Search results for "ECR"

showing 10 items of 3689 documents

Diversity of extracellular vesicles from different developmental stages of Fasciola hepatica

2020

The secretion of extracellular vesicles (EVs) in Fasciola hepatica adult worms was described by our group in 2012. Since then, EVs have been found in other helminths, thus providing a new paradigm for the complete understanding of host-parasite communication. However, information was lacking regarding the possible existence and role of EVs from other developmental stages of the parasite. In this study, we confirm the secretion of EVs by F. hepatica eggs and juvenile forms. EVs were isolated by size exclusion chromatography and characterised by nanoparticle tracking analysis and electron microscopy. We observed a large diversity in the morphologies of these EVs, suggesting specific functions…

0301 basic medicineFuture studies030231 tropical medicineNanoparticle tracking analysisExtracellular vesicles03 medical and health sciencesExtracellular Vesicles0302 clinical medicineHepaticaSize exclusion chromatographyparasitic diseasesFasciola hepaticaParasite hostingAnimalsSecretionLife Cycle StagesbiologyVesicleFasciola hepaticaExtracellular vesiclesbiology.organism_classificationCell biology030104 developmental biologyInfectious DiseasesFasciola hepatica juvenilesParasitology
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Potential benefits of colostrum in gastrointestinal diseases

2016

This paper reviews the composition of colostrum and the potential preventive and therapeutic use of this "first milk" for treating various gastrointestinal disorders in humans. Colostrum is a complex biological liquid that is richer in antimicrobial peptides, immune-regulating compounds and growth factors than the subsequent mature milk. The main functions of colostrum are to provide essential nutritional components, strengthen the natural defense system, modulate immune response, balance intestinal microbiota and enhance the growth and repair of several tissues. Several studies and clinical trials carried out both in vitro and in vivo on humans and animals suggest the clinical benefits of …

0301 basic medicineGastrointestinal Diseasesanimal diseasesAntimicrobial peptidesPhysiologyGeneral Biochemistry Genetics and Molecular Biology03 medical and health sciencesImmune systemfluids and secretionsImmunityIn vivogastrointestinal diseases dysbiosis colostrumMedicineAnimalsHumansClinical significanceColostrum Anti-Microbical Factors Immunity Growth Factors Intestinal Disorders ReviewGeneral Immunology and Microbiologybusiness.industryColostrumfood and beveragesmedicine.diseaseClinical trial030104 developmental biologyDietary SupplementsColostrumCattleFemalebusinessDysbiosis
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Clinical efficacy of α4 integrin block with natalizumab in ankylosing spondylitis

2016

We describe the impact of α4-β1/7 blockade with natalizumab, a recombinant humanised immunoglobulin (Ig) G4κ monoclonal antibody (mAb) targeted to the α4 subunit of the α4β1 and α4β7 integrins, on the gut and spine inflammation in a patient with ankylosing spondylitis (AS) who developed multiple sclerosis after treatment with tumour necrosis factor (TNF)-blocking agents. A 45-year-old man with human leucocyte antigen (HLA)-B27-positive AS was admitted in January 2007. He had been diagnosed with AS 4 years earlier based on the presence of inflammatory back pain, peripheral arthritis, radiographic bilateral grade 2 sacroiliitis, HLA-B27 positivity. At that time, he had evidence of chronic int…

0301 basic medicineGenetics and Molecular Biology (all)medicine.drug_classImmunologyHuman leukocyte antigenMonoclonal antibodyBiochemistryGeneral Biochemistry Genetics and Molecular Biology03 medical and health sciences0302 clinical medicineNatalizumabRheumatologymedicineAdalimumabImmunology and Allergy030203 arthritis & rheumatologyInflammationAnkylosing spondylitisBiochemistry Genetics and Molecular Biology (all)business.industryMultiple sclerosisMedicine (all)Sacroiliitismedicine.diseaseTreatmentSettore MED/16 - Reumatologia030104 developmental biologyImmunologyTumor necrosis factor alphaSpondyloarthritibusinessmedicine.drug
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Nearly Complete Genome Sequences of Human Norovirus Belonging to Several Genotypes from Valencia, Spain

2019

Human noroviruses are responsible for most nonbacterial acute gastroenteritis cases. The GII.2, GII.4, and GII.17 genotypes of human noroviruses have recently arisen as the most frequent genotypes found in humans worldwide. We report here seven nearly complete genomes of these genotypes from patients with acute gastroenteritis in Valencia, Spain.

0301 basic medicineGeneticsMolecular epidemiologyVirus RNAvirusesGenome Sequences030106 microbiologyMicrobiologiavirus diseasesBiologyAcute gastroenteritisGenoma humàmedicine.disease_causeGenome03 medical and health sciencesfluids and secretions030104 developmental biologyImmunology and Microbiology (miscellaneous)Genetic variationGenotypeGeneticsNorovirusmedicineMolecular BiologyMicrobiology Resource Announcements
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A dual role of caspase-8 in triggering and sensing proliferation-associated DNA damage, a key determinant of liver cancer development.

