Search results for "DNA replication"

showing 10 items of 107 documents

Resveratrol, a chemopreventive agent, disrupts the cell cycle control of human SW480 colorectal tumor cells

2002

Resveratrol is a natural polyphenolic compound produced by a number of plants and found in high amount in peanuts, seeds, grapes or berries as source of human nutrition. Epidemiological studies strongly suggest that resveratrol may act as a cancer chemopreventive compound. The mechanism by which resveratrol inhibits cell proliferation was studied in human colorectal tumor SW480 cell line. The results show that resveratrol strongly inhibits cell proliferation at the micromolar range in a time- and dose-dependent manner. Resveratrol appears to block the cell cycle at the transition --> G2/M since inhibition of [(3)H]-thymidine incorporation is not observed, while there is an increase of the c…

DNA Replicationendocrine system diseasesCellCyclin AAdenocarcinomaCyclin BProtein Serine-Threonine KinasesResveratrolS Phasechemistry.chemical_compoundCDC2 Protein KinaseStilbenesCDC2-CDC28 KinasesTumor Cells CulturedGeneticsmedicineAnticarcinogenic AgentsHumansCyclin B1Phosphorylationskin and connective tissue diseasesCyclinCyclin-dependent kinase 1biologyKinaseCell growthorganic chemicalsCell CycleCyclin-Dependent Kinase 2Cyclin-dependent kinase 2food and beveragesGeneral MedicineCell cycleFlow CytometryCyclin-Dependent KinasesGrowth InhibitorsNeoplasm ProteinsGene Expression Regulation Neoplasticmedicine.anatomical_structureBiochemistrychemistryResveratrolEnzyme Inductionbiology.proteinCancer researchColorectal NeoplasmsProtein Processing Post-TranslationalCell DivisionInternational Journal of Molecular Medicine
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Class 1 Histone Deacetylases and Ataxia-Telangiectasia Mutated Kinase Control the Survival of Murine Pancreatic Cancer Cells upon dNTP Depletion

2021

Pancreatic ductal adenocarcinoma (PDAC) is a highly aggressive disease with a dismal prognosis. Here, we show how an inhibition of de novo dNTP synthesis by the ribonucleotide reductase (RNR) inhibitor hydroxyurea and an inhibition of epigenetic modifiers of the histone deacetylase (HDAC) family affect short-term cultured primary murine PDAC cells. We used clinically relevant doses of hydroxyurea and the class 1 HDAC inhibitor entinostat. We analyzed the cells by flow cytometry and immunoblot. Regarding the induction of apoptosis and DNA replication stress, hydroxyurea and the novel RNR inhibitor COH29 are superior to the topoisomerase-1 inhibitor irinotecan which is used to treat PDAC. Ent…

DNA Replicationendocrine system diseasesDNA damagereplication stressQH301-705.5RNR Inhibitor COH29Antineoplastic AgentsCell Cycle ProteinsRNRAtaxia Telangiectasia Mutated ProteinsArticle03 medical and health scienceschemistry.chemical_compoundAtaxia TelangiectasiaMice0302 clinical medicineHDACAnimalscancerPDAC cellsRibonucleotide Reductase SubunitEnzyme InhibitorsBiology (General)030304 developmental biology0303 health sciencesbiologyChemistryEntinostatDNA replicationapoptosisGeneral Medicine3. Good healthPancreatic NeoplasmsHistoneRibonucleotide reductase030220 oncology & carcinogenesisATMbiology.proteinCancer researchDNA damageHistone deacetylaseCells
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Identification of Gip as a novel phage‐encoded gyrase inhibitor protein of Corynebacterium glutamicum

2021

By targeting key regulatory hubs of their host, bacteriophages represent a powerful source for the identification of novel antimicrobial proteins. Here, a screening of small cytoplasmic proteins encoded by the CGP3 prophage of Corynebacterium glutamicum resulted in the identification of the gyrase-inhibiting protein Cg1978, termed Gip. Pull-down assays and surface plasmon resonance revealed a direct interaction of Gip with the gyrase subunit A (GyrA). The inhibitory activity of Gip was shown to be specific to the DNA gyrase of its bacterial host C. glutamicum. Overproduction of Gip in C. glutamicum resulted in a severe growth defect as well as an induction of the SOS response. Furthermore, …

DNA Replicationendocrine systemProtein subunitProphagesBiologyMicrobiologyDNA gyraseCorynebacterium glutamicum03 medical and health scienceschemistry.chemical_compoundViral Proteinsddc:570Topoisomerase II InhibitorsSOS responseMolecular BiologyProphage030304 developmental biology0303 health sciences030306 microbiologyDNA replicationAnti-Bacterial AgentsHigh-Throughput Screening AssaysCorynebacterium glutamicumchemistryBiochemistrybacteriaTopoisomerase-II InhibitorDNAhormones hormone substitutes and hormone antagonistsMolecular Microbiology
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The effects of glucocorticoids on thymidine kinase and nucleoside phosphotransferase during development of chicken embryo retina.

