Search results for "DNA Damage"

showing 10 items of 534 documents

Oxidative stress and antioxidant response in fibroblasts from Werner and Atypical Werner Syndromes

2014

Werner Syndrome (WS, ICD-10 E34.8, ORPHA902) and Atypical Werner Syndrome (AWS, ICD-10 E34.8, ORPHA79474) are very rare inherited syndromes characterized by premature aging. While approximately 90% of WS individuals have any of a range of mutations in theWRN gene, there exists a clinical subgroup in which the mutation occurs in the LMNA/C gene in heterozygosity. Although both syndromes exhibit an age-related pleiotropic phenotype, AWS manifests the onset of the disease during childhood, while major symptoms in WS appear between the ages of 20 and 30. To study the molecular mechanisms of progeroid diseases provides a useful insight into the normal aging process. Main changes found were the d…

AdultMalePremature agingAgingWerner Syndrome HelicaseAdolescentBiologymedicine.disease_causeAntioxidantsCell LineWerner Syndrome HelicaseLMNAProgeriaSuperoxide Dismutase-1antioxidant enzymesmedicineoxidative stressHumansRNA MessengerAtypical Werner syndromeChildeducationCell ProliferationWerner syndromeeducation.field_of_studyProgeriaAtypical Werner SyndromeRecQ Helicasespremature agingSuperoxide DismutaseAging PrematurethioredoxinglutaredoxinCell BiologyFibroblastsLamin Type Amedicine.diseaseGlutathioneMolecular biologyExodeoxyribonucleasesCase-Control StudiesMutationDNA damageFemaleWerner SyndromeThioredoxinOxidative stressResearch PaperAging
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Cigarette smoke causes caspase-independent apoptosis of bronchial epithelial cells from asthmatic donors

2015

BackgroundEpidemiologic studies have demonstrated important links between air pollution and asthma. Amongst these pollutants, environmental cigarette smoke is a risk factor both for asthma pathogenesis and exacerbation. As the barrier to the inhaled environment, the bronchial epithelium is a key structure that is exposed to cigarette smoke.ObjectivesSince primary bronchial epithelial cells (PBECs) from asthmatic donors are more susceptible to oxidant-induced apoptosis, we hypothesized that they would be susceptible to cigarette smoke-induced cell death.MethodsPBECs from normal and asthmatic donors were exposed to cigarette smoke extract (CSE); cell survival and apoptosis were assessed by fl…

AdultMaleProgrammed cell deathDNA damageScienceCaspase 3ApoptosisBronchiBiologyTobacco smokeAntioxidantschemistry.chemical_compoundYoung Adultparasitic diseasesHumansAgricultural and Biological Sciences (all); Biochemistry Genetics and Molecular Biology (all); Medicine (all)MultidisciplinaryBiochemistry Genetics and Molecular Biology (all)Caspase-Independent ApoptosisCaspase 3Medicine (all)QSmokingREpithelial CellsGlutathioneMiddle AgedAscorbic acid3. Good healthchemistryAgricultural and Biological Sciences (all)13. Climate actionApoptosisImmunologyMedicineFemaleResearch Article
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Primary DNA damage in peripheral mononuclear blood cells of workers exposed to bitumen-based products.

1996

The genotoxic effect of occupational exposure to bitumen-based products was determined by the extent of DNA strand breaks and alkali-labile sites of the DNA of peripheral mononuclear blood cells from seven roofers, 18 road paving workers, and nine bitumen painters. In order to evaluate short-term genotoxic effect the workers were investigated on Fridays and on Mondays after a weekend free of occupational exposure. The roofers (all cigarette smokers) showed a significantly (P < 0.002) 43% higher mean level of alkaline DNA strand breaks on Friday than did the ten smoking controls included in this study. Also, comparison of the individual levels of alkaline strand breaks on Mondays and on Frid…

AdultMaleRoad constructionDNA damagebusiness.industryeducationSignificant differencePublic Health Environmental and Occupational HealthMutagenMiddle Agedmedicine.disease_causeHydrocarbonsAndrologyRisk FactorsGermanyOccupational ExposuremedicineLeukocytes MononuclearHumansOccupational exposurebusinesshuman activitiesDNA DamageInternational archives of occupational and environmental health
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Chronic occupational noise exposure: Effects on DNA damage, blood pressure, and serum biochemistry

2019

Abstract Noise levels experienced by industrial workers may cause both auditory and non-auditory harmful effects. We have studied the effects of chronic industrial noise exposure on DNA damage, blood pressure, and serum biochemistry in factory workers. Male workers (109 individuals) in three parts of a food factory in Shahroud, Iran were enrolled as the exposed group and male office workers (123 individuals) were the unexposed control group. Noise exposure was measured (dosimetry) and the comet assay was used to evaluate DNA damage in peripheral blood mononuclear cells. Glutathione peroxidase (GPx) and ceruloplasmin (Cp) levels were measured in serum samples. GPx levels, systolic and diasto…

