Search results for "FACTOR"

showing 10 items of 17757 documents

Frequency of polymorphisms of signal peptide of TGF-beta1 and -1082G/A SNP at the promoter region of Il-10 gene in patients with carotid stenosis

2006

The role of inflammation in atherosclerosis is well recognized. We have evaluated the allele frequencies of the +869T/C and +915G/C polymorphisms (SNPs) at the TGF-beta1 gene and -1082G/A SNP at IL-10 promoter sequence, two well-known immunosuppressive and anti-inflammatory cytokines, in patients with carotid stenosis. Our data suggest a lack of association between these SNPs and the susceptibility to atherosclerosis although other reports have demonstrated this association. These results may be due to the pleiotropic effects of the cytokines and/or differences in haplotype combination that should be investigated to elucidate the role of TGF-beta1 and IL-10 polymorphisms in atherosclerosis.

medicine.medical_treatmentSNPSingle-nucleotide polymorphismInflammationProtein Sorting SignalsBioinformaticsPolymorphism Single NucleotideGeneral Biochemistry Genetics and Molecular BiologyTransforming Growth Factor beta1atherosclerosiHistory and Philosophy of ScienceGene FrequencyPolymorphism (computer science)Transforming Growth Factor betacytokineMedicineSNPHumansCarotid StenosisPromoter Regions GeneticAllele frequencyAgedAged 80 and overPolymorphism Geneticbusiness.industryGeneral NeuroscienceHaplotypePromoterSequence Analysis DNAMiddle AgedInterleukin-10carotid stenosiCytokineImmunologyIL-10medicine.symptombusinessTGF-beta 1
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Differential requirements for antigen or homeostatic cytokines for proliferation and differentiation of human Vgamma9Vdelta2 naive, memory and effect…

2005

We have compared four human subsets of Vgamma9Vdelta2 T cells, naive (T(naive), CD45RA(+)CD27(+)), central memory (T(CM), CD45RA(-)CD27(+)), effector memory (T(EM), CD45RA(-)CD27(-)) and terminally differentiated (T(EMRA), CD45RA(+)CD27(-)), for their capacity to proliferate and differentiate in response to antigen or homeostatic cytokines. Cytokine responsiveness and IL-15R expression were low in T(naive) cells and progressively increased from T(CM) to T(EM) and T(EMRA) cells. In contrast, the capacity to expand in response to antigen or cytokine stimulation showed a reciprocal pattern and was associated with resistance to cell death and Bcl-2 expression. Whereas antigen-stimulated cells a…

medicine.medical_treatmentT cellCellular differentiationImmunologychemical and pharmacologic phenomenaBiologyLymphocyte ActivationAntigenimmune system diseasesT-Lymphocyte SubsetsmedicineImmunology and AllergyHomeostasisHumansAntigensReceptorCells CulturedInterleukin-15Receptors Interleukin-15virus diseaseshemic and immune systemsCell DifferentiationReceptors Antigen T-Cell gamma-deltaReceptors Interleukin-2In vitroCell biologyTumor Necrosis Factor Receptor Superfamily Member 7Cytokinemedicine.anatomical_structureInterleukin 15CytokinesLeukocyte Common AntigensImmunologic MemoryEx vivoEuropean journal of immunology
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Mast cell growth-enhancing activity (MEA) is structurally related and functionally identical to the novel mouse T cell growth factor P40/TCGFIII (int…

1990

We have previously shown that certain bone marrow-derived mast cell (BMMC) lines proliferate in response to a mast cell growth-enhancing activity (MEA) that is distinct from interleukin (IL) 3 and IL 4. Here we provide evidence that MEA is identical with the recently cloned mouse T cell growth factor P40. The evidence is as follows: (a) recombinant P40 displayed all the biological activities ascribed to MEA: it supported the growth of MEA-sensitive BMMC lines, it induced IL 6 secretion by these cells, and it enhanced survival of primary mast cell cultures; (b) highly purified MEA stimulated the growth of P40-dependent cell lines; (c) a rabbit monospecific antiserum directed against P40 spec…

medicine.medical_treatmentT-LymphocytesImmunologyBone Marrow CellsBiologyIn Vitro TechniquesBinding CompetitiveMicemedicineImmunology and AllergyAnimalsInterleukin 9Mast CellsGrowth SubstancesInterleukin 4Cell growthGrowth factorImmune SeraInterleukinsInterleukin-9Interleukinfood and beveragesMast cellCell biologyCytokinemedicine.anatomical_structureCell cultureImmunologyEuropean journal of immunology
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Is COVID‐19 infection more severe in kidney transplant recipients?

