Search results for "Cyclic compound"

showing 10 items of 819 documents

International Normalized Ratio and Mortality Risk in Acute Heart Failure and Nonvalvular Atrial Fibrillation Patients Receiving Vitamin K Antagonists

2019

Introduction and objectives: Heart failure patients with nonvalvular atrial fibrillation (NVAF) on treatment with vitamin K antagonists (VKA) often have suboptimal international normalized ratio (INR) values. Our aim was to evaluate the association between INR values at admission due to acute heart failure and mortality risk during follow-up. Methods: In this observational study, we retrospectively assessed INR on admission in 1137 consecutive patients with acute heart failure and NVAF who were receiving VKA treatment. INR was categorized into optimal values (INR = 2-3, n = 210), subtherapeutic (INR 3, n = 267). Because INR did not meet the proportional hazards assumption for mortality, res…

Maleendocrine systemmedicine.medical_specialtyTime FactorsVitamin KHeart failure030204 cardiovascular system & hematologyVitamin kRisk Assessment03 medical and health sciences0302 clinical medicineRisk FactorsInterquartile rangeCause of DeathThromboembolismhealth services administrationMean Survival TimeInternal medicineAtrial FibrillationmedicineHumansheterocyclic compoundsInternational Normalized Ratiocardiovascular diseasesInternational normalized ratioNormal rangeAgedRetrospective StudiesHeart Failurebusiness.industryIncidencefungiAnticoagulantsAtrial fibrillationGeneral MedicinePrognosismedicine.diseaseAtrial fibrillationSurvival RateSpainHeart failureAcute DiseaseCardiologyFemaleObservational studybusinessFollow-Up StudiesRevista Española de Cardiología (English Edition)
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Interrelationship between demethylation of p-nitroanisole and conjugation of p-nitrophenol in rat liver

1973

The metabolism of p-nitroanisole (pNA) and p-nitrophenol (pNP) was studied in isolated rat livers perfused with a hemoglobin-free medium. The activity and viability of the surviving organ was tested by recording pH, “arterial” and “venous” oxygen tension as well as the disappearance of added pNP. pNA is converted to its primary metabolite pNP which, in turn, is excreted into the perfusion medium as conjugates. The coordination of pNA oxidation and the conjugation reactions of pNP were investigated. When 50 μM pNA is added as substrate 0.4±0.1 nmoles×ml−1×(g liver)−1 are excreted as pNP-glucuronide and 3.5±0.2 nmoles×ml−1×(g liver)−1 as the sulphate within 90 min. When pNP itself (50 μM) is …

Maleinorganic chemicalsGlucuronidationGlucuronatesAnisolesIn Vitro TechniquesMethylationNitrophenolsOrganophosphorus CompoundsSulfationAnimalsheterocyclic compoundsGlucuronosyltransferaseGlucuronidaseDemethylationPharmacologyChromatographyChemistryHydrolysisLiver cellSubstrate (chemistry)General MedicineMetabolismHydrogen-Ion ConcentrationNitro CompoundsRatsOxygen tensionOxygenenzymes and coenzymes (carbohydrates)LiverBiochemistryMicrosomes LiverSulfatasesGlucuronideOxidation-ReductionGlucosidasesNaunyn-Schmiedeberg's Archives of Pharmacology
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Does the NO/sGC/cGMP/PKG pathway play a stimulatory role in platelets?

