Search results for "DNA Damage"

showing 10 items of 534 documents

Fibroblasts from bank voles inhabiting Chernobyl have increased resistance against oxidative and DNA stresses

2018

Background Elevated levels of environmental ionizing radiation can be a selective pressure for wildlife by producing reactive oxygen species and DNA damage. However, the underlying molecular mechanisms that are affected are not known. Results We isolated skin fibroblasts from bank voles (Myodes glareolus) inhabiting the Chernobyl nuclear power plant accident site where background radiation levels are about 100 times greater than in uncontaminated areas. After a 10 Gy dose of gamma radiation fibroblasts from Chernobyl animals recovered faster than fibroblasts isolated from bank voles living in uncontaminated control area. The Chernobyl fibroblasts were able to sustain significantly higher do…

Male0301 basic medicinep53Antioxidantmedicine.medical_treatment010501 environmental sciences01 natural sciencesAntioxidantsIonizing radiationsäteilybiologiachemistry.chemical_classificationCell DeathbiologyArvicolinaelcsh:CytologyBank volefibroblastitOxidantshumanitiesCell biologyAntioxidant capacityBank voleResearch ArticleG2 PhaseCell SurvivalDNA damagemetsämyyräOxidative phosphorylationCell LineMicrobiologyChernobyl03 medical and health sciencesmedicineAnimalsEnvironmental ionizing radiationlcsh:QH573-671Geneoksidatiivinen stressi0105 earth and related environmental sciencesantioksidantitReactive oxygen speciesCell Cycle CheckpointsDNACell BiologyFibroblastsbiology.organism_classificationresistenssiOxidative Stress030104 developmental biologyChernobyl Nuclear AccidentchemistryGamma RaysApoptosisbank volesDNA damageTumor Suppressor Protein p53BMC Cell Biology
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Is the Mean Blood Leukocyte Telomere Length a Predictor for Sporadic Thoracic Aortic Aneurysm? Data from a Preliminary Study

2012

Telomeres have been postulated as a universal clock that shortens in parallel with cellular aging. They are specialized DNA-protein structures at the ends of chromosome with remarkable functions--preventing their recognition as double-stranded DNA breaks, protecting their recombination and degradation, and avoiding a DNA damage cellular response. Telomere shortening is currently considered the best aging marker, but is also a predictor for age-related diseases, including cardiovascular diseases. Biological age clearly seems to be a better predictor of vascular risk rather than chronological age. This concept is supported by key assumptions that peripheral blood leukocyte telomere content ac…

MaleAgingPathologymedicine.medical_specialtyThoracicBiological ageVascular riskBiologyBioinformaticsThoracic aortic aneurysmGeneticLeukocytesmedicineHumansSettore MED/05 - Patologia ClinicaAged; Aging; Aortic Aneurysm Thoracic; Case-Control Studies; Cellular Senescence; DNA; DNA Damage; Female; Humans; Leukocytes; Male; Middle Aged; Recombination Genetic; Telomere; Vascular DiseasesVascular DiseasesCellular Senescencevascular ageingAgedRecombination GenetictelomereAortic Aneurysm ThoracicVascular diseaseChromosomeSettore MED/23 - Chirurgia CardiacaDNAMiddle Agedmedicine.diseaseRecombinationPeripheral bloodAortic AneurysmTAATelomereCellular AgingCase-Control StudiesFemaleGeriatrics and GerontologyDNA DamageRejuvenation Research
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Effect of nut consumption on oxidative stress and the endothelial function in metabolic syndrome

2010

Effect of nut consumption on oxidative stress and the endothelial function in metabolic syndrome BACKGROUND & AIMS: Oxidative stress has a key role in atherosclerosis, cancer and other chronic diseases. Some bioactive compounds in nuts have been implicated in antioxidant activities. OBJECTIVE: We assessed how nut consumption affected several markers of oxidation and endothelial function (EF) in metabolic syndrome (MetS) patients. PATIENTS AND METHODS: A randomized, controlled, parallel feeding trial was conducted on 50 MetS adults who were recommended a healthy diet supplemented or not with 30 g of mixed nuts (Nut and Control groups, respectively) every day for 12 weeks. The plasma anti…

