Search results for "Growth factor"

showing 10 items of 1300 documents

Effects of bovine colostrum supplementation on serum IGF-I, IgG, hormone, and saliva IgA during training.

1997

Mero, Antti, Heidi Miikkulainen, Jarmo Riski, Raimo Pakkanen, Jouni Aalto, and Timo Takala. Effects of bovine colostrum supplementation on serum IGF-I, IgG, hormone, and saliva IgA during training. J. Appl. Physiol.83(4): 1144–1151, 1997.—The purpose of this study was to examine the effects of bovine colostrum supplementation (Bioenervi) on serum insulin-like growth factor I (IGF-I), immunoglobulin G, hormone, and amino acid and saliva immunoglobulin A concentrations during a strength and speed training period. Nine male sprinters and jumpers underwent three randomized experimental training treatments of 8 days separated by 13 days. The only difference in the treatments was the drink of 125…

Immunoglobulin AAdultMalemedicine.medical_specialtySalivaPhysiologymedicine.medical_treatmentPlaceboImmunoglobulin GDouble-Blind MethodPhysiology (medical)Internal medicineMedicineAnimalsHumansNutritional Physiological PhenomenaInsulin-Like Growth Factor ISalivaCross-Over Studiesbiologybusiness.industryInsulinColostrumTrack and FieldCrossover studyHormonesImmunoglobulin AEndocrinologyPhysical FitnessImmunoglobulin Gbiology.proteinColostrumCattlebusinessHormoneJournal of applied physiology (Bethesda, Md. : 1985)
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MicroRNAs play a central role in molecular dysfunctions linking inflammation with cancer.

2013

It is now largely admitted that a pro-inflammatory environment may curtail anti-tumor immunity and favor cancer initiation and progression. The discovery that small non-coding regulatory RNAs, namely microRNAs (miRNAs), regulate all aspects of cell proliferation, differentiation, and function has shed a new light on regulatory mechanisms linking inflammation and cancer. Thus, miRNAs such as miR-21, miR-125b, miR-155, miR-196, and miR-210 that are critical for the immune response or hypoxia are often overexpressed in cancers and leukemias. Given the high number of their target transcripts, their deregulation may have a number of deleterious consequences, depending on the cellular context. In…

ImmunologyBiologyBioinformaticsmedicine.disease_causemiR-155Transforming Growth Factor betaNeoplasmsmicroRNAmedicineImmunology and AllergyAnimalsHumansTranscription factorCell ProliferationRegulation of gene expressionInflammationImmunityBiological TherapyGene Expression Regulation NeoplasticMicroRNAsCell Transformation NeoplasticTumor EscapeCancer researchTumor EscapeSignal transductionCarcinogenesisTransforming growth factorSignal TransductionTranscription FactorsImmunological reviews
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Inhibition of human allergic T-helper type 2 immune responses by induced regulatory T cells requires the combination of interleukin-10-treated dendri…

2006

Summary Background In grass pollen-allergic individuals, T cell anergy can be induced by IL-10-treated dendritic cells (IL-10-DC) resulting in the suppression of T helper type 1 (Th1) as well as Th2 cells. This study was performed to analyse whether such IL-10-DC-treated T cells are able to act as regulatory T cells (Treg) suppressing the function of other T cells in the periphery. As transforming growth factor (TGF)-β is also a potential inducer of Treg, we additionally analysed the inhibitory capacity of TGF-β-treated T cells in this system. Materials and Methods Freshly isolated CD4+ or CD4+CD25− T cells from grass pollen-allergic donors were stimulated with autologous mature monocyte-de…

ImmunologyEnzyme-Linked Immunosorbent AssayCell CommunicationBiologyPoaceaeT-Lymphocytes RegulatoryInterleukin 21Interferon-gammaTh2 CellsAntigens CDTransforming Growth Factor betaHypersensitivityImmunology and AllergyCytotoxic T cellHumansCTLA-4 AntigenIL-2 receptorAntigen-presenting cellCells CulturedInterleukin 3Cell ProliferationDendritic cellDendritic CellsAllergensNatural killer T cellFlow CytometryAntigens DifferentiationCell biologyInterleukin-10ImmunologyInterleukin 12PollenImmunizationInterleukin-4Interleukin-5Clinical and experimental allergy : journal of the British Society for Allergy and Clinical Immunology
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Reciprocal IFN-gamma and TGF-beta responses regulate the occurrence of mucosal inflammation.

