Search results for "FATE"

showing 10 items of 765 documents

Comparison of the metabolism of 7-ethoxycoumarin and coumarin in precision-cut rat liver and lung slices

1995

The metabolism of 7-ethoxycoumarin and [3-(14)C]coumarin was compared in precision-cut rat liver and lung slices. The lung slices were prepared using an agarose gel instilling technique enabling the production of tissue cylinders followed by lung slices employing a Krumdieck tissue slicer. Both 50 microM 7-ethoxycoumarin and 50 microM [3-(14)C]coumarin were metabolized by rat liver and lung slices. 7-Ethoxycoumarin was converted to 7-hydroxycoumarin (7-HC) which was conjugated with both D-glucuronic acid and sulfate. 7-HC sulfate was the major metabolite formed by both liver and lung slices. [3-(14)C]Coumarin was metabolized by rat liver and lung slices to both polar products and to metabol…

MaleMetaboliteGlucuronatesBiologyToxicologyRats Sprague-Dawleychemistry.chemical_compoundGlucuronic AcidCoumarinsCulture TechniquesmedicineAnimalsUmbelliferonesLungLungSulfatesCytochrome P450General MedicineMetabolismCoumarinGlucuronic acidRatsmedicine.anatomical_structureLiverchemistryBiochemistryIsotope Labelingbiology.proteinAgaroseDrug metabolismFood ScienceFood and Chemical Toxicology
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Stage-specific germ-cell marker genes are expressed in all mouse pluripotent cell types and emerge early during induced pluripotency.

2011

Embryonic stem cells (ESCs) generated from the in-vitro culture of blastocyst stage embryos are known as equivalent to blastocyst inner cell mass (ICM) in-vivo. Though several reports have shown the expression of germ cell/pre-meiotic (GC/PrM) markers in ESCs, their functional relevance for the pluripotency and germ line commitment are largely unknown. In the present study, we used mouse as a model system and systematically analyzed the RNA and protein expression of GC/PrM markers in ESCs and found them to be comparable to the expression of cultured pluripotent cells originated from the germ line. Further, siRNA knockdown experiments have demonstrated the parallel maintenance and independen…

MaleMouselcsh:MedicineGene ExpressionEmbryoid bodyCell Fate DeterminationMice0302 clinical medicineMolecular Cell BiologyNuclear Reprogramminglcsh:ScienceInduced pluripotent stem cellPromoter Regions Genetic0303 health sciencesMultidisciplinaryStem CellsGene Expression Regulation DevelopmentalAnimal ModelsCellular ReprogrammingChromatinChromatinMeiosismedicine.anatomical_structureBlastocyst Inner Cell Massembryonic structuresEpigeneticsBiological MarkersFemaleGerm cellResearch ArticleBivalent chromatinInduced Pluripotent Stem CellsBiologyCell Line03 medical and health sciencesModel OrganismsGeneticsmedicineAnimalsRNA MessengerGene NetworksEmbryonic stem cells (ESCs); germ layer cell typesBiology030304 developmental biologylcsh:RMolecular DevelopmentMolecular biologyEmbryonic stem cellGerm Cellslcsh:QGene FunctionChromatin immunoprecipitationBiomarkers030217 neurology & neurosurgeryDevelopmental Biology
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ZBTB20 is crucial for the specification of a subset of callosal projection neurons and astrocytes in the mammalian neocortex

2021

ABSTRACT Neocortical progenitor cells generate subtypes of excitatory projection neurons in sequential order followed by the generation of astrocytes. The transcription factor zinc finger and BTB domain-containing protein 20 (ZBTB20) has been implicated in regulation of cell specification during neocortical development. Here, we show that ZBTB20 instructs the generation of a subset of callosal projections neurons in cortical layers II/III in mouse. Conditional deletion of Zbtb20 in cortical progenitors, and to a lesser degree in differentiating neurons, leads to an increase in the number of layer IV neurons at the expense of layer II/III neurons. Astrogliogenesis is also affected in the mut…

MaleNeurogenesisCèl·lulesCellMutation MissenseNeocortexNeuronesCell fate determinationBiologyGene Knockout TechniquesMiceIntellectual DisabilitymedicineAnimalsAbnormalities MultipleProgenitor cellEar DiseasesMolecular BiologyTranscription factorMice KnockoutNeuronsZinc fingerNeocortexStem CellsCalcinosisCell biologyMice Inbred C57BLMuscular Atrophymedicine.anatomical_structurenervous systemAstrocytesExcitatory postsynaptic potentialFemaleSignal TransductionTranscription FactorsResearch ArticleDevelopmental BiologyAstrocyte
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Intussusceptive Remodeling of Vascular Branch Angles in Chemically-Induced Murine Colitis

