Search results for "F2-Isoprostane"

showing 7 items of 7 documents

Even free radicals should follow some rules: a guide to free radical research terminology and methodology.

2014

Free radicals and oxidants are now implicated in physiological responses and in several diseases. Given the wide range of expertise of free radical researchers, application of the greater understanding of chemistry has not been uniformly applied to biological studies. We suggest that some widely used methodologies and terminologies hamper progress and need to be addressed. We make the case for abandonment and judicious use of several methods and terms and suggest practical and viable alternatives. These changes are suggested in four areas: use of fluorescent dyes to identify and quantify reactive species, methods for measurement of lipid peroxidation in complex biological systems, claims of…

Biological studiesFree RadicalsChemistryRadicalRADICAIS LIVRESFree Radical ScavengersBiochemistryReactive Nitrogen SpeciesThiobarbituric Acid Reactive SubstancesPhysiological responsesAntioxidantsTerminologyLipid peroxidationF2-Isoprostanechemistry.chemical_compoundBiochemistryPhysiology (medical)Terminology as TopicAnimalsHumansBiochemical engineeringLipid PeroxidationReactive Oxygen SpeciesFluorescent DyesFree radical biologymedicine
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An inter-laboratory validation of methods of lipid peroxidation measurement in UVA-treated human plasma samples

2010

Lipid peroxidation products like malondialdehyde, 4-hydroxynonenal and F2-isoprostanes are widely used as markers of oxidative stress in vitro and in vivo. This study reports the results of a multi-laboratory validation study by COST Action B35 to assess inter-laboratory and intra-laboratory variation in the measurement of lipid peroxidation. Human plasma samples were exposed to UVA irradiation at different doses (0, 15 J, 20 J), encoded and shipped to 15 laboratories, where analyses of malondialdehyde, 4-hydroxynonenal and isoprostanes were conducted. The results demonstrate a low within-day-variation and a good correlation of results observed on two different days. However, high coefficie…

Ultraviolet RaysClinical Chemistry TestsEnzyme-Linked Immunosorbent AssayIsoprostanesmedicine.disease_causeF2-isoprostanesSensitivity and SpecificityBiochemistryHigh-performance liquid chromatographyMass Spectrometry4-HydroxynonenalLipid peroxidationPlasmachemistry.chemical_compoundIn vivoMalondialdehydemedicineHumansChromatography High Pressure LiquidAldehydesChromatographyChemistryReproducibility of Resultsoxidative stress; F2-Isoprostanes; 4.-hydroxynonenal; malondialdehydeGeneral MedicineOxidative stress; F2-isoprostanes; 4-hydroxynonenal; malondialdehydeMalondialdehydeIsoprostanes4-hydroxynonenalF2-IsoprostanesBiochemistryOxidative stressLipid PeroxidationOxidative stressChromatography Liquid
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The Mediterranean diet improves the systemic lipid and DNA oxidative damage in metabolic syndrome individuals. A randomized, controlled, trial.

2013

Summary Background & aims Metabolic syndrome (MetS), in which a non-classic feature is an increase in systemic oxidative biomarkers, presents a high risk of diabetes and cardiovascular disease (CVD). Adherence to the Mediterranean Diet (MedDiet) is associated with a reduced risk of MetS. However, the effect of the MedDiet on biomarkers for oxidative damage has not been assessed in MetS individuals. We have investigated the effect of the MedDiet on systemic oxidative biomarkers in MetS individuals. Methods Randomized, controlled, parallel clinical trial in which 110 female with MetS, aged 55–80, were recruited into a large trial (PREDIMED Study) to test the efficacy of the traditional MedDie…

medicine.medical_specialtyMediterranean dietUrinary systemCritical Care and Intensive Care Medicinemedicine.disease_causeDiet Mediterraneanlaw.inventionRandomized controlled triallawRisk FactorsInternal medicineDiabetes mellitusmedicineHumansNutsPlant OilsDiet Fat-RestrictedOlive OilAgedAged 80 and overMetabolic SyndromeF2-IsoprostanesNutrition and Dieteticsbusiness.industryDeoxyguanosineMiddle Agedmedicine.diseaseLipid MetabolismClinical trialOxidative StressEndocrinology8-Hydroxy-2'-DeoxyguanosineCardiovascular DiseasesFemaleMetabolic syndromebusinessBody mass indexRisk Reduction BehaviorOxidative stressBiomarkersDNA DamageClinical nutrition (Edinburgh, Scotland)
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F-2-isoprostanes: review of analytical methods