2017

Summary Concomitant hepatocyte apoptosis and regeneration is a hallmark of chronic liver diseases (CLDs) predisposing to hepatocellular carcinoma (HCC). Here, we mechanistically link caspase-8-dependent apoptosis to HCC development via proliferation- and replication-associated DNA damage. Proliferation-associated replication stress, DNA damage, and genetic instability are detectable in CLDs before any neoplastic changes occur. Accumulated levels of hepatocyte apoptosis determine and predict subsequent hepatocarcinogenesis. Proliferation-associated DNA damage is sensed by a complex comprising caspase-8, FADD, c-FLIP, and a kinase-dependent function of RIPK1. This platform requires a non-apop…

0301 basic medicineGenome instabilityMaleliver; Hepatocellular carcinoma; DNA damage response; replication stress; apoptosisCancer ResearchDNA RepairCarcinogenesisFas-Associated Death Domain ProteinApoptosisurologic and male genital diseasesDNA damage responseDna Damage Response ; Apoptosis ; Hepatocellular Carcinoma ; Liver ; Replication StressHistonesMice0302 clinical medicineRisk FactorsFADDPhosphorylationCellular SenescenceCaspase 8biologyLiver Neoplasmshepatocellular carcinomaLiver regeneration3. Good healthHistoneOncologyReceptors Tumor Necrosis Factor Type I030220 oncology & carcinogenesisReceptor-Interacting Protein Serine-Threonine KinasesFemalebiological phenomena cell phenomena and immunityCell agingCarcinoma HepatocellularDNA damageDNA repairreplication stressCaspase 8liverArticleGenomic Instability03 medical and health sciencesAnimalsHepatectomyHumansCrosses GeneticCell ProliferationJNK Mitogen-Activated Protein KinasesCell BiologyLiver Regeneration030104 developmental biologyImmunologyChronic Diseasebiology.proteinCancer researchHepatocytesMyeloid Cell Leukemia Sequence 1 ProteinDNA Damage
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With mouse age comes wisdom : a review and suggestions of relevant mouse models for age-related conditions

2016

Ageing is a complex multifactorial process that results in many changes in physiological changes processes that ultimately increase susceptibility to a wide range of diseases. As such an ageing population is resulting in a pressing need for more and improved treatments across an assortment of diseases. Such treatments can come from a better understanding of the pathogenic pathways which, in turn, can be derived from models of disease. Therefore the more closely the model resembles the disease situation the more likely relevant the data will be that is generated from them. Here we review the state of knowledge of mouse models of a range of diseases and aspects of an ageing physiology that ar…

0301 basic medicineGerontologyAgingPopulation ageingProcess (engineering)TRAUMATIC BRAIN-INJURYDiseaseBiologyMouse modelsMice03 medical and health sciences0302 clinical medicineAge relatedMedicine and Health SciencesAnimalsHumansCLOSED-BONE-FRACTURESENESCENCE-ACCELERATED MOUSEE-DEFICIENT MICECELL-MEDIATED-IMMUNITYTRIPLE-TRANSGENIC MODELBiology and Life SciencesNECROSIS-FACTOR-ALPHAAged patientsCell mediated immunityC-REACTIVE PROTEINACTIVATION IN-VIVODisease Models AnimalPatient populationAgeing030104 developmental biologyAgeingPhenotypingMouse models ; ageing ; phenotypingLONG-TERM POTENTIATION030217 neurology & neurosurgeryCognitive psychologyDevelopmental Biology
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Role of the DNA repair glycosylase OGG1 in the activation of murine splenocytes

2017

OGG1 (8-oxoguanine-DNA glycosylase) is the major DNA repair glycosylase removing the premutagenic DNA base modification 8-oxo-7,8-dihydroguanine (8-oxoG) from the genome of mammalian cells. In addition, there is accumulating evidence that OGG1 and its substrate 8-oxoG might function in the regulation of certain genes, which could account for an attenuated immune response observed in Ogg1-/- mice in several settings. Indications for at least two different mechanisms have been obtained. Thus, OGG1 could either act as an ancillary transcription factor cooperating with the lysine-specific demethylase LSD1 or as an activator of small GTPases. Here, we analysed the activation by lipopolysaccaride…

0301 basic medicineGuanineDNA RepairDNA repairp38 mitogen-activated protein kinasesBiologyBiochemistryDNA GlycosylasesMice03 medical and health sciencesAnimalsMolecular BiologyTranscription factorTumor Necrosis Factor-alphaKinaseActivator (genetics)MacrophagesDNACell BiologyBase excision repairMolecular biology030104 developmental biologyGene Expression RegulationDNA glycosylaseTumor necrosis factor alphaSpleenDNA DamageTranscription FactorsDNA Repair
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Disentangling the effect of host genetics and gut microbiota on resistance to an intestinal parasite

