaging techniques in inflammatory bowel disease: recent trends, questions and answers

2009

Summary Imaging techniques have undergone substantial progress in recent years and contribute significantly in the diagnosis of inflammatory bowel diseases in conjunction with patient history, clinical and laboratory examination. Modern cross-sectional imaging modalities such as computed tomography and magnetic resonance imaging allow an evaluation not only of the complete bowel wall of the small intestine, but also of extraluminal structures. They constitute a major diagnostic component in the initial workup, in stricturing or fistulizing disease and in suspected abscess. Transabdominal ultrasonography has been re-appreciated in these settings as an easy- and ready-to-use tool yielding rea…

Diagnostic Imagingmedicine.medical_specialtyContrast MediaColonoscopySensitivity and SpecificityInflammatory bowel diseaseChromoendoscopyDiagnosis DifferentialCrohn DiseasePredictive Value of TestsmedicineEndomicroscopyHumansMedical historyClinical Trials as TopicMicroscopy Confocalmedicine.diagnostic_testbusiness.industryGastroenterologyMagnetic resonance imagingColonoscopyGeneral MedicineInflammatory Bowel Diseasesmedicine.diseaseMagnetic Resonance ImagingUlcerative colitisPositron emission tomographyPositron-Emission TomographyColitis UlcerativeRadiologyTomography X-Ray ComputedbusinessGastroentérologie Clinique et Biologique
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Clostridium difficile heterogeneously impacts intestinal community architecture but drives stable metabolome responses

2015

Clostridium difficile-associated diarrhoea (CDAD) is caused by C. difficile toxins A and B and represents a serious emerging health problem. Yet, its progression and functional consequences are unclear. We hypothesised that C. difficile can drive major measurable metabolic changes in the gut microbiota and that a relationship with the production or absence of toxins may be established. We tested this hypothesis by performing metabolic profiling on the gut microbiota of patients with C. difficile that produced (n=6) or did not produce (n=4) toxins and on non-colonised control patients (n=6), all of whom were experiencing diarrhoea. We report a statistically significant separation (P-value o0…

DiarrheaMaleBacterial ToxinsDiseasePathogenesisGut floraMicrobiologyMicrobiologyFecesClostridiumMetabolomicsRNA Ribosomal 16SmedicineMetabolomeHumansMetabolomicsColitisEcology Evolution Behavior and SystematicsbiologyClostridioides difficileClostridium difficilebiology.organism_classificationmedicine.diseaseColitisIntestinesRNA BacterialDiarrheaClostridium InfectionsMetabolomeFemaleOriginal Articlemedicine.symptomBacterial infection
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Hyaluronic Acid Increases Anti-Inflammatory Efficacy of Rectal 5-Amino Salicylic Acid Administration in a Murine Colitis Model

2021

5-amino salicylic acid (5-ASA) is a standard therapy for the treatment of mild to moderate forms of inflammatory bowel diseases (IBD) whereas more severe forms involve the use of steroids and immunosuppressive drugs. Hyaluronic acid (HA) is a naturally occurring non-sulfated glycosaminoglycan that has shown epithelium protective effects in experimental colitis recently. In this study, both 5-ASA (30 mg/kg) and HA (15 mg/kg or 30 mg/kg) were administered rectally and investigated for their potential complementary therapeutic effects in moderate or severe murine colitis models. Intrarectal treatment of moderate and severe colitis with 5-ASA alone or HA alone at a dose of 30 mg/kg led to a sig…

Drug5-amino salicylic acidmedicine.drug_classHyaluronic acidmedia_common.quotation_subjectInflammationPharmacologyBiochemistryInflammatory bowel diseaseInflammatory bowel diseaseAnti-inflammatorychemistry.chemical_compoundDrug DiscoveryHyaluronic acidmedicineColitismedia_commonInflammationPharmacologybusiness.industryTherapeutic effectColitismedicine.diseasechemistryMolecular MedicineOriginal Articlemedicine.symptombusinessSalicylic acidBiomolecules & Therapeutics
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Efficacy of budesonide-loaded mesoporous silica microparticles capped with a bulky azo derivative in rats with TNBS-induced colitis.

2019

Abstract A colon targeted drug delivery system for inflammatory bowel diseases (IBD), consisting in budesonide loaded mesoporous silica microparticles functionalized with a selective azo-molecular gate (M-Bud), has been evaluated for in vivo efficacy. Experimental colitis in male Wistar rats was induced by rectal instillation of 2,4,6-trinitrobenzenesulfonic acid (TNBS). M-Bud was orally administered to the rats as a suspension in water. Colon/body weight ratio, clinical activity score, and histological evaluation were used as inflammatory indices to measure the performance of the microparticles. The formulation was compared with a suspension prepared from the commercial drug Entocord®. Sta…

DrugBudesonideMalemedia_common.quotation_subjectPharmaceutical Science02 engineering and technologyPharmacology030226 pharmacology & pharmacy03 medical and health sciences0302 clinical medicineDrug Delivery SystemsIn vivomedicineAnimalsColitisBudesonideTnbs colitismedia_commonChemistryMesoporous silica021001 nanoscience & nanotechnologymedicine.diseaseColitisSilicon DioxideControlled releasedigestive system diseasesRatsTargeted drug deliveryTrinitrobenzenesulfonic Acid0210 nano-technologyAzo Compoundsmedicine.drugInternational journal of pharmaceutics
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5-Aminosalicylsäure-assoziierte Pankreatitis

