Search results for "FEM"

showing 10 items of 28965 documents

Autonomic nervous control of the urinary bladder

2013

The autonomic nervous system plays an important role in the regulation of the urinary bladder function. Under physiological circumstances, noradrenaline, acting mainly on β(3) -adrenoceptors in the detrusor and on α(1) (A) -adrenoceptors in the bladder outflow tract, promotes urine storage, whereas neuronally released acetylcholine acting mainly on M(3) receptors promotes bladder emptying. Under pathophysiological conditions, however, this system may change in several ways. Firstly, there may be plasticity at the levels of innervation and receptor expression and function. Secondly, non-neuronal acetylcholine synthesis and release from the urothelium may occur during the storage phase, leadi…

medicine.medical_specialtyPhysiologyReceptor expressionUrinary Bladder030232 urology & nephrologyAdrenergicBiologyurologic and male genital diseasesAutonomic Nervous System03 medical and health sciences0302 clinical medicineInternal medicineMuscarinic acetylcholine receptorReceptors Adrenergic betamedicineAnimalsHumansUrotheliumUrinary bladderNeuronal PlasticityUrinary Bladder DiseasesMuscarinic acetylcholine receptor M3Receptors Muscarinicfemale genital diseases and pregnancy complicationsAutonomic nervous systemmedicine.anatomical_structureEndocrinology030217 neurology & neurosurgeryAcetylcholinemedicine.drugActa Physiologica
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The effect of hormone replacement therapy and/or exercise on skeletal muscle attenuation in postmenopausal women: a yearlong intervention

2005

Hormone replacement therapy (HRT) has been reported to exert a positive effect on preserving muscle strength following the menopause, however, the mechanism of action remains unclear. We examined whether the mechanism involved preservation of muscle composition as determined by skeletal muscle attenuation. Eighty women aged 50-57 years were randomly assigned to either: HRT, exercise (Ex), HRT + exercise (ExHRT), and control (Co) for 1 year. The study was double-blinded with subjects receiving oestradiol and norethisterone acetate (Kliogest) or placebo. Exercise included progressive high-impact training for the lower limbs. Skeletal muscle attenuation in Hounsfield units (HU) was determined …

medicine.medical_specialtyPhysiologymedia_common.quotation_subjectAdipose tissuePhysical exerciseVertical jumpDouble-Blind MethodPhysiology (medical)Internal medicinemedicineHormone replacement therapy (male-to-female)Body SizeHumansMuscle SkeletalExerciseMenstrual cyclemedia_commonAnalysis of Variancebusiness.industryBody WeightEstrogen Replacement TherapySkeletal muscleGeneral MedicineMiddle Agedmedicine.diseaseNorethisterone acetatePostmenopauseMenopauseEndocrinologymedicine.anatomical_structureAdipose TissueFemaleTomography X-Ray Computedbusinessmedicine.drugClinical Physiology and Functional Imaging
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Combination of hormone replacement therapy and high physical activity is associated with differences in Achilles tendon size in monozygotic female tw…

2009

Estrogen concentration has been suggested to play a role in tendon abnormalities and injury. In physically active postmenopausal women, hormone replacement therapy (HRT) has been suggested to decrease tendon diameter. We hypothesized that HRT use and physical activity are associated with Achilles tendon size and tissue structure. The study applied cotwin analysis of fourteen 54- to 62-yr-old identical female twin pairs with current discordance for HRT use for an average of 7 yr. Achilles tendon thickness and cross-sectional areas were determined by ultrasonography, and tendon structural organization was analyzed from the images using linear discriminant analysis (LDA). Maximal voluntary and…

medicine.medical_specialtyPhysiologymedicine.drug_classEstroneHormone Replacement TherapyAnterior cruciate ligamentPhysical activityMonozygotic twinPhysical exerciseMotor ActivityAchilles Tendon03 medical and health sciences0302 clinical medicinePhysiology (medical)Internal medicineSex Hormone-Binding GlobulinMedicineHumansTestosteroneMuscle StrengthAgedUltrasonographyAchilles tendonbusiness.industryEstrogens030229 sport sciencesTwins MonozygoticMiddle AgedTendonmedicine.anatomical_structureEndocrinologyCholesterolTransgender hormone therapyEstrogenFemaleMenopausebusiness030217 neurology & neurosurgeryMuscle ContractionJournal of applied physiology (Bethesda, Md. : 1985)
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Early, but not late onset estrogen replacement therapy prevents oxidative stress and metabolic alterations caused by ovariectomy.

