Search results for "Gliogenesis"

showing 8 items of 8 documents

Engineering of Adult Neurogenesis and Gliogenesis

2016

Neural stem/progenitor cells (NSPCs) retain their ability to generate newborn neurons throughout life in the mammalian brain. Here, we describe how recently developed virus- and transgenesis-based techniques will help us (1) to understand the functional effects of neurogenesis in health and disease, (2) to design novel approaches to harness the potential for NSPC-associated endogenous repair, and (3) to induce the generation of neurons outside the main neurogenic niches in the adult brain.

0301 basic medicineNeurogenesisCellular differentiationPhysiology610 Medicine & healthBiologyGeneral Biochemistry Genetics and Molecular BiologyMice03 medical and health sciencesNeural Stem Cells1300 General Biochemistry Genetics and Molecular BiologyAnimalsHumansProgenitor cellCell ProliferationGliogenesis10242 Brain Research InstituteNeurogenesisBrainCell DifferentiationMammalian brainTechniquesNeural stem cell030104 developmental biology570 Life sciences; biologyNeuroscience
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Rat CNS cell culture. Enhancement of neuronal survival and delay of glial proliferation by serum from patients with multiple sclerosis. A morphologic…

1984

The addition of serum from multiple sclerosis (MS) patients to the culture medium of dissociated cells from cerebral hemispheres of rat embryos caused a delay in glial proliferation and an enhancement of neuronal survival. Sera from normal individuals and patients with other neurological diseases failed to show this effect. These morphological observations are interpreted as the outcome of inhibition of in vitro gliogenesis.

Central Nervous Systemmedicine.medical_specialtyPathologyNeurologyMultiple SclerosisDermatologyBiologyGliotoxinmedicineAnimalsCells CulturedGliogenesisNeuronsGeneral NeuroscienceMultiple sclerosisEmbryoCell DifferentiationGeneral MedicineMycotoxinsmedicine.diseaseEmbryo MammalianIn vitroRatsPsychiatry and Mental healthCell cultureOrgan SpecificityImmunologyNeurology (clinical)NeurogliaItalian journal of neurological sciences
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Chromatin remodelling factor Mll1 is essential for neurogenesis from postnatal neural stem cells

2009

Epigenetic mechanisms that maintain neurogenesis throughout adult life remain poorly understood(1). Trithorax group (trxG) and Polycomb group (PcG) gene products are part of an evolutionarily conserved chromatin remodelling system that activate or silence gene expression, respectively(2). Although PcG member Bmi1 has been shown to be required for postnatal neural stem cell self-renewal(3,4), the role of trxG genes remains unknown. Here we show that the trxG member Mll1 (mixed-lineage leukaemia 1) is required for neurogenesis in the mouse postnatal brain. Mll1-deficient subventricular zone neural stem cells survive, proliferate and efficiently differentiate into glial lineages; however, neur…

Chromatin ImmunoprecipitationEpigenetic regulation of neurogenesisCell SurvivalNeurogenesisCellular differentiationSubventricular zoneNerve Tissue ProteinsBiologyMethylationArticleHistonesMiceBasic Helix-Loop-Helix Transcription FactorsmedicineAnimalsCell LineageCells CulturedCell ProliferationGliogenesisHomeodomain ProteinsNeuronsMultidisciplinaryStem CellsNeurogenesisCell DifferentiationHistone-Lysine N-MethyltransferaseOligodendrocyte Transcription Factor 2Chromatin Assembly and DisassemblyOlfactory BulbMolecular biologyChromatinNeural stem cellCell biologyChromatinmedicine.anatomical_structureAnimals NewbornStem cellNeurogliaMyeloid-Lymphoid Leukemia ProteinTranscription Factors
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Dual roles of Aβ in proliferative processes in an amyloidogenic model of Alzheimer’s disease

2017

Alzheimer’s disease is a major neurodegenerative disorder that leads to severe cognitive deficits in the elderly population. Over the past two decades, multiple studies have focused on elucidating the causative factors underlying memory defects in Alzheimer’s patients. In this regard, new evidence linking Alzheimer’s disease-related pathology and neuronal stem cells suggests that hippocampal neurogenesis impairment is an important factor underlying these cognitive deficits. However, because of conflicting results, the impact of Aβ pathology on neurogenesis/gliogenesis remains unclear. Here, we investigated the effect of Aβ on neuronal and glial proliferation by using an APP/PS1 transgenic m…