1997

The above new findings concerning the immunological mechanisms governing mucosa, immune responses and oral tolerance in TCR-transgenic mice, as well as those operative in mice with experimental colitis, greatly expand our understanding of the processes that normally control mucosal inflammation and possibly other types of inflammation as well (Fig. 1). They indicate that, in the nondiseased mouse, ingested proteins evoke a Th1-cell (IFN-gamma) response in the mucosal follicles that is quickly counter-regulated by induction of T-cell anergy/deletion, if this Th1-cell response is inhibited (experimentally by anti-IL-12), TGF beta-producing cells appear, and these are capable of active immune …

ImmunologyMucosal inflammationGastrointestinal inflammationExperimental colitisBiologyColitisProinflammatory cytokineInterferon-gammaInterferon γTransforming Growth Factor betaImmunologyAnimalsHumansIntestinal MucosaOral toleranceTransforming growth factorImmunology today
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Physico-chemical and mechanical characterization of in-situ forming xyloglucan gels incorporating a growth factor to promote cartilage reconstruction

2016

Abstract The development of growth factors is very promising in the field of tissue regeneration but specifically designed formulations have to be developed in order to enable such new biological entities (NBEs). In particular, the range of therapeutic concentrations is usually very low compared to other active proteins and the confinement in the target site can be of crucial importance. In-situ forming scaffolds are very promising solutions for minimally invasive intervention in cartilage reconstruction and targeting of NBEs. In this work injectable, in-situ forming gels of a temperature responsive partially degalactosylated xyloglucan (Deg-XG) incorporating the growth factor FGF-18 are fo…

In situInjectionFibroblast Growth FactorChemical Phenomenamedicine.medical_treatment02 engineering and technologyFibroblast growth factor01 natural sciencesViscositychemistry.chemical_compoundTissue ScaffoldSettore BIO/10 - BiochimicaComposite materialGlucansGelTissue ScaffoldsIn-situ forming gelsViscosityGrowth factor021001 nanoscience & nanotechnologyGlucanXyloglucanmedicine.anatomical_structureSettore ING-IND/22 - Scienza E Tecnologia Dei MaterialiMechanics of MaterialsXylansMaterials Science (all)0210 nano-technologyMaterials scienceMechanical PhenomenaInjectable scaffoldsBioengineeringCondensed Matter Physic010402 general chemistryInjectable scaffoldInjectionsBiomaterialsShear modulusXylanChondrocytesmedicineAnimalsMechanics of MaterialXyloglucanCartilage reconstructionCell ProliferationMechanical PhenomenaAnimalCartilageGrowth factorMechanical EngineeringIn-situ forming gelChondrocyte0104 chemical sciencesFibroblast Growth FactorsMolecular WeightCartilagechemistryBiophysicsCattleSettore CHIM/07 - Fondamenti Chimici Delle TecnologieTemperature-responsiveGels
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Total RNA-isolation of abdominal hernia of rats for quantitative real-time reverse transcription (RT) PCR assays.

2007

Abstract Increasing complications in incisional hernia surgery call for novel treatments. A gene expression analysis of injured tissues displays important parameters for tissue regeneration. Until today, no reliable method has been described for a quantitative gene expression analysis of hernia tissues. In this work, a protocol is described for the isolation of DNA‐free total RNA of incisional hernias for the first time. Moreover, real‐time RT PCR assays for collagen type I and III and TGF‐β1 are demonstrated for relative gene expression analyses. Both methods enable relative gene expression analyses of hernia tissues for the first time.

Incisional herniaAbdominal HerniaBiologyBiochemistryCollagen Type ITransforming Growth Factor beta1Gene expressionmedicineAnimalsHerniaGeneBase SequenceReverse Transcriptase Polymerase Chain ReactionGene Expression ProfilingGeneral Medicinemedicine.diseaseMolecular biologyReverse transcriptaseHernia AbdominalRatssurgical procedures operativeReal-time polymerase chain reactionCollagen Type IIIRNABiological AssayRNA extractionBiotechnologyPreparative biochemistrybiotechnology
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Profile and molecular modeling of 3-(indole-3-yl)-4-(3,4,5-trimethoxyphenyl)-1 H-pyrrole-2,5-dione (1) as a highly selective VEGF-R2/3 inhibitor.

2006

We report on selectivity profiling of 1 in a panel of 20 protein kinases and molecular modeling indicating 1 to be highly active and selective for VEGF-R2/3. Sequence alignment analysis and detailed insights into the ATP binding pockets of targeted protein kinases from the panel result in a unique structural architecture of VEGF-R2 mainly caused by the hydrophobic pocket I, determining the molecular basis for activity and selectivity of 1.