1983

AbstractThymidine kinase in chick embryo retina reaches its highest values on the 8–10th day of development, then declines reaching the lowest value at hatching. The rate of DNA synthesis essentially follows this activity while, in contrast, nucleoside phosphotransferase increases progressively during development. Glucocorticoids at 5 × 10−6M lower the level of thymidine kinase in isolated retinas of chick embryo. The most effective steroid was hydrocortisone. The effect was observed in retinas from 8–18-day-old chick embryo and, except on the 18th day, was always of the same magnitude. We suggest that a glucocorticoid can be the natural factor responsible for the marked fall in thymidine k…

DNA Replicationmedicine.medical_specialtyanimal structuresNucleoside phosphotransferase activityHydrocortisonePrednisoloneBiophysicsChick EmbryoBiologyDevelopmentBiochemistryThymidine KinaseRetinachemistry.chemical_compoundGlucocorticoidThe effects of glucocorticoidsStructural BiologyCorticosteroneSettore BIO/10 - BiochimicaInternal medicineNucleoside phosphotransferaseGeneticsmedicineAnimalsMolecular BiologyGlucocorticoidsDNA synthesisEmbryogenesisPhosphotransferasesEmbryoCell BiologyCortisoneKineticsEndocrinologyNucleoside phosphotransferasechemistryThymidine kinaseembryonic structuresPrednisoneCorticosteroneGlucocorticoidmedicine.drugFEBS letters
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Mutations in CTC1, encoding conserved telomere maintenance component 1, cause Coats plus

2012

Coats plus is a highly pleiotropic disorder particularly affecting the eye, brain, bone and gastrointestinal tract. Here, we show that Coats plus results from mutations in CTC1, encoding conserved telomere maintenance component 1, a member of the mammalian homolog of the yeast heterotrimeric CST telomeric capping complex. Consistent with the observation of shortened telomeres in an Arabidopsis CTC1 mutant and the phenotypic overlap of Coats plus with the telomeric maintenance disorders comprising dyskeratosis congenita, we observed shortened telomeres in three individuals with Coats plus and an increase in spontaneous γ 3H2AX-positive cells in cell lines derived from two affected individual…

DNA polymeraseMolecular Sequence DataTelomere-Binding ProteinsHistones/metabolismHDE GENHDE NEU PEDCST complexCEREBRORETINAL MICROANGIOPATHY FAMILIAL SYNDROME CALCIFICATIONS CYSTS PROTEIN DNA LEUKOENCEPHALOPATHY EVOLUTION DEFECTSHistoneschemistry.chemical_compoundAbnormalities Multiple/geneticsGeneticsmedicineAbnormalities MultipleGenetic Predisposition to DiseaseGeneticsTelomere-binding proteinTelomere/pathologyddc:618biologyBase SequenceGenetic Predisposition to Disease/geneticsDNA replicationSequence Analysis DNATelomeremedicine.diseaseFlow CytometryTelomereCell biologyRetinal Telangiectasis/genetics/pathologychemistrySequence Analysis DNA/methodsbiology.proteinRetinal TelangiectasisPrimaseTelomere-Binding Proteins/geneticsDNADyskeratosis congenitaNature Genetics
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DNA damage-induced cell death: From specific DNA lesions to the DNA damage response and apoptosis

2011

DNA damaging agents are potent inducers of cell death triggered by apoptosis. Since these agents induce a plethora of different DNA lesions, it is firstly important to identify the specific lesions responsible for initiating apoptosis before the apoptotic executing pathways can be elucidated. Here, we describe specific DNA lesions that have been identified as apoptosis triggers, their repair and the signaling provoked by them. We discuss methylating agents such as temozolomide, ionizing radiation and cisplatin, all of them are important in cancer therapy. We show that the potentially lethal events for the cell are O(6)-methylguanine adducts that are converted by mismatch repair into DNA dou…

DNA re-replicationCancer ResearchGuanineDNA RepairDNA repairDNA damageSurvivinAntineoplastic AgentsApoptosisBiologyInhibitor of Apoptosis ProteinsDNA AdductsNeoplasmsRadiation IonizingmedicineAnimalsHumansPhosphorylationCisplatinCell DeathCell CycleNF-kappa BDNA replicationDNAG2-M DNA damage checkpointCell cycleOncologyCancer researchDNA mismatch repairProto-Oncogene Proteins c-aktDNA DamageSignal Transductionmedicine.drugCancer Letters
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Transglutaminase and polyamine dependence of effector functions of human immunocompetent cells

1982

AbstractThe effects of the transglutaminase inhibitor dansyl cadaverine (DC) and the polyamine antagonist methyl glyoxal-bis-(guanylhydrazone) (MeGbG) on the response of lymphocytes towards allogeneic and lectin stimulation and on the zymosan-induced chemiluminescence of neutrophilic granulocytes was studied. Application of DC resulted in dose-dependent suppression of chemiluminiscence and lymphocyte proliferation; no difference of inhibitory potential occurred with variation of incubation time in the latter system. MeGbG was inactive in granulocytes, but inhibited lymphocyte proliferation; its effect increased with time. The experiments provide further evidence for the importance of transg…

