Search results for "Gastrin"

showing 10 items of 46 documents

European disparities in malignant digestive endocrine tumours survival.

2009

The aim of this study was to report on malignant digestive endocrine tumours (MDET) prognosis in several European countries. We analysed survival data from 19 cancer registries in 12 European countries on 3,715 MDET diagnosed between 1985 and 1994. The overall 5-year survival rate was 47.5%. It was 58.1% for differentiated MDET and 8.1% for small-cell MDET (p < 0.001), 55.9% for patients under 65 and 37.0% for older patients. Survival rates for small intestinal and colorectal were higher than for the other sites. The 5-year relative survival rates were 60.3% in Northern Europe, 53.6% in Western Continental Europe, 42.5% in the UK, 37.6% in Eastern Europe (p < 0.001). Among well-differentiat…

MaleCancer Researchmedicine.medical_specialtyPathologyGlucagonomaDigestive System NeoplasmsGastroenterologydigestive endocrine tumours survivalInternal medicineEndocrine Gland NeoplasmsmedicineHumansCarcinoid tumourRegistriesSurvival rateAgedNeoplasm StagingGastrinomaRelative survivalbusiness.industryAbsolute risk reductionCancerMiddle Agedmedicine.diseasePrognosisCancer registryEuropeSurvival RateOncologyFemalebusinessInternational journal of cancer
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Local barrier dysfunction identified by confocal laser endomicroscopy predicts relapse in inflammatory bowel disease

2011

Objectives: Loss of intestinal barrier function plays an important role in the pathogenesis of inflammatory bowel disease (IBD). Shedding of intestinal epithelial cells is a potential cause of barrier loss during inflammation. The objectives of the study were (1) to determine whether cell shedding and barrier loss in humans can be detected by confocal endomicroscopy and (2) whether these parameters predict relapse of IBD. Methods: Confocal endomicroscopy was performed in IBD and control patients using intravenous fluorescein to determine the relationship between cell shedding and local barrier dysfunction. A grading system based on appearances at confocal endomicroscopy in humans was devise…

MalePathologyfluoresceintight junctionPilot ProjectsCrohn's DiseaseInflammatory bowel diseaseGastroenterologyEndoscopy Gastrointestinaltumour necrosis factor0302 clinical medicineIntestinal mucosaRecurrencecolonoscopyMedizinische Fakultätgut differentiationProspective Studies1506Intestinal MucosaConfocal laser endomicroscopyIBD modelsBarrier function0303 health sciencesCrohn's diseaseMicroscopy ConfocalapoptosisGastroenterologyMiddle AgedPrognosisUlcerative colitisBarrett's oesophagus3. Good healthcell deathDisease ProgressionFemalecell shedding030211 gastroenterology & hepatologyBarrett's metaplasiagastrointestinal physiologyAdultmedicine.medical_specialtySubsequent RelapseConfocalcolorectal cancer-mucosal healing03 medical and health sciencesPredictive Value of Testscolorectal metastasesInternal medicinegastrinmedicineEndomicroscopyHumansddc:610endoscopyFluorescent Dyesulcerative colitis030304 developmental biologymagnifying colonoscopybusiness.industryInflammatory Bowel DiseaseInflammatory Bowel Diseasesmedicine.diseaseIBD basic researchbarrier functionbusiness
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Progastrin Represses the Alternative Activation of Human Macrophages and Modulates Their Influence on Colon Cancer Epithelial Cells

2014

Macrophage infiltration is a negative prognostic factor for most cancers but gastrointestinal tumors seem to be an exception. The effect of macrophages on cancer progression depends on their phenotype, which may vary between M1 (pro-inflammatory, defensive) to M2 (tolerogenic, pro-tumoral). Gastrointestinal cancers often become an ectopic source of gastrins and macrophages present receptors for these peptides. The aim of the present study is to analyze whether gastrins can affect the pattern of macrophage infiltration in colorectal tumors. We have evaluated the relationship between gastrin expression and the pattern of macrophage infiltration in samples from colorectal cancer and the influe…

Malelcsh:MedicineCell CountLigandsMonocytesWhite Blood CellsCell SignalingAnimal CellsMolecular Cell BiologyGastrointestinal CancersBasic Cancer ResearchMedicine and Health SciencesIntestinal Mucosalcsh:ScienceImmune ResponseWNT Signaling CascadeGastrinAged 80 and overMultidisciplinaryCD68Middle AgedImmunohistochemistrySignaling CascadesInterleukin 10PhenotypeOncologyColonic NeoplasmsInterleukin 12FemaleCellular TypesResearch ArticleSignal Transductionmedicine.medical_specialtyDrug Research and DevelopmentImmune CellsAdipose tissue macrophagesImmunologyAntigen-Presenting CellsGastroenterology and HepatologyBiologyCell Line TumorInternal medicineGastrinsGastrointestinal TumorsmedicineHumansProtein PrecursorsInterleukin 4AgedNeoplasm StagingInflammationPharmacologyCD86Blood CellsMacrophageslcsh:RImmunityBiology and Life SciencesCancers and NeoplasmsCancerCell BiologyMacrophage Activationmedicine.diseaseWnt ProteinsEndocrinologyCancer researchClinical Immunologylcsh:QNeoplasm GradingClinical MedicinePLoS ONE
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Involvement of endogenous nitric oxide in the inhibition by endotoxin and interleukin-1 beta of gastric acid secretion.

