Search results for "Genesis"

showing 10 items of 12136 documents

FRET biosensor allows spatio-temporal observation of shear stress-induced polar RhoGDIα activation

2018

Rho GDP-dissociation inhibitor α (RhoGDIα) is a known negative regulator of the Rho family that shuts off GDP/GTP cycling and cytoplasm/membrane translocation to regulate cell migration. However, to our knowledge, no reports are available that focus on how the RhoGDIα-Rho GTPases complex is activated by laminar flow through exploring the activation of RhoGDIα itself. Here, we constructed a new biosensor using fluorescence resonance energy transfer (FRET) technology to measure the spatio-temporal activation of RhoGDIα in its binding with Rho GTPases in living HeLa cells. Using this biosensor, we find that the dissociation of the RhoGDIα-Rho GTPases complex is increased by shear stress, and i…

0301 basic medicineGTP'ChemistryMedicine (miscellaneous)Cell migrationGTPasebiosensoritbiosensorsArticleGeneral Biochemistry Genetics and Molecular Biology03 medical and health sciences030104 developmental biology0302 clinical medicineFörster resonance energy transferlcsh:Biology (General)Cytoplasm030220 oncology & carcinogenesisMembrane fluidityBiophysicsGeneral Agricultural and Biological SciencesCytoskeletonlcsh:QH301-705.5Proto-oncogene tyrosine-protein kinase SrcCommunications Biology
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Photoactivatable Adhesive Ligands for Light-Guided Neuronal Growth

2018

Neuro-regeneration after trauma requires growth and reconnection of neurons to reestablish information flow in particular directions across the damaged tissue. To support this process, biomaterials for nerve tissue regeneration need to provide spatial information to adhesion receptors on the cell membrane and to provide directionality to growing neurites. Here, photoactivatable adhesive peptides based on the CASIKVAVSADR laminin peptidomimetic are presented and applied to spatiotemporal control of neuronal growth to biomaterials in vitro. The introduction of a photoremovable group [6-nitroveratryl (NVOC), 3-(4,5-dimethoxy-2-nitrophenyl)butan-2-yl (DMNPB), or 2,2′-((3′-(1-hydroxypropan-2-yl)…

0301 basic medicineGUIDED AXON GROWTHNeuritePeptidomimeticNeurogenesisPeptideBiocompatible Materials02 engineering and technologyNEUROCHEMISTRYLigandsBiochemistryPHOTOTRIGGERED CELL ADHESIONCell membrane03 medical and health sciencesLamininmedicineCell AdhesionAnimalsAmino Acid SequenceCell adhesionMolecular BiologyCells Culturedchemistry.chemical_classificationNeuronsPhotolysisbiologyChemistryCELL ADHESIONOrganic ChemistryCiencias QuímicasAdhesion021001 nanoscience & nanotechnologyIn vitroLAMININ PEPTIDOMIMETICSMice Inbred C57BL030104 developmental biologymedicine.anatomical_structureQuímica OrgánicaBiophysicsbiology.proteinMolecular MedicineLaminin0210 nano-technologyPeptidesCIENCIAS NATURALES Y EXACTAS
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Effects of exposure to gadolinium on the development of geographically and phylogenetically distant sea urchins species.

2016

Gadolinium (Gd), a metal of the lanthanide series used as contrast agent for magnetic resonance imaging, is released into the aquatic environment. We investigated the effects of Gd on the development of four sea urchin species: two from Europe, Paracentrotus lividus and Arbacia lixula, and two from Australia, Heliocidaris tuberculata and Centrostephanus rodgersii. Exposure to Gd from fertilization resulted in inhibition or alteration of skeleton growth in the plutei. The similar morphological response to Gd in the four species indicates a similar mechanism underlying abnormal skeletogenesis. Sensitivity to Gd greatly varied, with the EC50 ranging from 56 nM to 132 μM across the four species…