2012

Nitric oxide (NO) stimulates cGMP synthesis by activating its intracellular receptor, soluble guanylyl cyclase (sGC). It is a currently prevailing concept that No and cGMP inhibits platelet function. However, the data supporting the inhibitory role of NO/sGC/cGMP in platelets have been obtained either in vitro or using whole body gene deletion that affects vessel wall function. Here we have generated mice with sGC gene deleted only in megakaryocytes and platelets. Using the megakaryocyte- and platelet-specific sGC-deficient mice, we identify a stimulatory role of sGC in platelet activation and in thrombosis in vivo. Deletion of sGC in platelets abolished cGMP production induced by either NO…

Maleinorganic chemicalsbusiness.industryImmunologyReceptors Cytoplasmic and NuclearCell BiologyHematologyPlatelet ActivationPlatelets and ThrombopoiesisBiochemistryCell biologyCyclic gmpGuanylate Cyclasecardiovascular systemAnimalsMedicineFemaleheterocyclic compoundsPlateletbusinessSoluble guanylyl cyclaseBlood
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Biosynthesis and transformation of 20α 21-dihydroxycholesterol by rat adrenal preparations

1979

Abstract The biosynthesis of [ 3 H]-20α, 21 dihydroxycholestderol from [ 3 H]-20α-hydroxycholesterol and its transformation to [ 3 H]-21-hydroxypregnenolone by rat adrenal preparations has been demonstrated. 20α-Hydroxycholesterol was transformed to 20α, 21-dihydroxycholesterol by microsomal preparations in the presence of NADPH and 20α-21-dihydroxycholesterol was metabolized to 21-hydroxypregnenolone by mitochondrial preparations in the presence of a NADPH-generating-system. Comparison of the Michaelis-Menten-Kinetics of the steps “20α, 21-dihydroxycholesterol → 21-hydroxycholesterol” and “20α-hydroxycholesterol → pregnenolone” revealed that both compounds behaved as analogue substrates of…

Malemedicine.drug_classStereochemistryBiology17-alpha-HydroxypregnenoloneBiochemistrychemistry.chemical_compoundEndocrinologyBiosynthesisMicrosomesAdrenal Glandspolycyclic compoundsmedicineAnimalsCholesterol Side-Chain Cleavage EnzymeCholesterol side-chain cleavage enzymeHydroxycholesterolsMitochondriaRatsTransformation (genetics)BiochemistrychemistryAlternative complement pathwayPregnenoloneMicrosomeCorticosteroidlipids (amino acids peptides and proteins)NADPmedicine.drugJournal of Steroid Biochemistry
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Gastric relaxation induced by apigenin and quercetin: Analysis of the mechanism of action

2009

Abstract Aims Recently, flavonoids have been shown to cause murine gastric relaxation. In the present study we examined the mechanism of action underlying gastric relaxation induced by apigenin and quercetin in isolated mouse stomach. Main methods The mechanical activity from the whole stomach was detected as changes in the endoluminal pressure and the response to increasing concentrations of both flavonoids were tested before and after different pharmacological treatments. Key findings Apigenin and quercetin-induced a concentration-dependent gastric relaxation, apigenin being more potent than quercetin. The responses were unaffected by 2′5′dideoxyadenosine, an inhibitor of adenylate cyclas…

Malemedicine.medical_specialtyCarbacholNifedipineMuscle Relaxationchemistry.chemical_elementCalcium antagonistIn Vitro TechniquesMuscarinic AgonistsCalciumPharmacologySettore BIO/09 - FisiologiaGeneral Biochemistry Genetics and Molecular BiologyPotassium ChlorideMicechemistry.chemical_compoundSmooth muscleInternal medicineCyclic AMPmedicineAnimalsheterocyclic compoundsApigeninGeneral Pharmacology Toxicology and PharmaceuticsCyclic GMPCyclic nucleotide phosphodiesteraseChemistryRyanodine receptorStomachMuscle SmoothGeneral MedicineCalcium Channel BlockersMice Inbred C57BLEndocrinologyMechanism of actionGastric toneApigeninFlavonoidCalciumCarbacholQuercetinmedicine.symptomQuercetinIntracellularMuscle Contractionmedicine.drugLife Sciences
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Effects of dopamine antagonists with different receptor blockade profiles on morphine-induced place preference in male mice.