1994

Administration of Escherichia coli endotoxin abolished the acid secretory response induced by a bolus injection of pentagastrin in the continuously perfused stomach of the anaesthetized rat. Likewise, acid secretion stimulated by the continuous intravenous perfusion of pentagastrin was inhibited by administration of interleukin-1 beta (IL-1 beta). In both cases pretreatment with NG-nitro-L-arginine methyl ester (L-NAME) but not dexamethasone or indomethacin substantially restored the secretory responses to pentagastrin. The actions of L-NAME were reversed by the prior administration of L-arginine but not by its enantiomer D-arginine. Even though L-NAME increased blood pressure, this does no…

Malemedicine.medical_specialtyArginineIn Vitro TechniquesArginineNitric OxideNitric oxideGastric Acidchemistry.chemical_compoundInternal medicineEscherichia coliMedicineAnimalsSecretionRats WistarPhenylephrineHepatologybusiness.industryStomachdigestive oral and skin physiologyGastroenterologyInterleukinRatsPentagastrinEndotoxinsEndocrinologymedicine.anatomical_structureNG-Nitroarginine Methyl EsterchemistryGastric acidFemalePentagastrinbusinessmedicine.drugInterleukin-1Journal of gastroenterology and hepatology
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Nitric oxide mediates the inhibition by interleukin-1β of pentagastrin-stimulated rat gastric acid secretion

1993

Bolus injection of interleukin-1 beta (2 micrograms kg-1, i.v.) inhibited acid secretion induced by intravenous infusion of pentagastrin (8 micrograms kg-1 h-1) in the continuously perfused stomach of the anaesthetized rat. Administration of interleukin-1 beta did not modify mean systemic arterial blood pressure. Pretreatment with NG-nitro-L-arginine methyl ester (L-NAME, 2-10 mg kg-1, i.v.), but not dexamethasone (5 mg kg-1, s.c. twice over 16 h), restored the acid secretory responses to pentagastrin. The actions of L-NAME were reversed by the prior administration of L-arginine (100 mg kg-1, i.v.), but not by its enantiomer D-arginine (100 mg kg-1, i.v.). L-NAME (5 mg kg-1, i.v.) increased…

Malemedicine.medical_specialtyBlood PressureBiologyPeptide hormoneArginineNitric OxideNitric oxideGastric Acidchemistry.chemical_compoundInternal medicinemedicineAnimalsRats WistarInfusions IntravenousPhenylephrineDexamethasonePharmacologyStomachStereoisomerismRatsPentagastrinNG-Nitroarginine Methyl Estermedicine.anatomical_structureEndocrinologychemistryGastrointestinal hormoneGastric acidFemalePentagastrinResearch ArticleInterleukin-1medicine.drugBritish Journal of Pharmacology
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Effects of calcium channel blockers on gastric emptying and acid secretion of the rat in vivo.

1986

Abstract Experiments were designed to evaluate the effects of three calcium channel blockers (verapamil, diltiazem and cinnarizine) on gastric emptying and secretion in the rat. Pretreatment with the calcium blockers delayed gastric emptying of phenol red in a dose-dependent manner. Verapamil was the most effective of the agents tested. Verapamil and diltiazem inhibited gastric acid secretion in the pylorus-ligated rat without affecting pepsin output. Cinnarizine was ineffective in this model. When the perfused lumen of the anaesthetized rat was used, verapamil was found to inhibit responses to carbachol or histamine more than those to pentagastrin. Further, we found a greater sensitivity t…

Malemedicine.medical_specialtyGastric motilitychemistry.chemical_elementBlood PressureCalciumBiologyGastric AcidInternal medicinemedicineAnimalsAnesthesiaDiltiazemPylorusPharmacologyGastric emptyingDose-Response Relationship DrugCalcium channeldigestive oral and skin physiologyRats Inbred StrainsCalcium Channel BlockersRatsPentagastrinPerfusionEndocrinologychemistryGastric Emptyingcardiovascular systemGastric acidVerapamilFemalemedicine.drugResearch ArticleBritish journal of pharmacology
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Modulation by peripheral opioids of basal and distension-stimulated gastric acid secretion in the rat.