0301 basic medicineGadoliniumved/biology.organism_classification_rank.specieschemistry.chemical_elementGadolinium010501 environmental sciencesAquatic ScienceEcotoxicologyOceanography01 natural sciencesParacentrotus lividusEnvironmental impact03 medical and health sciencesDevelopmental abnormalityEchinoid; Embryos; Skeletogenesis; Developmental abnormality; Medical drugs; Ecotoxicology; Environmental impactbiology.animalEcotoxicologyAnimalsCentrostephanus rodgersiiSettore BIO/06 - Anatomia Comparata E CitologiaArbacia lixulaSea urchin0105 earth and related environmental sciencesArbaciaLarvabiologyEcologyved/biologyGeneral Medicinebiology.organism_classificationPollutionPhylogeographySkeletogenesi030104 developmental biologyMedical drugchemistryEmbryoAquatic environmentSea UrchinsParacentrotusEchinoidWater Pollutants ChemicalEnvironmental MonitoringMarine environmental research
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Association of Long Non-Coding RNA Polymorphisms with Gastric Cancer and Atrophic Gastritis

2020

Long non-coding RNAs (lncRNA) play an important role in the carcinogenesis of various tumours, including gastric cancer. This study aimed to assess the associations of lncRNA ANRIL, H19, MALAT1, MEG3, HOTAIR single-nucleotide polymorphisms (SNPs) with gastric cancer and atrophic gastritis. SNPs were analyzed in 613 gastric cancer patients, 118 patients with atrophic gastritis and 476 controls from three tertiary centers in Germany, Lithuania and Latvia. Genomic DNA was extracted from peripheral blood leukocytes. SNPs were genotyped by the real-time polymerase chain reaction. Results showed that carriers of MALAT1 rs3200401 CT genotype had the significantly higher odds of atrophic gastritis …

0301 basic medicineGastritis AtrophicMalemedicine.medical_specialtylcsh:QH426-470GenotypeAtrophic gastritisSingle-nucleotide polymorphismmedicine.disease_causeGastroenterologyPolymorphism Single NucleotideArticleTertiary Care Centers03 medical and health sciences0302 clinical medicineGene FrequencyStomach NeoplasmsInternal medicineGermanyatrophic gastritisGenotypeGeneticsmedicineOdds RatioHumansGenetic Predisposition to DiseaseRNA NeoplasmGenetics (clinical)AllelesAgedMALAT1long non-coding RNAbusiness.industrylong non-coding RNA ; single-nucleotide polymorphism ; gastric cancer ; atrophic gastritisgastric cancerCancerHOTAIRMiddle Agedsingle-nucleotide polymorphismmedicine.diseaseLong non-coding RNAlcsh:Genetics030104 developmental biology030220 oncology & carcinogenesisFemaleRNA Long NoncodingCarcinogenesisbusinessGenes
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Bioactive potential of silica coatings and its effect on the adhesion of proteins to titanium implants

2018

There is an ever-increasing need to develop dental implants with ideal characteristics to achieve specific and desired biological response in the scope of improve the healing process post-implantation. Following that premise, enhancing and optimizing titanium implants through superficial treatments, like silica sol-gel hybrid coatings, are regarded as a route of future research in this area. These coatings change the physicochemical properties of the implant, ultimately affecting its biological characteristics. Sandblasted acid-etched titanium (SAE-Ti) and a silica hybrid sol-gel coating (35M35G30T) applied onto the Ti substrate were examined. The results of in vitro and in vivo tests and t…

0301 basic medicineGene Expression02 engineering and technologychemistry.chemical_compoundMiceColloid and Surface ChemistryCoatingCoated Materials Biocompatiblebone regenerationOsteogenesisTitaniumChemistrySurfaces and InterfacesGeneral MedicineAdhesion021001 nanoscience & nanotechnologySilicon DioxideRabbits0210 nano-technologyBiotechnologyTitaniumSilicon dioxideSurface Propertieschemistry.chemical_elementengineering.materialOsseointegrationPhase Transitionosteogenesis03 medical and health sciencesproteomicsIn vivoOsseointegrationCell Line Tumordental implantsAnimalsPhysical and Theoretical ChemistryBone regenerationDental ImplantsosteoimmunologyOsteoblastsTibiaInterleukin-6Complement System ProteinsAlkaline Phosphatase030104 developmental biologyengineeringBiophysicsImplantapolipoproteinsBiomarkers
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ESC-Derived BDNF-Overexpressing Neural Progenitors Differentially Promote Recovery in Huntington's Disease Models by Enhanced Striatal Differentiation