AdultMaleSerum0301 basic medicineDNA damageHealth Toxicology and MutagenesisPhysiologyBlood PressureIran010501 environmental sciencesmedicine.disease_cause01 natural sciencesPeripheral blood mononuclear cellYoung Adult03 medical and health scienceschemistry.chemical_compoundOccupational ExposureGeneticsmedicineHumansIndustry0105 earth and related environmental scienceschemistry.chemical_classificationGlutathione Peroxidasebiologybusiness.industryGlutathione peroxidaseCeruloplasminGlutathioneMiddle AgedComet assay030104 developmental biologyBlood pressurechemistryLeukocytes MononuclearNoise Occupationalbiology.proteinComet AssayCeruloplasminbusinessOxidative stressDNA DamageMutation Research/Genetic Toxicology and Environmental Mutagenesis
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DNA single strand break analysis in mononuclear blood cells of petrol pump attendants

1995

DNA single strand breaks, including DNA adducts that lead to alkali-labile sites, were measured in peripheral mononuclear blood cells of 35 petrol pump attendants by alkaline filter elution. Blood samples from petrol pump attendants were taken on Monday and Friday. Additionally, DNA single strand breaks of smoking and non-smoking control persons were examined. For the smoking (n = 12) and the non-smoking controls (n = 20) a mean normalized elution rate of 1.49 +/- 0.52 (mean value +/- 95% confidence interval) and 1.32 +/- 0.28, respectively, was obtained. The difference between smoking and non-smoking controls was not statistically significant (U test). An increase in DNA single strand brea…

AdultMaleeducationAnimal scienceCigarette smokingRisk FactorsOccupational ExposureHumansMedicineDNA Single Strand BreakDNA single strandElution ratebusiness.industryOrganic solventSmokingMean valuePublic Health Environmental and Occupational HealthDNAMiddle AgedConfidence intervalPetroleumLeukocytes MononuclearOccupational exposurebusinesshuman activitiesDNA DamageEnvironmental MonitoringInternational Archives of Occupational and Environmental Health
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Glutathione levels in blood from ataxia telangiectasia patients suggest in vivo adaptive mechanisms to oxidative stress

2007

Objective: To evaluate an in vivo pro-oxidant state in patients with ataxia telangiectasia (AT). Methods: A set of oxidative stress endpoints were measured in 9 AT homozygotes, 16 AT heterozygotes (parents) and 83 controls (grouped in age ranges as for patients and parents, respectively). The following analytes were measured: (a) leukocyte 8-hydroxy-2-deoxyguanosine (8-OHdG); (b) blood glutathione (GSSG and GSH); and (c) plasma levels of glyoxal (Glx) and methylglyoxal (MGlx). Results: AT patients displayed a significant decrease in blood GSSG (p=0.012) and in MGlx plasma concentrations (P=0.012). A nonsignificant decrease in the GSSG:GSH ratio (p = 0.1) and a non-significant increase in 8-…

AdultMalemedicine.medical_specialtyAdolescentglyoxalClinical Biochemistryataxia telengiectasiamedicine.disease_causeAtaxia Telangiectasiachemistry.chemical_compoundIn vivoInternal medicinemethylglyoxalmedicineHumansataxia telangectasiaoxidative stressglutathioneChildoxidative streMethylglyoxalDeoxyguanosine8-Hydroxy-2'-deoxyguanosineHeterozygote advantageGeneral MedicineGlutathionePyruvaldehydemedicine.diseaseAdaptation Physiological8-Hydroxy-2'-deoxyguanosineEndocrinologychemistryBiochemistryChild PreschoolAtaxia-telangiectasiaFemaleataxia telangectasia; oxidative stress; glutathione; glyoxal; methylglyoxal; 8-Hydroxy-2'-deoxyguanosineOxidative stressTarget organDNA DamageClinical Biochemistry
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Factors related to the impact of antihypertensive treatment in antioxidant activities and oxidative stress by-products in human hypertension

2004

The objective was to study factors related to the changes induced by antihypertensive treatment on oxidative status, antioxidant activities, and reactive oxygen species by-products in whole blood and mononuclear peripheral cells. Eighty-nine hypertensive patients (mean age 46 years, 46 men, average 24-h blood pressure 139/88 mm Hg, body mass index 29) were included. After 3 months of nonrandomized allocation to antihypertensive treatment (20 nonpharmacologic, 36 beta-blockers, 33 angiotensin receptor blocker), oxidized/reduced glutathione ratio and malondialdehyde were significantly reduced, and the activity of superoxide dismutase, catalase, and glutathione peroxidase was significantly inc…

AdultMalemedicine.medical_specialtyAntioxidantmedicine.medical_treatmentAngiotensin-Converting Enzyme Inhibitorsmedicine.disease_causeBenzoatesAntioxidantsSuperoxide dismutasechemistry.chemical_compoundInternal medicineInternal MedicinemedicineHumansTelmisartanAntihypertensive AgentsWhole bloodchemistry.chemical_classificationReactive oxygen speciesbiologybusiness.industryGlutathione peroxidaseGlutathioneMiddle AgedMalondialdehydeOxidative StressHydrochlorothiazideTreatment OutcomeEndocrinologyAtenololchemistryHypertensionbiology.proteinBenzimidazolesDrug Therapy CombinationFemalebusinessOxidative stressDNA DamageAmerican Journal of Hypertension
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AZT treatment induces molecular and ultrastructural oxidative damage to muscle mitochondria. Prevention by antioxidant vitamins.