2014

Aims: The usefulness of estrogen replacement therapy (ERT) in preventing oxidative stress associated with menopause is controversial. We aimed to study if there is a critical time window for effective treatment of the effects of ovariectomy with estrogens at the molecular, metabolic, and cellular level. Results: Our main finding is that early, but not late onset of ERT prevents an ovariectomy-associated increase in mitochondrial hydrogen peroxide levels, oxidative damage to lipids and proteins, and a decrease in glutathione peroxidase and catalase activity in rats. This may be due to a change in the estrogen receptor (ER) expression profile: ovariectomy increases the ER α/β ratio and immedi…

medicine.medical_specialtyPhysiologymedicine.drug_classGlucose uptakeOvariectomyClinical BiochemistryGlucose Transport Proteins FacilitativeEstrogen receptorMitochondria LiverBiologymedicine.disease_causeBiochemistryAntioxidantsInternal medicinemedicineAnimalsMetabolomicsMolecular BiologyGeneral Environmental Sciencechemistry.chemical_classificationEstradiolGlutathione peroxidaseEstrogen Replacement TherapyGlucose transporterBrainCell BiologyHydrogen Peroxidemedicine.diseaseRatsMenopauseOxidative StressOriginal Research CommunicationsEndocrinologyGlucosechemistryEstrogenCatalasebiology.proteinGeneral Earth and Planetary SciencesFemaleOxidative stressAntioxidantsredox signaling
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Renal protection by antihypertensive drugs

1998

During the last few years there has been a renewed interest in blood-pressure (BP)-induced kidney damage, owing to a progressive increase in the incidence and prevalence of hypertension and vascular diseases as a cause of end-stage renal disease (ESRD). The need to prevent ESRD demands continued efforts so as to identify early those people with hypertension who are at risk and to provide them with effective antihypertensive therapy. This review analyses what is needed in terms of surrogate endpoints for monitoring kidney damage and what is known about the impact of antihypertensive treatments in reducing the BP burden on the kidney in non-diabetic subjects. Although glomerular filtration ra…

medicine.medical_specialtyPhysiologymedicine.drug_classRenal functionBlood PressureKidneyurologic and male genital diseasesEssential hypertensionNephropathyRisk FactorsInternal medicineInternal MedicinemedicineAlbuminuriaHumansAntihypertensive drugAntihypertensive AgentsKidneyProteinuriabusiness.industrySurrogate endpointmedicine.diseasefemale genital diseases and pregnancy complicationsEndocrinologymedicine.anatomical_structureHypertensionCardiologyKidney Failure ChronicMicroalbuminuriamedicine.symptomCardiology and Cardiovascular MedicinebusinessJournal of Hypertension
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Release of non-neuronal acetylcholine from the human placenta: difference to neuronal acetylcholine

2001

The synthesis and release of non-neuronal acetylcholine, a widely expressed signaling molecule, were investigated in the human placenta. This tissue is free of cholinergic neurons, i.e. a contamination of neuronal acetylcholine can be excluded. The villus showed a choline acetyltransferase (ChAT) activity of 0.65 nmol/mg protein per h and contained 500 nmol acetylcholine/g dry weight. In the absence of cholinesterase inhibitors the release of acetylcholine from isolated villus pieces amounted to 1.3 nmol/g wet weight per 10 min corresponding to a fractional release rate of 0.13% per min. The following substances did not significantly modify the release of acetylcholine: oxotremorine (1 micr…

medicine.medical_specialtyPhysostigminePlacentaReceptors NicotinicCholine O-AcetyltransferaseNicotineInternal medicineOxotremorinemedicineHumansDrug InteractionsCholinergic neuronCholinesterasePharmacologybiologyChemistryColforsinGeneral MedicineCholine acetyltransferaseAcetylcholineElectric StimulationNeostigmineEndocrinologybiology.proteinFemaleCholinesterase InhibitorsAcetylcholinemedicine.drugNaunyn-Schmiedeberg's Archives of Pharmacology
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Lack of autoreceptor mediated regulation of the spontaneous dopamine turnover in the isolated neurointermediate lobe of the rat pituitary gland in vi…

1990

Isolated neurointermediate lobes of the rat pituitary gland were incubated in Krebs-HEPES solution and the spontaneous outflow of endogenous dopamine and its metabolites (DOPAC, HVA and MOPET) was determined by HPLC with electrochemical detection. The spontaneous outflow of dopamine metabolites (about 1500 fmol/10 min) largely exceeded that of dopamine (about 60 fmol/10 min). Apomorphine concentration-dependently (IC50, 205 nmol/l) reduced the spontaneous outflow of the dopamine metabolites. The effect of apomorphine developed slowly and was progressive over an observation period of 70 min. After 1 h of exposure to a maximall effective concentration of apomorphine (10 mumol/l), the outflow …

