Search results for "Frontal cortex"

showing 10 items of 355 documents

Role of the amygdala in antidepressant effects on hippocampal cell proliferation and survival and on depression-like behavior in the rat

2021

The stimulation of adult hippocampal neurogenesis by antidepressants has been associated with multiple molecular pathways, but the potential influence exerted by other brain areas has received much less attention. The basolateral complex of the amygdala (BLA), a region involved in anxiety and a site of action of antidepressants, has been implicated in both basal and stress-induced changes in neural plasticity in the dentate gyrus. We investigated here whether the BLA modulates the effects of the SSRI antidepressant fluoxetine on hippocampal cell proliferation and survival in relation to a behavioral index of depression-like behavior (forced swim test). We used a lesion approach targeting th…

MaleLong-Term Potentiationlcsh:MedicineHippocampal formationElement-Binding ProteinAmygdala/*drug effects/physiopathologyHippocampusMemory FormationRats Sprague-Dawleyddc:616.890302 clinical medicineMedial Prefrontal CortexElevated Plus-MazeSerotonin Uptake Inhibitors/*pharmacologylcsh:ScienceBasolateral Amygdala0303 health sciencesMultidisciplinaryNeuroscience/Behavioral NeuroscienceDepressionNeurogenesisBLAAmygdalaImmunohistochemistryChronic FluoxetineAdult-RatNeuroscience/Psychologymedicine.anatomical_structureFluoxetine/*pharmacologyDepression/*pathologyAntidepressantAntidepressive Agents Second-GenerationSelective Serotonin Reuptake InhibitorsResearch ArticleEstrèsElevated plus mazemedicine.medical_specialtyAnimal-ModelAntidepressive Agents Second-Generation/*pharmacologyCell SurvivalAmygdala03 medical and health sciencesFluoxetineNeuroplasticityHippocampus/cytology/*drug effectsmedicineAnimalsPsychiatryMaze Learning030304 developmental biologyCell Proliferationbusiness.industryDentate gyrusMental Health/Mood Disorderslcsh:RBasolateral complex of the amygdaleRatsCell Proliferation/*drug effectsDentate Gyruslcsh:QCell Survival/*drug effectsbusinessNeuroscience030217 neurology & neurosurgeryBasolateral amygdala
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Intermittent cortical stimulation evokes sensitization to cocaine and enduring changes in matrix and striosome neuron responsiveness

2005

Both the behavioral sensitization syndrome and the changes in the responsiveness of striatal neurons evoked by chronic cocaine exposure may be linked to enhanced neocortical activity, yet a direct demonstration of the effects of cortical stimulation on these parameters is lacking. We have found that repeated stimulation of the rat prelimbic cortex with picrotoxin (0.25 microg/0.25 microl, five injections on alternate days followed by 7-day withdrawal) contributed to increase c-Fos protein expression in the striosomes of the dorsolateral striatum, while producing the opposite effect in the matrix compartment, after a single exposure to cocaine (25 mg/kg). Moreover, rats exposed to cortical s…

MaleMicroinjectionsStriosomeInfralimbic cortexPrefrontal CortexStimulationStriatumMotor ActivityGABA AntagonistsRats Sprague-DawleyCellular and Molecular Neurosciencechemistry.chemical_compoundCocainemedicineAnimalsPicrotoxinPrefrontal cortexNeuronsBehavior AnimalImmunohistochemistryStimulation ChemicalRatsNeostriatumStereotypy (non-human)medicine.anatomical_structurechemistryNeuronStereotyped BehaviorPsychologyProto-Oncogene Proteins c-fosNeurosciencePicrotoxinSynapse
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Dysfunction of attention switching networks in amyotrophic lateral sclerosis

2019

Objective To localise and characterise changes in cognitive networks in Amyotrophic Lateral Sclerosis (ALS) using source analysis of mismatch negativity (MMN) waveforms. Rationale The MMN waveform has an increased average delay in ALS. MMN has been attributed to change detection and involuntary attention switching. This therefore indicates pathological impairment of the neural network components which generate these functions. Source localisation can mitigate the poor spatial resolution of sensor-level EEG analysis by associating the sensor-level signals to the contributing brain sources. The functional activity in each generating source can therefore be individually measured and investigat…

