Search results for "Fibril"

showing 10 items of 836 documents

A new familial congenital myopathy in children with desmin and dystrophin reacting plaques.

1995

In 5 children with a progressive congenital myopathy representing 3 different families, unusual histological, immunohistochemical and ultrastructural changes in skeletal muscle have been found. Histologically, this myopathy was characterized by the presence of fine hyaline plaques devoid of oxidative as well as ATPase enzyme activities. At the ultrastructural level plaques were composed of helical filaments and amorphous dense material. Helical filament storage corresponded to strong desmin as well as ubiquitin immunoreactivity. In addition they were also dystrophin positive. The exclusive appearance of desmin, ubiquitin and dystrophin positive plaques in muscle specimens from 5 children em…

MalePathologymedicine.medical_specialtyAdolescentImmunocytochemistryBiologyDesminDystrophinMyofibrilsBiopsymedicineHumansMyopathyChildUbiquitinsHyalinemedicine.diagnostic_testMusclesSkeletal muscleNeuromuscular Diseasesmedicine.diseaseCongenital myopathyImmunohistochemistryMicroscopy Electronmedicine.anatomical_structureNeurologyChild Preschoolbiology.proteinDesminFemaleNeurology (clinical)medicine.symptomDystrophinJournal of the neurological sciences
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Allocortical neurofibrillary changes in progressive supranuclear palsy.

1992

Silver techniques for intraneuronal cytoskeleton abnormalities (neurofibrillary tangles and neuropil threads) and extracellular A4-amyloid deposits were used to examine lesions of the cerebral cortex in six cases of progressive supranuclear palsy (three were mentally unimpaired and three showed moderate degrees of dementia). Deposits of A4-amyloid protein occurred in small numbers or were absent. Neurofibrillary tangles and neuropil threads were present in all cases and were largely confined to the allocortex. A characteristic pattern of changes was found in the entorhinal cortex. The three mentally unimpaired individuals had mild cortical changes virtually confined to the transentorhinal r…

MalePathologymedicine.medical_specialtyAmyloidSilver StainingHippocampusBiologyHippocampal formationHippocampusPathology and Forensic MedicineProgressive supranuclear palsyCellular and Molecular NeuroscienceAlzheimer DiseasemedicineHumansAgedCerebral CortexAllocortexBrainNeurofibrillary tangleNeurofibrillary TanglesMiddle AgedPerforant pathmedicine.diseaseEntorhinal cortexmedicine.anatomical_structureCerebral cortexFemaleNeurology (clinical)Supranuclear Palsy ProgressiveActa neuropathologica
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Identification to cardiac conduction cells in humans and pigs according to their zonal distribution, using histological, immunohistochemical and morp…

2021

Abstract Histologically, the cardiac conduction network is formed of electrically isolated subendocardial fibers that comprise specialized cells with fewer myofibrils and mitochondria than cardiomyocytes. Our aim is to uncover regional variations of cardiac conduction fibers through histological and morphometric study in a porcine and human model. We analyzed five male adult human hearts and five male pig hearts. The left ventricles were dissected and sectioned in the axial plane into three parts: basal, middle third and apex regions. Cardiac conduction fibers study was carried out using hematoxylin-eosin and Masson's trichrome staining, and cardiac conduction cells and their junctions were…

MalePathologymedicine.medical_specialtyGeneral VeterinaryStaining and LabelingChemistryHeart VentriclesMyocardiumSus scrofaHeartMasson's trichrome stainHeart Conduction SystemCardiac conductioncardiovascular systemmedicineDistribution (pharmacology)ImmunohistochemistryAnimalsHumansDesminElectrical conduction system of the heartMyofibrilEndocardiumResearch in veterinary science
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Diethylene glycol (DEG)-associated myocardial changes: a pilot investigation of chronic intoxication in guinea-pigs.

1991

The myocardium of guinea pigs fed on sublethal doses of diethylene glycol (DEG) over a period of 2–11 days was examined for microscopical and ultrastructural changes. Coagulative myocytolysis and loss of myofibrils, not observed in the controls, was patchily distributed throughout the myocardium. The accompanying ultrastructural features included swelling, pleomorphism and hyperplasia of mitochondria with an associated distension of the interfibrillary spaces and a displacement, distortion and rupture of adjacent myofibrils.

MalePathologymedicine.medical_specialtyGuinea PigsAdministration OralPilot ProjectsDistensionMitochondria HeartPathology and Forensic MedicineGuinea pigchemistry.chemical_compoundmedicineAnimalsMyocytolysisChemistryMyocardiumDiethylene glycolAnatomyHyperplasiamedicine.diseaseMicroscopy ElectronCoagulative necrosisPleomorphism (microbiology)Ethylene GlycolsMyofibrilCardiomyopathiesInternational journal of legal medicine
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Upregulation of antibody response to heat shock proteins and tissue antigens in an ocular ischemia model.