2016

Summary Huntington's disease (HD) is characterized by fatal motoric failures induced by loss of striatal medium spiny neurons. Neuronal cell death has been linked to impaired expression and axonal transport of the neurotrophin BDNF (brain-derived neurotrophic factor). By transplanting embryonic stem cell-derived neural progenitors overexpressing BDNF, we combined cell replacement and BDNF supply as a potential HD therapy approach. Transplantation of purified neural progenitors was analyzed in a quinolinic acid (QA) chemical and two genetic HD mouse models (R6/2 and N171-82Q) on the basis of distinct behavioral parameters, including CatWalk gait analysis. Explicit rescue of motor function by…

0301 basic medicineGene ExpressionBiochemistrychemistry.chemical_compoundMice0302 clinical medicineNeural Stem CellsNeurotrophic factorsGenes Reporterlcsh:QH301-705.5Neuronslcsh:R5-920NeurogenesisCell DifferentiationAnatomyembryonic stem cellsHuntington Diseaselcsh:Medicine (General)NeurogliaLocomotionNeurotrophinHuntington’s diseaseCell SurvivalBiologyMedium spiny neuronArticle03 medical and health sciencesHuntington's diseaseGeneticsmedicinestriatal differentiationAnimalsBrain-derived neurotrophic factorBrain-Derived Neurotrophic FactorCell Biologymedicine.diseaseCorpus StriatumTransplantationDisease Models Animal030104 developmental biologylcsh:Biology (General)chemistrynervous systembiology.proteinNeuroscience030217 neurology & neurosurgeryBiomarkersDevelopmental BiologyQuinolinic acidStem Cell TransplantationStem Cell Reports
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Oxidative stress-mediated alterations in histone post-translational modifications

2021

Abstract Epigenetic regulation of gene expression provides a finely tuned response capacity for cells when undergoing environmental changes. However, in the context of human physiology or disease, any cellular imbalance that modulates homeostasis has the potential to trigger molecular changes that result either in physiological adaptation to a new situation or pathological conditions. These effects are partly due to alterations in the functionality of epigenetic regulators, which cause long-term and often heritable changes in cell lineages. As such, free radicals resulting from unbalanced/extended oxidative stress have been proved to act as modulators of epigenetic agents, resulting in alte…

0301 basic medicineGene ExpressionContext (language use)Biologymedicine.disease_causeBiochemistryEpigenesis GeneticHistones03 medical and health sciences0302 clinical medicinePhysiology (medical)Gene expressionmedicineHumansHistone codeEpigeneticsRegulation of gene expressionDNA MethylationChromatinCell biologyOxidative Stress030104 developmental biologyHistonebiology.proteinProtein Processing Post-Translational030217 neurology & neurosurgeryOxidative stressFree Radical Biology and Medicine
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Use of deep learning methods to translate drug-induced gene expression changes from rat to human primary hepatocytes

2020

In clinical trials, animal and cell line models are often used to evaluate the potential toxic effects of a novel compound or candidate drug before progressing to human trials. However, relating the results of animal and in vitro model exposures to relevant clinical outcomes in the human in vivo system still proves challenging, relying on often putative orthologs. In recent years, multiple studies have demonstrated that the repeated dose rodent bioassay, the current gold standard in the field, lacks sufficient sensitivity and specificity in predicting toxic effects of pharmaceuticals in humans. In this study, we evaluate the potential of deep learning techniques to translate the pattern of …

