Search results for "HEPATITIS"

showing 10 items of 1578 documents

Serological pattern of Hepatitis B, C, and HIV infections among immigrants in Sicily: epidemiological aspects and implication on public health.

2011

The objective of this study was to describe the prevalence of Hepatitis B virus (HBV), Hepatitis C virus (HCV), and human immunodeficiency virus (HIV) infections in a cohort of immigrants living in Palermo, Sicily. The study was carried out in the period May 2006-June 2010 and recruited a total of 393 patients (59.8% males-median age of 32.6 years). All patients were tested for serological markers of HBV, HCV, and HIV infection. One-hundred thirty-eight (35.1%) individuals did not show any HBV/HCV/HIV serological marker, while 186 (47.3%) were indicative of past or current HBV infection. A total of 42 (10.7%) subjects were HBsAg positive, 59 (15.0%) showed the serological profile "anti-HBc …

AdultMaleHBsAgmedicine.medical_specialtyHealth (social science)HIV AntigensHepatitis C virusEmigrants and ImmigrantsHIV InfectionsSettore MED/42 - Igiene Generale E Applicatamedicine.disease_causeEpidemiologymedicinePrevalenceHumansHuman immunodeficiencySicilyRetrospective StudiesHepatitis B virusHepatitis B Surface Antigensbusiness.industryPublic healthPublic Health Environmental and Occupational Healthvirus diseasesHepatitis CHepatitis BMiddle Agedmedicine.diseaseHepatitis BVirologyHepatitis B Core AntigensHepatitis Cdigestive system diseasesCross-Sectional StudiesCohortFemalePublic HealthHepatitis C AntigensbusinessBiomarkersImmigrantJournal of community health
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Randomised study comparing 48 and 96 weeks peginterferon α-2a therapy in genotype D HBeAg-negative chronic hepatitis B

2013

Treatment with peginterferon α-2a (PegIFN) for 48 weeks is the standard of care for selected HBeAg-negative patients chronically infected with hepatitis B virus (HBV), but with limited treatment efficacy. A study was undertaken to investigate whether treatment extension to 96 weeks improves the outcome in this patient population.128 HBeAg-negative patients (120 genotype D) were randomised to weekly 180 μg PegIFN for 48 weeks (group A, n=51), 180 μg PegIFN for 48 weeks followed by 135 μg weekly for an additional 48 weeks (group B, n=52) or 180 μg PegIFN plus lamivudine (100 mg/day) for 48 weeks then 135 μg PegIFN for 48 weeks (group C, n=25). Endpoints were alanine aminotransferase normalisa…

AdultMaleHBsAgmedicine.medical_specialtyHepatitis B virusTime FactorsAnti-HIV Agentsmedicine.disease_causeGastroenterologyAntiviral AgentsGroup Blaw.inventionPolyethylene GlycolsPharmacotherapyHepatitis B ChronicRandomized controlled triallawPegylated interferonInternal medicinemedicineHumansHepatitis B e AntigensHepatitis B virusbusiness.industryGastroenterologyLamivudineInterferon-alphaAlanine TransaminaseHepatitis BMiddle Agedmedicine.diseaseHepatitis BRecombinant ProteinsTreatment OutcomeLamivudineImmunologyDNA ViralInterferonDrug Therapy CombinationFemaleHepatitis B; Interferonbusinessmedicine.drug
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Familiar clustering and spreading of hepatitis delta virus infection

1985

The prevalence of hepatitis delta virus (HDV) infection was significantly higher among the relatives of 79 carriers of HBsAg with antibody to HDV (index cases) than among relatives of 111 carriers without serological evidence of HDV infection (controls). Antibody to HDV was found in 45 of the 80 (56%) carriers of HBsAg in families of index cases but only in 2 of 59 (3%) carriers in families of controls (P less than 0.0001). During follow-up new HDV infection developed in 31% of 13 susceptible carriers in families of index cases, but only in 1.2% of 162 susceptible carriers in families of controls (P less than 0.001). None of the family members previously unexposed to the hepatitis B virus h…

