Search results for "FATE"

showing 10 items of 765 documents

PCF11 links alternative polyadenylation to formation and spontaneous regression of neuroblastoma

2018

AbstractDiversification at the transcriptome 3’end is an important and evolutionarily conserved layer of gene regulation associated with differentiation and dedifferentiation processes. However the underlying mechanisms and functional consequences are poorly defined. Here, we identify extensive transcriptome-3’end-alterations in neuroblastoma, a tumour entity with a paucity of recurrent somatic mutations and an unusually high frequency of spontaneous regression. Utilising extensive RNAi-screening we reveal the landscape and drivers of transcriptome-3’end-diversification, discovering PCF11 as critical regulator, directing alternative polyadenylation (APA) of hundreds of transcripts including…

TranscriptomeRegulation of gene expressionPolyadenylationMechanism (biology)RegulatorComputational biologyBiologyCell fate determinationCell cycleReprogramming
researchProduct

CCDC 819694: Experimental Crystal Structure Determination

2012

Related Article: G.J.Merten, C.Neis, S.Stucky, V.Huch, E.Rentschler, H.Natter, R.Hempelmann, K.Stowe, K.Hegetschweiler|2012|Eur.J.Inorg.Chem.||31|doi:10.1002/ejic.201100914

Triaqua-(1-hydroxycyclohexan-246-triamine)-nickel(ii) sulfate dihydrateSpace GroupCrystallographyCrystal SystemCrystal StructureCell ParametersExperimental 3D Coordinates
researchProduct

Contribution of the gas-phase reaction between hydroxyl radical and sulfur dioxide to the sulfate aerosol over West Pacific

2021

Sulfate is among the major components of atmospheric aerosols or fine particulate matters. Aerosols loaded with sulfate result in low air quality, damage to ecosystems, and influences on climate change. Sulfate aerosols could originate from that directly emitted to the atmosphere and that produced by atmospheric physicochemical processes. The latter is generated from sulfur dioxide (SO2) via oxidation either in the gas phase reactions or in the aqueous phase. Several mechanisms of SO2 oxidation have been proposed, but the differentiation of the various mechanisms and identification of the sources remain challenging. To meet this need, a new method to estimate the contribution of the gas-pha…

TroposphereAtmospherechemistry.chemical_compoundchemistryEnvironmental chemistryRadicalSulfate aerosolHydroxyl radicalSulfatecomplex mixturesSulfur dioxideTrace gas
researchProduct

The dynamics of tropospheric aerosols

1966

After a brief review of the present knowledge of the Stratospheric sulfate layer, several possible mechanisms of formation are discussed in detail. A direct transport of low tropospheric particles into the stratosphere by convective clouds penetrating the tropopause is not very likely because these penetrations are not high enough and because of the chemical composition of the stratospheric particles. DOI: 10.1111/j.2153-3490.1966.tb00287.x

TroposphereConvectionchemistry.chemical_compoundchemistryClimatologyEnvironmental scienceGeneral MedicineTropopauseSulfateAtmospheric sciencesChemical compositionStratosphereTellus
researchProduct

The Terminal Complement Complex (TCC) in the Plasma of Patients with Type 1 Diabetes Mellitus. Relationship with Albumin Excretion Rate

1991

Increased TCC levels were found to be present in the plasma of patients with type 1 diabetes associated with the presence of anti-heparan sulfate antibodies and increased urinary excretion of albumin. The authors suggest a role of terminal complement activation in the pathogenesis of diabetic nephropathy.

Type 1 diabetesmedicine.medical_specialtybiologybusiness.industryAlbuminHeparan sulfatemedicine.diseaseComplement systemPathogenesisDiabetic nephropathychemistry.chemical_compoundEndocrinologychemistryTerminal complement complexInternal medicinemedicinebiology.proteinAntibodybusiness
researchProduct

Sulfate-Templated 2D Anion-Layered Supramolecular Self-Assemblies

2019

Summary Using solution and solid-state analyses, we demonstrate that the tripodal N-methylated(1,3,5-benzene-tricarboxamide)-tris(phenylurea) BTA ligands, possessing urea functionalities in the meta position, are able to form extended self-assembly 2D networks via hydrogen bonding templated by sulfate (SO42–). The divergence of the urea binding sites confers a propeller-like conformation to the ligands and is key to formation of the self-assemblies. Studies in solution and in the solid state as well as scanning electron microscopy (SEM) on the self-assembly properties of the ligands showed that the convergence also leads to the formation of hierarchical structures, including porous films an…

Urea bindingScanning electron microscopeChemistryHydrogen bondGeneral Chemical EngineeringBiochemistry (medical)Supramolecular chemistryGeneral ChemistryBiochemistrySolventMeta-Crystallographychemistry.chemical_compoundMaterials ChemistryEnvironmental ChemistrySelf-assemblySulfateChem
researchProduct

