Search results for "DNA"

showing 10 items of 6803 documents

Distribution of type I and type II collagen gene expression during the development of human long bones

1990

The temporal and spatial gene expression of collagen type I and type II during the development of the human long bones was studied by the technique of in situ hybridization covering the period from the cartilagenous bone anlage to the formation of a regular growth plate in the newborn. Analysis of the early stages around the seventh week of gestation revealed for type II collagen a strong hybridization signal limited to the chondrogenic tissue. The surrounding connective tissue and the perichondrium showed weak type I collagen expression, while the zones of desmal ossification like the clavicle gave a strong signal. Beginning with the eighth week of gestation, type I collagen mRNA was detec…

medicine.medical_specialtyPathologyHistologyPhysiologyEndocrinology Diabetes and MetabolismType II collagenGene ExpressionConnective tissueBiologyBone and BonesInternal medicinemedicineHumansPerichondriumBone DevelopmentOssificationCartilageNucleic Acid HybridizationDNAHypertrophymedicine.diseaseCollagen type I alpha 1Cartilagemedicine.anatomical_structureEndocrinologyCollagenmedicine.symptomDNA ProbesType I collagenCalcificationBone
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Study of Cellular DIMA Content by Flow Cytometry in Primary Bladder Carcinomas

1992

A prospective study of cellular DNA content by flow cytometry was performed on a nonconsecutive series of 67 patients undergoing diagnostic and/or therapeutic transurethral resection for primary urothelial bladder carcinoma. DNA-aneuploidy was present in 82% of the cases (55/67), while multiclonality was found in 45% of the DNA-aneuploid cases (25/55). DNA-ploidy was much more strictly correlated with histological grading (p less than 0.005) than with papillary or non-papillary growth pattern (p less than 0.05) or T staging (p less than 0.05). Of 26 patients with a minimum follow-up of 24 months, 100% (6/6) of cases with DNA-diploid neoplasias showed no signs of disease relapse, versus 10% …

medicine.medical_specialtyPathologyUrinary bladdermedicine.diagnostic_testbusiness.industryUrologyCytogeneticsmedicine.diseaseFlow cytometrymedicine.anatomical_structureCellular dnaMonoclonalmedicineCarcinomaProspective cohort studybusinessGrading (tumors)European Urology
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Is there a role for recombinant tumor necrosis factor alpha in the intravesical treatment of superficial bladder tumors?--a phase II study

1995

Clinical use of recombinant tumor necrosis factor-alpha is strongly limited by its severe toxicity, mainly cardiovascular, when systemically administered. Recent studies suggest that topical (intrapleural, intraperitoneal, intratumoral) administration is free of significant toxicity. Human recombinant tumor necrosis factor-alpha was administered intravesically, at a dose of 500 mg dissolved in 30 ml of phosphate buffer (pH7.6-7.8) plus 0.25% human albumin, weekly for two months to 18 patients with papillary transitional cell carcinoma of the bladder. Of the 15 evaluable patients, four (26%) achieved a complete response. Systemic and local tolerability were excellent. Int J Urol 1995;2:100-1…

medicine.medical_specialtyPathologyUrologyUrologyPhases of clinical researchlaw.inventionlawRecurrencemedicineHumansPapillary transitional cell carcinomaCarcinoma Transitional Cellbusiness.industryTumor Necrosis Factor-alphaRecombinant ProteinRecombinant ProteinsRecombinant Tumor Necrosis Factor-AlphaIntravesical treatmentAdministration IntravesicalTolerabilityUrinary Bladder NeoplasmsToxicityRecombinant DNATumor necrosis factor alphabusinessHuman
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Exercise restores decreased physical activity levels and increases markers of autophagy and oxidative capacity in myostatin/activin blocked mdx mice

2013

The importance of adequate levels of muscle size and function and physical activity is widely recognized. Myostatin/activin blocking increases skeletal muscle mass but may decrease muscle oxidative capacity and can thus be hypothesized to affect voluntary physical activity. Soluble activin receptor IIB (sActRIIB-Fc) was produced to block myostatin/activins. Modestly dystrophic mdx mice were injected with sActRIIB-Fc or PBS with or without voluntary wheel running exercise for 7 wk. Healthy mice served as controls. Running for 7 wk attenuated the sActRIIB-Fc-induced increase in body mass by decreasing fat mass. Running also enhanced/restored the markers of muscle oxidative capacity and autoph…

medicine.medical_specialtyPhysiologyActivin Receptors Type IIEndocrinology Diabetes and MetabolismBlotting WesternCitrate (si)-SynthaseMyostatinMotor ActivityHematocritMuscle hypertrophyEatingHemoglobinsMice03 medical and health sciences0302 clinical medicinePhysical Conditioning AnimalPhysiology (medical)Internal medicineAutophagymedicineAnimalsMuscle Skeletalta315Creatine KinaseAdiposity030304 developmental biology0303 health sciencesbiologymedicine.diagnostic_testTumor Necrosis Factor-alphaBody WeightAutophagySkeletal muscleDNAActivin receptorMyostatinActivinsMice Inbred C57BLmedicine.anatomical_structureEndocrinologyHematocritMice Inbred mdxbiology.proteinCreatine kinaseTumor necrosis factor alphaOxidation-Reduction030217 neurology & neurosurgeryAmerican Journal of Physiology-Endocrinology and Metabolism
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Insulin-like growth factors in chick embryo retina during development.

