Search results for "DAMAGE"

showing 10 items of 1289 documents

Recommendations for the management of patients with aneurysmal subarachnoid hemorrhage

2005

After SAH, primary and secondary complications are frequent and often require neurosurgical interventions to avoid secondary brain damage. The authors of the present paper have summarized the available data about the treatment modalities often used for patients with SAH. The present recommendations have been developed as a neurosurgical and neuroanestesiological consensus. Evidence from prospective, randomized, double blind, placebo-controlled studies support grade A recommendations (standard) for the prophylaxis and treatment of cerebral vasospasm with oral Nimodipine in good grade patients. For intravenous Nimodipine or for oral nimodipine treatment in poor grade patients, available data …

medicine.medical_specialtySubarachnoid hemorrhagemedicine.medical_treatmentNeurosurgeryBrain damageCerebral vasospasmGermanyAngioplastyHypovolemiaHumansVasospasm IntracranialMedicineProspective cohort studyNimodipineRandomized Controlled Trials as Topicbusiness.industrySubarachnoid Hemorrhagemedicine.diseaseSurgeryAnesthesiaSurgeryNeurology (clinical)medicine.symptombusinessHypervolemiaAngioplasty Balloonmedicine.drugNeurochirurgie
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Mouse embryonic stem cells are hypersensitive to apoptosis triggered by the DNA damage O(6)-methylguanine due to high E2F1 regulated mismatch repair.

2007

Exposure of stem cells to genotoxins may lead to embryonic lethality or teratogenic effects. This can be prevented by efficient DNA repair or by eliminating genetically damaged cells. Using undifferentiated mouse embryonic stem (ES) cells as a pluripotent model system, we compared ES cells with differentiated cells, with regard to apoptosis induction by alkylating agents forming the highly mutagenic and killing DNA adduct O(6)-methylguanine. Upon treatment with N-methyl-N'-nitro-N-nitrosoguanidine (MNNG), ES cells undergo apoptosis at much higher frequency than differentiated cells, although they express a high level of the repair protein O(6)-methylguanine-DNA methyltransferase (MGMT). Apo…

Pluripotent Stem CellsMethylnitronitrosoguanidineDNA ComplementaryGuanineDNA damageDNA repairCellular differentiationApoptosisBiologyDNA Mismatch RepairModels BiologicalDNA AdductsMiceO(6)-Methylguanine-DNA MethyltransferaseDNA adductAnimalsMolecular BiologyEmbryonic Stem CellsSwiss 3T3 CellsBase SequenceCell DifferentiationCell BiologyDNA MethylationFibroblastsEmbryonic stem cellMolecular biologyDNA-Binding ProteinsMutS Homolog 2 ProteinDNA methylationDNA mismatch repairStem cellE2F1 Transcription FactorDNA DamageCell death and differentiation
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Danno all'ambiente e tutela della persona

2012

Il libro propone una riflessione sul danno ambientale, inquadrandola nella più ampia prospettiva della tutela della persona, alla quale la figura del danno ambientale sembra potersi ascrivere. Delineata la disciplina in tema di danno all'ambiente, quale emergente dal lungo iter normativo, dottrinale e giurisprudenziale, che ne ha segnato la sua evoluzione, emerge l'idea di un danno ambientale, che potrebbe definirsi esistenziale, in grado di suggerire riflessioni interessanti, tanto in tema di responsabilità ambientale, quanto riguardo all'area del danno alla persona. La parabola del danno ambientale, se continua forse oggi ad evocare pericolosi "mostriciattoli", è figlia della storia di un…

Liability Enviroment Existential damageResponsabilità Ambiente danno esistenzialeSettore IUS/01 - Diritto Privato
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Le cause di giustificazione della responsabilità civile e la nuova legittima difesa

2020

the essay addresses the problem of wrongfulness in torts and analyzes the recent reform of self-defense. In particular, the study highlights the effects on the structure of tort liability of the expansion of the area of necessary and proportional behavior. The essay aims to offer a systematic picture of the justifications in tort liability

Self-defense tort liability wrongfulness justifications damages danno ingiustoSettore IUS/01 - Diritto Privato
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Redox regulation of genome stability by effects on gene expression, epigenetic pathways and DNA damage/repair

2015

Reactive oxygen and nitrogen species (e.g. H2O2, nitric oxide) confer redox regulation of essential cellular signaling pathways such as cell differentiation, proliferation, migration and apoptosis. In addition, classical regulation of gene expression or activity, including gene transcription to RNA followed by translation to the protein level, by transcription factors (e.g. NF-κB, HIF-1α) and mRNA binding proteins (e.g. GAPDH, HuR) is subject to redox regulation. This review will give an update of recent discoveries in this field, and specifically highlight the impact of reactive oxygen and nitrogen species on DNA repair systems that contribute to genomic stability. Emphasis will be placed …

