Search results for "DOPAMINERGIC"

showing 10 items of 200 documents

Dopamine receptor 2 activation inhibits ovarian vascular endothelial growth factor secretion in vitro: implications for treatment of ovarian hypersti…

2013

Objective To ascertain whether vascular endothelial growth factor (VEGF) secretion by luteinized granulosa cells (GCs) is modulated by the dopaminergic system in a dose-dependent fashion and how this is related to the differential efficacy of dopamine receptor 2 (D2)-agonists (D2-ag) in preventing ovarian hyperstimulation syndrome (OHSS). Design The relationship between the dopaminergic system and VEGF secretion in luteinized GCs was evaluated. Archived human ovaries were immunostained to characterize D2 expression. Setting University affiliated infertility center. Patient(s) Premenopausal women and egg donors. Intervention(s) Luteinized GCs were cultured with the D2-ag cabergoline. Human o…

AdultVascular Endothelial Growth Factor Aendocrine systemmedicine.medical_specialtyOvarian hyperstimulation syndromePilot ProjectsLuteal phaseBiologyOvarian Hyperstimulation Syndromechemistry.chemical_compoundCabergolineInternal medicinemedicineHumansSecretionCells CulturedGranulosa CellsReceptors Dopamine D2OvaryDopaminergicObstetrics and GynecologyMiddle Agedmedicine.diseaseVascular endothelial growth factorTreatment OutcomeEndocrinologyReproductive MedicinechemistryDopamine receptorDopamine AgonistsImmunohistochemistryFemalesense organsmedicine.drugFertility and Sterility
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A53T-Alpha-Synuclein Overexpression Impairs Dopamine Signaling and Striatal Synaptic Plasticity in Old Mice

2010

BACKGROUND: Parkinson's disease (PD), the second most frequent neurodegenerative disorder at old age, can be caused by elevated expression or the A53T missense mutation of the presynaptic protein alpha-synuclein (SNCA). PD is characterized pathologically by the preferential vulnerability of the dopaminergic nigrostriatal projection neurons. METHODOLOGY/PRINCIPAL FINDINGS: Here, we used two mouse lines overexpressing human A53T-SNCA and studied striatal dysfunction in the absence of neurodegeneration to understand early disease mechanisms. To characterize the progression, we employed young adult as well as old mice. Analysis of striatal neurotransmitter content demonstrated that dopamine (DA…

AgingDopaminelcsh:MedicineMicechemistry.chemical_compoundHomer Scaffolding ProteinsReceptor Cannabinoid CB1lcsh:ScienceLong-term depressionNeurotransmitterChromatography High Pressure LiquidIn Situ Hybridization FluorescenceOligonucleotide Array Sequence AnalysisMice KnockoutNeuronal PlasticityMultidisciplinaryReverse Transcriptase Polymerase Chain ReactionDopaminergicNeurodegenerationGenetics and Genomics/Gene ExpressionElectrophysiologyalpha-SynucleinResearch ArticleRadioimmunoprecipitation Assaymedicine.medical_specialtyNeuronal Calcium-Sensor ProteinsHOMER1Substantia nigraNeurotransmissionBiologyNeurological DisordersInternal medicinemedicineAnimalsHumansddc:610Cyclic Nucleotide Phosphodiesterases Type 7Activating Transcription Factor 2lcsh:RNeuropeptidesmedicine.diseaseMolecular biologyCorpus StriatumMice Mutant StrainsEndocrinologyGenetics and Genomics/Disease ModelschemistrySynaptic plasticitylcsh:QCarrier ProteinsPLoS ONE
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Progerin expression induces a significant downregulation of transcription from human repetitive sequences in iPSC-derived dopaminergic neurons.