1996

Evidence exists supporting an important role for insulin-like growth factors (IGFs) during fetal growth. In the present report we performed studies to define whether developing chick retina contains IGFs and whether IGFs play a role in the growth of this tissue. We have shown that both IGF-I and IGF-II are present in chick embryo retina throughout development (7th-18th day). The highest values, when expressed as ng/g of tissue, were found in the youngest retinas studied (7th-9th day) and at 16th-18th day of development. During whole development the content of IGF-II was about two to three times higher than that ascertained for IGF-I. The tissue also contains cell-surface binding for IGFs. H…

medicine.medical_specialtyPhysiologymedicine.medical_treatmentCellular differentiationClinical BiochemistryChick EmbryoBiologyInsulin-Like Growth Factor ReceptorBiochemistryRetinaInsulin-likeCellular and Molecular NeuroscienceInsulin-like growth factorEndocrinologyInsulin-Like Growth Factor IIInternal medicineCulture TechniquesmedicineAnimalsInsulin-Like Growth Factor IReceptorRetinaAffinity labelingEmbryoCell DifferentiationDNAEndocrinologymedicine.anatomical_structureInsulin-like growth factor 2Culture Media Conditionedbiology.proteinCell DivisionRegulatory peptides
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Ethanol inhibits astroglial cell proliferation by disruption of phospholipase D-mediated signaling.

2002

The activation of phospholipase D (PLD) is a common response to mitogenic stimuli in various cell types. As PLD-mediated signaling is known to be disrupted in the presence of ethanol, we tested whether PLD is involved in the ethanol-induced inhibition of cell proliferation in rat cortical primary astrocytes. Readdition of fetal calf serum (FCS) to serum-deprived astroglial cultures caused a rapid, threefold increase of PLD activity and a strong mitogenic response; both effects were dependent on tyrosine kinases but not on protein kinase C. Ethanol (0.1-2%) suppressed the FCS-induced, PLD-mediated formation of phosphatidic acid (PA) as well as astroglial cell proliferation in a concentration…

medicine.medical_specialtyPlatelet-derived growth factorIndolestert-Butyl Alcoholmedicine.medical_treatmentButanolsBecaplerminPhosphatidic AcidsNerve Tissue ProteinsBiologyBiochemistryCulture Media Serum-FreeCellular and Molecular Neurosciencechemistry.chemical_compound1-ButanolInternal medicineLysophosphatidic acidmedicinePhospholipase DAnimalsPhosphorylationProtein kinase APlatelet-Derived Growth FactorEndothelin-1EthanolPhospholipase DCell growthGrowth factorPhosphatidic acidDNAProto-Oncogene Proteins c-sisProtein-Tyrosine KinasesGenisteinGrowth InhibitorsCell biologyRatsEndocrinologychemistryFetal Alcohol Spectrum DisordersAstrocyteslipids (amino acids peptides and proteins)Signal transductionVanadatesProtein Processing Post-TranslationalCell DivisionSignal TransductionJournal of neurochemistry
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Endocannabinoid anandamide mediates hypoxic pulmonary vasoconstriction

2013

Endocannabinoids are important regulators of organ homeostasis. Although their role in systemic vasculature has been extensively studied, their impact on pulmonary vessels remains less clear. Herein, we show that the endocannabinoid anandamide (AEA) is a key mediator of hypoxic pulmonary vasoconstriction (HPV) via fatty acid amide hydrolase (FAAH)-dependent metabolites. This is underscored by the prominent vasoconstrictive effect of AEA on pulmonary arteries and strongly reduced HPV in FAAH(-/-) mice and wild-type mice upon pharmacological treatment with FAAH inhibitor URB597. In addition, mass spectrometry measurements revealed a clear increase of AEA and the FAAH-dependent metabolite arac…

medicine.medical_specialtyPolyunsaturated Alkamidesmedicine.medical_treatmentHypertension PulmonaryBlotting WesternMyocytes Smooth MuscleArachidonic AcidsBiologyAmidohydrolaseschemistry.chemical_compoundMiceFatty acid amide hydrolaseInternal medicineHypoxic pulmonary vasoconstrictionmedicineAnimalsHypoxiaLungDNA PrimersMice KnockoutAnalysis of VarianceMultidisciplinaryReverse Transcriptase Polymerase Chain ReactionAnandamideHypoxia (medical)URB597Biological Sciencesmedicine.diseaseEndocannabinoid systemPulmonary hypertensionImmunohistochemistryEndocrinologynervous systemchemistryVasoconstrictionBenzamideslipids (amino acids peptides and proteins)CannabinoidCarbamatesmedicine.symptompsychological phenomena and processesChromatography LiquidEndocannabinoidsSignal Transduction
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Pharmacogenetics and Pharmacotherapy of Military Personnel Suffering from Post-traumatic Stress Disorder