MaleAntioxidantmedicine.medical_treatmentantioxidant capacityIsoprostanesCritical Care and Intensive Care Medicinemedicine.disease_causeAntioxidantsLipid peroxidationchemistry.chemical_compoundendothelial functionMedicineNutschemistry.chemical_classificationMetabolic SyndromeBioquímica y tecnologíaNutrition and Dieteticsdigestive oral and skin physiologyfood and beveragesArteriesMiddle Aged0261-5614Lipoproteins LDLBiochemistry and technologymedicine.anatomical_structure8-Hydroxy-2'-DeoxyguanosineFatty Acids UnsaturatedPolyunsaturated fatty acidAdultmedicine.medical_specialtyEndotheliumAdolescentDNA damageEstrès oxidatiuFruita seca -- Aspectes nutritiusBioquímica i biotecnologiaYoung AdultLipid oxidationInternal medicineHumansAgedbusiness.industryDeoxyguanosineEndoteli vascularmedicine.diseaseDietOxidative StressEndocrinologychemistryDNA damageVascular ResistanceEndothelium VascularMetabolic syndromebusinessOxidative stressBiomarkers
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Deregulation of the G1 to S-phase cell cycle checkpoint is involved in the pathogenesis of human osteosarcoma.

2004

Osteosarcoma (OS) displays complex karyotypes with numerical changes as well as structural abnormalities suggesting that several oncogenes and tumor suppressor genes may be implicated in the biology of OS. The aim of our study was to investigate the possible implication of the molecular alterations of the G1 to S-phase checkpoint genes in the pathogenesis of OS. We analyzed samples from 29 patients and found molecular alterations of the RB and TP53 genes in 6 (21%) and 3 (10%) cases, respectively. Homozygous deletion of the INK4A/ARF locus and methylation of INK4A was detected in 3 (10%) and 2 (7%) cases, respectively. CDK4 and MDM2 co-amplification was observed in 1 case (3%). Cyclin D3 is…

MaleCell cycle checkpointAdolescentLocus (genetics)Bone NeoplasmsBiologyPathology and Forensic MedicineS PhasePathogenesisGene duplicationmedicineHumansCHEK1Cyclin D3ChildMolecular BiologyAgedOsteosarcomaReverse Transcriptase Polymerase Chain ReactionCell CycleAge FactorsG1 PhaseGene AmplificationCell BiologyG2-M DNA damage checkpointMiddle Agedmedicine.diseaseGenes cdcHistory 16th CenturyCancer researchOsteosarcomaFemaleChromosomes Human Pair 9Diagnostic molecular pathology : the American journal of surgical pathology, part B
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Assessment of the mutagenic potency of sewage sludges contaminated with polycyclic aromatic hydrocarbons by an Ames sludges for fluctuation assay

2003

9 pages, 1 figure, 6 tables.-- PMID: 14587895 [PubMed].

MaleCost-Benefit AnalysisHealth Toxicology and MutagenesisSewageFractionationGene mutationSensitivity and SpecificityWaste Disposal FluidAmes testReference ValuesMutagenicityAnimalsEnvironmental ChemistryCitiesRats WistarSewage sludgeChromatographySewageMutagenicity Testsbusiness.industryChemistryReproducibility of ResultsAmes fluctuation assayPolycyclic aromatic hydrocarbonsRatsEnvironmental chemistryGas chromatography-mass spectrometrySewage treatmentGas chromatography–mass spectrometrybusinessSludgeDNA DamageWaste disposal
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DNA repair activity of 8-oxoguanine DNA glycosylase 1 (OGG1) in human lymphocytes is not dependent on genetic polymorphism Ser326/Cys326.

2001

8-oxoguanine DNA glycosylase 1 (OGG1) is a DNA repair enzyme that excises 7,8-dihydro-8-oxoguanine (8oxoG) from DNA. Since 8oxoG is a highly mispairing lesion, decreased OGG1 expression level could lead to a higher background mutation frequency and could possibly increase the cancer risk of an individual under oxidative stress. In order to analyse the natural variation of OGG1, we measured the DNA repair activity in human lymphocytes of healthy individuals by means of an 8oxoG-containing oligonucleotide assay. The data obtained revealed a two fold interindividual variation of OGG1 activity in lymphocytes. There was no difference in OGG1 activity due to gender and smoking behaviour. Transcri…

MaleDNA RepairDNA damageDNA repairBiologyIn Vitro TechniquesToxicologyDNA-formamidopyrimidine glycosylasechemistry.chemical_compoundGene FrequencyMUTYHGeneticsHumansAmino Acid SequenceLymphocytesMolecular BiologyGeneN-Glycosyl HydrolasesAllelesDNA PrimersPolymorphism GeneticBase SequenceOligonucleotideReverse Transcriptase Polymerase Chain ReactionMolecular biologyIsoenzymeschemistryDNA-Formamidopyrimidine GlycosylaseDNA glycosylaseDNADNA DamageMutation research
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Expression of somatic DNA repair genes in human testes