2001

The effects of dopamine (DA) antagonists with different selectivity for the DA receptors (SCH 23390, 0.5, 0.25, 0.125 mg/kg; haloperidol, 0.2, 0.1 mg/kg; raclopride, 1.2, 0.6, 0.3 mg/kg; risperidone, 0.4, 0.2, 0.1 mg/kg; U-99194A maleate, 40, 20 mg/kg; clozapine, 2.5, 1.25, 0.625 mg/kg) on the acquisition of place conditioning and morphine-induced conditioned place preference (CPP) were explored in male mice. Morphine (40 mg/kg) produced CPP while SCH 23390, haloperidol and clozapine (highest dose) and risperidone (lowest dose) produced conditioned place aversion (CPA). Raclopride and U-99194A maleate did not produce CPP or CPA. Morphine-induced CPP was reversed by the administration of SCH…

Malemedicine.medical_specialtyConditioning ClassicalPharmacologyChoice BehaviorReceptors DopamineBehavioral Neurosciencechemistry.chemical_compoundMiceDopamineInternal medicineOrientationpolycyclic compoundsmedicineHaloperidolAvoidance LearningAnimalsRacloprideSCH-23390MotivationDose-Response Relationship DrugMorphineChemistryAntagonistBrainConditioned place preferenceEndocrinologyDopamine receptorMorphineDopamine Antagonistsmedicine.drugBehavioural brain research
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Safety of meglumine gadoterate (Gd-DOTA)-enhanced MRI compared to unenhanced MRI in patients with chronic kidney disease (RESCUE study).

2012

To prospectively compare the renal safety of meglumine gadoterate (Gd-DOTA)-enhanced magnetic resonance imaging (MRI) to a control group (unenhanced MRI) in high-risk patients.Patients with chronic kidney disease (CKD) scheduled for MRI procedures were screened. The primary endpoint was the percentage of patients with an elevation of serum creatinine levels, measured 72 ± 24 h after the MRI procedure, by at least 25 % or 44.2 μmol/l (0.5 mg/dl) from baseline. A non-inferiority margin of the between-group difference was set at -15 % for statistical analysis of the primary endpoint. Main secondary endpoints were the variation in serum creatinine and eGFR values between baseline and 72 ± 24 h …

Malemedicine.medical_specialtyDrug-Related Side Effects and Adverse ReactionsContrast MediaNephrogenic Fibrosing DermopathyHeterocyclic CompoundsRisk FactorsOrganometallic CompoundsPrevalenceMedicineHumansRadiology Nuclear Medicine and imagingRenal Insufficiency ChronicneoplasmsNeuroradiologyAgedmedicine.diagnostic_testMegluminebusiness.industryAcute kidney injuryMagnetic resonance imagingInterventional radiologyGeneral MedicineAcute Kidney Injurymedicine.diseaseMagnetic Resonance ImagingEuropeTreatment OutcomeRadiology Nuclear Medicine and imagingNephrogenic systemic fibrosisAngiographyFemaleRadiologybusinessmedicine.drugKidney diseaseEuropean radiology
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Studies on the disposition, metabolism and hepatotoxicity of coumarin in the rat and Syrian hamster.

2002

The hepatotoxicity, metabolism and disposition of coumarin has been compared in male Sprague-Dawley rats and Syrian hamsters. The treatment of rats for 12, 24 and 42 weeks with diets containing 0.2 and 0.5% coumarin resulted in hepatotoxicity and increased relative liver weights. While levels of cytochrome P450 (CYP) and CYP-dependent enzymes were decreased, levels of reduced glutathione (GSH) and activities of UDP glucuronosyltransferase, gamma-glutamyltransferase and GSH S-transferase were increased. In contrast, coumarin produced few hepatic changes in the Syrian hamster. Following a single oral dose of 25 mg/kg [3-14C]coumarin, radioactivity was rapidly excreted by the rat and Syrian ha…