2019

Repetitive DNA sequences represent about half of the human genome. They have a central role in human biology, especially neurobiology, but are notoriously difficult to study. The purpose of this study was to quantify the transcription from repetitive sequences in a progerin-expressing cellular model of neuronal aging. Progerin is a nuclear protein causative of the Hutchinson–Gilford progeria syndrome that is also incrementally expressed during the normal aging process. A dedicated pipeline of analysis allowed to quantify transcripts containing repetitive sequences from RNAseq datasets oblivious of their genomic localization, tolerating a sufficient degree of mutational noise, all with low c…

AgingRetroelementsTranscription GeneticAluInduced Pluripotent Stem CellsAlu elementDown-RegulationSettore BIO/11 - Biologia MolecolareRetrotransposonComputational biologyBiologySettore BIO/19 - Microbiologia GeneraleProgerinProgeriaSettore BIO/13 - Biologia ApplicataAlu ElementsRepetitive sequencemedicineRetrotransposonHumansDNA transposonRepeated sequenceGeneCellular SenescenceProgeriaintegumentary systemDopaminergic NeuronsFibroblastsmedicine.diseaseProgerinLamin Type ASettore BIO/18 - GeneticaSatelliteHuman genomeOriginal ArticleGeriatrics and GerontologyGeroScience
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Study of the Expression of Angiogenic Factors and Dopaminergic Receptors: Correlation with ZAP70, CD38 and the Mutational State in Chronic Lymphocyti…

2006

Abstract The angiogenesis is a well known process implicate in the progression of CLL. In this disease has been described an increase of angiogenic and pro-angiogenic factors in serum and angiogenic receptors in B cells, mainly in advances stages. Recently had been known that the agonist of the dopaminergic receptor D2 can inhibit the tumour growth and a possible mechanism mediator is the internalization of VEGFR-2. The aim of this work had been to analyze the expression of the dopaminergic receptors (D1, D2, D3, D4 and D5), the angiogenic receptors and its correlation with the main biological factor implicated in the illness (ZAP-70, CD38 and the mutational status of IgVH/BCL-6). We had de…

Agonistmedicine.medical_specialtymedicine.drug_classAngiogenesisZAP70Chronic lymphocytic leukemiaImmunologyDopaminergicCell BiologyHematologyBiologyCD38medicine.diseaseBiochemistryEndocrinologyDopamine receptorInternal medicinemedicineReceptorBlood
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112 Workshop Summary: DOPAMINERGIC MECHANISMS OF PAIN CONTROL

2007

Anesthesiology and Pain MedicinePain controlbusiness.industryMedicineDopaminergic mechanismsbusinessNeuroscienceEuropean Journal of Pain
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A Methanol Extract ofBrugmansia arboreaAffects the Reinforcing and Motor Effects of Morphine and Cocaine in Mice

2013

Previous reports have shown that several of the effects of morphine, including the development of tolerance and physical withdrawal symptoms, are reduced by extracts ofBrugmansia arborea(L.) Lagerheim (Solanaceae) (B. arborea). In the present study we evaluate the action of the methanol extract ofB. arborea(7.5–60 mg/kg) on the motor and reinforcing effects of morphine (20 and 40 mg/kg) and cocaine (25 mg/kg) using the conditioned place preference (CPP) procedure. At the doses employed,B. arboreadid not affect motor activity or induce any effect on CPP. The extract partially counteracted morphine-induced motor activity and completely blocked the CPP induced by 20 mg/kg morphine. On the othe…

Article Subjectbiologybusiness.industryDopaminergiclcsh:Other systems of medicinePharmacologylcsh:RZ201-999biology.organism_classificationConditioned place preferenceComplementary and alternative medicineMechanism of actionBrugmansiaMorphinemedicineCholinergicMotor activitymedicine.symptombusinessCocaine abuseResearch Articlemedicine.drugEvidence-Based Complementary and Alternative Medicine
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Preparation of dopaminergic N-alkyl-benzyltetrahydroisoquinolines using a 'one-pot' procedure in acid medium.

