Search results for "Endocrine System"

showing 10 items of 1530 documents

Visceral adiposity index (VAI) is predictive of an altered adipokine profile in patients with type 2 diabetes.

2014

Aims Although there is still no clear definition of “adipose tissue dysfunction” or ATD, the identification of a clinical marker of altered fat distribution and function may provide the needed tools for early identification of a condition of cardiometabolic risk. Our aim was to evaluate the correlations among various anthropometric indices [BMI, Waist Circumference (WC), Hip Circumference (HC), Waist/Hip ratio (WHR), Body Adiposity Index (BAI) and Visceral adiposity Index (VAI)] and several adipocytokines [Visfatin, Resistin, Leptin, Soluble leptin receptors (sOB-R), Adiponectin, Ghrelin, Adipsin, PAI-1, vascular endothelial growth factor (VEGF), Hepatocyte growth factor (HGF) TNF-α, hs-CRP…

MalePhysiologyEpidemiologylcsh:MedicineType 2 diabetesSettore MED/13 - EndocrinologiaEndocrinologyImmune PhysiologyMedicine and Health SciencesCluster AnalysisClinical Epidemiologylcsh:ScienceAdiposityMultidisciplinaryAnthropometryLeptinVAI adipokine diabetesMiddle AgedLipidsType 2 DiabetesPhysiological ParametersResearch DesignCytokinesFemaleAnatomyResearch Articlemedicine.medical_specialtyWaistClinical Research DesignImmunologyAdipokineEndocrine SystemIntra-Abdominal FatBody adiposity indexResearch and Analysis MethodsAdipokinesInternal medicineDiabetes MellitusmedicineHumansObesityAgedNutritionDiabetic EndocrinologyAdiponectinbusiness.industryBody Weightlcsh:RBiology and Life SciencesMolecular Developmentmedicine.diseaseEndocrinologyDiabetes Mellitus Type 2ROC CurveImmune SystemMetabolic DisordersClinical ImmunologyResistinlcsh:QMetabolic syndromebusinessDevelopmental BiologyPLoS ONE
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Association of placental concentrations of phenolic endocrine disrupting chemicals with cognitive functioning in preschool children from the Environm…

2020

Developmental exposure to bisphenol A (BPA) and other phenolic endocrine disrupting chemicals (EDCs) may affect child neurodevelopment, but data on the effects of prenatal exposure to phenols on cognitive function remain sparse. Our aim was to examine the association of placental concentrations of several phenolic EDCs, including BPA, parabens (PBs), and benzophenones (BzPs), with cognitive development in preschool children from the Environment and Childhood (INMA) Project in Spain. Concentrations of BPA, four PBs (methylparaben [MePB], ethylparaben [EtPB], propylparaben [PrPB], and butylparaben [BuPB]), and six BzPs (BzP-1, BzP-2, BzP-3, BzP-6, BzP-8, and 4-hydroxybenzophenone [4-OH-BzP]) …

MalePlacentaPhysiology010501 environmental sciencesEndocrine DisruptorsNeuropsychological Tests01 natural sciencesMotor function03 medical and health scienceschemistry.chemical_compound0302 clinical medicineCognitionPhenolsPregnancyEndocrine systemMedicineHumans030212 general & internal medicineNeuropsychological assessmentCognitive skillBenzhydryl CompoundsAssociation (psychology)ChildPrenatal exposure0105 earth and related environmental sciencesGeneral Environmental ScienceButylparabenmedicine.diagnostic_testbusiness.industryConfoundingPublic Health Environmental and Occupational HealthchemistryChild PreschoolGeneral Earth and Planetary SciencesFemalebusinessClinical psychologyInternational journal of hygiene and environmental health
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Quantitative mass spectrometry for human melanocortin peptides in vitro and in vivo suggests prominent roles for β-MSH and desacetyl α-MSH in energy …

2018

Objective The lack of pro-opiomelanocortin (POMC)-derived melanocortin peptides results in hypoadrenalism and severe obesity in both humans and rodents that is treatable with synthetic melanocortins. However, there are significant differences in POMC processing between humans and rodents, and little is known about the relative physiological importance of POMC products in the human brain. The aim of this study was to determine which POMC-derived peptides are present in the human brain, to establish their relative concentrations, and to test if their production is dynamically regulated. Methods We analysed both fresh post-mortem human hypothalamic tissue and hypothalamic neurons derived from …