2006

International audience; F2-isoprostanes (F2-isoPs) represent a new family of biomarkers for oxidative stress generated by free radical attack of membrane-bounded arachidonic acid. Esterified F2-isoPs can be found in tissue or plasma lipids whereas the free form F2-isoPs, hydrolyzed by phospholipase, is mainly present in body fluids. The extent of systematic damage due to oxidative stress within the body can be assessed by the determination of plasma or urine F2-isoPs. The determination of F2-isoPs in clinical practice is not often used due to the complexity to extract the compounds from their biologic matrixes before the analysis step. In most of published protocols, extraction procedure is…

MASS SPECTROMETRYIsoprostaneBiophysicsPharmaceutical ScienceMass spectrometry01 natural sciencesBiochemistryIMMUNOASSAYchemistry.chemical_compound[ CHIM.ORGA ] Chemical Sciences/Organic chemistryPlasma lipidsQUANTITATIVE ANALYSISNEUROPROSTANESample preparationComputingMilieux_MISCELLANEOUSChromatography010405 organic chemistryChemistry[CHIM.ORGA]Chemical Sciences/Organic chemistryL IPID PEROXIDATION010401 analytical chemistryExtraction (chemistry)[SDV.SP]Life Sciences [q-bio]/Pharmaceutical sciences[CHIM.ORGA] Chemical Sciences/Organic chemistry3. Good health0104 chemical sciencesClinical Practice[SDV.SP] Life Sciences [q-bio]/Pharmaceutical sciencesF2-IsoprostanesMolecular MedicineFree formISOPROSTANE
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Enhanced Lipid Peroxidation and Platelet Activation in the Early Phase of Type 1 Diabetes Mellitus

2003

Background— To investigate early events possibly related to the development of diabetic angiopathy, we examined whether 8-iso-prostaglandin F 2α (8-iso-PGF 2α ) formation, a marker of in vivo oxidant stress, is altered in different stages of type 1 diabetes (T1DM) and whether it correlates with the rate of thromboxane (TX) A 2 biosynthesis, a marker of in vivo platelet activation. We also investigated the relationship between inflammatory markers and F 2 -isoprostane formation in this setting. Methods and Results— A cross-sectional study was performed in 23 insulin-treated patients aged <18 years with new-onset T1DM (≤6 weeks, group A), matched for age and gender with 23 patients with s…

diabetes mellitus; inflammation; plateletsMalemedicine.medical_specialtyAdolescentThromboxaneInflammationDiabetic angiopathyDinoprostTimeThromboxane A2Reference ValuesPhysiology (medical)Internal medicineDiabetes mellitusHumansInsulinMedicinePlatelet activationChildInterleukin 6InflammationF2-IsoprostanesType 1 diabetesbiologyInterleukin-6Tumor Necrosis Factor-alphabusiness.industryPlatelet Activationmedicine.diseaseThromboxane B2Oxidative StressC-Reactive ProteinCross-Sectional StudiesDiabetes Mellitus Type 1Endocrinologydiabetes mellitusplateletsDisease Progressionbiology.proteinFemaleTumor necrosis factor alphaLipid Peroxidationmedicine.symptomCardiology and Cardiovascular MedicinebusinessBiomarkersFollow-Up StudiesCirculation
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Correlation between a marker of oxidative stress and few indexes of endothelial dysfunction in essential hypertensive patients

2005

medicine.medical_specialtyEndotheliumbusiness.industrymedicine.diseasemedicine.disease_causeNitric oxidechemistry.chemical_compoundF2-Isoprostanesmedicine.anatomical_structureEndocrinologychemistryPlasma drug concentrationInternal medicineInternal MedicinemedicineEndothelial dysfunctionbusinessOxidative stressAmerican Journal of Hypertension
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Therapeutic dosages of raloxifene do not modify myeloperoxidase and F2alpha-isoprostane levels in postmenopausal women.

2005

We investigated the effect of a therapeutic dose of raloxifene on the plasma levels of myeloperoxidase and F2alpha-isoprostanes, two markers of oxidative stress recently described as reliable indicators of coronary heart disease. Contrary to changes described in the literature for estrogens (E), raloxifene did not modify the levels of either myeloproxidase or F2alpha-isoprostanes after 3 or 6 months of treatment.

Selective Estrogen Receptor Modulatorsmedicine.medical_specialtyIsoprostaneNeutrophilsEstrogen receptormedicine.disease_causeAntioxidantschemistry.chemical_compoundTherapeutic indexInternal medicineMedicineHumansRaloxifenePeroxidaseF2-Isoprostanesbiologybusiness.industryObstetrics and GynecologyMiddle AgedAntiestrogenIsoprostanesPostmenopauseOxidative StressEndocrinologyReproductive MedicinechemistryCardiovascular DiseasesMyeloperoxidaseRaloxifene Hydrochloridebiology.proteinFemalebusinessOxidative stressBiomarkersmedicine.drugFertility and sterility
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