Search results for "Cricetulu"

showing 10 items of 123 documents

Thyroid Stimulating Antibodies Are Highly Prevalent in Hashimoto's Thyroiditis and Associated Orbitopathy

2016

Thyroid-associated orbitopathy (TAO) rarely occurs in patients with Hashimoto's thyroiditis (HT).There is evidence that TSH receptor stimulating antibodies (TSAb) play a role in the pathogenesis of TAO. In this report, the prevalence of TSAb in HT patients with and without TAO was studied.This is a longitudinal observational study.The study took place in an academic joint thyroid-eye clinic.A total of 1055 subjects were included.TSAb was measured with a Food and Drug Administration-cleared bioassay that uses Chinese hamster ovary cells expressing a chimeric TSH receptor and a cAMP response element-dependent luciferase. Results of TSAb activity were reported as percentage of specimen-to-refe…

AdultMaleendocrine systemmedicine.medical_specialtyAdolescentendocrine system diseasesEndocrinology Diabetes and MetabolismClinical Biochemistry030209 endocrinology & metabolismContext (language use)CHO CellsHashimoto DiseaseBiochemistryThyroiditisPathogenesisYoung Adult03 medical and health sciencesCricetulus0302 clinical medicineEndocrinologyCricetinaeInternal medicineAnimalsHumansMedicineYoung adultReceptorbiologybusiness.industryBiochemistry (medical)ThyroidOutcome measuresReceptors ThyrotropinMiddle Agedmedicine.diseaseeye diseasesGraves OphthalmopathyEndocrinologymedicine.anatomical_structure030220 oncology & carcinogenesisbiology.proteinFemaleAntibodybusinessImmunoglobulins Thyroid-StimulatingThe Journal of Clinical Endocrinology & Metabolism
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Prevalence and clinical relevance of thyroid stimulating hormone receptor-blocking antibodies in autoimmune thyroid disease

2017

Summary The prevalence and clinical relevance of thyroid stimulating hormone (TSH) receptor (TSHR) blocking antibodies (TBAb) in patients with autoimmune thyroid disease (AITD) was investigated. Serum TBAb were measured with a reporter gene bioassay using Chinese hamster ovary cells. Blocking activity was defined as percentage inhibition of luciferase expression relative to induction with bovine TSH alone (cut-off 40% inhibition). All samples were measured for TSHR stimulatory antibody (TSAb) and TSHR binding inhibiting immunoglobulins (TBII). A total of 1079 unselected, consecutive patients with AITD and 302 healthy controls were included. All unselected controls were negative for TBAb and…

AdultMaleendocrine systemmedicine.medical_specialtyAdolescentendocrine system diseasesGraves' diseaseImmunologyThyroid Gland030209 endocrinology & metabolismCHO CellsHashimoto DiseaseThyroiditisYoung Adult03 medical and health sciencesCricetulus0302 clinical medicineCricetinaeInternal medicineBlocking antibodyPrevalencemedicineAnimalsHumansImmunology and AllergyEuthyroidClinical significanceAutoantibodiesbiologybusiness.industryChinese hamster ovary cellThyroidThyroiditis AutoimmuneReceptors ThyrotropinOriginal ArticlesMiddle Agedmedicine.diseaseeye diseasesGraves Diseasemedicine.anatomical_structureEndocrinology030220 oncology & carcinogenesisImmunologybiology.proteinBiological AssayFemaleAntibodybusinesshormones hormone substitutes and hormone antagonistsClinical and Experimental Immunology
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Analytical performance and clinical utility of a bioassay for thyroid-stimulating immunoglobulins.

2013

Abstract The analytical performance and the clinical utility of a thyrotropin receptor (TSHR)–stimulating immunoglobulin (TSI) bioassay were compared with those of a TSHR-binding inhibitory immunoglobulin (TBII) assay. Limits of detection (LoD) and quantitation (LoQ), assay cutoff, and the half-maximal effective concentration (EC50) were measured. Dilution analysis was performed in sera of hyperthyroid patients with Graves disease (GD) during antithyroid treatment (ATD). Titer was defined as the first dilution step at which measurement of TSI or TBII fell below the assay cutoff. The LoD, LoQ, cutoff, and EC50 of the bioassay were 251-, 298-, 814-, and 827-fold lower than for the TBII assay.…

AdultMaleendocrine systemmedicine.medical_specialtyendocrine system diseasesSerial dilutionGraves' diseaseCHO CellsThyrotropin receptorYoung AdultCricetulusAntithyroid AgentsLimit of DetectionInternal medicineCricetinaemedicineBioassayAnimalsHumansProspective StudiesAgedDetection limitbiologyDose-Response Relationship DrugChemistryReproducibility of ResultsGeneral MedicineMiddle Agedmedicine.diseaseeye diseasesGraves DiseaseTiterEndocrinologybiology.proteinThyroid Stimulating ImmunoglobulinBiological AssayFemaleAntibodyBiomarkersImmunoglobulins Thyroid-StimulatingAmerican journal of clinical pathology
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Cytogenetic effects of promutagens in genetically engineered V79 Chinese hamster cells expressing cytochromes P450.