MalePluripotent Stem CellsLeptinlcsh:Internal medicineendocrine systemhPSC human pluripotent stem cellsPro-Opiomelanocortin[SDV.NEU.NB]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/NeurobiologyHypothalamusMass SpectrometryTandem Mass Spectrometry[SDV.BBM.GTP]Life Sciences [q-bio]/Biochemistry Molecular Biology/Genomics [q-bio.GN]beta-MSHHomeostasisHumansHuman pluripotent stem cellObesitylcsh:RC31-1245MSHNeuronsintegumentary systemReceptors MelanocortinLC-MS/MS liquid chromatography tandem mass spectrometryNeuropeptidesdigestive oral and skin physiologyPOMCPVH the paraventricular nucleus of the hypothalamusCTX cerebral cortexMelanocortinsNeuropeptidealpha-MSHOriginal ArticleFemalehormones hormone substitutes and hormone antagonistsChromatography Liquid
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Effects of Vildagliptin/Metformin Therapy on Patient-Reported Outcomes: Work Productivity, Patient Satisfaction, and Resource Utilization

2013

Introduction: Type 2 diabetes mellitus (T2DM) is associated not only with high direct healthcare costs, but also with indirect costs, as diabetic complications and the disease itself result in loss of productivity. Vildagliptin is a novel dipeptidyl peptidase-4 inhibitor that is given either alone or in combination with oral hypoglycemic drugs, including metformin. The study was designed to assess the hypothesis that fixed-combination vildagliptin/metformin improves work productivity measured as Work Productivity and Activity Impairment (WPAI) scores. Secondary objectives were the assessment of patient satisfaction by means of the Diabetes Treatment Satisfaction Questionnaire (DTSQs), the c…

MalePyrrolidinesSettore MED/09 - Medicina Internaendocrine system diseasesAdamantaneEfficiencyoutcomeschemistry.chemical_compoundIndirect costsDiabetes mellitusVildagliptinPharmacology (medical)Prospective StudiesPatient-reported outcomeProductivityAged 80 and overVildagliptinMedicine(all)Health Care CostsGeneral MedicineHealth ServicesMiddle AgedMetforminMetforminType 2 diabetes mellituDrug CombinationsTreatment OutcomeItalyPatient SatisfactionFemalemedicine.drugAdultEmploymentResourcemedicine.medical_specialtyDiabetes mellitus; oral antidiabetics; vildagliptin; outcomesoral antidiabeticsPatient satisfactionInternal medicineDiabetes mellitusNitrilesmedicineHumansHypoglycemic AgentsHealthcare costFixed combinationAgedbusiness.industryType 2 Diabetes Mellitusnutritional and metabolic diseasesmedicine.diseaseEndocrinologyDiabetes Mellitus Type 2chemistryEmergency medicineObservational studyGlycated hemoglobinbusinessAdvances in Therapy
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Induction of RAGE Shedding by Activation of G Protein-Coupled Receptors

2011

The multiligand Receptor for Advanced Glycation End products (RAGE) is involved in various pathophysiological processes, including diabetic inflammatory conditions and Alzheimers disease. Full-length RAGE, a cell surface-located type I membrane protein, can proteolytically be converted by metalloproteinases ADAM10 and MMP9 into a soluble RAGE form. Moreover, administration of recombinant soluble RAGE suppresses activation of cell surface-located RAGE by trapping RAGE ligands. Therefore stimulation of RAGE shedding might have a therapeutic value regarding inflammatory diseases. We aimed to investigate whether RAGE shedding is inducible via ligand-induced activation of G protein-coupled recep…

