Search results for "G6"

showing 10 items of 19 documents

Integration of a Dirac comb and the Bernoulli polynomials

2016

Abstract For any positive integer n , we consider the ordinary differential equations of the form y ( n ) = 1 − Ш + F where Ш denotes the Dirac comb distribution and F is a piecewise- C ∞ periodic function with null average integral. We prove the existence and uniqueness of periodic solutions of maximal regularity. Above all, these solutions are given by means of finite explicit formulae involving a minimal number of Bernoulli polynomials. We generalize this approach to a larger class of differential equations for which the computation of periodic solutions is also sharp, finite and effective.

Bernoulli differential equationDifferential equations[ MATH ] Mathematics [math]Differential equationGeneral MathematicsBernoulli polynomials010102 general mathematicsMathematical analysisDirac combPiecewise-smooth01 natural sciencesDirac comb010305 fluids & plasmasBernoulli polynomialsPeriodic functionsymbols.namesakeDistribution (mathematics)Ordinary differential equation0103 physical sciencessymbols[MATH]Mathematics [math]0101 mathematicsBernoulli processMathematicsMSC: 34A36 37B55 11B68 70G60
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Dissecting the Phenotype and Genotype of PLA2G6-Related Parkinsonism.

2022

Background Complex parkinsonism is the commonest phenotype in late-onset PLA2G6-associated neurodegeneration. Objectives The aim of this study was to deeply characterize phenogenotypically PLA2G6-related parkinsonism in the largest cohort ever reported. Methods We report 14 new cases of PLA2G6-related parkinsonism and perform a systematic literature review. Results PLA2G6-related parkinsonism shows a fairly distinct phenotype based on 86 cases from 68 pedigrees. Young onset (median age, 23.0 years) with parkinsonism/dystonia, gait/balance, and/or psychiatric/cognitive symptoms were common presenting features. Dystonia occurred in 69.4%, pyramidal signs in 77.2%, myoclonus in 65.2%, and cere…

LevodopaPediatricsmedicine.medical_specialtyMovement disordersDeep brain stimulationGenotypemedicine.medical_treatmentPLANPLA2G6Group VI Phospholipases A2Atrophysystematic reviewParkinsonian DisordersmedicineHumansAge of OnsetparkinsonismDystoniaNBIAbusiness.industryParkinsonismmedicine.diseasenervous system diseasesPedigreeDystoniaPhenotypeNeurologyMutationCerebellar atrophyNeurology (clinical)medicine.symptomAtrophybusinessMyoclonusmedicine.drugMovement disorders : official journal of the Movement Disorder SocietyReferences
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Key features and clinical variability of COG6-CDG

2015

The conserved oligomeric Golgi (COG) complex consists of eight subunits and plays a crucial role in Golgi trafficking and positioning of glycosylation enzymes. Mutations in all COG subunits, except subunit 3, have been detected in patients with congenital disorders of glycosylation (CDG) of variable severity. So far, 3 families with a total of 10 individuals with biallelic COG6 mutations have been described, showing a broad clinical spectrum. Here we present 7 additional patients with 4 novel COG6 mutations. In spite of clinical variability, we delineate the core features of COG6-CDG i.e. liver involvement (9/10), microcephaly (8/10), developmental disability (8/10), recurrent infections (7…

MaleMicrocephalyGlycosylationAdolescentEndocrinology Diabetes and MetabolismProtein subunitHyperkeratosisMolecular Sequence DataGolgi ApparatusCase ReportsResearch SupportBiochemistryConserved oligomeric Golgi complexYoung AdultEndocrinologyCogCongenital Disorders of GlycosylationGeneticsJournal ArticleMedicineHumansNon-U.S. Gov'tChildMolecular BiologyExome sequencingGenetic Association StudiesGeneticsbusiness.industryConserved oligomeric Golgi complexResearch Support Non-U.S. Gov'tHigh-Throughput Nucleotide SequencingInfantCongenital disorder of glycosylationmedicine.diseasePhenotypeAdaptor Proteins Vesicular TransportPhenotypeCOG6MutationMicrocephalyFemaleCDGbusinessCongenital disorder of glycosylation
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Molecular characterization of genotype G6 human rotavirus strains detected in Italy from 1986 to 2009.