2006

Meiosis is the key process for recombination and reduction of the diploid chromosome set to a haploid one. Many genes that have been found in yeast or mouse models to play a role in meiosis are also important for the repair of DNA damage in somatic cells. To study the DNA repair gene transcriptome during male germ cell development, we have developed a specialized cDNA microarray with 181 human genes which are involved in different somatic DNA repair pathways and/or cell cycle control and 45 control house-keeping genes. This DNA repair gene chip was used to quantify the mRNA expression levels in three human testes samples versus a fibroblast RNA pool. Two hundred twenty genes on the chip (in…

MaleDNA RepairDNA damageSomatic cellDNA repairBiologyBiochemistryTranscriptomeTestismedicineHumansMolecular BiologyGeneCells CulturedOligonucleotide Array Sequence AnalysisSkinReverse Transcriptase Polymerase Chain ReactionGene Expression ProfilingCell BiologyFibroblastsDNA repair protein XRCC4Molecular biologyMeiosismedicine.anatomical_structureGene Expression RegulationHuman genomeBiomarkersGerm cellJournal of Cellular Biochemistry
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Coffee and its chemopreventive components Kahweol and Cafestol increase the activity of O6-methylguanine-DNA methyltransferase in rat liver—compariso…

2003

Abstract A lower rate of colon cancer was observed in consumers of coffee with a high content of the diterpenes Kahweol and Cafestol (K/C). In animal models, K/C have been found to protect against the mutagenic/carcinogenic effects of compounds such as 2-amino-1-methyl-6-phenylimidazo[4,5-b]pyridine (PhIP), aflatoxin B1, and 7,12-dimethylbenz[a]anthracene. Thus far, such chemoprotection by K/C has been attributed to modifications of xenobiotic metabolism, e.g. enhanced detoxification by UDP-glucuronosyltransferase (UDPGT) and/or glutathione transferase (GST). In the present study, we investigated the potential of several coffee-related treatments (K/C [1:1], Cafestol-alone, Turkish coffee) …

MaleDNA damageHealth Toxicology and MutagenesisCafestolBiologyCoffeeXenobioticschemistry.chemical_compoundGeneticsmedicineAnimalsDNA Modification MethylasesMolecular BiologyAnticarcinogenCarcinogenKahweolGlutathioneMolecular biologyRatsLiverchemistryBiochemistryDiterpenesAntimutagenDrug metabolismmedicine.drugMutation Research/Fundamental and Molecular Mechanisms of Mutagenesis
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Increased Oxidative Damage Associated with Unfavorable Cytogenetic Subgroups in Chronic Lymphocytic Leukemia

2014

Oxidative stress contributes to genomic instability in chronic lymphocytic leukemia (CLL), but its relationship with the acquisition of specific chromosomal abnormalities is unknown. We recruited 55 untreated CLL patients and assessed 8-oxo-2′-deoxyguanosine (8-oxo-dG), glutathione, and malondialdehyde (MDA) levels, and we compared them among the cytogenetic subgroups established using fluorescence in situ hybridization (FISH). Significant increases in 8-oxo-dG and/or MDA were observed in patients with unfavorable cytogenetic aberrations (17p and 11q deletions) compared to the 13q deletion group.TP53deletion patients exhibited a diminished DNA repair efficiency. Finally, cases with normal F…

MaleGenome instabilityArticle SubjectDNA RepairDNA damageDNA repairChronic lymphocytic leukemialcsh:MedicineBiologymedicine.disease_causeGeneral Biochemistry Genetics and Molecular BiologyCohort Studieschemistry.chemical_compoundMalondialdehydemedicineHumansLymphocytesIn Situ Hybridization FluorescenceAgedAged 80 and overChromosome AberrationsGeneral Immunology and Microbiologymedicine.diagnostic_testlcsh:RDeoxyguanosineGeneral MedicineGlutathioneMiddle AgedMalondialdehydemedicine.diseaseGlutathioneLeukemia Lymphocytic Chronic B-CellOxidative Stresschemistry8-Hydroxy-2'-DeoxyguanosineImmunologyFemaleLipid PeroxidationReactive Oxygen SpeciesGene DeletionOxidative stressDNA DamageResearch ArticleFluorescence in situ hybridizationBioMed Research International
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Genotoxic effects of the herbicides alachlor, atrazine, pendimethaline, and simazine in mammalian cells

1994

MaleHealth Toxicology and MutagenesisSimazineSimazineToxicologyRats Sprague-Dawleychemistry.chemical_compoundAcetamidesAnimalsHumansEcotoxicologyLymphocytesAtrazineCells CulturedAniline CompoundsHerbicidesMutagenicity TestsChemistrybusiness.industryAlachlorGeneral MedicinePesticidePollutionRatsBiotechnologyLiverAtrazinebusinessSister Chromatid ExchangeDNA DamageBulletin of Environmental Contamination and Toxicology
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