Search results for "FAD"

showing 10 items of 292 documents

On the Statistical Analysis of Equal Gain Combining over Multiple Double Rice Fading Channels in Cooperative Networks

2010

This article analyzes the statistical properties of narrowband mobile-to-mobile (M2M) fading channels with equal gain combining (EGC) under line-of-sight (LOS) propagation conditions. Here, we study a dual-hop amplify-and-forward (AF)relay network. It is assumed that there can exist LOS components in the transmission links between the source mobile station and the destination mobile station via K mobile relays. In order to cater for asymmetric fading conditions in the relay links, the received signal envelope at the output of the equal gain (EG) combiner is thus modeled as a sum of K double Rice processes. These processes are considered to be independent but not necessarily identically dist…

Independent and identically distributed random variablesbusiness.industryComputer scienceCumulative distribution functionComputerSystemsOrganization_COMPUTER-COMMUNICATIONNETWORKSData_CODINGANDINFORMATIONTHEORYTopologylaw.inventionNarrowbandRelaylawMobile stationComputer Science::Networking and Internet ArchitectureElectronic engineeringWirelessProbability distributionFadingbusinessComputer Science::Information TheoryCommunication channel2010 IEEE 72nd Vehicular Technology Conference - Fall
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Performance Enhancement of Multimedia Broadcast Multicast Service (MBMS) with Receive Diversity

2007

The aim of this paper is to evaluate the effect of receive diversity on multimedia broadcast multicast service (MBMS) performance in wideband code division multiple access (WCDMA) networks. In addition to time diversity provided by long interleaving and receive diversity provided by multiple receive antennas, 3GPP release 6 specifications for MBMS introduce two macro diversity schemes: soft and selective combining. Diversity techniques are introduced in order to improve the MBMS performance. Improvements help especially the cell edge MBMS users who suffer most from the fading channel conditions. As receive diversity is one of the most efficient diversity techniques this paper examines the s…

Interleavingbusiness.industryCode division multiple accessComputer scienceFadingEnhanced Data Rates for GSM EvolutionRadio resource managementWidebandbusinessMultimedia Broadcast Multicast ServiceTime diversityComputer networkCooperative diversitySixth International Conference on Networking (ICN'07)
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Level-Crossing Rate and Average Duration of Fades in Non-Isotropic Hoyt Fading Channels with Applications to Selection Combining Diversity

2015

In this paper, we investigate the second-order statistics of Hoyt fading channels under non isotropic scattering scenarios. Assuming an asymmetrical Doppler power spectral density (PSD), we derive, in the form of single finite-range integrals, expressions for the level-crossing rate (LCR) and average duration of fades (ADF). These new results are then applied to obtain the LCR and ADF of selection combining (SC) diversity over non-isotropic Hoyt channels. In addition to their importance for studying the system performance and characterizing the dynamic behavior of multipath fading channels, the formulas derived are general in that they can be applied to many non-isotropic scattering situati…

IsotropySpectral densitysymbols.namesakeAngle of arrivalComputer Science::Networking and Internet Architecturevon Mises distributionsymbolsElectronic engineeringFadingStatistical physicsRayleigh scatteringGaussian processMultipath propagationComputer Science::Information TheoryMathematics2015 IEEE 82nd Vehicular Technology Conference (VTC2015-Fall)
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cFLIPL Inhibits Tumor Necrosis Factor-related Apoptosis-inducing Ligand-mediated NF-κB Activation at the Death-inducing Signaling Complex in Human Ke…

2004

Human keratinocytes undergo apoptosis following treatment with tumor necrosis factor-related apoptosis-inducing ligand (TRAIL) via surface-expressed TRAIL receptors 1 and 2. In addition, TRAIL triggers nonapoptotic signaling pathways including activation of the transcription factor NF-kappaB, in particular when TRAIL-induced apoptosis is blocked. The intracellular protein cFLIP(L) interferes with TRAIL-induced apoptosis at the death-inducing signaling complex (DISC) in many cell types. To study the role of cFLIP(L) in TRAIL signaling, we established stable HaCaT keratinocyte cell lines expressing varying levels of cFLIP(L). Functional analysis revealed that relative cFLIP(L) levels correlat…