MaleReceptors Vasopressinendocrine system diseasesReceptor for Advanced Glycation End Productslcsh:MedicineHydroxamic Acids570 Life sciencesRAGE (receptor)Adenylyl cyclaseADAM10 ProteinMicePhosphatidylinositol 3-Kinaseschemistry.chemical_compoundMolecular Cell BiologyNeurobiology of Disease and RegenerationSignaling in Cellular ProcessesMembrane Receptor SignalingReceptors Immunologiclcsh:ScienceReceptorLungCellular Stress ResponsesCalcium signalingMultidisciplinaryKinaseDipeptidesHormone Receptor SignalingCell biologyMatrix Metalloproteinase 9NeurologyReceptors OxytocinGene Knockdown Techniquescardiovascular systemMatrix Metalloproteinase 2Pituitary Adenylate Cyclase-Activating PolypeptideMedicineRNA InterferenceAdenylyl CyclasesResearch ArticleSignal Transduction570 Biowissenschaftenmedicine.medical_specialtyMAP Kinase Signaling SystemADAM17 ProteinBiologyAlzheimer DiseaseCa2+/calmodulin-dependent protein kinaseInternal medicinemedicineAnimalsHumansProtease InhibitorsCalcium Signalingcardiovascular diseasesBiologyG protein-coupled receptorlcsh:RHEK 293 cellsMembrane Proteinsnutritional and metabolic diseasesCyclic AMP-Dependent Protein KinasesADAM ProteinsG-Protein SignalingHEK293 CellsEndocrinologychemistryProteolysisDementialcsh:QAmyloid Precursor Protein SecretasesMolecular Neurosciencehuman activitiesReceptors Pituitary Adenylate Cyclase-Activating Polypeptide Type INeurosciencePLoS ONE
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Integrated and diurnal indices of maternal pregnancy cortisol in relation to sex-specific parasympathetic responsivity to stress in infants.

2020

Maternal hypothalamic-pituitary-adrenal (HPA) axis activity may prenatally program sex-specific stress response pathways. We investigated associations between maternal cortisol during pregnancy and infant parasympathetic responsivity to stress among 204 mother-infant pairs. Cortisol indices included 3(rd) trimester hair cortisol, as well as diurnal slope and area under the curve, derived from saliva samples collected during pregnancy. Mother-infant dyads participated in the Repeated Still-Face Paradigm (SFP-R) at age 6 months. We calculated respiration-adjusted respiratory sinus arrhythmia (RSA(c)), an indicator of parasympathetic activation, from infant respiration and cardiac activity mea…

MaleSalivaendocrine systemHypothalamo-Hypophyseal SystemHydrocortisonePhysiologyMothersPituitary-Adrenal System3rd trimesterArticle03 medical and health sciencesBehavioral Neuroscience0302 clinical medicineDevelopmental NeurosciencePregnancyRespirationDevelopmental and Educational PsychologymedicineHumans0501 psychology and cognitive sciencesVagal toneSalivaPregnancybusiness.industry05 social sciencesArea under the curveInfantmedicine.diseaseSex specificPrenatal Exposure Delayed EffectsAutonomic reactivityFemalebusiness030217 neurology & neurosurgeryhormones hormone substitutes and hormone antagonistsStress Psychological050104 developmental & child psychologyDevelopmental BiologyDevelopmental psychobiology
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Biphenyl and fluorinated derivatives: liver enzyme-mediated mutagenicity detected in Salmonella typhimurium and Chinese hamster V79 cells.

1992

Abstract Hepatocarcinogenic polychlorinated and polybrominated biphenyls usually show negative results in in vitro mutagenicity assays. Problems in their testing result from their low water solubility and their slow rate of metabolism. We therefore investigated better soluble model compounds, namely biphenyl and its 3 possible monofiuorinated derivatives. In the direct test, these compounds proved tobe nonmutagenic in Salmonella typhimurium TA98 and TA100 (reversion to histidine prototrophy) and in Chinese hamster V79 cells (acquisition of resistance to 6-thioguanine). However, when the exposure was carried out in the presence of NADPH-fortified postmitochondrial fraction of liver homogenat…

