Search results for "Escape"

showing 10 items of 119 documents

Neuroprotective effects of behavioural training and nicotine on age-related deficits in spatial learning.

2006

Studies in humans and animals show a clear decline in spatial memory with age and several approaches have been adopted to alleviate this impairment. The purpose of our review is to assess the studies that have suggested the possible neuroprotective actions of behavioural training and nicotine-applied both independently and in conjunction-on age-related deficits in spatial learning. Both spatial pretraining and nonspatial experiences influence an animal's performance in spatial tasks. In aged rats, the experience of training in the water maze task increases the number of newly generated neurons in the hippocampus. The neuroprotective effects of nicotine have been demonstrated in both in-vitr…

AgingNicotineHippocampusWater mazeReceptors NicotinicNeuroprotectionSpatial memoryHippocampusNicotineBehavior TherapyEscape ReactionOrientationmedicineAnimalsHumansMaze LearningProblem SolvingPharmacologyConfoundingNeurodegenerative DiseasesSpatial cognitionRatsPsychiatry and Mental healthNeuroprotective AgentsPractice PsychologicalMental RecallSpatial learningSeptum PellucidumPsychologyCognitive psychologymedicine.drugBehavioural pharmacology
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CD39 is highly involved in mediating the suppression activity of tumor-infiltrating CD8+ T regulatory lymphocytes

2013

CD39 is an ectoenzyme, present on different immune cell subsets, which mediates immunosuppressive functions catalyzing ATP degradation. It is not known whether CD39 is expressed and implicated in the activity of CD8+ regulatory T lymphocytes (Treg). In this study, CD39 expression and function was analyzed in both CD8+ and CD4+CD25hi Treg from the peripheral blood of healthy donors as well as from tumor specimens. CD39 was found expressed by both CD8+ (from the majority of healthy donors and tumor patients) and CD4+CD25 hi Treg, and CD39 expression correlated with suppression activity mediated by CD8+ Treg. Importantly, CD39 counteraction remarkably inhibited the suppression activity of CD8+…

CD4-Positive T-LymphocytesCancer ResearchImmunologyAntineoplastic Agentschemical and pharmacologic phenomenaCD8-Positive T-LymphocytesBiologyT-Lymphocytes RegulatoryImmune toleranceAntineoplastic AgentLymphocytes Tumor-InfiltratingImmune systemAntigenAntigens CDImmune TolerancemedicineHumansIndoleamine-Pyrrole 23-DioxygenaseImmunology and AllergyCell ProliferationCD39Tumor microenvironmentCell growthApyraseInterleukin-2 Receptor alpha SubunitCD8-Positive T-LymphocyteTumor immune escapePhenotypePhenotypeCD39 CD8+ Treg Tumor immune escape ToleranceMicroscopy FluorescenceOncologyMechanism of actionCD4-Positive T-LymphocyteImmunologyCD8+ Tregmedicine.symptomToleranceCD8HumanCancer Immunology, Immunotherapy
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miR-155expression in antitumor immunity: The higher the better?

2019

MicroRNAs are small noncoding RNAs that modulate gene expression either directly, by impairing the stability and/or translation of transcripts that contain their specific target sequence, or indirectly through the targeting of transcripts that encode transcription factors, factors implicated in signal transduction pathways, or epigenetic regulators. Abnormal expression of micro-RNAs has been found in nearly all types of pathologies, including cancers. MiR-155 has been the first microRNA to be implicated in the regulation of the innate and adaptative immune responses, and its expression is either increased or decreased in a variety of liquid and solid malignancies. In this review, we examine…

Cancer ResearchLeukemiaCarcinogenesisBiologymiR-155MicroRNAs03 medical and health sciences0302 clinical medicineImmune system030220 oncology & carcinogenesisGene expressionmicroRNAGeneticsCancer researchAnimalsHumansCytotoxic T cellTumor EscapeImmunotherapyEpigeneticsDown SyndromeSignal transductionTranscription factorGenes, Chromosomes and Cancer
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Altered CD94/NKG2A and perforin expression reduce the cytotoxic activity in malignant pleural effusions.