2021

International audience; There are no studies which have compared the risk of severe Covid-19 and related mortality between transplant recipients and non-transplant patients. We enrolled two groups of patients hospitalized for Covid-19, i.e., kidney transplant recipients from the French Registry of Solid Organ Transplant (n=306) and a single-center cohort of non-transplant patients (n=795). An analysis was performed among subgroups matched for age and risk factors for severe Covid-19 or mortality. Severe Covid-19 was defined as admission (or transfer) to an intensive care unit, need for mechanical ventilation, or death.Transplant recipients were younger and had more comorbidities compared to…

medicine.medical_treatment[SDV]Life Sciences [q-bio]MESH: Registries*AucunMESH: Comorbidity030230 surgerylaw.inventionchemistry.chemical_compound0302 clinical medicinelawcardiovascular diseaseMESH: Risk Factors[SDV.MHEP.MI]Life Sciences [q-bio]/Human health and pathology/Infectious diseasesclinical research / practiceImmunology and AllergyCumulative incidencePharmacology (medical)kidney transplantation / nephrologyMESH: IncidenceMESH: AgedUnivariate analysisMESH: France / epidemiologyMESH: Middle AgedMESH: Transplant Recipients / statistics & numerical data*Acute kidney injuryIntensive care unit3. Good healthMESH: COVID-19 / epidemiologyCohort[SDV.MHEP.MI] Life Sciences [q-bio]/Human health and pathology/Infectious diseasesglomerular filtration rate (GFR)kidney failure / injurymedicine.medical_specialtyinfection and infectious agents - viralinfectious diseaseBrief CommunicationMESH: Graft Rejection / prevention & control03 medical and health sciencesInternal medicineDiabetes mellitusMESH: Severity of Illness IndexMESH: COVID-19 / diagnosis*medicineHumansMESH: SARS-CoV-2Mechanical ventilationCreatinineTransplantationMESH: Humansbusiness.industrySARS-CoV-2MESH: Graft Rejection / epidemiology*COVID-19MESH: Retrospective Studiesmedicine.diseaseKidney TransplantationTransplant RecipientsMESH: Maleimmunosuppressive regimensMESH: Immunosuppressive Agents / therapeutic useMESH: Pandemics*MESH: Propensity Score*chemistryReinfectionMESH: Immunosuppression / methodsMESH: Intensive Care UnitsbusinessMESH: FemaleMESH: Kidney Transplantation*
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Perfluoroalkylated amphiphilic MUC1 glycopeptide antigens as tools for cancer immunotherapy.

2010

The synthesis of perfluoroalkylated glycopeptide antigens and their specific binding to anti-MUC1 mouse antibodies is reported.

medicine.medical_treatmentdigestive systemCatalysisAntibodiesAntigen-Antibody ReactionsMiceAntigenCancer immunotherapyNeoplasmsAmphiphileMaterials ChemistrymedicineAnimalsAntigensskin and connective tissue diseasesneoplasmsMUC1Mice Inbred BALB CBinding SitesbiologyMolecular StructureChemistryMucin-1Metals and AlloysGlycopeptidesGeneral Chemistrybiological factorsdigestive system diseasesGlycopeptideSurfaces Coatings and FilmsElectronic Optical and Magnetic MaterialsBiochemistryCeramics and Compositesbiology.proteinImmunotherapyAntibodyChemical communications (Cambridge, England)
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What we learn about bipolar disorder from large-scale neuroimaging

2020

Abstract MRI‐derived brain measures offer a link between genes, the environment and behavior and have been widely studied in bipolar disorder (BD). However, many neuroimaging studies of BD have been underpowered, leading to varied results and uncertainty regarding effects. The Enhancing Neuro Imaging Genetics through Meta‐Analysis (ENIGMA) Bipolar Disorder Working Group was formed in 2012 to empower discoveries, generate consensus findings and inform future hypothesis‐driven studies of BD. Through this effort, over 150 researchers from 20 countries and 55 institutions pool data and resources to produce the largest neuroimaging studies of BD ever conducted. The ENIGMA Bipolar Disorder Workin…

mega-analysisStress-related disorders Donders Center for Medical Neuroscience [Radboudumc 13]cortical surface areaReview Article0302 clinical medicineManic-depressive illnessMulticenter Studies as TopicSpectrum disorderReview Articlesbipolar disorderCerebral CortexTrastorn bipolarneuroimagingRadiological and Ultrasound Technology05 social sciencesENIGMAHUMAN BRAINMagnetic Resonance Imagingpsychiatry3. Good healthNeurologyMeta-analysisScale (social sciences)AnatomyPsychologyClinical risk factorClinical psychologyMRIMAJOR PSYCHIATRIC-DISORDERSSchizoaffective disorder050105 experimental psychology03 medical and health sciencesMagnetic resonance imagingNeuroimagingMeta-Analysis as TopicSDG 3 - Good Health and Well-beingImatges per ressonància magnèticamedicineHumans0501 psychology and cognitive sciencesRadiology Nuclear Medicine and imagingBipolar disorderHIPPOCAMPAL VOLUMESmega‐analysisGRAY-MATTER VOLUMESPECTRUM DISORDERvolumeDIABETES-MELLITUScortical thicknessCOGNITIVE IMPAIRMENTmedicine.diseaseMental illnessmeta-analysismeta‐analysisRC0321Neurology (clinical)SCHIZOAFFECTIVE DISORDERPSYCHOTIC FEATURES030217 neurology & neurosurgeryHuman Brain Mapping
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Mesenchymal Stem Cells Provide Better Results Than Hematopoietic Precursors for the Treatment of Myocardial Infarction