2011

PURPOSE. The aim of this study was to characterize the serum antibody reactivities occurring after ocular ischemia reperfusion. The time course of serum antibody responses was examined. METHODS. Wistar rats were exposed to transient ocular ischemia by elevating intraocular pressure to 130 mm Hg for 60 minutes. Axonal damage was evaluated on optic-nerve sections 2 and 4 weeks later. Blood samples collected before and several times after ischemia were used for antibody detection via customized protein microarrays. Different tissue antigens, including heat shock proteins (HSPs) and crystallins, were selected based on previous identification of antibody reactivities in studies on ischemic event…

MalePathologymedicine.medical_specialtyIschemiaHSP27 Heat-Shock ProteinsProtein Array AnalysisVimentinBiologyAutoantigensDownregulation and upregulationAntigenRetinal DiseasesHeat shock proteinGlial Fibrillary Acidic ProteinmedicineAnimalsRats WistarEye ProteinsIntraocular PressureAutoantibodiesGlial fibrillary acidic proteinRetinal VesselsSpectrinMyelin Basic ProteinOptic Nervemedicine.diseaseAxonsRatsUp-RegulationMyelin-Associated GlycoproteinShock (circulatory)Immunoglobulin GReperfusion Injurybiology.proteinMyelin-Oligodendrocyte Glycoproteinmedicine.symptomAntibodyMyelin ProteinsInvestigative ophthalmologyvisual science
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Pleomorphic pineocytoma with extensive neuronal differentiation: report of two cases

1994

Two pineal parenchymal tumors are presented, arising in a 54-year-old man and a 72-year-old woman; respectively. They showed isomorphic, cellular areas of small cells, often with characteristic pineocytomatous rosettes, and of medium-sized cells, as well as less cellular regions with highly pleomorphic, often ganglioid large cells. Immunohistochemistry disclosed extensive neuronal differentiation. There was intense positivity for neurofilament protein and microtubule-associated protein 2 in the pleomorphic areas and more variable expression in the isomorphic regions. Diffuse synaptophysin positivity was seen, accentuated along the borders of pleomorphic cells and in the rosettes, as well as…

MalePathologymedicine.medical_specialtyNeurofilamentEnolasePineal GlandPathology and Forensic MedicineCellular and Molecular NeurosciencemedicineCentral neurocytomaHumansAgedNeuronsPineoblastomaGlial fibrillary acidic proteinbiologyBrain NeoplasmsPineocytomaMiddle Agedmedicine.diseaseImmunohistochemistrybiology.proteinSynaptophysinImmunohistochemistryFemaleNeurology (clinical)PinealomaActa Neuropathologica
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Esthesioneuroblastoma: Ultrastructural, immunohistological and biochemical investigation of one case

1984

A case of esthesioneuroblastoma, the pathological diagnosis of which almost always causes great difficulties, was investigated ultrastructurally, biochemically, and immunohistologically, using antibodies against the five known types of intermediate filaments [keratin, vimentin, desmin, glial fibrillary acidic protein (GFAP) and neurofilaments]. The tumour cells did not react with antibodies against any of the five intermediate filament proteins. Ultrastructural investigations showed dense cored secretory granules in the cytoplasm and cell processes. Thus, immunohistology offers by "exclusion" a differential diagnosis to avoid often misdiagnosed tumours (undifferentiated carcinomas, embryona…

MalePathologymedicine.medical_specialtyNeurofilamentVimentinmacromolecular substancesCytoplasmic GranulesImmunofluorescenceDiagnosis DifferentialVanilmandelic Acid03 medical and health sciences0302 clinical medicineEsthesioneuroblastomaKeratinmedicineHumansNeuroectodermal Tumors Primitive Peripheral030223 otorhinolaryngologyIntermediate filamentchemistry.chemical_classificationbiologymedicine.diagnostic_testGlial fibrillary acidic proteinHomovanillic AcidGeneral MedicineMiddle Agedmedicine.disease3. Good healthOtorhinolaryngologychemistry030220 oncology & carcinogenesisbiology.proteinDesminParanasal Sinus NeoplasmsArchives of Oto-Rhino-Laryngology
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Myofibrillar disorganization characterizes myopathy of camptocormia in Parkinson’s disease

2011

Camptocormia is a highly disabling syndrome that occurs in various diseases but is particularly associated with Parkinson’s disease (PD). Although first described nearly 200 years ago, the morphological changes associated with camptocormia are still under debate and the pathophysiology is unknown. We analyzed paraspinal muscle biopsies of 14 PD patients with camptocormia and compared the findings to sex-matched postmortem controls of comparable age to exclude biopsy site-specific changes. Camptocormia in PD showed a consistent lesion pattern composed of myopathic changes with type-1 fiber hypertrophy, loss of type-2 fibers, loss of oxidative enzyme activity, and acid phosphatase reactivity …