0301 basic medicineGene ExpressionGene Expression Regulation/drug effectsPathology and Laboratory MedicineConvolutional neural networkTOXICITYMachine LearningVoeding Metabolisme en GenomicaTime Measurement0302 clinical medicineGene expressionMedicine and Health SciencesMeasurementClinical Trials as TopicMultidisciplinaryArtificial neural networkPharmaceuticsQRMetabolism and GenomicsTOXICOGENOMICS030220 oncology & carcinogenesisMetabolisme en GenomicaMedicineEngineering and TechnologyNutrition Metabolism and GenomicsHepatocytes/drug effectsAlgorithmsResearch ArticleComputer and Information SciencesClinical Trials as Topic/statistics & numerical dataNeural NetworksGenetic ToxicologyTOXICOLOGYSciencePredictive ToxicologyComputational biologyBiologyComputer03 medical and health sciencesDose Prediction MethodsDeep LearningVoedingArtificial IntelligenceIn vivoGeneticsLife ScienceAnimalsHumansGeneNutritionbusiness.industryDeep learningBiology and Life SciencesGold standard (test)REPRESENTATIONSRats030104 developmental biologyGene Expression RegulationHepatocytesArtificial intelligenceNeural Networks ComputerToxicogenomicsbusinessNeuroscience
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Analysis of the expression of SDF-1 splicing variants in human colorectal cancer and normal mucosa tissues

2016

C-X-C motif chemokine ligand 12 (CXCL12), also termed stromal cell-derived factor-1 (SDF-1) is a small protein 8-14 kDa in length that is expressed as six isoforms, consisting of SDF-1α, SDF-1β, SDF-1γ, SDF-1δ, SDF-1ε and SDF-1θ. All six isoforms are encoded by the single CXCL12 gene on chromosome 10. This gene regulates leukocyte trafficking and is variably expressed in a number of normal and cancer tissues. The potential role of the novel CXCL12 splice variants as components of the CXCR4 axis in cancer development is not fully understood. The present study aimed to analyze the expression profile of the various SDF-1 isoforms and SDF-1 polymorphisms, and the association with the clinicopat…

0301 basic medicineGene isoformCancer ResearchPathologymedicine.medical_specialtyOncogeneArticlesBiologymedicine.diseasePrimary tumorCXCR403 medical and health sciences030104 developmental biology0302 clinical medicineOncologyTumor progression030220 oncology & carcinogenesisGenotypemedicineCancer researchGene polymorphismTumor markerOncology Letters
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ΔNp63 drives metastasis in breast cancer cells via PI3K/CD44v6 axis

2016

P63 is a transcription factor belonging to the family of p53, essential for the development and differentiation of epithelia. In recent years, it has become clear that altered expression of the different isoforms of this gene can play an important role in carcinogenesis. The p63 gene encodes for two main isoforms known as TA and ΔN p63 with different functions. The role of these different isoforms in sustaining tumor progression and metastatic spreading however has not entirely been clarified. Here we show that breast cancer initiating cells express ΔNp63 isoform that supports a more mesenchymal phenotype associated with a higher tumorigenic and metastatic potential. On the contrary, the ma…

0301 basic medicineGene isoformEpithelial-Mesenchymal TransitionBreast Neoplasmsmedicine.disease_causeMetastasisMicePhosphatidylinositol 3-Kinases03 medical and health sciencesBreast cancerTumor MicroenvironmentmedicineAnimalsHumansmetastasisEpithelial–mesenchymal transitionNeoplasm MetastasisPI3K/AKT/mTOR pathwayAgedAged 80 and overTumor microenvironmentp63breast cancer initiating cellsbusiness.industryMembrane ProteinsCD44v6Middle Agedmedicine.diseasePI3K/AKT pathwayHyaluronan Receptors030104 developmental biologyOncologyDrug Resistance NeoplasmTumor progressionImmunologyCancer researchFemalebreast cancer initiating cellmetastasibusinessCarcinogenesisProto-Oncogene Proteins c-aktSignal TransductionPriority Research Paper
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