The pea sulfate transporter, PsSULTR4, contributes to seed yield and quality

2023

To investigate the role of vacuolar sulfate in seed yield and quality, we have targeted the single pea SULTR4 gene (PsSULTR4), which encodes a transporter homologous to Arabidopsis SULTR4;1 and 4;2 that allow sulfate efflux from the vacuole to the cytosol. By simulating the 3D structure of PsSULTR4, we observed that it is similar to that of SULTR4;1 in Arabidopsis. Furthermore, a phylogenetic analysis revealed a high level of conservation of SULTR4 protein motifs across land species. A fluorescent protein fusion experiment confirmed that PsSULTR4 localizes to the vacuolar membrane.Five sultr4 mutants were identified by TILLING (Targeting Induced Local Lesions IN Genomes), two of which showe…

Vacuolar sulfateSeed yieldSulfate transporter SULTR4[SDV.BBM] Life Sciences [q-bio]/Biochemistry Molecular BiologySeed qualitySulfur amino acidsSulfur deficiencyPisum sativumStorage proteins
researchProduct

VEGF receptor signaling links inflammation and tumorigenesis in colitis-associated cancer.

2010

Inflammation drives expression of VEGFR2, which is expressed on and drives growth of tumor cells in colitis-associated cancer.

Vascular Endothelial Growth Factor AColorectal cancerGene Expressionmedicine.disease_causechemistry.chemical_compoundMice0302 clinical medicineImmunology and AllergyDecoy receptorsCells CulturedMice Knockout0303 health sciencesMicroscopy ConfocalReverse Transcriptase Polymerase Chain ReactionDextran Sulfaterespiratory systemColitisImmunohistochemistry3. Good healthUp-RegulationVascular endothelial growth factorVascular endothelial growth factor A030220 oncology & carcinogenesisColonic Neoplasmscardiovascular systemcirculatory and respiratory physiologySignal TransductionSTAT3 Transcription FactorImmunologyBlotting WesternMice TransgenicBiologyArticle03 medical and health sciencesmedicineAnimalsHumans030304 developmental biologyCell ProliferationVascular Endothelial Growth Factor Receptor-1CancerEndothelial CellsKinase insert domain receptorEpithelial CellsCell Biologymedicine.diseaseInflammatory Bowel DiseasesVascular Endothelial Growth Factor Receptor-2Mice Inbred C57BLHIF1AchemistryCancer researchCarcinogenesis030215 immunologyThe Journal of experimental medicine
researchProduct

Integrin-dependent and -independent functions of astrocytic fibronectin in retinal angiogenesis

2011

Fibronectin (FN) is a major component of the extracellular matrix and functions in cell adhesion, cell spreading and cell migration. In the retina, FN is transiently expressed and assembled on astrocytes (ACs), which guide sprouting tip cells and deposit a provisional matrix for sprouting angiogenesis. The precise function of FN in retinal angiogenesis is largely unknown. Using genetic tools, we show that astrocytes are the major source of cellular FN during angiogenesis in the mouse retina. Deletion of astrocytic FN reduces radial endothelial migration during vascular plexus formation in a gene dose-dependent manner. This effect correlates with reduced VEGF receptor 2 and PI3K/AKT signalli…

Vascular Endothelial Growth Factor AIntegrinsAngiogenesisIntegrinNeovascularization PhysiologicMice TransgenicExtracellular matrixMicePhosphatidylinositol 3-KinasesCell MovementAnimalsProtein Interaction Domains and MotifsMolecular BiologyResearch ArticlesMice KnockoutSprouting angiogenesisbiologyRetinal VesselsCell migrationKinase insert domain receptorVascular Endothelial Growth Factor Receptor-2Extracellular MatrixFibronectinsCell biologyMice Inbred C57BLFibronectinVascular endothelial growth factor AAstrocytesbiology.proteinHeparitin SulfateOligopeptidesProto-Oncogene Proteins c-aktIntegrin alpha5beta1Signal TransductionDevelopmental BiologyDevelopment
researchProduct

Microphthalmia, persistent hyperplastic hyaloid vasculature and lens anomalies following overexpression of VEGF-A188 from the αA-crystallin promoter

2007

Purpose During growth of the embryonic eye, dose- and site-specific expression of heparin-binding growth factors is critical for the formation of an appropriate vascular supply. Overexpression of vascular endothelial growth factor-A188 (VEGF-A188), a strongly heparin-binding, endothelial-specific mitogen, leads to severe disturbance of vascular and overall ocular morphology. This study aimed to evaluate the effects of VEGF-A188 overexpression on growth of ocular tissue components. Methods Stereological and immunohistochemical methods were employed to identify the vascular profiles, ocular tissue proportions, and cell types in VEGF-A188 transgenic mice and compare them with wild-type mice. R…

Vascular Endothelial Growth Factor Agenetic structuresMyocytes Smooth MuscleCell CountMice TransgenicEyealpha-Crystallin A ChainCongenital AbnormalitiesCorneaMiceLens CrystallineAnimalsMicrophthalmosVascular DiseasesPromoter Regions GeneticHyperplasiaEndothelial CellsHypertrophyEmbryo MammalianAntigens DifferentiationImmunohistochemistryeye diseasesActinsDisease Models AnimalAnimals NewbornBlood Vesselssense organsPericytesHeparan Sulfate ProteoglycansResearch ArticleMolecular Vision
researchProduct