Malemedicine.medical_specialtyHamsterToxicologyRats Sprague-Dawleychemistry.chemical_compoundCytosolPharmacokineticsLiver Function TestsSpecies SpecificityOral administrationCoumarinsInternal medicineCricetinaemedicineToxicokineticsAnimalsheterocyclic compoundsTissue DistributionNutritionbiologyMesocricetusBody WeightCytochrome P450AnticoagulantsGeneral MedicineGlutathioneOrgan SizeCoumarinGlutathioneDietRatsEndocrinologychemistryLiverToxicitybiology.proteinChemical and Drug Induced Liver InjuryOxidation-ReductionFood ScienceFood and chemical toxicology : an international journal published for the British Industrial Biological Research Association
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Protein gene product (PGP) 9.5 immunoreactivity in nerve fibres and pinealocytes of guinea-pig pineal gland: interrelationship with tyrosine- hydroxy…

1993

This light-microscopic (LM) immunohistochemical study has evaluated the presence and distribution of the pan-neural and neuroendocrine marker protein gene product (PGP) 9.5 in pinealocytes and nerve fibres of guinea-pig pineal gland. The pattern of PGP 9.5-immunoreactive (ir) nerve fibres has been compared with that of fibres staining for tyrosine hydroxylase (TH) or neuropeptide Y (NPY). The vast majority of pinealocytes stained for PGP 9.5, although with variable intensity. PGP 9.5 immunoreactivity was localized in pinealocytic cell bodies and processes. Double-immunofluorescence revealed that PGP 9.5 immunoreactivity was absent from glial cells identified with a monoclonal antibody again…

Malemedicine.medical_specialtyHistologyendocrine system diseasesTyrosine 3-MonooxygenaseImmunocytochemistryGuinea PigsNerve fiberBiologyPineal GlandPathology and Forensic MedicinePinealocytePineal glandNerve FibersInternal medicinepolycyclic compoundsmedicineAnimalsNeuropeptide Yintegumentary systemTyrosine hydroxylaseGlial fibrillary acidic proteinCell BiologyNeuropeptide Y receptorMolecular biologyImmunohistochemistrymedicine.anatomical_structureEndocrinologybiology.proteinImmunohistochemistryThiolester HydrolasesUbiquitin ThiolesteraseCell and tissue research
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Paradoxically, iron overload does not potentiate doxorubicin-induced cardiotoxicity in vitro in cardiomyocytes and in vivo in mice

2015

Doxorubicin (DOX) is known to induce serious cardiotoxicity, which is believed to be mediated by oxidative stress and complex interactions with iron. However, the relationship between iron and DOX-induced cardiotoxicity remains controversial and the role of iron chelation therapy to prevent cardiotoxicity is called into question. Firstly, we evaluated in vitro the effects of DOX in combination with dextran-iron on cell viability in cultured H9c2 cardiomyocytes and EMT-6 cancer cells. Secondly, we used an in vivo murine model of iron overloading (IO) in which male C57BL/6 mice received a daily intra-peritoneal injection of dextran-iron (15mg/kg) for 3weeks (D0-D20) and then (D21) a single su…

Malemedicine.medical_specialtyIron OverloadCell SurvivalHeart VentriclesIronCardiomegaly030204 cardiovascular system & hematologyToxicologymedicine.disease_causeCell LineVentricular MyosinsMice03 medical and health sciences0302 clinical medicine[SDV.MHEP.CSC]Life Sciences [q-bio]/Human health and pathology/Cardiology and cardiovascular systemAtrial natriuretic peptideIn vivoCell Line TumorInternal medicineNatriuretic Peptide Brainpolycyclic compoundsmedicineAnimalsMyocytes CardiacDoxorubicinViability assay030304 developmental biologyPharmacology0303 health sciencesCardiotoxicityCell growthChemistryDextransBrain natriuretic peptideCardiotoxicity[SDV.MHEP.CSC] Life Sciences [q-bio]/Human health and pathology/Cardiology and cardiovascular systemUp-Regulation3. Good healthMice Inbred C57BLOxidative Stresscell proliferationEndocrinologyDoxorubicincardiovascular systemOxidative stressmedicine.drugToxicology and Applied Pharmacology
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