1993

Abstract V79 Chinese hamster cell lines genetically engineered to express rat CYP2B1, CYP1A1, CYP1A2, and their parental cell lines V79-MZ, without acetyltransferase, and V79-NH, with acetyltransferase, were studied for chromosome aberrations and sister chromatid exchange induced by aflatoxin B 1 , cyclophosphamide, benzo[a]pyrene, 7,12-dimethylbenz[a]anthracene and dimethylnitrosamine. The parental V79 cell lines did not show clastogenic effects. Significant clastogenic effects were observed after an 18 h exposure to aflatoxin B 1 and cyclophosphamide in CYP2B1 expressing cells, to benzo[a]pyrene in CYP1A1 and CYP1A2 expressing cells, to 7,12-dimethylbenz[a]anthracene and dimethylnitrosami…

Aflatoxin B1910-Dimethyl-12-benzanthraceneHamsterSister chromatid exchangeMutagenToxicologymedicine.disease_causeChinese hamsterCell LineDimethylnitrosamineClastogenCricetulusCytochrome P-450 Enzyme SystemCricetinaepolycyclic compoundsmedicineBenzo(a)pyreneAnimalsCyclophosphamideBiotransformationPharmacologyChromosome Aberrationsbiologyrespiratory systembiology.organism_classificationPollutionMolecular biologyIn vitroRatsCell cultureAcetyltransferaseGenetic EngineeringSister Chromatid ExchangeMutagensEuropean journal of pharmacology
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Characterization of an epithelial, nearly diploid liver cell strain, from Chinese hamster, able to activate promutagens

1987

Epithelial liver cells of the Chinese hamster (CHEL cells) were propagated in culture for 35 passages. At favourable cell densities, the population doubling time in normal medium, was 20 h. L-Tyrosine amino transferase activity was retained at a measurable level, but its enhancement by dexamethasone was detected solely in cells of early passages. Pyruvate kinase was strongly activated by fructose-1,6-biphosphate at low substrate concentrations. These enzymatic properties suggest that the CHEL cells are derived from a sub-population of parenchymal hepatocytes or from cells closely related to parenchymal hepatocytes. With a lag period of a few hours, CHEL cultures metabolized benzo[a]pyrene. …

Aflatoxin B1910-Dimethyl-12-benzanthraceneHealth Toxicology and MutagenesisPyruvate KinaseCellToxicologyEpitheliumChinese hamsterCricetulusAflatoxinsCricetinaeBenzo(a)pyreneGeneticsmedicineAnimalsDoubling timeBiotransformationCells CulturedGenetics (clinical)Tyrosine TransaminaseGeneticsbiologyLiver cellEpithelial CellsMonooxygenasebiology.organism_classificationMolecular biologyClone CellsEpoxide hydrolase activitymedicine.anatomical_structureLiverKaryotypingPloidyCell DivisionPyruvate kinaseMutagensMutagenesis
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Selection and characterization of a novel agonistic human recombinant anti-Trail-R2 minibody with anti-leukemic activity

2009

Tumor necrosis factor-related apoptosis-inducing ligand (TRAIL) is a promising natural anticancer therapeutic agent because through its “death receptors”, TRAIL-R1 and TRAIL-R2, it induces apoptosis in many transformed tumor cells, but not in the majority of normal cells. Hence, agonistic compounds directed against TRAIL death receptors have the potential of being excellent cancer therapeutic agents, with minimal cytotoxicity in normal tissues. Here, we report the selection and characterization of a new single-chain fragment variable (scFv) to TRAIL-R2 receptor isolated from a human phage-display library, produced as minibody (MB), and characterized for the in vitro anti-leukemic tumoricid…

Agonistmedicine.drug_classTRAIL; TRAIL-R2; minibody; anticancer therapyImmunologylymphoma; therapy; recombinant antibodyTRAILApoptosislymphomaCHO CellsCricetulusPeptide LibraryTRAIL-R2CricetinaeImmunoglobulin FragmentmedicineAnimalsHumansImmunology and Allergyrecombinant antibodyanticancer therapyReceptorCytotoxicityImmunoglobulin FragmentsPharmacologytherapyLeukemiaChemistryAnimalChinese hamster ovary cellAntibody-Dependent Cell CytotoxicityminibodyApoptosiIn vitroRecombinant ProteinsReceptors TNF-Related Apoptosis-Inducing LigandCHO CellCell cultureApoptosisImmunologyCancer researchTumor necrosis factor alphaCricetuluHuman
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Karyotype Abnormalities in a Variant Chinese Hamster Cell Line Resistant to Methyl Methanesulphonate