MaleMismatch negativitySource localisationEEG ElectroencephalographyMismatch negativityNetworkElectroencephalographylcsh:RC346-429PET Positron emission tomographyCognition0302 clinical medicineC9orf72AttentionEEGAUROC Area under receiver operating characteristic curveAmyotrophic lateral sclerosisAged 80 and overmedicine.diagnostic_test05 social sciencesCognitive flexibilityBrainRegular ArticleElectroencephalographyCognitionMiddle AgedSTG Superior temporal gyrusNeurologyMTG Mid temporal gyrusDLPFC Dorsolateral prefrontal cortexlcsh:R858-859.7FemaleLCMV Linearly constrained minimum varianceIFG Inferior frontal gyrusAdultCognitive Neurosciencelcsh:Computer applications to medicine. Medical informatics050105 experimental psychologyCWIT Colour-word interference test03 medical and health sciencesfMRI Functional magnetic resonance imagingMEG MagnetoencephalographymedicineMMN Mismatch negativityHumans0501 psychology and cognitive sciencesRadiology Nuclear Medicine and imagingLS Amyotrophic Lateral SclerosisAAL Automated Anatomical Labellinglcsh:Neurology. Diseases of the nervous systemAEP Auditory evoked potentialAgedbusiness.industryAmyotrophic Lateral SclerosisIQR Interquartile rangeNeurophysiologyqEEG Quantitative EEGmedicine.diseaseNeurology (clinical)Nerve NetFunctional magnetic resonance imagingbusinessNeuroscience030217 neurology & neurosurgeryeLORETA Exact low-resolution brain electromagnetic tomographyNeuroImage: Clinical
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Functional networks of motor inhibition in conversion disorder patients and feigning subjects

2016

The neural correlates of motor inhibition leading to paresis in conversion disorder are not well known. The key question is whether they are different of those of normal subjects feigning the symptoms. Thirteen conversion disorder patients with hemiparesis and twelve healthy controls were investigated using functional magnetic resonance tomography under conditions of passive motor stimulation of the paretic/feigned paretic and the non-paretic hand. Healthy controls were also investigated in a non-feigning condition. During passive movement of the affected right hand conversion disorder patients exhibited activations in the bilateral triangular part of the inferior frontal gyri (IFG), with a…

MaleMotor DisordersAudiologylcsh:RC346-4290302 clinical medicineddc:150Neural PathwaysImage Processing Computer-AssistedYoung adultPrefrontal cortexFeigningParesismedicine.diagnostic_test05 social sciencesfMRIRegular ArticleMiddle AgedMagnetic Resonance ImagingInhibition PsychologicalTreatment OutcomeNeurologyMotor inhibitionlcsh:R858-859.7Femalemedicine.symptomPsychologyAdultmedicine.medical_specialtyImagery PsychotherapyCognitive Neurosciencelcsh:Computer applications to medicine. Medical informatics050105 experimental psychologyFunctional networks03 medical and health sciencesYoung AdultmedicineHumans0501 psychology and cognitive sciencesRadiology Nuclear Medicine and imagingIn patientConversion disorderlcsh:Neurology. Diseases of the nervous systemMagnetic resonance imagingmedicine.diseaseOxygenHemiparesisConversion disorderMotor paresisNeurology (clinical)NeuroscienceConversion disorder ; Motor inhibition ; Feigning ; fMRI ; Motor paresis030217 neurology & neurosurgery
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Collaborative roles of Temporoparietal Junction and Dorsolateral Prefrontal Cortex in Different Types of Behavioural Flexibility

2017

AbstractBehavioural flexibility is essential for everyday life. This involves shifting attention between different perspectives. Previous studies suggest that flexibility is mainly subserved by the dorsolateral prefrontal cortex (DLPFC). However, although rarely emphasized, the temporoparietal junction (TPJ) is frequently recruited during flexible behaviour. A crucial question is whether TPJ plays a role in different types of flexibility, compared to its limited role in perceptual flexibility. We hypothesized that TPJ activity during diverse flexibility tasks plays a common role in stimulus-driven attention-shifting, thereby contributing to different types of flexibility, and thus the colla…