1992

1. The influence of opioids in modulating gastric acid secretory responses has been investigated in the continuously perfused stomach of the anaesthetized rat. 2. Intravenous administration of morphine (0.75-3 mg kg-1) or the peripherally acting enkephalin analogue, BW443C (0.75-3 mg kg-1), substantially augmented acid secretion in basal conditions. These effects were significantly inhibited by the opioid antagonists naloxone (1 mg kg-1) and the peripherally acting N-methylnalorphine (2 mg kg-1). When administered alone, neither opioid antagonist influenced basal acid output. 3. Acid secretory responses to different levels of gastric distension (5-20 cmH2O) were significantly and dose-depen…

Malemedicine.medical_specialtyNarcotic AntagonistsNalorphine(+)-NaloxoneDistensionDeoxyglucoseGastric AcidInternal medicineNalorphineGastrinsmedicineAnimalsInsulinGastrinPharmacologyMorphinebusiness.industryNaloxoneGastric distensionRats Inbred StrainsRatsPentagastrinEndocrinologyOpioidInjections IntravenousGastric acidFemalePentagastrinmedicine.symptombusinessOligopeptidesmedicine.drugResearch ArticleHistamine
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Gastric acid secretory responses induced by peptone are mediated by capsaicin-sensitive sensory afferent neurons

1992

The involvement of capsaicin-sensitive afferent neurons in modulating acid-secretory responses to peptone, a product of protein digestion, has been investigated in the continuously perfused stomach of the urethan-anesthetized rat. Systemic neonatal pretreatment with capsaicin, which destroys primary afferent neurons, does not modify basal levels of acid secretion. Acid responses to intragastric perfusion with isotonic (0.5, 1, and 2.4%) or hypertonic (10 and 20%) solutions of peptone were reduced in capsaicin-treated rats. Intragastric perfusion with hypertonic mannitol (18%) did not stimulate secretion of acid. Systemic capsaicin pretreatment did not modify acid responses to intraperitone…

Malemedicine.medical_specialtyPhysiologyProtein digestionmedicine.medical_treatmentVagotomyGastric Acidchemistry.chemical_compoundPhysiology (medical)Internal medicineAnimalsMedicineNeurons AfferentGanglionectomyGanglia SympatheticHepatologybusiness.industryGastroenterologyRats Inbred StrainsVagotomyGanglionectomyRatsVagus nervePerfusionPentagastrinEndocrinologyHypotonic SolutionschemistryCapsaicinPeptonesReflexGastric acidFemaleCapsaicinbusinessmedicine.drug
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Gastric antisecretory drugs induce leukocyte-endothelial cell interactions through gastrin release and activation of CCK-2 receptors.

2007

Antisecretory drugs are effective antiulcer agents, but its chronic use generates hypergastrinemia and accelerates the development of atrophic gastritis in Helicobacter pylori-positive patients. We have recently shown that gastrin exerts a proinflammatory effect in rats through CCK-2 receptor activation that contributes to the inflammation induced by H. pylori. The present study was designed to examine whether gastrin hypersecretion in response to treatment with antisecretory drugs induces an inflammatory response that could promote mucosal atrophy. The effects of omeprazole or famotidine on leukocyte/endothelial cell interactions in vivo were analyzed in rat mesenteric venules using intrav…

Malemedicine.medical_specialtyProglumidemedicine.drug_classInflammationCD18Cell CommunicationProinflammatory cytokineRats Sprague-DawleyInternal medicineGastrinsmedicineLeukocytesAnimalsOmeprazoleGastrinPharmacologyInflammationMicroscopy Videobusiness.industryEndothelial CellsProton Pump InhibitorsReceptor antagonistAnti-Ulcer AgentsFlow CytometryImmunohistochemistryReceptor Cholecystokinin BRatsFamotidineChemotaxis LeukocyteEndocrinologyGastric MucosaMolecular Medicinemedicine.symptombusinessmedicine.drugThe Journal of pharmacology and experimental therapeutics
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Alpha 2-receptor-mediated inhibition of intraluminal release of gastric somatostatin in anaesthetized rats.

1992

Alino, S. F., Garcia, D. & Uvnas-Moberg, K. 1992. Alpha2-receptor-mediated inhibition of intraluminal release of gastric somatostatin in anaesthetized rats. Acta Physiol Scand144, 233–238. Received 22 February 1991, accepted 11 October 1991. ISSN 00014772. Department of Pharmacology and Pharmaceutics, University of Valencia, Valencia, Spain, Department of Cell Biology and Morphology Science, University of Pais Vasco, Leioa, Spain and Department of Pharmacology, Karolinska Institute, Stockholm, Sweden. The aim of the present study was to investigate how the sympathetic nervous system affects the vagally induced intragastric release of somatostatin and gastrin. Experiments were performed on a…

Malemedicine.medical_specialtySympathetic Nervous SystemPhysiologyNeuropeptideClonidinePhentolamineInternal medicineGastrinsmedicineAnimalsAnesthesiaPhentolamineGastrinbusiness.industryStomachdigestive oral and skin physiologyRats Inbred StrainsVagus NerveHydrogen-Ion ConcentrationReceptors Adrenergic alphaElectric StimulationVagus nerveRatsEndocrinologymedicine.anatomical_structureSomatostatinGastrointestinal hormoneGastric MucosabusinessSomatostatinPerfusionhormones hormone substitutes and hormone antagonistsmedicine.drugActa physiologica Scandinavica
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