medicine.medical_specialtyPituitary gland3-Methoxy-4-hydroxyphenylethanol34-Dihydroxyphenylacetic acidApomorphineDopamineDopamine AgentsIn Vitro TechniquesBiologyReceptors Dopaminechemistry.chemical_compoundDopamineInternal medicineElectrochemistrymedicineAnimalsChromatography High Pressure LiquidPharmacologyHomovanillic acidHomovanillic AcidRats Inbred StrainsGeneral MedicineRatsApomorphineEndocrinologymedicine.anatomical_structurenervous systemchemistryDopamine receptorPituitary GlandAutoreceptor34-Dihydroxyphenylacetic AcidFemaleSulpiridemedicine.drugNaunyn-Schmiedeberg's Archives of Pharmacology
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Release of endogenous 3,4-dihydroxyphenylethylamine and its metabolites from the isolated neurointermediate lobe of the rat pituitary gland. Effects …

1986

: Isolated rat neurointermediate lobes were incubated in vitro. The release of 3,4-dihydroxyphenylethylamine (dopamine, DA), dihydroxyphenylacetic acid (DOPAC), homovanillic acid (HVA), and methoxyphenylethanol (MOPET) was determined by HPLC with electrochemical detection. Under resting conditions, the outflow of metabolites was 35–50 times that of DA. HVA accounted for 50%, DOPAC for 45%, and MOPET for 5% of the metabolites. Although an equivalent of 40–50% of the tissue DA content was released per hour as metabolites, the tissue DA content was not reduced after 110 min of incubation. The spontaneous outflow of DA and its metabolites was not affected by the DA uptake inhibitor GBR 12921 (1…

medicine.medical_specialtyPituitary gland3-Methoxy-4-hydroxyphenylethanolMonoamine Oxidase InhibitorsMonoamine oxidaseDopamineStimulationBiochemistryPiperazinesReuptakeCellular and Molecular Neurosciencechemistry.chemical_compoundPituitary Gland PosteriorDopamineInternal medicinemedicineAnimalsChromatography High Pressure LiquidChemistryCatabolismHomovanillic acidHomovanillic AcidPargylineElectric StimulationRatsKineticsmedicine.anatomical_structureEndocrinologyPargyline34-Dihydroxyphenylacetic AcidFemalemedicine.drugJournal of neurochemistry
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Isoprenaline and forskolin increase evoked vasopressin release from rat pituitary

1982

Isolated neurointermediate lobes of rat pituitaries were incubated in Krebs solution. The vasopressin release evoked by electrical stimulation (0.2 ms, 80 V, 15 Hz, 10 s trains at 10 s intervals for a total of 10 min) was completely inhibited by tetrodotoxin. Isoprenaline increased the evoked vasopressin release to a maximum of 60% (EC50 10 nM) and this effect was antagonized surmountably by propranolol. Forskolin increased the vasopressin release by 98%. These results suggest the presence within the neurohypophysis of a beta-adrenoceptor-linked adenylate cyclase facilitating vasopressin secretion.

medicine.medical_specialtyPituitary glandVasopressinVasopressinsStimulationPropranololchemistry.chemical_compoundPituitary Gland PosteriorInternal medicineIsoprenalinemedicineAnimalsPharmacologyForskolinColforsinIsoproterenolRats Inbred StrainsElectric StimulationStimulation ChemicalRatsEndocrinologymedicine.anatomical_structureVasopressin secretionchemistryPituitary GlandTetrodotoxinFemaleDiterpeneshormones hormone substitutes and hormone antagonistsmedicine.drugEuropean Journal of Pharmacology
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Pityriasis rosea Gibert triggered by SARS-CoV-2 infection: A case report.

2021

RATIONALE: Pityriasis rosea Gibert is an erythematous-papulosquamous dermatosis that frequently occurs in young adults. The etiopathogenesis of PR is still unknown, but is frequently associated with episodes of upper respiratory tract infections. It is likely that a new viral trigger of pityriasis rosea is the severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2). PATIENT CONCERNS: We present the case of a female patient in whom the diagnosis of pityriasis rosea led to the investigation and diagnosis of the SARS-CoV-2 infection. The patient presented to the Department of Dermatology for a 3 week duration of an extremely pruritic erythematous-squamous lesion, initially on the trunk …

medicine.medical_specialtyPityriasis RoseaContext (language use)Lesion03 medical and health sciencescoronavirus disease 20190302 clinical medicineAdrenal Cortex HormonesmedicineHumans030212 general & internal medicineClinical Case ReportRespiratory tract infectionsbusiness.industrySARS-CoV-24900COVID-19pityriasis rosea GibertGeneral MedicineMiddle Agedmedicine.diseaseDermatologyRashTopical medicationmedicine.anatomical_structure030220 oncology & carcinogenesisPityriasis roseaEtiologyFemalemedicine.symptombusinessRespiratory tractResearch Articlesevere acute respiratory syndrome coronavirus 2Medicine
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