MaleNEURAL BASISBrain activity and meditationDecisiontemporoparietal junctionBRAIN ACTIVITYNeuropsychological Tests3124 Neurology and psychiatry0302 clinical medicineParietal LobeAttentionmedia_commonprefrontal cortexMultidisciplinaryShifting attention05 social sciencesQCognitive flexibilityRFlexibility (personality)Magnetic Resonance ImagingTemporal Lobemedicine.anatomical_structureSocial behaviourECONOMIC DECISION-MAKINGMedicineFemalePsychologyULTIMATUM GAMECognitive psychology515 Psychologymedia_common.quotation_subjectScienceTemporoparietal junctionSPATIAL ATTENTIONPrefrontal CortexMorals050105 experimental psychologyArticle03 medical and health sciencesYoung AdultPerceptionmedicineHumans0501 psychology and cognitive sciencesSocial BehaviorPARIETAL JUNCTIONMechanism (biology)collaborative rolesSTIMULUS-DRIVEN ATTENTIONDorsolateral prefrontal cortexMORAL JUDGMENTTASKCOGNITIVE FLEXIBILITY030217 neurology & neurosurgeryScientific Reports
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Effects of chronic fluoxetine treatment on the rat somatosensory cortex: Activation and induction of neuronal structural plasticity

2009

Recent hypotheses support the idea that disruption of normal neuronal plasticity mechanisms underlies depression and other psychiatric disorders, and that antidepressant treatment may counteract these changes. In a previous report we found that chronic fluoxetine treatment increases the expression of the polysialylated form of the neural cell adhesion molecule (PSA-NCAM), a molecule involved in neuronal structural plasticity, in the somatosensory cortex. In the present study we intended to find whether, in fact, cell activation and neuronal structural remodeling occur in parallel to changes in the expression of this molecule. Using immunohistochemistry, we found that chronic fluoxetine trea…

MaleNeuronsNeuronal PlasticityDose-Response Relationship DrugGeneral NeuroscienceCentral nervous systemHippocampusSomatosensory CortexBiologySomatosensory systemRatsRats Sprague-Dawleymedicine.anatomical_structurenervous systemFluoxetineApical dendriteNeuroplasticitymedicineAnimalsAntidepressive Agents Second-GenerationNeural cell adhesion moleculeCell activationPrefrontal cortexNeuroscienceNeuroscience Letters
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PSA-NCAM expression in the rat medial prefrontal cortex

2005

The rat medial prefrontal cortex, an area considered homologous to the human prefrontal cortex, is a region in which neuronal structural plasticity has been described during adulthood. Some plastic processes such as neurite outgrowth and synaptogenesis are known to be regulated by the polysialylated form of the neural cell adhesion molecule (PSA-NCAM). Since PSA-NCAM is present in regions of the adult CNS which are undergoing structural remodeling, such as the hypothalamus or the hippocampus, we have analyzed the expression of this molecule in the medial prefrontal cortex of adult rats using immunohistochemistry. PSA-NCAM immunoreactivity was found both in cell bodies and in the neuropil of…

MaleNeuropilNeuriteInterneuronAntimetabolitesCell SurvivalSynaptophysinSynaptogenesisPrefrontal CortexHippocampusNeural Cell Adhesion Molecule L1BiologyRats Sprague-DawleyNeuroplasticityNeuropilmedicineAnimalsFluorescent Antibody Technique IndirectPrefrontal cortexNeuronsNeuronal PlasticityGlutamate DecarboxylasePyramidal CellsGeneral NeuroscienceImmunohistochemistryRatsPhenotypemedicine.anatomical_structureBromodeoxyuridinenervous systemSialic AcidsNeural cell adhesion moleculeNeuroscienceNeuroscience
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Chronic fluoxetine treatment alters the structure, connectivity and plasticity of cortical interneurons

2014

Novel hypotheses suggest that antidepressants, such as the selective serotonin reuptake inhibitor fluoxetine, induce neuronal structural plasticity, resembling that of the juvenile brain, although the underlying mechanisms of this reopening of the critical periods still remain unclear. However, recent studies suggest that inhibitory networks play an important role in this structural plasticity induced by fluoxetine. For this reason we have analysed the effects of a chronic fluoxetine treatment in the hippocampus and medial prefrontal cortex (mPFC) of transgenic mice displaying eGFP labelled interneurons. We have found an increase in the expression of molecules related to critical period pla…