Doublecortin Domain ProteinsMale0301 basic medicineCellular pathologyPathologymedicine.medical_specialtyNeurogenesisGene ExpressionHippocampuslcsh:MedicineMice TransgenicBiologyHippocampusArticleAmyloid beta-Protein PrecursorMice03 medical and health sciences0302 clinical medicineNeural Stem CellsAlzheimer DiseaseSpheroids CellularNeurospheremedicineAnimalsHumansProgenitor celllcsh:ScienceCells CulturedCell ProliferationGliogenesisNeuronsAmyloid beta-PeptidesMultidisciplinaryNeuropeptidesNeurogenesislcsh:RCell DifferentiationNeural stem cellDisease Models Animal030104 developmental biologynervous systemOrgan Specificitylcsh:QStem cellMicrotubule-Associated ProteinsNeurogliaNeuroscience030217 neurology & neurosurgery
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Adult Neurogenesis in Reptiles

2011

Adult neurogenesis in reptiles is a well-documented phenomenon and exists in many telencephalic areas. The newly generated neurons originate along the walls of the lateral ventricles, mainly in the sulci. The putative neural progenitors are radial glial cells. These glial cells give rise to neuroblasts that migrate to their final destination. In general, the new neurons are born in the portion of the ventricular zone (VZ) adjacent to the telencephalic area where they will be recruited and migrate radially through the brain parenchyma along the processes of radial glial cells to their final destination, although migration to the olfactory bulbs (OB) is different. Specifically, it seems that …

Glial fibrillary acidic proteinNeurogenesisBiologyOlfactory bulbLateral ventriclesmedicine.anatomical_structurenervous systemNeuroblastParenchymamedicinebiology.proteinSomaNeuroscienceGliogenesis
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Neuroprotection of lipoic acid treatment promotes angiogenesis and reduces the glial scar formation after brain injury

2012

After trauma brain injury, a large number of cells die, releasing neurotoxic chemicals into the extracellular medium, decreasing cellular glutathione levels and increasing reactive oxygen species that affect cell survival and provoke an enlargement of the initial lesion. Alpha-lipoic acid is a potent antioxidant commonly used as a treatment of many degenerative diseases such as multiple sclerosis or diabetic neuropathy. Herein, the antioxidant effects of lipoic acid treatment after brain cryo-injury in rat have been studied, as well as cell survival, proliferation in the injured area, gliogenesis and angiogenesis. Thus, it is shown that newborn cells, mostly corresponded with blood vessels …

MaleProgrammed cell deathAngiogenesisBlotting WesternNeovascularization PhysiologicPharmacologyBiologyNeuroprotectionAntioxidantsGlial scarNeovascularizationLesionCicatrixchemistry.chemical_compoundMicroscopy Electron TransmissionIn Situ Nick-End LabelingmedicineAnimalsRats WistarChromatography High Pressure LiquidGliogenesisThioctic AcidGeneral NeuroscienceImmunohistochemistryRatsLipoic acidNeuroprotective AgentschemistryBrain Injuriesmedicine.symptomNeurogliaNeuroscienceNeuroscience
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Stage-Specific Transcription Factors Drive Astrogliogenesis by Remodeling Gene Regulatory Landscapes.

2017

Summary A broad molecular framework of how neural stem cells are specified toward astrocyte fate during brain development has proven elusive. Here we perform comprehensive and integrated transcriptomic and epigenomic analyses to delineate gene regulatory programs that drive the developmental trajectory from mouse embryonic stem cells to astrocytes. We report molecularly distinct phases of astrogliogenesis that exhibit stage- and lineage-specific transcriptomic and epigenetic signatures with unique primed and active chromatin regions, thereby revealing regulatory elements and transcriptional programs underlying astrocyte generation and maturation. By searching for transcription factors that …

Malecell fateActivating Transcription Factor 3Neurogenesisepigenetic mechanismsCore Binding Factor Alpha 1 SubunitArticleMice Inbred C57BLastrogliogenesisMiceNFI Transcription FactorsastrocyteGene Expression Regulationtranscription factorsAnimalsgene regulationCells Culturedneural stem cellsCell stem cell
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In vitro differentiation of E-N-CAM expressing rat neural precursor cells isolated by FACS during prenatal development

1994

Most fetal rat brain cells expressing the embryonal, highly sialylated form of the cell adhesion molecule N-CAM (E-N-CAM) are precursor cells, as judged from the absence of marker molecules specific for mature neural cell types. However, the detection of E-N-CAM+ cells in frozen sections does not provide information on the lineage-specific differentiation of these cells during development. To investigate their differentiation behaviour in vitro, E-N-CAM+ cells were isolated at different times of brain development by fluorescence-activated cell sorting (FACS), using a monoclonal antibody (Mab RB21-7) which specifically recognizes polysialic acid (PSA) residues on E-N-CAM. Double-immunofluore…

animal structuresPolysialic acidCell adhesion moleculeBiologyCell sortingCell biologyCellular and Molecular Neurosciencemedicine.anatomical_structureCell culturePrecursor cellImmunologymedicineProgenitor cellAstrocyteGliogenesisJournal of Neuroscience Research
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