2013

Intussusceptive angiogenesis is a developmental process linked to both blood vessel replication and remodeling in development. To investigate the prediction that the process of intussusceptive angiogenesis is associated with vessel angle remodeling in adult mice, we systematically evaluated corrosion casts of the mucosal plexus in mice with trinitrobenzesulphonic acid (TNBS)-induced and dextran sodium sulfate (DSS)-induced colitis. The mice demonstrated a significant decrease in vessel angles in both TNBS-induced and DSS-induced colitis within 4 weeks of the onset of colitis (p<.001). Corrosion casts 28–30 days after DSS treatment were studied for a variety of detailed morphometric changes.…

MalePathologymedicine.medical_specialtyColonCorrosion CastingBiochemistryArticleDescending colonNeovascularizationMicemedicineAscending colonAnimalsColitisIntestinal MucosaIntussusceptive angiogenesisPlexusMice Inbred BALB CNeovascularization PathologicChemistryDextran SulfateCell BiologyAnatomyBlood flowmedicine.diseaseColitisDisease Models Animalmedicine.anatomical_structureTrinitrobenzenesulfonic AcidMicroscopy Electron ScanningBlood Vesselsmedicine.symptomCardiology and Cardiovascular MedicineBlood vessel
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Protection by Almagate of Ethanol-induced Gastric Mucosal Damage in Rats

1995

Abstract The study was designed to analyse the protective effects of almagate on a model of gastric injury, ethanol-induced mucosal damage, in which acid plays little, if any, role. Pretreatment with almagate dose-dependently reduced the level of gastric damage induced by oral administration of 1mL 100% ethanol. Administration of 12 μmol kg−1 almagate 30 min before ethanol significantly reduced the area of mucosal damage by 65 ± 10%, and the maximum level of inhibition (74 ± 11%) was obtained with 150 μmol kg−1 almagate. Administration of higher doses of almagate (200–250 μmol kg−1) did not result in any further increase in the level of protection against ethanol-induced gastric damage. Adm…

MalePathologymedicine.medical_specialtyMagnesium HydroxideSucralfateIndomethacinCarbonatesAdministration OralPharmaceutical ScienceAluminum HydroxidePharmacologychemistry.chemical_compoundOral administrationGastric mucosamedicineAnimalsStomach UlcerRats WistarPharmacologyDiminutionAlmagateDose-Response Relationship DrugEthanolbusiness.industryStomachRatsDisease Models AnimalSucralfateDose–response relationshipmedicine.anatomical_structurechemistryGastric MucosaToxicityFemaleAntacidsbusinessmedicine.drugJournal of Pharmacy and Pharmacology
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The effects of three absorption-modifying critical excipients on the in vivo intestinal absorption of six model compounds in rats and dogs.

2018

Pharmaceutical excipients that may affect gastrointestinal (GI) drug absorption are called critical pharmaceutical excipients, or absorption-modifying excipients (AMEs) if they act by altering the integrity of the intestinal epithelial cell membrane. Some of these excipients increase intestinal permeability, and subsequently the absorption and bioavailability of the drug. This could have implications for both the assessment of bioequivalence and the efficacy of the absorption-enhancing drug delivery system. The absorption-enhancing effects of AMEs with different mechanisms (chitosan, sodium caprate, sodium dodecyl sulfate (SDS)) have previously been evaluated in the rat single-pass intestin…

MalePharmaceutical ScienceExcipientBiological Availability02 engineering and technologyBioequivalencePharmacology030226 pharmacology & pharmacyIntestinal absorptionPermeabilityExcipients03 medical and health sciences0302 clinical medicineDogsIn vivomedicineAnimalsPharmaceutical sciencesIntestinal MucosaChitosanIntestinal permeabilityChemistrySodium Dodecyl Sulfate021001 nanoscience & nanotechnologymedicine.diseaseBioavailabilityRatsIntestinesIntestinal AbsorptionPharmaceutical PreparationsDrug delivery0210 nano-technologyDecanoic Acidsmedicine.drugInternational journal of pharmaceutics
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Different mechanisms of the inhibition of the transient outward current in rat ventricular myocytes.

