Search results for "Core"

showing 10 items of 1999 documents

GPRS Security for Smart Meters

2013

Part 1: Cross-Domain Conference and Workshop on Multidisciplinary Research and Practice for Information Systems (CD-ARES 2013); International audience; Many Smart Grid installations rely on General Packet Radio Service (GPRS) for wireless communication in Advanced Metering Infrastructures (AMI). In this paper we describe security functions available in GPRS, explaining authentication and encryption options, and evaluate how suitable it is for use in a Smart Grid environment. We conclude that suitability of GPRS depends on the chosen authentication and encryption functions, and on selecting a reliable and trustworthy mobile network operator.

GPRSComputer science[SHS.INFO]Humanities and Social Sciences/Library and information sciences02 engineering and technology[INFO] Computer Science [cs]Encryption[SHS.INFO] Humanities and Social Sciences/Library and information sciencesGPRS core networkAMI0202 electrical engineering electronic engineering information engineeringWirelessMetering mode[INFO]Computer Science [cs]General Packet Radio ServiceSmartgridMobile network operatorAuthenticationbusiness.industryComputerSystemsOrganization_COMPUTER-COMMUNICATIONNETWORKS020208 electrical & electronic engineering020206 networking & telecommunicationsSmart gridSecuritybusinessSmart MeteringComputer network
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No-Reference 3D Mesh Quality Assessment Based on Dihedral Angles Model and Support Vector Regression

2016

International audience; 3D meshes are subject to various visual distortions during their transmission and geometrical processing. Several works have tried to evaluate the visual quality using either full reference or reduced reference approaches. However, these approaches require the presence of the reference mesh which is not available in such practical situations. In this paper, the main contribution lies in the design of a computational method to automatically predict the perceived mesh quality without reference and without knowing beforehand the distortion type. Following the no-reference (NR) quality assessment principle, the proposed method focuses only on the distorted mesh. Specific…

Gamma distribution[ INFO.INFO-TS ] Computer Science [cs]/Signal and Image Processing[ INFO ] Computer Science [cs]Computer science02 engineering and technologycomputer.software_genre[INFO.INFO-AI]Computer Science [cs]/Artificial Intelligence [cs.AI]Quality (physics)[INFO.INFO-TS]Computer Science [cs]/Signal and Image ProcessingVisual maskingDistortion0202 electrical engineering electronic engineering information engineeringGamma distribution[INFO]Computer Science [cs]Polygon mesh[ INFO.INFO-AI ] Computer Science [cs]/Artificial Intelligence [cs.AI]No-reference mesh quality assessmentVisual masking effect020207 software engineeringSupport vector machineSupport vector regressionQuality ScoreHuman visual system modelDihedral angles020201 artificial intelligence & image processingData miningAlgorithmcomputer
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THE SHAPLEY-SOLIDARITY VALUE FOR GAMES WITH A COALITION STRUCTURE

2013

A value for games with a coalition structure is introduced, where the rules guiding cooperation among the members of the same coalition are different from the interaction rules among coalitions. In particular, players inside a coalition exhibit a greater degree of solidarity than they are willing to use with players outside their coalition. The Shapley value is therefore used to compute the aggregate payoffs for the coalitions, and the solidarity value to obtain the payoffs for the players inside each coalition.

General Computer ScienceCoalitional value Shapley value Owen value Solidarity value C71ComputingMilieux_PERSONALCOMPUTINGMathematicsofComputing_GENERALStructure (category theory)TheoryofComputation_GENERALComputingMethodologies_ARTIFICIALINTELLIGENCEShapley valueSolidarityjel:M2MicroeconomicsCore (game theory)Value (economics)jel:C0Economicsjel:D5Statistics Probability and UncertaintyBusiness and International Managementjel:B4jel:C6jel:D7Mathematical economicsjel:C7International Game Theory Review
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Evaluation of linkage of bipolar affective disorder to chromosome 18 in a sample of 57 German families.

1999

Previously reported linkage of bipolar affective disorder to DNA markers on chromosome 18 was reexamined in a large sample of German bipolar families. Twenty-three short tandem repeat markers were investigated in 57 families containing 103 individuals with bipolar I disorder (BPI), 26 with bipolar II disorder (BPII), nine with schizoaffective disorder of the bipolar type (SA/BP), and 38 individuals with recurrent unipolar depression (UPR). Evidence for linkage was tested with parametric and non-parametric methods under two definitions of the affected phenotype. Analysis of all 57 families revealed no robust evidence for linkage. Following previous reports we performed separate analyses afte…

Genetic MarkersMaleBipolar I disorderBipolar DisorderGenetic LinkageSchizoaffective disorderGenes RecessiveGenetic determinismNuclear FamilyCellular and Molecular NeuroscienceBipolar II disorderGenomic ImprintingChromosome 18GermanymedicineHumansFamilyBipolar disorderMolecular BiologyGenes DominantLinkage (software)GeneticsRecombination GeneticSex CharacteristicsModels GeneticChromosome Mappingmedicine.diseasePsychiatry and Mental healthChromosomal regionFemaleLod ScorePsychologyChromosomes Human Pair 18Molecular psychiatry
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Linkage studies of bipolar disorder in the region of the Darier's disease gene on chromosome 12q23-24.1.