Indole testModels MolecularBinding SitesIndolesMolecular modelStereochemistryChemistryKinaseMolecular Sequence DataBiological activitySequence alignmentCrystallography X-RayVascular Endothelial Growth Factor Receptor-3Vascular Endothelial Growth Factor Receptor-2Drug DiscoveryMolecular MedicinePyrrolesAmino Acid SequenceBinding siteSelectivityPeptide sequenceHydrophobic and Hydrophilic InteractionsJournal of medicinal chemistry
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Geldanamycin and its derivatives as Hsp90 inhibitors

2012

The Hsp90 molecule, one of the most abundant heat shock proteins in mammalian cells, maintains homeostasis and prevents stress-induced cellular damage. Hsp90 is expressed under normal conditions at a level of about 1-2 Percent of total proteins, while its expression increases 2-10 fold in cancer cells. The two main constitutively expressed isoforms of Hsp90 are known as Hsp90-alpha and Hsp90-beta, and their upregulation is associated with tumor progression, invasion and formation of metastases, as well as development of drug resistance. The Hsp90 is a key target for many newly established, potent anticancer agents containing Hsp90 N-terminal ATP binding inhibitors, such as geldanamycin, and…

IndolesLactams MacrocyclicCyclin-Dependent KinaseAntineoplastic AgentsTanespimycinBenzoquinoneModels BiologicalAntineoplastic Agentchemistry.chemical_compoundDownregulation and upregulationTransforming Growth Factor betaCyclin-dependent kinaseHeat shock proteinBenzoquinonespolycyclic compoundsAnimalsHumansHSP90 Heat-Shock ProteinsbiologyAnimalTriazolesGeldanamycinHsp90Cyclin-Dependent KinasesProto-Oncogene Proteins c-rafHSP90 Heat-Shock Proteinsrc-Family KinaseschemistryTumor progressionMutationCancer cellbiology.proteinCancer researchMacrolidesMacrolideTriazoleTumor Suppressor Protein p53Animals; Antineoplastic Agents; Benzoquinones; Cyclin-Dependent Kinases; HSP90 Heat-Shock Proteins; Humans; Lactams Macrocyclic; Macrolides; Models Biological; Mutation; Novobiocin; Proto-Oncogene Proteins c-raf; Transforming Growth Factor beta; Triazoles; Tumor Suppressor Protein p53; src-Family KinasesNovobiocinHumanFrontiers in Bioscience
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Intrinsic fluorescence of the clinically approved multikinase inhibitor nintedanib reveals lysosomal sequestration as resistance mechanism in FGFR-dr…

2017

Background Studying the intracellular distribution of pharmacological agents, including anticancer compounds, is of central importance in biomedical research. It constitutes a prerequisite for a better understanding of the molecular mechanisms underlying drug action and resistance development. Hyperactivated fibroblast growth factor receptors (FGFRs) constitute a promising therapy target in several types of malignancies including lung cancer. The clinically approved small-molecule FGFR inhibitor nintedanib exerts strong cytotoxicity in FGFR-driven lung cancer cells. However, subcellular pharmacokinetics of this compound and its impact on therapeutic efficacy remain obscure. Methods 3-dimens…

IndolesLung NeoplasmsNintedanibResistancelcsh:RC254-282FluorescenceMiceCell Line TumorAntineoplastic Combined Chemotherapy ProtocolsAnimalsHumansPhosphorylationLungCell ProliferationAntineoplastic Combined Chemotherapy ProtocolAnimalResearchDrug Synergismlcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogensLysosomeReceptors Fibroblast Growth FactorXenograft Model Antitumor AssaysLung NeoplasmFGFR1IndoleSettore CHIM/03 - Chimica Generale E InorganicaMacrolidesMacrolideLysosomesHumanSignal Transduction
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Cytokines and Neurotrophins Interact in Normal and Diseased States

2006

Neurotrophins (NTs) such as nerve growth factor (NGF) as well as cytokines, for example, interleukin-6 (IL-6), are communicators between the nervous and immune systems. There is evidence for mutual interactions between NTs and cytokines. Strategies are being developed to elucidate the molecular mechanism/s of interactions and to understand how cytokines are involved in health and disease. Analysis of underlying signaling pathways in glial cells indicates that different transcription factors, such as NF-kappa B, cAMP-responsive-element binding protein (CREB), and activator protein 1 (AP-1), are involved in NT induction. IL-6 and NTs of the NGF family are coexpressed at sites of nerve injury.…

InflammationbiologyNeuroimmunomodulationGeneral Neurosciencerespiratory systemNerve injuryCREBGeneral Biochemistry Genetics and Molecular BiologyImmune systemNerve growth factornervous systemHistory and Philosophy of Sciencebiology.proteinmedicineAnimalsCytokinesHumansNerve Growth FactorsSignal transductionmedicine.symptomReceptorNeuroscienceTranscription factorNeurotrophinAnnals of the New York Academy of Sciences
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