2010

Objectives The purpose of this study was to compare the ability of human CD34(+) hematopoietic stem cells and bone marrow mesenchymal stem cells (MSC) to treat myocardial infarction (MI) in a model of permanent left descendent coronary artery (LDA) ligation in nude rats. Background Transplantation of human CD34(+) cells and MSC has been proved to be effective in treating MI, but no comparative studies have been performed to elucidate which treatment prevents left ventricular (LV) remodelling more efficiently. Methods Human bone marrow MSC or freshly isolated CD34(+) cells from umbilical cord blood were injected intramyocardially in infarcted nude rats. Cardiac function was analyzed by echoc…

mesenchymal stem cellsPathologymedicine.medical_specialtybusiness.industrymedicine.medical_treatmentMesenchymal stem cellCD34hematopoietic precursorsHematopoietic stem cell transplantationmedicine.diseaseTransplantationleft ventricular functionHaematopoiesismyocardial infarctionmedicine.anatomical_structureImmunologymedicineMyocardial infarctionparacrine factorsStem cellCardiology and Cardiovascular MedicinebusinessArteryJournal of the American College of Cardiology
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Stem Cell Therapy

2019

The necessity of improving the health and quality of life of subjects affected by diverse injuries and chronic diseases, has led to develop a new branch of translational medicine, called Regenerative Medicine (RegMed). RegMed has the principal objects of restoring, maintaining or enhancing structures and functions of tissues and organs. In order to achieve these goals, the RegMed researchers have proposed and tested diverse approaches. The Stem Cell Therapy represents the central focus of RegMed, even if its applications may imply the onset of adverse conditions. Here, an overview of this topic will be reported, by pointing advantages and disadvantages.

mesenchymal stem cellsbusiness.industrymedicine.medical_treatmentfibrosisregenerative medicineStem-cell therapybenefits and limitationsstem cell therapyangiogenesistranslational medicinegrowth factors/cytokinesCancer researchSettore MED/05 - Patologia ClinicaMedicinebusinessangiogenesis; benefits and limitations; fibrosis; growth factors/cytokines; mesenchymal stem cells; regenerative medicine; stem cell therapy; translational medicine
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Chronic Mesenteric Ischemia: Critical review and guidelines for management

2011

Background CMI is caused by chronic occlusive disease of mesenteric arteries. In such an uncommon disease, clear recommendations are strongly needed. Unfortunately, treatment options for symptomatic CMI are still controversial and no guidelines exist. Methods A systematic literature review of the last 25-years was conducted through MEDLINE, Embase, and Cochrane Review/Trials register to identify studies reporting on CMI treatment with more than 10 patients. Primary outcomes were perioperative mortality and morbidity rates. Secondary outcomes were survival rates, primary and secondary patency rates, vessels treated, CMI recurrence, follow-up (FU), technical success (TS), and in-hospital leng…

mesenteric chronic ischemia vascular surgeryPediatricsmedicine.medical_specialtyTime FactorsMEDLINE610 Medicine & healthDiseaseSettore MED/22 - Chirurgia Vascolare2705 Cardiology and Cardiovascular MedicineIschemiaRecurrenceRisk FactorsMesenteric Vascular OcclusionmedicineHumansVascular PatencyVascular DiseasesSurvival rateVascular PatencyChi-Square Distributionbusiness.industry10042 Clinic for Diagnostic and Interventional RadiologyPatient Selection10031 Clinic for AngiologyEndovascular ProceduresGeneral MedicinePerioperativeLength of Staymedicine.disease10020 Clinic for Cardiac Surgery2746 SurgeryTreatment OutcomeSystematic reviewMesenteric ischemiaMesenteric IschemiaPractice Guidelines as TopicSurgeryCardiology and Cardiovascular MedicinebusinessVascular Surgical ProceduresChi-squared distributionAlgorithms
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Observation of the rare decay K-S -> pi(0)e(+)e(-)

2003

A search for the decay Ks->pi0e+e- has been made by the NA48/1 experiment at the CERN SPS accelerator. Using data collected during 89 days in 2002 with a high-intensity Ks beam, 7 events were found with a background of 0.15 events. The branching fraction BR(Ks->pi0e+e-, m(ee) > 0.165 GeV/c^2) = (3.0^{+1.5}_{-1.2}(stat) +/-0.2 (syst)) x 10^{-9} has been measured. Using a vector matrix element and a form factor equal to one, the measurement gives BR(Ks->pi0e+e-) = (5.8^{+2.9}_{-2.4}) x 10^{-9}.

mesoni K; decadimenti rariPhysicsNuclear and High Energy Physics010308 nuclear & particles physicsBranching fractionK-LForm factor (quantum field theory)Analytical chemistryKAON DECAYS01 natural sciences3. Good healthHigh Energy Physics - Experimentmesoni Kdecadimenti rariCP VIOLATION; KAON DECAYS; K-L; SEARCHSEARCH0103 physical sciencesMatrix elementCP VIOLATIONAtomic physics010306 general physics
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