KeratinocytesCytoplasmReceptor complexCell SurvivalCASP8 and FADD-Like Apoptosis Regulating ProteinApoptosisCell SeparationBiologyCaspase 8Sensitivity and SpecificityBiochemistryProinflammatory cytokineTNF-Related Apoptosis-Inducing LigandRibonucleasesCell Line TumorHumansEnzyme InhibitorsMolecular BiologyTranscription factorSkinInflammationCaspase 8Membrane GlycoproteinsTumor Necrosis Factor-alphaIntracellular Signaling Peptides and ProteinsNF-kappa BCell BiologyFlow CytometryRecombinant ProteinsCell biologyRetroviridaeApoptosisCaspasesDeath-inducing signaling complexRNATumor necrosis factor alphaSignal transductionApoptosis Regulatory ProteinsPropidiumProtein BindingSignal TransductionJournal of Biological Chemistry
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Ķermeņa masas indeksa ietekme uz paplašinātās limfadenektomijas kvalitāti kunģa vēža pacientiem

2017

Aktualitāte. Saslimstība ar kuņģa vēzi katru gadu pieaug. Arvien vairāk kuņģa vēzis tiek diagnosticēts novēlotās stadijās, kad pacienta dzīvildze un dzīves kvalitāte ir ievērojami samazināta. Mūsdienās pieaug arī aptaukošanās risks, kas ir viens no riska faktoriem kuņģa vēža attīstībai. Liela daļa pacienti pie ārsta ierodas jau ieilgušā stadija ar ievērojamu aptaukošanos, kas apgrūtina ķirurģisko ārstēšanu. Veicot radikālu operāciju, papildus noteikti ir jāveic limfadenektomija, lai uzlabotu ārstēšanas rezultātus un nepieļautu metastāžu veidošanos. Darba mērķis: izpētīt kā ķermeņa masas indekss ietekmē paplašinātās limfadenektomijas kvalitāti ( izdalīto limfmezglu skaits), pēcoperāciju komp…

LimfadenektomijaPalielinātsĶermeņa masas indekssMedicīnaKuņģa vēzis
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El aprendizaje de los rasgos fonopragmáticos del enfado en español como lengua extranjera

2020

espanolIdentificar la expresion de enfado en una lengua extranjera y reproducirla con adecuacion y precision son destrezas cuya relevancia y falta de propuestas satisfactorias han sido senaladas por las investigaciones sobre la ensenanza de emociones (Dewaele, 2010; Toya & Kodis, 1996). Este trabajo combina la identificacion de carencias en la didactica de la entonacion emocional con el analisis de muestras espontaneas de enfado como base de unas pautas didacticas para introducir la ensenanza contextualizada de la expresion de enfado en espanol. A traves de muestras de un corpus coloquial, se identifican los rasgos caracteristicos de la expresion de enfado en espanol. Una seleccion de rasgo…

Linguistics and LanguageLiterature and Literary TheoryemocionesfonopragmáticaPhilosophylcsh:Philosophy (General)Key featuresLanguage and LinguisticsPhilosophyprosodiaELEAnger expressionenfadoAnger inlcsh:B1-5802HumanitiesLogos: Revista de Lingüística, Literatura y Filosofía
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A Multi-Phase Decode-and-Forward Transmission Protocol in Cognitive Relay Networks: Outage Analysis and Relay Power Allocation

2015

Consider an underlay cognitive relay network with multiple source and destination pairs and that a decode-and-forward scheme is adopted at the relay. We propose a multi-phase transmission protocol in this paper which involves cooperation among source, relay and destination nodes. Given perfect interference elimination before decoding at the destination nodes, we derive closed-form expressions for outage probability calculation over Rayleigh fading channels. Furthermore, the optimal relay power allocation factor which leads to minimal outage probability is determined. Numerical results reveal the conditions under which satisfactory performance can be achieved based on the proposed scheme.

Link Access Procedure for Frame Relaybusiness.industryComputer scienceComputerSystemsOrganization_COMPUTER-COMMUNICATIONNETWORKSData_CODINGANDINFORMATIONTHEORYlaw.inventionPower (physics)RelaylawComputer Science::Networking and Internet ArchitectureUnderlayTransmission protocolbusinessDecoding methodsRelay channelComputer Science::Information TheoryCommunication channelRayleigh fadingComputer network2015 IEEE 81st Vehicular Technology Conference (VTC Spring)
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Ablation of c-FLIP in hepatocytes enhances death-receptor mediated apoptosis and toxic liver injury in vivo