2010

CD94/NKG2A is an inhibitory receptor expressed by NK cells and cytotoxic lymphocytes and, upon activation by HLA-E, downregulates the cytolytic activities of these cells thus representing a tumour immune escape mechanism. This study was aimed at assessing whether cytotoxic lymphocytes (CD8+) and NK cells from malignant pleural effusions have a deregulated expression of CD94/NKG2A. The expression of membrane CD94/NKG2A and perforin was evaluated by flow-cytometry in CD8+ and NK cells from pleural effusions and autologous peripheral blood of cancer (n=19) and congestive heart failure (CHF) (n=11) patients. Intracellular CD94/NKG2A expression was evaluated by flow-cytometry in pleural effusion…

Cancer ResearchPleural effusionLymphocyteSettore MED/10 - Malattie Dell'Apparato RespiratorioCD8-Positive T-LymphocytesInterleukin 21Pleural diseaseNeoplasmsmedicineMalignant pleural effusionCytotoxic T cellHumansAgedHeart Failurebiologybusiness.industryPerforinCancerCD8-Positive T-LymphocyteMiddle Agedmedicine.diseaseNatural Killer T-CellPleural Effusion Malignantmedicine.anatomical_structureOncologyPerforinCase-Control StudiesImmunologybiology.proteinNeoplasmNatural Killer T-CellsTumor EscapebusinessCase-Control StudieNK Cell Lectin-Like Receptor Subfamily DHumanT-Lymphocytes CytotoxicEuropean journal of cancer (Oxford, England : 1990)
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Dendritic cells as mediators of tumor-induced tolerance in metastatic melanoma.

1997

Escape from immune surveillance is critical for tumor progression in metastatic melanoma. We assessed the function of melanoma-derived dendritic cells (DCs) in patients presenting simultaneously with responding (rM) or progressing (pM) melanoma metastases. These rare coincidences allowed us to compare syngeneically the function of tumor DCs. CD83+ DCs were purified freshly from large responding (rDCs) or progressing (pDCs) metastases following chemo-immunotherapy. rDCs were 5 times more potent inducers of allogeneic T-cell proliferation than the pDCs that were used as control. Phenotypic analysis showed a marked depression of CD86 expression on pDCs. Culture supernatants from pM showed prod…

Cancer ResearchT-LymphocytesImmune toleranceImmune systemAntigens CDAntigens NeoplasmAntineoplastic Combined Chemotherapy ProtocolsmedicineHumansAntigen-presenting cellMelanomaCD86Membrane Glycoproteinsbusiness.industryMelanomaInterferon-alphahemic and immune systemsDendritic cellDendritic Cellsmedicine.diseaseInterleukin-10Neoplasm ProteinsTolerance inductionOncologyTumor progressionImmunologyCytokinesInterleukin-2Tumor EscapeB7-2 AntigenCisplatinbusinessCell DivisionInternational journal of cancer
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Toll-like receptors: Expression and involvement in Multiple Myeloma

2010

Multiple Myeloma (MM) cells express and respond to a broad range of TLRs. Accumulating evidences suggest that TLRs act as double-edged sword in MM biology. Indeed, TLR9 or TLR3 ligands could enhance immunity against MM cells or directly induce cell apoptosis, whereas various TLR agonists could induce MM survival, proliferation, and immune escape. This review is focused on the heterogeneous expression and function of TLRs in MM and on the potential implication of TLR ligands of infectious or endogenous origin in MM emergence, resistance, or progression.

Cancer ResearchTLR9ApoptosisEndogenyHematologyBiologyLigandsmedicine.diseaseToll-Like Receptor 3Cell biologyGene Expression Regulation NeoplasticImmune systemOncologyApoptosisImmunityToll-Like Receptor 9TLR3medicineAnimalsHumansTumor EscapeMultiple MyelomaReceptorMultiple myelomaLeukemia Research
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Lactate: A Metabolic Key Player in Cancer

2011

Abstract Increased glucose uptake and accumulation of lactate, even under normoxic conditions (i.e., aerobic glycolysis or the Warburg Effect), is a common feature of cancer cells. This phenomenon clearly indicates that lactate is not a surrogate of tumor hypoxia. Tumor lactate can predict for metastases and overall survival of patients, as shown by several studies of different entities. Metastasis of tumors is promoted by lactate-induced secretion of hyaluronan by tumor-associated fibroblasts that create a milieu favorable for migration. Lactate itself has been found to induce the migration of cells and cell clusters. Furthermore, radioresistance has been positively correlated with lactate…