MalePERINEURONAL NET EXPRESSIONTime FactorsDendritic spinePSA-NCAMCritical period plasticityHippocampusCell CountADULT BRAIN PLASTICITYTREATMENT INCREASESHippocampusMice0302 clinical medicinePharmacology (medical)Prefrontal cortexCerebral Cortex0303 health sciencesNeuronal PlasticitybiologyGlutamate DecarboxylaseMEDIAL PREFRONTAL CORTEXPOLYSIALIC ACIDmusculoskeletal neural and ocular physiologyPerineuronal net3. Good healthPsychiatry and Mental healthParvalbuminsmedicine.anatomical_structureCerebral cortexCELL-ADHESION MOLECULEAntidepressive Agents Second-GenerationDendritic SpinesGreen Fluorescent ProteinseducationMice TransgenicNerve Tissue ProteinsNeural Cell Adhesion Molecule L1Inhibitory postsynaptic potentialRAT HIPPOCAMPUS03 medical and health sciencesmedicineAnimalsPSA-NCAM EXPRESSION030304 developmental biologyPharmacologyperineuronal netsinterneuronsCENTRAL-NERVOUS-SYSTEMfluoxetine3112 NeurosciencesGene Expression Regulationnervous systemVesicular Glutamate Transport Protein 1Sialic Acidsbiology.proteinNeural cell adhesion moleculeNerve NetNeuroscience030217 neurology & neurosurgeryParvalbuminThe International Journal of Neuropsychopharmacology
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Cognitive control in auditory working memory is enhanced in musicians

2010

Musical competence may confer cognitive advantages that extend beyond processing of familiar musical sounds. Behavioural evidence indicates a general enhancement of both working memory and attention in musicians. It is possible that musicians, due to their training, are better able to maintain focus on task-relevant stimuli, a skill which is crucial to working memory. We measured the blood oxygenation-level dependent (BOLD) activation signal in musicians and non-musicians during working memory of musical sounds to determine the relation among performance, musical competence and generally enhanced cognition. All participants easily distinguished the stimuli. We tested the hypothesis that mus…

MalePITCH PERCEPTIONAuditory PathwaysBrain activity and meditationlcsh:MedicinePREFRONTAL CORTEXSpatial memoryCognition0302 clinical medicineTerveystiede - Health care scienceBRAIN ACTIVATION311 Basic medicinelcsh:SciencePrefrontal cortexta515Neuroscience/Behavioral NeuroscienceMultidisciplinaryGENERAL FLUID INTELLIGENCEMusic psychology05 social sciencesCognitionmedicine.anatomical_structureRegression AnalysisFemaleResearch ArticleCognitive psychologyAdultPosterior parietal cortexBiologyta3112INDIVIDUAL-DIFFERENCESbehavioral disciplines and activities050105 experimental psychology03 medical and health sciencesMemoryBROCAS AREAmedicineNONMUSICIANSHumans0501 psychology and cognitive sciencesNEURAL MECHANISMSAnterior cingulate cortexta217ta113Neuroscience/Cognitive Neuroscienceta114Working memoryNeuroscience/Sensory Systemslcsh:Rta3124Acoustic StimulationANTERIOR CINGULATE CORTEXTASKlcsh:QNerve NetMusic030217 neurology & neurosurgery
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Amygdala pathology in Parkinson's disease.

1994

The amygdala undergoes severe pathological changes during the course of Parkinson's disease (PD). Lewy bodies and Lewy neurites are distributed in a specific manner throughout the nuclear complex. The lesional pattern displays only minor interindividual variation. The most prominent changes occur in the accessory cortical and central nuclei. The cortical, accessory basal and granular nuclei show less severe alterations, while the basal and lateral nuclei, as well as the intercalated cell masses, generally remain uninvolved. The amygdala receives a broad range of afferents, allowing integration of exteroceptive information with interoceptive data. It generates major projections to the isocor…

MalePathologymedicine.medical_specialtyParkinson's diseaseLewy bodyHippocampusParkinson DiseaseBiologyMiddle Agedmedicine.diseaseAmygdalaAmygdalaPathology and Forensic MedicineCellular and Molecular NeuroscienceBasal (phylogenetics)Limbic systemmedicine.anatomical_structureDegenerative diseasemedicineHumansFemaleNeurology (clinical)Prefrontal cortexAgedActa neuropathologica
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