2004

A variant cell population, isolated from V79-CI 3 Chinese hamster cells after two consecutive treatments with methyl methanesulphonate (MMS), was found to be highly resistant to killing by this alkylating agent. The resistant cell line was cytogenetically characterized both by the presence of a stable translocation involving metacentric chromosome 2 and acrocentric chromosome 6 and by a supernumerary chromosome originated by the duplication of a small telocentric chromosome. This cell population also showed a transient transformed phenotype, seen as formation of transformed foci containing cells with high chromosomes counts and multiple chromosomal aberrations. As MMS-resistance and karyoty…

Alkylating AgentsCellPopulationDrug ResistanceChromosome DisordersChromosomal translocationChinese hamsterCell LineCricetulusCricetinaeGene duplicationGeneticsmedicineAnimalseducationChromosome AberrationsGeneticseducation.field_of_studybiologyChromosome MappingChromosomeKaryotypeGeneral MedicineMethyl Methanesulfonatebiology.organism_classificationMolecular biologyChromosome Bandingmedicine.anatomical_structureCell cultureKaryotypingHereditas
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Kinetics of gamma-H2AX focus formation upon treatment of cells with UV light and alkylating agents.

2008

Histone H2AX is rapidly phosphorylated in response to DNA double-strand breaks (DSBs) induced by ionizing radiation (IR). Here we show that DNA damage induced by alkylating agents [methyl methanesulfonate (MMS) and N-methyl-N'-nitro-N-nitrosoguanidine (MNNG)] and ultraviolet light (UV-C) leads to a dose and time dependent accumulation of phosphorylated H2AX (gamma-H2AX). Time course experiments revealed that the number of gamma-H2AX foci reached peak levels 8 hr after MMS or MNNG treatment and declined to almost control values within 24 hr after exposure. Upon UV-C treatment, a biphasic response was observed with a maximum 12 hr after treatment. In 43-3B cells deficient in nucleotide excisi…

Alkylating AgentsMethylnitronitrosoguanidineTime FactorsDNA RepairEpidemiologyDNA damageMethylnitronitrosoguanidineDNA repairUltraviolet RayscellsHealth Toxicology and MutagenesisFluorescent Antibody TechniqueCHO CellsBiologyenvironment and public healthHistoneschemistry.chemical_compoundCricetulusCricetinaeUltraviolet lightAnimalsPhosphorylationGenetics (clinical)DNA replicationMethyl MethanesulfonateMolecular biologyMethyl methanesulfonateenzymes and coenzymes (carbohydrates)KineticschemistryBiochemistrybiological phenomena cell phenomena and immunityDNANucleotide excision repairDNA DamageEnvironmental and molecular mutagenesis
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Hypertonic Stress and Amino Acid Deprivation Both Increase Expression of mRNA for Amino Acid Transport System A

2004

The activity of amino acid transport system A ([Oxender and Christensen, 1963][1]) is regulated in a variety of different ways, the best studied being the increases of its activity caused by starving cells of amino acids or by exposing them to hypertonicity (for review see [McGivan and Pastor-

Amino Acid Transport System APhysiologyCHO CellsBiologyCricetulusOsmotic PressureCricetinaeAnimalsHumansOsmotic pressureRNA MessengerAmino AcidsLetter to the Editorchemistry.chemical_classificationRegulation of gene expressionMessenger RNAChinese hamster ovary cellbiology.organism_classificationAmino acidGene Expression RegulationHypotonic SolutionschemistryBiochemistryHypertonic StressTonicityCricetulusJournal of General Physiology
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The DNA methylation inhibitor 5-azacytidine modulates 6-thioguanine toxicity in mammalian cells

2003

In order to assess the effects of combining two antimetabolites used separately to treat human leukemias, we carried out an experimental study by exposing V79 Chinese hamster cells, a 6-thioguanine (6-tG)-sensitive cell line, to sequential and concurrent treatments with 5-azacytidine (5-azaC) and 6-tG. In this paper, we demonstrate that there is a clear dependency for the way in which this combination was tested. Pre-treatment with 5-azaC made V79 cells more resistant to 6-tG by a substantial reduction in 6-tG incorporation into DNA; this effect could still be detected for several cell divisions after the removal of 5-azaC, and was achieved neither by reduced cell growth nor by the inductio…

Antimetabolites AntineoplasticHypoxanthine Phosphoribosyltransferasemedicine.drug_classCell SurvivalCellHamsterToxicologyAntimetaboliteChinese hamster6-thioguanineCricetulus5-azacytidineCricetinaeAntineoplastic Combined Chemotherapy ProtocolsmedicineAnimalsThioguanineCells CulturedbiologyCell growthtoxicityDrug SynergismGeneral MedicineDNAProdrugDNA Methylationbiology.organism_classificationMolecular biologySettore BIO/18 - Geneticamedicine.anatomical_structureBiochemistryCell cultureToxicityAzacitidineSister Chromatid ExchangeCell Division
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