Cancer ResearchTumor hypoxiaGlucose uptakeCellCancerBiologymedicine.diseaseRadiation ToleranceMetastasismedicine.anatomical_structureImmune systemOncologyCell MovementNeoplasmsImmunologymedicineCancer researchAnimalsHumansTumor EscapeGlycolysisLactic AcidWound healingGlycolysisCancer Research
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Immune Control in Hepatocellular Carcinoma Development and Progression: Role of Stromal Cells

2014

Immune control of hepatocellular carcinoma (HCC) is executed by effector immune cells, which efficiently eliminate malignant transformed cells. However, progression of HCC clearly documents failure of tumor immune control, which led to the concept of immune subversion by the tumor environment. Particularly tumor-associated stromal cells cooperate within an inflammatory network, which is responsible for immune privilege. The stromal cell composition matures during tumor growth and is derived from surrounding noncancerous tissue or from circulating cells recruited to the tumor site. Therefore, immunosuppressive stromal cells represent heterogeneous cell lineages, including myeloid cells, lymp…

Carcinoma HepatocellularStromal cellmedicine.medical_treatmentAdaptive ImmunityBiologyLymphocytes Tumor-InfiltratingImmune systemCancer immunotherapyImmune privilegeTumor MicroenvironmentLymph node stromal cellmedicineAnimalsHumansTumor microenvironmentHepatologyLiver Neoplasmsbiochemical phenomena metabolism and nutritionAcquired immune systemImmunity InnateB-1 cellImmunologyCytokinesbacteriaTumor EscapeImmunotherapyStromal CellsSignal TransductionSeminars in Liver Disease
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Coping with continual danger: assessing alertness to visual disturbances in crucian carp following long-term exposure to chemical alarm signals.

2013

Chemical alarm signals in fish are passively released into the water from ruptured epidermal cells, and induce instant fright responses in conspecifics. Fish also display alarm responses to injured heterospecific skin, as well as to scent of piscivorous predators that have ingested prey. A conspicuous alertness to visual disturbances has also been observed in fish following long-term exposure to extracts of filtered, homogenized skin, but the chemical inducers of such vigilance are actually unknown. We tested if a continual exposure to water-soluble alarm signals, from either conspecifics or heterospecifics, affects alertness of fish. Based on previous experience, it was assumed that alertn…

CarpsTime Factorsmedia_common.quotation_subjectMovementZoologyExperimental and Cognitive PsychologyPheromonesPredationBehavioral NeuroscienceALARMEscape ReactionAnimalsVision Ocularmedia_commonSkinPerchbiologyEcologybiology.organism_classificationStimulation ChemicalTroutAlertnessVisual DisturbancePredatory BehaviorOdorantsCrucian carpSensory DeprivationVigilance (psychology)Physiologybehavior
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Tumor-derived lactic acid modulates dendritic cell activation and antigen expression.

2005

The tumor milieu can influence dendritic cell (DC) differentiation. We analyzed DC differentiation in a 3-dimensional tumor model and propose a new mechanism of DC modulation by the tumor environment. Monocytes were cultured in the presence of IL-4 and GM-CSF within multicellular tumor spheroids (MCTSs) generated from different tumor cell lines. Monocytes invaded the MCTSs and differentiated into tumor-associated dendritic cells (TADCs). The antigen expression was altered on TADCs independent of the culture conditions (immature/mature DCs, Langerhans cells) and IL-12 secretion was reduced. Supernatants of MCTSs could partially transfer the suppressive effect. Conditioned media from urotheli…

Cellular differentiationImmunologyBiologyBiochemistryMonocyteschemistry.chemical_compoundCell Line TumorNeoplasmsSpheroids CellularmedicineHumansSecretionLactic AcidMelanomaCell DifferentiationCell BiologyHematologyDendritic cellDendritic CellsTumor-Derivedmedicine.diseaseCoculture TechniquesCell biologyLactic acidTumor EscapechemistryCell cultureCytokinesTumor EscapeBlood
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