1998

AIDS patients who receive zidovudine (AZT) frequently suffer from myopathy. This has been attributed to mitochondrial (mt) damage, and specifically to the loss of mtDNA. This study examines whether AZT causes oxidative damage to DNA in patients and to skeletal muscle mitochondria in mice, and whether this damage may be prevented by supranutritional doses of antioxidant vitamins. Asymptomatic HIV-infected patients treated with AZT have a higher urinary excretion (355+/-100 pmol/kg/d) of 8-oxo-7, 8-dihydro-2'-deoxyguanosine (8-oxo-dG) (a marker of oxidative damage to DNA) than untreated controls (asymptomatic HIV-infected patients) (182+/-29 pmol/kg/d). This was prevented (110+/-79 pmol/kg/d)…

AdultMalemedicine.medical_specialtyDNA damageAnti-HIV Agentsmedicine.medical_treatmentAscorbic AcidBiologyDNA MitochondrialAntioxidantsZidovudinechemistry.chemical_compoundMiceInternal medicinemedicineDeoxyguanosineAnimalsHumansVitamin Eheterocyclic compoundsMyopathyVitamin ESkeletal musclevirus diseasesDeoxyguanosineGeneral MedicineGlutathioneHydrogen PeroxideAscorbic acidMitochondria Musclemedicine.anatomical_structureEndocrinologychemistryBiochemistry8-Hydroxy-2'-Deoxyguanosinemedicine.symptomZidovudinemedicine.drugDNA DamageResearch ArticleThe Journal of clinical investigation
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DNA damage in leukocytes and serum nitrite concentration are negatively associated in type 1 diabetes.

2020

Abstract Chronic hyperglycaemia leads to DNA damage in diabetes and might be associated with nitrosative stress. In this study, we aimed at assessing the level of DNA strand breaks in leukocytes, serum nitrite and nitrate in patients with type 1 diabetes and healthy controls and associations of these parameters with diabetes-related outcomes in a prospective study. The level of DNA damage was determined in 71 patients with type 1 diabetes and 57 healthy controls by comet assay and scored with arbitrary units (AU). The chemiluminescence method was used to measure nitrite and nitrate. Clinical information and data on consumption of alcohol, physical activity and smoking were collected. Progre…

AdultMalemedicine.medical_specialtyDNA damageHealth Toxicology and MutagenesisArbitrary unit030204 cardiovascular system & hematologyToxicologyDiabetes Complications03 medical and health scienceschemistry.chemical_compound0302 clinical medicineInternal medicineDiabetes mellitusGeneticsmedicineLeukocytesHumansProspective StudiesNitriteProspective cohort studyGenetics (clinical)Nitrites030304 developmental biology0303 health sciencesType 1 diabetesbusiness.industrymedicine.diseaseComet assayEndocrinologyDiabetes Mellitus Type 1chemistryRelative riskFemaleComet AssaybusinessDNA DamageMutagenesis
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Urinary 8-oxo-7,8-dihydro-2′-deoxyguanosine (8-oxo-dG), a reliable oxidative stress marker in hypertension

2007

The potential use of oxidative stress products as disease markers and progression is an important aspect of biomedical research. In the present study, the quantification of urine 8-oxo-7,8-dihydro-2'-deoxyguanosine (8-oxo-dG) concentration has been used to express the oxidation status of hypertensive subjects. 8-oxo-dG has been simultaneously isolated and assayed in nuclear (nDNA) and mitochondrial DNA (mtDNA). In addition, oxidative stress of mononuclear cells has been estimated by means of GSH and GSSG levels and GSSG/GSH ratio in hypertensive subjects before and after antihypertensive treatment. It is shown that oxidative stress decreases significantly in hypertensive patients after trea…

AdultMalemedicine.medical_specialtyDNA damageUrinary systemUrinemedicine.disease_causeDNA MitochondrialBiochemistryPeripheral blood mononuclear cellchemistry.chemical_compoundInternal medicinemedicineHumansDeoxyguanosineChromatography High Pressure LiquidCell NucleusGlutathione DisulfideDeoxyguanosineGeneral MedicineGlutathioneGlutathioneOxidative StressEndocrinologychemistryBiochemistry8-Hydroxy-2'-DeoxyguanosineHypertensionGlutathione disulfideFemaleBiomarkersOxidative stressFree Radical Research
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