Genome instabilityRedox signalingRNA UntranslatedEpigenetic regulation of neurogenesisDNA RepairHuR mRNA-binding protein in the 3′-untranslated regionClinical BiochemistryHDAC histone deacetylaseReview ArticleAP-1 activator protein 1BiochemistryApe-1 apurinic/apyrimidinic endonuclease 1GPx-1 glutathione peroxidase-1Epigenesis GeneticHistonesTrx thioredoxinPHD prolylhydroxylaseBER base excision repairlcsh:QH301-705.5HO-1 heme oxygenase-1EpigenomicsGeneticsRegulation of gene expressionNox member of the NADPH oxidase familylcsh:R5-920JmjC Jumonji C domain-containing histone demethylasesHIF-1α hypoxia inducible factor-1α5-hmC 5-hydroxymethylcytosineddc:Cell biologyMMP matrix metalloproteinaseGrx glutaredoxinGAPDH glyceraldehyde-3-phosphate dehydrogenaseNrf2 nuclear factor erythroid related factor 2DNA methylationEpigeneticslcsh:Medicine (General)Oxidation-ReductionSignal Transduction5-mC 5-methylcytosineDNA repairDNA damageNF-κB nuclear factor-κBBiologyGenomic InstabilityRNS reactive nitrogen speciesROS reactive oxygen speciesNER nucleotide excision repairSOD superoxide dismutaseOxyR transcription factor (hydrogen peroxide-inducible genes activator)HumansEpigeneticsOrganic ChemistryPETN pentaerithrityl tetranitrateGene regulationOxidative StressDNMT DNA methyltransferaseGene Expression Regulationlcsh:Biology (General)AREs AU-rich elementsHAT histone acetyltransferaseKeap1 kelch-like ECH-associated protein 1BiomarkersCOPD chronic obstructive pulmonary disorderDNA DamageRedox Biology
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Sharing R&D investments in breakthrough technologies to control climate change

2017

This paper examines international cooperation on technological development as an alternative to international cooperation on GHG emission reductions. In order to analyze the scope of cooperation, a three-stage technology agreement formation game is solved. First, countries decide whether or not to sign up to the agreement. Then, in the second stage, the signatories (playing together) and the non-signatories (playing individually) select their investment in R&D. In this stage, it is assumed that the signatories not only coordinate their levels of R&D investment but also pool their R&D efforts to fully internalize the spillovers of their investment in innovation. Finally, in the third stage, …

021110 strategic defence & security studiesEconomics and EconometricsScope (project management)05 social sciencesControl (management)0211 other engineering and technologiesClimate change02 engineering and technologyInvestment (macroeconomics)MicroeconomicsOrder (exchange)Greenhouse gas0502 economics and businessDamagesEconomicsProduction (economics)050207 economicsIndustrial organizationOxford Economic Papers
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Cardiovascular risk assessment beyond Systemic Coronary Risk Estimation: A role for organ damage markers

2012

BACKGROUND: Cardiovascular risk assessment in the clinical practice is mostly based on risk charts, such as Framingham risk score and Systemic Coronary Risk Estimation (SCORE). These enable clinicians to estimate the impact of cardiovascular risk factors and assess individual cardiovascular risk profile. Risk charts, however, do not take into account subclinical organ damage, which exerts independent influence on risk and may amplify the estimated risk profile. Inclusion of organ damage markers in the assessment may thus contribute to improve this process. OBJECTIVE: Our aim was to evaluate the influence of implementation of SCORE charts with widely available indexes of organ damage, with t…

cardiovascular riskmedicine.medical_specialtyestimated glomerular filtration ratemicroalbuminuriaPhysiologyCardiovascular risk organ damage markersCoronary DiseaseLeft ventricular hypertrophyRisk Assessmentmetabolic syndromecardiovascular risk; estimated glomerular filtration rate; left ventricular hypertrophy; metabolic syndrome; microalbuminuria; prevention; score; target organ damagepreventiontarget organ damageSCOREInternal MedicineMedicineHumansAlbuminuriaRisk factorIntensive care medicineEstimationFramingham Risk Scorebusiness.industryMetabolic Syndrome XBiomarkermedicine.diseaseSurgeryleft ventricular hypertrophyAlbuminuriaMicroalbuminuriamedicine.symptomMetabolic syndromebusinessRisk assessmentCardiology and Cardiovascular MedicineBiomarkersGlomerular Filtration RateHuman
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Oxidative damage to mitochondrial DNA and glutathione oxidation in apoptosis: studiesin vivoandin vitro