2019

11 pages; International audience; Resistance to infection is a multifactorial trait, and recent work has suggested that the gut microbiota can also contribute to resistance. Here, we performed a fecal microbiota transplant to disentangle the contribution of the gut microbiota and host genetics as drivers of resistance to the intestinal nematode Heligmosomoides polygyrus. We transplanted the microbiota of a strain of mice (SJL), resistant to H. polygyrus, into a susceptible strain (CBA) and vice-versa. We predicted that if the microbiota shapes resistance to H. polygyrus, the FMT should reverse the pattern of resistance between the two host strains. The two host strains had different microbi…

0301 basic medicineHeligmosomoides polygyrusGut floramedicine.disease_causeFecal microbiota transplant0302 clinical medicinefluids and secretionsMESH: Fecal Microbiota TransplantationParasite hostingColonizationMESH: AnimalsMESH: Strongylida InfectionsDisease ResistanceGeneticsNematospiroides dubiusbiology[SDV.BA]Life Sciences [q-bio]/Animal biologyFecal Microbiota Transplantation3. Good healthInfectious DiseasesMESH: Nematospiroides dubiusGenetic Background030231 tropical medicineIntestinal parasiteHeterologousMice Inbred StrainsMESH: Disease ResistanceMESH: Host-Parasite InteractionsMESH: Mice Inbred Strainsdigestive systemMESH: Gastrointestinal MicrobiomeHost-Parasite Interactions03 medical and health sciencesImmunityparasitic diseasesmedicineAnimals[SDV.MP.PAR]Life Sciences [q-bio]/Microbiology and Parasitology/ParasitologyStrongylida InfectionsHost (biology)ImmunityLife history traitsMESH: Genetic Backgroundbiology.organism_classificationGastrointestinal MicrobiomeDisease Models Animalstomatognathic diseases030104 developmental biologyParasitologyHeligmosomoides polygyrusMESH: Disease Models Animal[SDV.AEN]Life Sciences [q-bio]/Food and Nutrition
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Gut-microbiota-derived extracellular vesicles: Overlooked mediators in host–helminth interactions?

2021

Helminth infections impact the composition of the mammalian gut microbiota; however, the mechanisms underpinning these interactions are, thus far, unknown. In this article, we propose that microbiota-derived extracellular vesicles might represent key players in host-helminth-microbiome crosstalk, and outline future directions to elucidate their role(s) in host-parasite relationships.

0301 basic medicineHelminth infectionsHost (biology)030231 tropical medicineHelminthiasisBiologyGut florabiology.organism_classificationdigestive systemExtracellular vesiclesGastrointestinal MicrobiomeHost-Parasite InteractionsCell biologyExtracellular Vesicles03 medical and health sciencesCrosstalk (biology)fluids and secretions030104 developmental biology0302 clinical medicineInfectious DiseasesHelminthsparasitic diseasesAnimalsHumansParasitologyTrends in Parasitology
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Rab33B Controls Hepatitis B Virus Assembly by Regulating Core Membrane Association and Nucleocapsid Processing

2017

Many viruses take advantage of cellular trafficking machineries to assemble and release new infectious particles. Using RNA interference (RNAi), we demonstrate that the Golgi/autophagosome-associated Rab33B is required for hepatitis B virus (HBV) propagation in hepatoma cell lines. While Rab33B is dispensable for the secretion of HBV subviral envelope particles, its knockdown reduced the virus yield to 20% and inhibited nucleocapsid (NC) formation and/or NC trafficking. The overexpression of a GDP-restricted Rab33B mutant phenocopied the effect of deficit Rab33B, indicating that Rab33B-specific effector proteins may be involved. Moreover, we found that HBV replication enhanced Rab33B expres…

0301 basic medicineHepatitis B virusBiologymedicine.disease_causeVirusArticleCell LineCell membraneRab33B03 medical and health sciencesnucleocapsid assemblyTranscription (biology)RNA interferenceVirologymedicineHumansSecretionNucleocapsidcore/capsid membrane associationHepatitis B virus030102 biochemistry & molecular biologyEffectorVirus AssemblyCell MembraneVirologyHepatitis B Core Antigenshepatitis B virus; Rab GTPase; Rab33B; core/capsid membrane association; nucleocapsid assembly; virus traffickingTransport proteinProtein Transport030104 developmental biologyInfectious Diseasesmedicine.anatomical_structurevirus traffickingrab GTP-Binding ProteinsHost-Pathogen InteractionsHepatocytesRab GTPaseViruses; Volume 9; Issue 6; Pages: 157
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