1995

We have recently described a family in which there is cosegregation of major affective disorder with Darier's disease and have mapped this autosomal dominant skin disorder to 12q23-q24.1. This has provided an interesting candidate region for genetic studies of bipolar disorder. We have studied the segregation of seven markers spanning the Darier's disease locus in 45 bipolar disorder pedigrees and found modest evidence in support of linkage under heterogeneity for 5 of these markers. Nonparametric analyses were suggestive of linkage with a marker at the gene encoding a secretory form of phospholipase A2. Our sample has relatively low power to detect linkage under heterogeneity and independe…

Genetic MarkersMaleCandidate geneBipolar DisorderCosegregationGenotypeGenetic LinkageLocus (genetics)BiologyPhospholipases AGene mappingDarier DiseaseGenetic linkageDarier's diseasemedicineHumansBipolar disorderGenetics (clinical)AllelesGenes DominantGeneticsChromosomes Human Pair 12Chromosome Mappingmedicine.diseasePedigreePhospholipases A2FemaleLod ScoreDarier DiseaseAmerican journal of medical genetics
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An autosomal dominant retinitis pigmentosa family with close linkage to D7S480 on 7q.

1995

Retinitis pigmentosa is the most prevalent inherited disorder of the retina. It can be autosomal dominant (adRP), autosomal recessive (arRP) or X-linked (XLRP). A form of adRP mapping to chromosome 7q was reported in a large Spanish pedigree. We have typed DNA from the members of another Spanish family for polymorphic markers from the known candidate genes. Positive lod scores were obtained only for the markers located on 7q31-35, giving a maximum lod score of 2.98 (3.01 by multipoint analysis) at theta = 0.00 for D7S480. A brief clinical evaluation is given.

Genetic MarkersMaleCandidate genecongenital hereditary and neonatal diseases and abnormalitiesgenetic structuresBiologyAutosomal dominant retinitis pigmentosaGene mappingRetinitis pigmentosaGeneticsmedicineHumansGeneGenetics (clinical)Genes DominantLinkage (software)GeneticsChromosome Mappingmedicine.diseaseHuman geneticseye diseasesPedigreeGenetic markerFemaleLod ScoreChromosomes Human Pair 7Retinitis PigmentosaHuman genetics
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Mutations in the β-tropomyosin (TPM2) gene – a rare cause of nemaline myopathy

2002

Nemaline myopathy is a clinically and genetically heterogeneous muscle disorder. In the nebulin gene we have detected a number of autosomal recessive mutations. Both autosomal dominant and recessive mutations have been detected in the genes for alpha -actin and alpha -tropomyosin 3. A recessive mutation causing nemaline myopathy among the Old Order Amish has recently been identified in the gene for slow skeletal muscle troponin T. As linkage studies had shown that at least one further gene exists for nemaline myopathy, we investigated another tropomyosin gene expressed in skeletal muscle, the beta -tropomyosin 2 gene. Screening 66 unrelated patients, using single strand conformation polymor…

Genetic MarkersMaleGenetic LinkageProtein ConformationBiopsyMolecular Sequence DataMutation MissenseTropomyosinmacromolecular substancesMuscle disorderMyopathies NemalineTPM203 medical and health sciencesNebulin0302 clinical medicineNemaline myopathymedicineAnimalsHumansAmino Acid SequenceMuscle SkeletalNemaline bodiesPolymorphism Single-Stranded ConformationalGenetics (clinical)DNA Primers030304 developmental biologyGenetics0303 health sciencesSequence Homology Amino AcidbiologyReverse Transcriptase Polymerase Chain Reactionmusculoskeletal systemmedicine.diseaseMolecular biologyTropomyosinCongenital myopathyPedigree3. Good healthHaplotypesNeurologyMutationPediatrics Perinatology and Child Healthbiology.proteinFemaleNeurology (clinical)Sequence Alignment030217 neurology & neurosurgeryCentral core diseaseNeuromuscular Disorders
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Detection of Hepatitis B Virus DNA in the Liver of Children with Chronic Hepatitis B by In Situ Hybridization and Its Relation to Other Viral Markers