2000

The preparation of N-methyl-BTHIQ (4) from N-phenylethyl-phenacetamide (1) by cyclization, reduction and N-alkylation in acid medium has been achieved in good yield in a 'one-pot' procedure. Acylation of imine (2) intermediate afforded the Z and E stereoselectivity in the enamide formation. 6-Hydroxy-BTHIQ (7) shows selectivity for D2 dopamine receptors, while its N-methylated homologue (8) displays higher affinities for both D1 and D2 receptor types, with an unexpected increase in D1 dopamine receptor affinity.

Bicyclic moleculeStereochemistryReceptors Dopamine D2Receptors Dopamine D1Spectrum AnalysisOrganic ChemistryClinical BiochemistryDopaminergicImineDopamine AgentsPharmaceutical ScienceIsoquinolinesBiochemistryChemical synthesisAcylationchemistry.chemical_compoundchemistryDopamine receptor D2Drug DiscoveryMolecular MedicineStereoselectivitySelectivityMolecular Biology
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Binge-like alcohol exposure in adolescence: behavioural, neuroendocrine and molecular evidence of abnormal neuroplasticity … and return

2021

Binge alcohol consumption among adolescents affects the developing neural networks underpinning reward and stress processing in the nucleus accumbens (NAc). This study explores in rats the long-lasting effects of early intermittent exposure to intoxicating alcohol levels at adolescence, on: (1) the response to natural positive stimuli and inescapable stress

Binge alcohol drinkingmedicine.medical_specialtySettore BIO/14 - FARMACOLOGIAQH301-705.5Medicine (miscellaneous)Nucleus accumbensArticleGeneral Biochemistry Genetics and Molecular Biology03 medical and health sciences0302 clinical medicineDopamineDopamine receptor D2Internal medicineNeuroplasticityMedicineCannabidiol[SDV.NEU] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]Biology (General)030304 developmental biologyDopamine transporter0303 health sciencesbiologyTyrosine hydroxylasebusiness.industryDopaminergicAdolescenceEndocrinologybiology.proteinNucleus accumbens[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]Adolescence Binge alcohol drinking Cannabidiol Nucleus accumbensbusinessCannabidiol030217 neurology & neurosurgerymedicine.drug
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Brunner syndrome associated MAOA dysfunction in human dopaminergic neurons results in NMDAR hyperfunction and increased network activity

2020

AbstractBackgroundMonoamine neurotransmitter abundance affects motor control, emotion, and cognitive function and is regulated by monoamine oxidases. Amongst these, monoamine oxidase A (MAOA) catalyzes the degradation of dopamine, norepinephrine, and serotonin into their inactive metabolites. Loss-of-function mutations in the X-linked MAOA gene cause Brunner syndrome, which is characterized by various forms of impulsivity, maladaptive externalizing behavior, and mild intellectual disability. Impaired MAOA activity in individuals with Brunner syndrome results in bioamine aberration, but it is currently unknown how this affects neuronal function.MethodsWe generated human induced pluripotent s…

Brunner syndromeDopaminergicBiologymedicine.diseaseNorepinephrineMonoamine neurotransmitterDopaminebiology.proteinmedicineGRIN2BSerotoninMonoamine oxidase ANeurosciencemedicine.drug
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Effects of motor cortex removal on circling behaviour.

1982

AbstractChemical stimulation of the dopaminergic nigro-striatal system induces circling behaviour also in animals in which an extensive telencephalic lesion has been performed. In order to avoid the effects linked to chemical stimulation, we have studied the circling behaviour means of electrical stimulation of the caudate nucleus in chronic cats with cortical lesions. The role of the motor cortex on the circling behaviour is emphasized.

CATSPhysiologyDopaminergicCaudate nucleusMotor CortexStimulationBiologyBiochemistryElectric StimulationLesionmedicine.anatomical_structuremedicineCatsAnimalsHumansmedicine.symptomCaudate NucleusStereotyped BehaviorNeuroscienceMotor cortexArchives internationales de physiologie et de biochimie
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