2011

Group A human rotavirus (HRV) strains with a bovine-like (G6) major outer capsid protein VP7 were first detected in Palermo, Italy, in the late 1980s, and subsequently worldwide. During a 25-year rotavirus surveillance period, additional HRV G6 strains, associated with either a P[9] or P[14] VP4 genotype, have been detected sporadically, but repeatedly, in Palermo. Whether these G6 HRVs were transmitted to humans directly from an animal reservoir or could have circulated at low prevalence in susceptible individuals is uncertain. Upon sequence analyses of the VP7, VP4, VP6, NSP4 and NSP5 gene segments, all the Italian HRV strains displayed a conserved genotype constellation, G6-P[9]/[14]-I2-…

Microbiology (medical)RotavirusGenotypingSettore MED/07 - Microbiologia E Microbiologia ClinicaLineage (genetic)GenotypevirusesPeriod (gene)Biologymedicine.disease_causeMicrobiologyGroup ARotavirus InfectionsViral ProteinsRotavirusGenotypeGeneticsmedicineAnimalsHumansMolecular BiologyGenotypingGeneG6Ecology Evolution Behavior and SystematicsPhylogenyGeneticsTransmission (medicine)Sequence Analysis RNAvirus diseasesRotaviruVirologyP[9]Infectious DiseasesItalyP[14]Infection, genetics and evolution : journal of molecular epidemiology and evolutionary genetics in infectious diseases
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Argan oil prevents down-regulation induced by endotoxin on liver fatty acid oxidation and gluconeogenesis and on peroxisome proliferator-activated re…

2015

In patients with sepsis, liver metabolism and its capacity to provide other organs with energetic substrates are impaired. This and many other pathophysiological changes seen in human patients are reproduced in mice injected with purified endotoxin (lipopolysaccharide, LPS). In the present study, down-regulation of genes involved in hepatic fatty acid oxidation (FAOx) and gluconeogenesis in mice exposed to LPS was challenged by nutritional intervention with Argan oil. Mice given a standard chow supplemented or not with either 6% (w/w) Argan oil (AO) or 6% (w/w) olive oil (OO) prior to exposure to LPS were explored for liver gene expressions assessed by mRNA transcript levels and/or enzyme a…

Peroxisome proliferator-activated receptor gammamedicine.medical_specialtyOO olive oilResearch paper[SDV]Life Sciences [q-bio]Peroxisome proliferator-activated receptorBiologyBiochemistryNuclear receptor 30lcsh:BiochemistryEstrogen-related receptorEstrogen-related receptor alphaInternal medicineACADS acyl CoA dehydrogenase short-chainACADL acyl CoA dehydrogenase long-chainmedicinePGC-1α peroxisome proliferator-activated receptor γ coactivator-1αlcsh:QD415-436ReceptorBeta oxidationHNF-4α hepatic nuclear factor-4αchemistry.chemical_classificationACADM acyl CoA dehydrogenase medium-chainPPARα peroxisome proliferator-activated receptor αERRα estrogen related receptor α[ SDV ] Life Sciences [q-bio]PEPCK phospoenolpyruvate carboxykinaseGluconeogenesisBeta-oxidationGlut4 glucose transporter 4[SDV] Life Sciences [q-bio]G6PH glucose-6-phosphataseEndocrinologyGlut2 glucose transporter 2chemistryNuclear receptorArgan oilAO Argan oilNuclear receptorACOX1 acyl-CoA oxidase 1CoactivatorLPS lipopolysaccharidePeroxisome proliferator-activated receptor alpha
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Ulrich bundles on K3 surfaces

2019

We show that any polarized K3 surface supports special Ulrich bundles of rank 2.

Pure mathematics14J60Algebra and Number TheoryMathematics::Commutative Algebra13C1414F05 13C14 14J60 16G60010102 general mathematics14F05acm bundlesACM vector sheaves and bundlesK3 surfaces01 natural sciencesUlrich sheavesMathematics - Algebraic GeometryMathematics::Algebraic Geometry0103 physical sciencesFOS: Mathematicssheaves010307 mathematical physics0101 mathematicsmoduli[MATH]Mathematics [math]Algebraic Geometry (math.AG)Mathematics
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Age-dependent regulation of antioxidant genes by p38α MAPK in the liver