1994

The mechanism of drug-induced inhibition of the transient outward current, Ito, has been investigated in rat ventricular myocytes using the whole cell patch clamp technique. Ito was activated by 300 ms depolarizing voltage clamp steps in 10 mV increments from −50 mV up to +40 mV. At +40 mV, Ito peaked after about 3 ms, and the time course of inactivation was appropriately described by two time constants, τfast = 17 ms and τslow = 203 ms. Verapamil, quinidine sulfate and nifedipine preferentially depressed Ito at the end of the 300 ms depolarizing voltage clamp step; the inactivation of Ito was accelerated by all drugs, whereas peak Ito was less affected. The time course of drug action at +4…

MalePotassium ChannelsVoltage clampHeart VentriclesPharmacologydigestive systemMembrane PotentialsRats Sprague-Dawleychemistry.chemical_compoundQuinidine SulfateNifedipinemedicineAnimalsVentricular FunctionPatch clampCells CulturedPharmacologyMembrane potentialCardiac transient outward potassium currentMyocardiumHeartGeneral MedicineTetraethylammonium chlorideRatsElectrophysiologychemistryBiophysicsVerapamilmedicine.drugNaunyn-Schmiedeberg's archives of pharmacology
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Neurosteroid modulation of the presynaptic NMDA receptors regulating hippocampal noradrenaline release in normal rats and those exposed prenatally to…

2003

Abstract Prenatal exposure to diazepam (DZ), a positive allosteric modulator of the γ-aminobutyric acidA (GABAA) receptor complex, exerts profound effects that become more evident during puberty and in many cases are sex-specific, suggesting that such exposure interferes with the activity of steroid hormones. Apart from their well known effects on the genome, the reduced metabolites of many steroid hormones also interact directly with membrane receptors, including those for N-methyl- d -aspartate (NMDA). In this study, we compared the effects of several neurosteroids on NMDA receptors from normal rats and those exposed in utero to DZ (1.25 mg/kg per day) from the 14th through the 20th day o…

MalePregnenolone sulfatemedicine.medical_specialtyReceptor complexNeuroactive steroidAllosteric modulatorGlycinePharmacologyHippocampusReceptors N-Methyl-D-AspartateNorepinephrineCellular and Molecular Neurosciencechemistry.chemical_compoundPregnancyInternal medicineNeurosteroidmedicinepregnenolone sulphateAnimalsRats WistarReceptorDiazepamGABAA receptorHippocampal synaptosomesCell BiologyRatsEndocrinologyNMDA/GLY-mediated [3H]NA releasechemistryPregnenolonePrenatal Exposure Delayed EffectsSettore BIO/14 - FarmacologiaNMDA receptorFemalePregnenolone sulfateSynaptosomesHormoneNeurochemistry International
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Perlecan controls neurogenesis in the developing telencephalon.

2006

This article is available from: http://www.biomedcentral.com/1471-213X/7/29

MaleTelencephalonLaminaOrganogenesisApoptosisCell CountNeocortexPerlecanExencephalyBiologyBasement MembraneMiceFetal Organ MaturityInterneuronsPregnancymedicineAnimalsHedgehog Proteinslcsh:QH301-705.5Embryonic Stem CellsCell ProliferationBasement membraneNeuronsCerebrumNeurogenesisAnatomymedicine.diseaseImmunohistochemistryCell biologyNeuroepithelial cellmedicine.anatomical_structurelcsh:Biology (General)embryonic structuresbiology.proteinMicrocephalyBasal laminaFemaleFibroblast Growth Factor 2Heparan Sulfate ProteoglycansDevelopmental BiologyResearch ArticleBMC developmental biology
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Mutations in the PDS Gene in German Families with Pendred’s Syndrome: V138F Is a Founder Mutation

2003

Pendred's syndrome, an autosomal-recessive condition characterized by congenital sensorineural hearing loss and goiter, is caused by mutations in the PDS gene. Located on chromosome 7q22-q31, it encodes a chloride-iodide transporter expressed in the thyroid, inner ear, and kidney. We investigated the PDS gene of six affected individuals from four unrelated families with Pendred's syndrome by direct sequencing. PDS mutations were identified in homozygous or compound heterozygous state in all six cases. A homozygous missense mutation leading to the amino acid substitution S133T was detected in a family of Turkish origin. The mutations found in the other affected individuals, who originate fro…

MaleThreoninemedicine.medical_specialtyAdolescentTurkeyHearing Loss SensorineuralEndocrinology Diabetes and MetabolismClinical BiochemistryMutation MissenseBiologyCompound heterozygositymedicine.disease_causeBiochemistryGenetic determinismEndocrinologyHypothyroidismGermanyInternal medicineSerinemedicineHumansMissense mutationAlleleChildPendred syndromeGeneticsMutationBase SequenceBiochemistry (medical)HaplotypeInfant NewbornMembrane Transport Proteinsfood and beveragesSyndromemedicine.diseaseFounder EffectPedigreeEndocrinologyAmino Acid SubstitutionHaplotypesSulfate TransportersChild PreschoolMicrosatelliteFemaleCarrier ProteinsThe Journal of Clinical Endocrinology &amp; Metabolism
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