2010

Background & Aims Apoptosis is crucially involved in acute and chronic liver injury, including viral, cholestatic, toxic, and metabolic liver disease. Additionally, dysregulation of apoptosis signaling pathways has been implicated in hepatocarcinogenesis. The most prominent members of the apoptosis-mediating tumor necrosis factor receptor superfamily are the TNF-R1 (CD120a) and the CD95 (Apo-1/Fas) receptor. Although extensively studied, the intracellular signaling events in hepatocytes are only incompletely understood. Methods To examine the role of the caspase-8 homolog cellular FLICE-inhibitory protein (c-FLIP) in liver injury, we generated mice with hepatocyte specific deletion of c-FLI…

LipopolysaccharidesProgrammed cell deathMAP Kinase Signaling SystemCASP8 and FADD-Like Apoptosis Regulating ProteinApoptosisGalactosamineBiologyCaspase 8MiceLiver diseaseConcanavalin AmedicineAnimalsfas ReceptorAnthracenesMice KnockoutLiver injuryHepatologyReceptors Death DomainFas receptormedicine.diseasemedicine.anatomical_structureApoptosisCaspasesHepatocyteDeath-inducing signaling complexHepatocytesCancer researchFemaleChemical and Drug Induced Liver InjuryJournal of Hepatology
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Dominant negative MORT1/FADD rescues mice from CD95 and TNF-induced liver failure

2002

Derangement of the apoptotic program is considered an important cause of liver disease. It became clear that receptor-mediated apoptosis is of specific interest in this context, and CD95 and CD120a, both members of the tumor necrosis factor (TNF) receptor superfamily, are the most prominent cell death receptors involved. The death signal is induced upon ligand binding by recruitment of caspases via the adapter molecule MORT1/FADD to the receptor and their subsequent activation. To investigate the role of MORT1/FADD in hepatocyte apoptosis, we generated transgenic mice expressing liver-specific dominant negative mutant. Mice looked grossly normal; breeding and liver development were not diff…

Lipopolysaccharidesmedicine.medical_specialtyProgrammed cell deathFas-Associated Death Domain ProteinOligonucleotidesMice TransgenicAntibodiesReceptors Tumor Necrosis FactorMiceLiver diseaseAntigens CDAlbuminsInternal medicinemedicineAnimalsfas ReceptorFADDPromoter Regions GeneticAdaptor Proteins Signal TransducingLiver injuryHepatitisMice Inbred BALB CHepatologybiologyTumor Necrosis Factor-alphamedicine.diseaseFas receptorMice Inbred C57BLEndocrinologyReceptors Tumor Necrosis Factor Type IApoptosisCaspasesbiology.proteinTumor necrosis factor alphaCarrier ProteinsLiver FailureHepatology
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Increased hepatic fibrosis and JNK2-dependent liver injury in mice exhibiting hepatocyte-specific deletion of cFLIP.

2012

Chronic liver disease promotes hepatocellular injury involving apoptosis and triggers compensatory regeneration that leads to the activation of quiescent stellate cells in the liver. The deposition of extracellular matrix from activated myofibroblasts promotes hepatic fibrosis and the progression to cirrhosis with deleterious effects on liver physiology. The role of apoptosis signaling pathways in the development of fibrosis remains undefined. The aim of the current study was to determine the involvement of the caspase-8 homologue cellular FLICE-inhibitory protein (cFLIP) during the initiation and progression of fibrosis. Liver injury and fibrosis from carbon tetrachloride (CCl4) and thioa…

Liver CirrhosisMalePathologymedicine.medical_specialtyTime FactorsGenotypePhysiologyCASP8 and FADD-Like Apoptosis Regulating ProteinApoptosisBiologyThioacetamideChronic liver diseaseMicePhysiology (medical)medicineAnimalsMitogen-Activated Protein Kinase 9PhosphorylationExtracellular Signal-Regulated MAP KinasesCarbon TetrachlorideCompensatory regenerationLiver injuryMice KnockoutHepatologyCaspase 3Gastroenterologymedicine.diseaseCaspase 9Enzyme ActivationDisease Models Animalmedicine.anatomical_structurePhenotypeLiverApoptosisHepatocyteHepatic stellate cellCancer researchDisease ProgressionHepatocytesHepatocellular injuryChemical and Drug Induced Liver InjuryHepatic fibrosisSignal TransductionAmerican journal of physiology. Gastrointestinal and liver physiology
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