2017

Background Posttraumatic stress disorder (PTSD) is a severe problem among soldiers with combating experience difficult to treat. The pathogenesis is still not fully understood at the psychological level. Therefore, genetic research became a focus of interest. The identification of single nucleotide polymorphisms (SNPs) may help to predict, which persons are at high risk to develop PTSD as a starting point to develop novel targeted drugs for treatment. Methods We conducted a systematic review on SNPs in genes related to PTSD pathology and development of targeted pharmacological treatment options based on PubMed database searches. We focused on clinical trials with military personnel. Results…

medicine.medical_specialtyPopulationTropomyosin receptor kinase BBioinformaticsArticleStress Disorders Post-Traumatic03 medical and health sciencessingle nucleotide polymorphisms0302 clinical medicinePharmacotherapyDopamineDopamine receptor D2medicineAnimalsHumansPharmacology (medical)geneticsGenetic Predisposition to DiseaseReceptorPsychiatryeducationeducation.field_of_studyClinical Trials as Topicbusiness.industryTraumatic stressGeneral MedicineDNAgene-environment interactions030227 psychiatryPsychiatry and Mental healthMilitary PersonnelNeurologyGene-Environment InteractionNeurology (clinical)pharmacologybusinessmental diseases030217 neurology & neurosurgeryPharmacogeneticsmedicine.drugCurrent Neuropharmacology
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Skin response to a carcinogen involves the xenobiotic receptor pregnane X receptor.

2015

Skin is in daily contact with potentially harmful molecules from the environment such as cigarette smoke, automobile emissions, industrial soot and groundwater. Pregnane X receptor (PXR) is a transcription factor expressed in liver and intestine that is activated by xenobiotic chemicals including drugs and environmental pollutants. Topical application of the tumor initiator 7,12-dimethylbenz(a)anthracene (DMBA) enhances Pxr, Cyp1a1, Cyp1b1 and Cyp3a11, but not Ahr expression in the skin. Surprisingly, DMBA-induced Pxr upregulation is largely impaired in Langerin(+) cell-depleted skin, suggesting that DMBA mainly triggers Pxr in Langerin(+) cells. Furthermore, PXR deficiency protects from DN…

medicine.medical_specialtyReceptors SteroidLangerinDNA damage910-Dimethyl-12-benzanthraceneDMBADermatologymedicine.disease_causeBiochemistrydigestive systemArticleDownregulation and upregulationCell MovementInternal medicinemedicineAnimalsMolecular BiologyCarcinogenSkinPregnane X receptorbiologyintegumentary systemPregnane X ReceptorAryl hydrocarbon receptordigestive system diseasesUp-RegulationMice Inbred C57BLEndocrinologyLangerhans CellsCancer researchbiology.proteinCarcinogensCarcinogenesisDNA DamageExperimental dermatology
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Cross-reactivity of anti-ssDNA antibodies with heparan sulfate in patients with type I diabetes mellitus

1989

Anti-single-stranded–DNA antibodies cross-reactive with heparan sulfate were detected in serums of patients with type I (insulin-dependent) diabetes mellitus. The results suggested that heparan sulfate, the major glycosaminoglycan constituent of the glomerular basement membrane, may serve as a target antigen in vivo for cross-reactive anti-DNA antibodies. These polyreactive antibodies, directed toward repeating negatively charged units, may neutralize the heparan sulfate–associated polyanionic sites in the glomerulus, leading to an abnormal permeability of anionic plasma proteins.

medicine.medical_specialtyRenal glomerulusEndocrinology Diabetes and MetabolismDNA Single-StrandedEnzyme-Linked Immunosorbent AssayPerlecanCross ReactionsBiologyGlycosaminoglycanchemistry.chemical_compoundInternal medicinemedicineInternal MedicineHumansChildGlycosaminoglycansBasement membraneGlomerular basement membraneHeparan sulfateBlood proteinsMolecular biologyDiabetes Mellitus Type 1Endocrinologymedicine.anatomical_structurechemistryImmunoglobulin Gbiology.proteinHeparitin SulfateAntibody
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