MaleSalmonella typhimuriumendocrine systemChinese hamsterAmes testCell LineToxicologychemistry.chemical_compoundStructure-Activity RelationshipCricetulusCricetinaeAnimalsBiotransformationBiphenylbiologyChemistryMutagenicity TestsBiphenyl CompoundsRats Inbred StrainsGeneral MedicineMetabolismbiology.organism_classificationEnterobacteriaceaeIn vitroRatsBiochemistryMicrosomal epoxide hydrolaseMicrosomes LiverPolybrominated BiphenylsMutation research
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Mutagenicity spectra in Salmonella typhimurium strains of glutathione, L-cysteine and active oxygen species

1989

Glutathione and L-cysteine, in the presence of rat kidney post-mitochondrial supernatant (S9) fraction, and various forms of active oxygen were investigated for mutagenicity in seven his- strains of Salmonella typhimurium. Glutathione and L-cysteine showed qualitatively and quantitatively virtually identical mutagenic activities. The number of mutants induced in strain TA97 was 3-4 times higher than in TA100, the strain in which the mutagenicity was originally detected. Mutagenic effects were also observed in strains TA92, TA102 and TA104, but not in TA1535 and TA1537. Hydrogen peroxide, superoxide and glucose/glucose oxidase in the presence and absence of kidney S9 fraction showed pronounc…

MaleSalmonella typhimuriumendocrine systemHealth Toxicology and MutagenesisIn Vitro TechniquesKidneyToxicologyAmes testSuperoxide dismutasechemistry.chemical_compoundSuperoxidesGeneticsAnimalsCysteineBiotransformationGenetics (clinical)chemistry.chemical_classificationReactive oxygen speciesbiologyMutagenicity TestsSuperoxide DismutaseSuperoxidefungifood and beveragesKidney metabolismRats Inbred StrainsHydrogen PeroxideGlutathioneCatalaseGlutathioneRatsOxygenchemistryS9 fractionBiochemistryCatalasebiology.proteinMutagensMutagenesis
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Inactivation of electrophilic metabolites by glutathione S-transferases and limitation of the system due to subcellular localization

1977

Benzo(a)pyrene was activated to metabolites mutagenic for Salmonella typhimurium TA 98 by liver microsomes from control and phenobarbital treated mice. Under these conditions benzo(a)pyrene 4,5-oxide accounts for most of the mutagenicity. We have therefore investigated (1) the conjugation of benzo(a)pyrene 4,5-oxide with glutathione and (2) the effect of glutathione on the mutagenicity of benzo(a)pyrene.

MaleSalmonella typhimuriumendocrine systemHealth Toxicology and MutagenesisMutagenToxicologymedicine.disease_causeMicechemistry.chemical_compoundCytosolBiotransformationpolycyclic compoundsmedicineAnimalsBenzopyrenesBiotransformationGlutathione Transferasebiologyfungifood and beveragesGeneral MedicineGlutathioneSubcellular localizationGlutathioneCytosolGlutathione S-transferaseBenzo(a)pyrenechemistryBiochemistryMicrosomes Liverbiology.proteinPyreneMutagensArchives of Toxicology
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Metabolic epoxidation of trans-4-acetylaminostilbene: A protective mechanism against its activation to a mutagen

1976

Abstract Trans -4-acetylaminostilbene is activated by liver preparations to mutagens for Salmonella typhimurium . Since this compound is metabolized to the trans -α,β-epoxide and since many epoxides are ultimate mutagens, this epoxide was tested for direct mutagenicity. It was, however, found to be non-mutagenic, and, in contrast to the parent compound, the epoxide was no longer activated by liver preparations to mutagens. The same was found for the β-ketone and for the threo -α,β-dihydrodiol, which are formed metabolically from trans -4-acetylaminostilbene and from its α,β-epoxide. 4-Acetylaminobibenzyl showed a very weak mutagenic activity in the presence of the liver preparation. Thus, i…

MaleSalmonella typhimuriumendocrine systemStereochemistryBiophysicsEpoxideMutagenmedicine.disease_causeBiochemistryMicechemistry.chemical_compoundStilbenesmedicineAnimalsMolecular BiologyMolecular Structurefungifood and beveragesCell BiologyTrans-4-acetylaminostilbeneLiverchemistryBiochemistryEpoxy CompoundsLiver preparationMutagensBiochemical and Biophysical Research Communications
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