2008

Acute pancreatitis with severe belt-like upper abdominal pain developed within 1-4 weeks of starting medication in three patients (29-year-old man with ulcerative colitis; 43-year-old woman and 22-year-old woman with Crohn's disease) treated, for the first time, with 5-aminosalicylic acid (mesalazine), 500 mg three times daily. Concentrations of lipase initially were 545, 1182 and 3000 U/l, and of amylase 243, 449 and 129 U/l, respectively. Symptoms receded within a few hours after the drug had been discontinued, enzyme levels returning to normal in the course of the next 2-3 weeks. On repeating the drug in two of the patients, in lower dosage, the pancreatitis recurred within a few days. T…

Drugmedicine.medical_specialtybiologybusiness.industrymedia_common.quotation_subjectGeneral Medicinemedicine.diseaseGastroenterologyUlcerative colitischemistry.chemical_compoundMesalazinechemistryInternal medicineUpper abdominal painbiology.proteinMedicinePancreatitisAcute pancreatitisAmylaseLipasebusinessmedia_commonDMW - Deutsche Medizinische Wochenschrift
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Guanosine prevents nuclear factor-κB nuclear translocation ameliorating experimental colitis in rats

2018

Background inflammatory bowel diseases (IBDs), including Crohn’s disease (CD) and ulcerative colitis (UC), are prevalent and debilitating health problems worldwide. Due to the adverse effects of classical treatment for IBD, therapeutic options and approaches for these diseases continue to evolve. Guanosine, a guanine-based purine, is an extracellular signalling molecule that seems to exert anti-inflammatory and antioxidative effects in several in vivo and in vitro injury models. The aim of the present study was to investigate whether exogenous guanosine may have protective effects on 2,4-dinitrobenzene sulfonic acid (DNBS)-induced Colitis in rat. Methods Experimental Colitis was induced by …

EXPERIMENTAL COLITIS GUANOSINE INFLAMMATORY BOWEL DISEASESettore BIO/09 - Fisiologia
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IL-12 family members in experimental colitis

2008

Interleukin (IL)-12 p35/p40 is a heterodimeric cytokine that plays an important role in T helper (Th) cell polarization and Th1 T-cell differentiation. Recent findings have shown that both p35 and p40 can form other cytokines with different proteins (IL-23: p19/p40; IL-35: p35/EBI3). Furthermore, the cytokine IL-27 (EBI3/p28) has been identified as a member of the IL-12 family. Here, we discuss the recent findings on the role of IL-12 family members in experimental colitis. In particular, the role of IL-23 as a master regulator of effector T-cell activation is highlighted. These findings have important implications for the design of new therapeutic approaches in chronic intestinal inflammat…

EffectorT-Lymphocytesmedicine.medical_treatmentImmunologyInterleukinMaster regulatorExperimental colitishemic and immune systemsEBI3BiologyColitisInflammatory Bowel DiseasesInterleukin-12CytokineIntestinal inflammationImmunologyInterleukin 12medicineAnimalsHumansImmunology and AllergySignal TransductionMucosal Immunology
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TGF-beta as a T cell regulator in colitis and colon cancer

2005

TGF-beta is a pleiotropic cytokine with powerful immunosuppressive functions. Mice deficient for TGF-beta1 show a dramatic phenotype with severe multiorgan inflammation and die shortly after birth. Recent investigations have highlighted the role of TGF-beta in suppression of T cell mediated autoimmune inflammation and anti-tumor immunity. In addition to its direct anti-inflammatory effects on T cells, TGF-beta has been implicated as central regulator of regulatory T cells. TGF-beta not only mediates the suppression of effector T cells by Tregs, recent evidence also reveals a role for TGF-beta along with TCR stimulation in the peripheral induction of regulatory T cells from naïve CD4+CD25- c…

Endocrinology Diabetes and MetabolismT cellImmunologyBiologyGeneral Biochemistry Genetics and Molecular BiologyTCIRG1Interleukin 21T-Lymphocyte SubsetsTransforming Growth Factor betamedicineAnimalsHumansImmunology and AllergyCytotoxic T cellIL-2 receptorIntestinal MucosaAntigen-presenting cellZAP70Cell DifferentiationColitisNatural killer T cellDisease Models Animalmedicine.anatomical_structureColonic NeoplasmsImmunologyInflammation MediatorsCytokine & Growth Factor Reviews
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Does uremic enterocolitis exist?

1981

In rats a severe but compensated chronic renal insufficiency was induced by stepwise 9/10 nephrectomy. Despite this severe chronic renal insufficiency we observed no relevant pathological changes in the intestinal mucosa. In particular, we found no evidence of mucosal erosions, ulceration or pseudomembranous colitis, findings which are traditionally thought to be characteristic of the uremic state. This was also true of those animals dying prematurely from uremia. Thus serious doubts arise about the existence of “uremic enterocolitis”, doubts which also proved justified after a critical review of the literature on human pathology.

EnterocolitisPathologymedicine.medical_specialtybusiness.industrymedicine.medical_treatmentPseudomembranous colitismedicine.diseaseIntestinal absorptionNephrectomyUremiaIntestinal mucosamedicinemedicine.symptombusinessHuman PathologyPathologicalVirchows Archiv B Cell Pathology Including Molecular Pathology
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Liver infarction in a patient with Clostridium Difficile colitis. A possible connection?

2019

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Enterocolitismedicine.medical_specialtyFatal outcomebusiness.industryGastroenterologyClostridium difficileLiver infarctionGastroenterologyConnection (mathematics)Clostridium Difficile ColitisInternal medicinemedicinemedicine.symptombusinessJournal of Gastrointestinal and Liver Diseases
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