2018

p38α is a redox sensitive MAPK activated by pro-inflammatory cytokines and environmental, genotoxic and endoplasmic reticulum stresses. The aim of this work was to assess whether p38α controls the antioxidant defense in the liver, and if so, to elucidate the mechanism(s) involved and the age-related changes. For this purpose, we used liver-specific p38α-deficient mice at two different ages: young-mice (4 months-old) and old-mice (24 months-old). The liver of young p38α knock-out mice exhibited a decrease in GSH levels and an increase in GSSG/GSH ratio and malondialdehyde levels. However, old mice deficient in p38α had higher hepatic GSH levels and lower GSSG/GSH ratio than young p38α knock-…

ROS Reactive oxygen species;RSK1 Ribosomal S6 kinase10301 basic medicineMAPK/ERK pathwayAgingHPLC High-performance liquid chromatographyAntioxidantmedicine.medical_treatmentTBP TATA-binding proteinClinical BiochemistryDEN Diethyl nitrosamine;MKP-1 MAPK phosphatase-1IκB kinaseGCLc Glutamate cysteine ligase catalytic subunitp38 Mitogen-Activated Protein KinasesG6PDH Glucose-6-phosphate dehydrogenaseBiochemistryAntioxidantsMicechemistry.chemical_compoundSuperoxide Dismutase-1Akt Protein kinase B0302 clinical medicineNrf2 Nuclear factor erythroid 2-related factor-2IL InterleukinSOD1 Cu/Zn-superoxide dismutaselcsh:QH301-705.5Mice KnockoutMK2 MAP-activated protein kinase 2;PGC-1α Peroxisome proliferator-activated receptor gamma coactivator 1-alphachemistry.chemical_classificationlcsh:R5-920Trx ThioredoxinGlutathione DisulfideTNF-α Tumor necrosis factor-alphabiologyLPS Lipopolysaccharide;GSSG Oxidized glutathione;MEF Mouse embryonic fibroblastsNF-kappa BGstm1 Glutathione S-transferase mu 1CatalaseEndoplasmic Reticulum StressGlutathioneLiverGSH Reduced glutathione;Catalase030220 oncology & carcinogenesisJNK c-Jun N-terminal kinaselcsh:Medicine (General)Research Papermedicine.medical_specialtyNF-E2-Related Factor 2Glutamate-Cysteine LigaseMKK MAPK kinaseAP-1 Activator protein-1IKK IƙB KinaseGene Expression Regulation EnzymologicSuperoxide dismutase03 medical and health sciencesInternal medicineGlutamate cysteine ligaseEGFR Epidermal growth factor receptormedicineAnimalsNuclear factor ƙBAnd catalaseChIP Chromatin immunoprecipitation;Protein kinase BNF-ƙB Nuclear factor kappa BSuperoxide DismutaseSuperoxide dismutase 1Superoxide dismutase 2Organic ChemistryGlutathioneASK1 Apoptosis signal-regulating kinase 1ATF2 activating transcription factor 2;030104 developmental biologyEndocrinologyEnzymeHsp Heat shock proteinlcsh:Biology (General)chemistrybiology.proteinSOD2 Mn-superoxide dismutaseMAPK mitogen activated protein kinaseNEM N-ethyl maleimide;Redox Biology
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Sporadic detection in children during a 25-year surveillance in Palermo, Italy, clarifies the zoonotic potential of G6 rotavirus strains

2011

Rotavirus G6 Italy
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IL PARTENOLIDE INDUCE MORTE CELLULARE CASPASI-INDIPENDENTE IN CELLULE DI OSTEOSARCOMA UMANO MG63

2011

Settore BIO/10 - BiochimicaCASPASIOSTEOSARCOMA UMANO MG63PARTENOLIDE
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WIN modulates osteosarcoma MG63 cell migration by inhibiting MMPs activity and adjusting intra- and extra-cellular SPARC differential expression

2014

Invasion of cancer cells into surrounding tissue is an initial step in tumor metastasis. This event, which requires migration of cancer cells and attachment to extracellular matrix (ECM), is regulated by elements of the local microenvironment, including ECM architecture. After having demonstrated the ability of the synthetic cannabinoid WIN55,512 to induce osteosarcoma MG63 cell death (1), we studied the effects of WIN on MG63 cell migration. Wound healing assay was performed to measure the ability of cells to migrate and fill the gap obtained by physical disruption of cell monolayer (2). We observed a significant delay in wound closure in 5 M WIN treated cells compared to untreated cells …

Settore BIO/10 - BiochimicaWIN osteosarcoma MG63 cell migration MMPs SPARC
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