Dansyl cadaverineDNA ReplicationPolyamineLuminescenceMitoguazoneTissue transglutaminaseLymphocyteBiophysicsStimulationLymphocyte proliferationDiaminesGranulocyteBiologyLymphocyte ActivationGuanidinesBiochemistrylaw.inventionMeGbGchemistry.chemical_compoundStructural BiologylawCadaverinePolyaminesGeneticsmedicineHumansLymphocytesMolecular BiologyChemiluminescenceCadaverineTransglutaminasesGranulocyteCell BiologyTransglutaminaseMolecular biologyKineticsmedicine.anatomical_structurechemistryBiochemistrybiology.proteinLymphocytePolyamineAcyltransferasesGranulocytesFEBS Letters
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Numerical analysis of density gradient centrifugation profiles from eukaryotic DNA

1990

A numerical method for the deconvolution of superimposed Gaussian distributions with a unique solution has been proposed by Medgyessy [10]. We have tested the usefulness of this method for the analysis of density gradient centrifugation profiles from eukaryotic DNA, which are normally composed from overlapping Gaussian distributed profiles of several subcomponents with different mean buoyant densities. From the analysis of human DNA and from model calculations we conclude that major subcomponents can be identified by this method, if they differ in their buoyant density by approximatly 0.005 g/ml. Minor components can only be identified if the total DNA has been fractionated according to buo…

Differential centrifugationPolymers and PlasticsChemistryGaussianNumerical analysisAnalytical chemistryBuoyant densityEukaryotic DNA replicationsymbols.namesakechemistry.chemical_compoundColloid and Surface ChemistryMaterials ChemistrysymbolsUltracentrifugeDeconvolutionPhysical and Theoretical ChemistryBiological systemDNAColloid & Polymer Science
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Pathway network inference from gene expression data

2014

[EN] Background: The development of high-throughput omics technologies enabled genome-wide measurements of the activity of cellular elements and provides the analytical resources for the progress of the Systems Biology discipline. Analysis and interpretation of gene expression data has evolved from the gene to the pathway and interaction level, i.e. from the detection of differentially expressed genes, to the establishment of gene interaction networks and the identification of enriched functional categories. Still, the understanding of biological systems requires a further level of analysis that addresses the characterization of the interaction between functional modules. Results: We presen…

ESTADISTICA E INVESTIGACION OPERATIVAGene regulatory networkGene ExpressionInferenceSister chromatidsOxidative Phosphorylation//purl.org/becyt/ford/1 [https]Structural BiologyEstadística e Investigación OperativaGene Regulatory NetworksTopology (chemistry)Alzheimers-DiseaseGeneticsDIBUJOBiological systemsApplied MathematicsSystems BiologyCell Cycle//purl.org/becyt/ford/1.2 [https]Computer Science ApplicationsMicroarray experimentsModeling and SimulationIdentification (biology)Functional assessmentDNA-replicationFunctional connectionsGlycolysisCIENCIAS NATURALES Y EXACTASPathway NetworkDNA ReplicationSaccharomyces-CervisiaeBioinformaticsS-phaseSystems biologyGenomicsComputational biologySaccharomyces cerevisiaeBiologyGene interactionAlzheimer DiseaseModelling and SimulationGenomic dataPANAPathwaysMolecular BiologyUbiquitinResearchGene Expression ProfilingR packageGluconeogenesisGene expression profilingComputingMethodologies_PATTERNRECOGNITIONPurinesCiencias de la Computación e InformaciónProteolysisGene expression dataCiencias de la Información y BioinformáticaUbiquitin conjugationPathwayBMC Systems Biology
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Comparison of the genotoxic and apoptosis-inducing properties of ganciclovir and penciclovir in Chinese hamster ovary cells transfected with the thym…

2000

We studied the genotoxic and apoptosis-inducing properties of ganciclovir (GCV) and penciclovir (PCV) using Chinese hamster ovary cells stably transfected with the thymidine kinase (tk) gene of herpes simplex virus-1 (HSV-1). Cells expressing HSVtk were 300 and 100 times more sensitive than their isogenic HSVtk- counterparts to the cytotoxic effects of GCV and PCV, respectively. Using radiolabeled drugs, GCV was found to be incorporated into the genomic DNA much more effectively than PCV. GCV was highly potent in inducing chromosomal aberrations compared with PCV, which provoked less sister chromatid exchanges and chromosomal changes using equimolar or equitoxic doses. For both agents, apop…

GanciclovirDNA ReplicationCancer ResearchGuaninevirusesAcyclovirApoptosisCHO CellsHerpesvirus 1 HumanBiologymedicine.disease_causeTransfectioncomplex mixturesThymidine KinaseNecrosisCricetinaemedicineAnimalsMolecular BiologyGanciclovirMutagenicity TestsChinese hamster ovary cellCell CycleDNAGenetic TherapySuicide geneCell cycleMolecular biologyCell killingThymidine kinasePenciclovirMolecular MedicineSister Chromatid ExchangeGenotoxicitymedicine.drugCancer gene therapy
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