1992

The aim of the study was to detect hepatitis B virus (HBV) DNA by in situ hybridization (ISH) with a 35S-labeled radioactive probe in frozen liver biopsy tissue sections of 63 hepatitis B virus surface antigen (HBsAg)-positive children. The results were compared to other markers of viral replication. HBV DNA was detected in 48 children. Of the 15 negative cases, four had hepatitis B envelope antigen (HBeAg), 10 anti-HBe, and one neither HBeAg nor anti-HBe. Free HBV DNA in serum and liver was positive in one patient. Forty of the positive children were HBeAg- and six anti-HBe-positive; two were negative for both. Of 45 36 had HBV DNA in serum. In 38 of 47 HBV DNA and in 31 of 42 HBcAg could …

Genetic MarkersMaleHepatitis B virusHBsAgAdolescentHepatitis B virus DNA polymerasemedicine.disease_causemedicineHumansChildHepatitis B virusbiologymedicine.diagnostic_testGastroenterologyInfantNucleic Acid Hybridizationvirus diseasesHepatitis BHepatitis Bbiology.organism_classificationmedicine.diseaseHepatitis B Core AntigensVirologydigestive system diseasesBlotting SouthernHBcAgLiverHepadnaviridaeHBeAgChild PreschoolLiver biopsyChronic DiseaseDNA ViralPediatrics Perinatology and Child HealthFemaleJournal of Pediatric Gastroenterology and Nutrition
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Mutations in SLC20A2 link familial idiopathic basal ganglia calcification with phosphate homeostasis.

2012

Familial idiopathic basal ganglia calcification (IBGC) is a genetic condition with a wide spectrum of neuropsychiatric symptoms, including parkinsonism and dementia. Here, we identified mutations in SLC20A2, encoding the type III sodium-dependent phosphate transporter 2 (PiT2), in IBGC-affected families of varied ancestry, and we observed significantly impaired phosphate transport activity for all assayed PiT2 mutants in Xenopus laevis oocytes. Our results implicate altered phosphate homeostasis in the etiology of IBGC.

Genetic Markersmedicine.medical_specialtyGenetic LinkageMolecular Sequence DataMutation MissenseXenopusBasal ganglia calcification610 Medicine & healthPhosphates10052 Institute of PhysiologyXenopus laevis03 medical and health scienceschemistry.chemical_compound0302 clinical medicineAsian PeopleBasal Ganglia Diseases1311 GeneticsCalcinosisGenetic linkageInternal medicineGeneticsmedicineAnimalsHomeostasisHumansBasal ganglia disease030304 developmental biology0303 health sciencesBase SequencebiologySodium-Phosphate Cotransporter Proteins Type IIIParkinsonismCalcinosisSequence Analysis DNAmedicine.diseasePhosphatebiology.organism_classificationPedigreeEndocrinologychemistry10076 Center for Integrative Human PhysiologyOocytes570 Life sciences; biologyLod Score030217 neurology & neurosurgeryHomeostasisChromosomes Human Pair 8Nature genetics
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Meta-analysis of genome-wide linkage scans of attention deficit hyperactivity disorder

2008

Contains fulltext : 69243.pdf (Publisher’s version ) (Closed access) Genetic contribution to the development of attention deficit hyperactivity disorder (ADHD) is well established. Seven independent genome-wide linkage scans have been performed to map loci that increase the risk for ADHD. Although significant linkage signals were identified in some of the studies, there has been limited replications between the various independent datasets. The current study gathered the results from all seven of the ADHD linkage scans and performed a Genome Scan Meta Analysis (GSMA) to identify the genomic region with most consistent linkage evidence across the studies. Genome-wide significant linkage (P(S…

Genetics and epigenetic pathways of disease [NCMLS 6]Genetic LinkageEuropean Continental Ancestry GroupMedizinGenome ScanBiologyNeuroinformatics [DCN 3]Mental health [NCEBP 9]Genetic determinismWhite PeopleArticleChromosomesGenomic disorders and inherited multi-system disorders [IGMD 3]03 medical and health sciencesCellular and Molecular Neuroscience0302 clinical medicineGene mappingCognitive neurosciences [UMCN 3.2]Genetic linkageGenetic predispositionmedicinePerception and Action [DCN 1]Attention deficit hyperactivity disorderHumansddc:610Medizinische Fakultät » Universitätsklinikum Essen » LVR-Klinikum Essen » Klinik für Psychiatrie Psychosomatik und Psychotherapie des Kindes- und JugendaltersGenetics (clinical)030304 developmental biologyProbabilityLinkage (software)Genetics0303 health sciencesGenomeGenome HumanPair 16Chromosome Mappingmedicine.diseasePsychiatry and Mental healthGenetic defects of metabolism [UMCN 5.1]Attention Deficit Disorder with HyperactivityMeta-analysisLod ScoreFunctional Neurogenomics [DCN 2]030217 neurology & neurosurgeryChromosomes Human Pair 16HumanAmerican Journal of Medical Genetics. Part B: Neuropsychiatric Genetics
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