1999

Free radicals may be involved in apoptosis although this is the subject of some controversy. Furthermore, the source of free radicals in apoptotic cells is not certain. The aim of this study was to elucidate the role of oxidative stress in the induction of apoptosis in serum-deprived fibroblast cultures and in weaned lactating mammary glands as in vitro and in vivo experimental models, respectively. Oxidative damage to mtDNA is higher in apoptotic cells than in controls. Oxidized glutathione (GSSG) levels in mitochondria from lactating mammary gland are also higher in apoptosis. There is a direct relationship between mtDNA damage and the GSSG/reduced glutathione (GSH) ratio. Furthermore, wh…

DNA damageApoptosisWeaningMitochondrionmedicine.disease_causeDNA MitochondrialBiochemistryCulture Media Serum-FreeMembrane Potentialschemistry.chemical_compoundCytosolMammary Glands AnimalIn vivoGeneticsmedicineAnimalsLactationAnaerobiosisRats WistarFragmentation (cell biology)Molecular BiologyCells CulturedGlutathione DisulfideGlutathioneFibroblastsGlutathioneIn vitroPeroxidesRatsCell biologyOxidative StresschemistryApoptosisFemaleReactive Oxygen SpeciesOxidative stressDNA DamageBiotechnologyThe FASEB Journal
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Radiation hard monolithic CMOS sensors with small electrodes for High Luminosity LHC

2019

Abstract The upgrade of the tracking detectors for the High Luminosity-LHC (HL-LHC) requires the development of novel radiation hard silicon sensors. The development of Depleted Monolithic Active Pixel Sensors targets the replacement of hybrid pixel detectors with radiation hard monolithic CMOS sensors. We designed, manufactured and tested radiation hard monolithic CMOS sensors in the TowerJazz 180 nm CMOS imaging technology with small electrodes pixel designs. These designs can achieve pixel pitches well below current hybrid pixel sensors (typically 50 ×  50 μ m ) for improved spatial resolution. Monolithic sensors in our design allow to reduce multiple scattering by thinning to a total si…

Nuclear and High Energy PhysicsParticle tracking detectors ; Radiation-hard detectors ; Electronic detector readout concepts ; CMOS sensors ; Monolithic active pixel sensorsPhysics::Instrumentation and DetectorscostsRadiationElectronic detector readout concepts01 natural sciences7. Clean energy030218 nuclear medicine & medical imaging03 medical and health sciences0302 clinical medicinesemiconductor detector: pixelElectronic detector readout conceptCMOS sensorselectrode: designParticle tracking detectors0103 physical sciences[PHYS.PHYS.PHYS-INS-DET]Physics [physics]/Physics [physics]/Instrumentation and Detectors [physics.ins-det]InstrumentationImage resolutionRadiation hardeningspatial resolutionradiation: damagePhysicsCMOS sensorsemiconductor detector: technologyMonolithic active pixel sensorPixelirradiation010308 nuclear & particles physicsbusiness.industrytracking detector: upgradeDetectorCMOS sensorParticle tracking detectorMonolithic active pixel sensorsUpgradeCERN LHC CollCMOSefficiencyOptoelectronicsbusinessperformanceRadiation-hard detectors
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Value of Home Blood Pressures as Predictor of Target Organ Damage in Mild Arterial Hypertension

2002

Home blood pressure measurement has gained increasing importance for the management of hypertensive patients. The aim of our study was to compare levels of clinic (CBP), ambulatory (ABP), and home blood pressure (HBP) measurements, and their relationships with various indexes of target organ damage in I-II grade essential hypertension.Thirty-eight essential hypertensives underwent evaluation of clinic, ambulatory and home blood pressures. Each patient recorded HBP for 2 days with a digital BP monitor three times daily, the first time on the same day during which ABP monitoring was simultaneously performed. Moreover, in all subjects electrocardiogram recording, echocardiographic study, micro…

AdultMalemedicine.medical_specialtyAmbulatory blood pressureEpidemiologyMultiple Organ FailureStatistics as TopicDiastoleAmbulatory Care FacilitiesSeverity of Illness IndexPrehypertensionPredictive Value of TestsInternal medicineAlbuminuriaHumansMedicinebusiness.industryBlood Pressure Monitoring AmbulatoryMiddle Agedmedicine.diseaseTarget organ damageSurgeryBlood pressureItalyHypertensionAmbulatoryCardiologyFemaleHypertrophy Left VentricularMicroalbuminuriaCardiology and Cardiovascular MedicinebusinessEuropean Journal of Cardiovascular Prevention & Rehabilitation
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