MalePathologymedicine.medical_specialtyParkinson's diseaseMyopathyClinical Neurology610BiologySpinal CurvaturesMuscle hypertrophyPathology and Forensic MedicineLesionMuscular Atrophy Spinal03 medical and health sciencesCamptocormiaMyofibrillar disorganizationCellular and Molecular Neuroscience0302 clinical medicineMyofibrilsBiopsymedicineHumansProspective StudiesMyopathyMuscle SkeletalParkinson’s disease; Camptocormia; Myopathy; Myofibrillar disorganization; ProprioceptionMyositis030304 developmental biologyAgedAged 80 and over0303 health sciencesOriginal Papermedicine.diagnostic_testParkinson DiseaseMiddle Agedmedicine.diseaseProprioceptionPathophysiologyCamptocormiaParkinson’s diseaseFemaleNeurology (clinical)medicine.symptomMedicine & Public Health; Neurosciences; Pathology030217 neurology & neurosurgeryActa Neuropathologica
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Fate of autologous dermal stem cells transplanted into the spinal cord after traumatic injury (TSCI)

2003

Rat dermis is a source of cells capable of growing in vitro and, in appropriate conditions, forming floating spheres constituted by nestin-positive cells. We have clonally grown these spheres up to the 15th generation. These spheres can be dissociated into cells that differentiate in vitro under appropriate conditions, these cells are labeled by antibodies to immature neuron markers such as nestin and beta-tubulin III and, later, to mature neuron markers such as microtubule-associated protein 2 and neurofilaments. However, most cells are positive to the astroglial marker glia fibrillary acidic protein (GFAP). When sphere-derived cells are transplanted into the spinal cord after traumatic in…

MalePathologymedicine.medical_specialtyTime Factorstiming of transplantationNeurofilamentCellular differentiationBlotting Westernstem cell migrationPolymerase Chain ReactionRats Sprague-DawleyCell MovementGlial Fibrillary Acidic ProteinmedicineAnimalsstem cell differentiationSpinal Cord InjuriesNeuronsrecovery from disabilityGlial fibrillary acidic proteinbiologystem cell migration; stem cell differentiation; timing of transplantation; recovery from disabilityStem CellsGeneral NeuroscienceCell DifferentiationDermisRecovery of FunctionNestinRatsTransplantationmedicine.anatomical_structureImmunologySettore BIO/14 - Farmacologiabiology.proteinSettore MED/26 - NeurologiaNeuronAntibodyStem cellStem Cell TransplantationNeuroscience
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Risk Stratification Using the CHA(2)DS(2)-VASc Score in Takotsubo Syndrome: Data From the Takotsubo Italian Network

2017

Background The CHA 2 DS 2 ‐VASc score predicts stroke in patients with atrial fibrillation and has been reported to have a prognostic role even in acute coronary syndrome patients. The Takotsubo syndrome is a condition that mimics acute coronary syndrome and may present several complications including stroke. We sought to assess the ability of CHA 2 DS 2 ‐VASc score to predict adverse events in Takotsubo syndrome patients. Methods and Results Overall, 371 Takotsubo syndrome patients were enrolled in a prospective registry. Patients were divided into 3 groups according to the CHA 2 DS 2 ‐VASc score: Group A (≤1), B (2–3), and C (≥4). The median CHA 2 DS 2 ‐VASc score was 3 (interquartile ra…

MalePediatricsArrhythmias030204 cardiovascular system & hematologySeverity of Illness Index0302 clinical medicineRisk FactorsInterquartile rangeOdds RatioCoronary Heart DiseaseProspective StudiesRegistries030212 general & internal medicineMyocardial infarctionDSStrokeTakotsuboOriginal ResearchIncidenceMortality rateanticoagulant cardiovascular events CHA2DS2-VASc score stroke Takotsubo Takotsubo cardiomyopathy. Takotsubo syndromeAtrial fibrillationMiddle AgedPrognosisAnticoagulant; Cardiovascular events; CHA2DS2-VASc score; Stroke; Takotsubo; Takotsubo cardiomyopathy; Takotsubo syndrome; Cardiology and Cardiovascular MedicineStrokeItalyCardiologyFemaleCHA2DS2‐VASc scoreTakotsubo syndromeCardiology and Cardiovascular Medicinemedicine.medical_specialtyAcute coronary syndromeCHA2DS2-VASc scoreCardiomyopathy2Cardiovascular eventRisk Assessment-VASc scorecardiovascular events03 medical and health sciencesInternal medicinemedicineHumansAgedbusiness.industryAnticoagulantAnticoagulantsThrombosisOdds ratiomedicine.diseaseCHA2DS2–VASc scoreCHACerebrovascular Disease/StrokeTakotsubo cardiomyopathybusinessFollow-Up Studies
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