AdultMaleHBsAgvirusesmedicine.disease_causeVirusmedicineHumansHepatitis B virusHepatologybiologyInfantvirus diseasesbiochemical phenomena metabolism and nutritionHepatitis BHepatitis Bmedicine.diseaseHepatitis DVirologyHepatitis DChild PreschoolCarrier StateImmunologybiology.proteinFemaleViral diseaseAntibodyViral hepatitisFollow-Up Studies
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HVR-1 quasispecies modifications occur early and are correlated to initial but not sustained response in HCV-infected patients treated with pegylated…

2003

Abstract Background/Aims HVR-1 quasispecies composition and evolution were investigated in patients chronically infected with genotype 1b HCV, treated with PEG-IFN α2b or STD-IFN α2b plus RBV. Methods HVR-1 heterogeneity was assessed by calculating nucleotidic complexity, diversity, synonymous (S) and non-synonymous (NS) substitutions at baseline, after 4 weeks of therapy ( T 1) and at follow-up ( T 18). Evolution of viral quasispecies was analysed by constructing phylogenetic trees. Results No correlation of baseline viremia with heterogeneity was observed. Nucleotidic complexity was lower in patients showing early virological response, and tended to be inversely correlated to viral load d…

AdultMaleHepacivirusHepatitis C virusViremiaHepacivirusViral quasispeciesInterferon alpha-2medicine.disease_causeAntiviral AgentsVirusPolyethylene GlycolsEvolution MolecularViral Proteinschemistry.chemical_compoundFlaviviridaeDrug Resistance ViralRibavirinmedicineHumansPhylogenyHepatologybiologyRibavirinGenetic VariationInterferon-alphaHepatitis C ChronicMiddle AgedPrognosisbiology.organism_classificationmedicine.diseaseVirologyRecombinant ProteinschemistryImmunologyDrug Therapy CombinationFemaleViral loadHCV Interferon Quasispecies HVR-1
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HCV replication in mononuclear cells stimulates anti-HCV-secreting B cells and reflects nonresponsiveness to interferon-α

1995

Recently, it was demonstrated in chronic hepatitis C that the release of IgG and IgM anti-HCV antibodies by mononuclear cells (PBMCs) correlated with inflammatory activity, HCV persistence in serum, and negative outcome from antiviral therapy. Thus, persistent antigenic stimulation of the antibody-secreting B cells has been suggested. In this study, PBMCs were derived from 13 patients with chronic hepatitis C. Nucleic acids were extracted by the guanidine-thiocyanate-method, and plus- and minus-stranded HCV-RNAs were determined using primers from the 5'-untranslated region of HCV. Simultaneously, unstimulated PBMCs were cultured for 8 days and anti-HCV antibodies were detected in the supern…

AdultMaleHepacivirusmedicine.medical_treatmentHepatitis C virusMolecular Sequence DataAlpha interferonHepacivirusInterferon alpha-2Virus Replicationmedicine.disease_causeAntiviral AgentsPeripheral blood mononuclear cellVirusVirologymedicineHumansCells CulturedInterferon alfaAgedDNA PrimersB-LymphocytesBase SequencebiologyInterferon-alphavirus diseasesHepatitis C AntibodiesMiddle Agedbiology.organism_classificationHepatitis CVirologyRecombinant Proteinsdigestive system diseasesTreatment OutcomeInfectious DiseasesCytokineChronic DiseaseImmunologyLeukocytes Mononuclearbiology.proteinRNA ViralFemaleAntibodymedicine.drugJournal of Medical Virology
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Lack of hepatitis B virus DNA sequences in sera from patients with acute and chronic liver diseases diagnosed as non-A, non-B-hepatitis

2008

— The sera of 15 individuals with transfusion-associated acute or chronic non A, non B hepatitis, which lacked hepatitis B virus markers, were tested for hepatitis B virus DNA by dot blot hybridization test. Three sera of two patients positive in this test, however, also gave positive results when the labeled plasmid was used as probe instead of labeled HBV-DNA, indicating false positive results in the initial test. In conclusion, the data indicated that sera of patients with confirmed non A, non B hepatitis do not contain DNA-sequences in the serum hybridizing with HBV-DNA.

AdultMaleHepatitis B virusAdolescentHepatitis Viral HumanDot blotmedicine.disease_causeChronic liver diseaseVirusPlasmidmedicineHumansFalse Positive ReactionsAgedHepatitis B virusHepatologybiologybusiness.industryMiddle AgedHepatitis Bmedicine.diseasebiology.organism_classificationHepatitis CVirologyHepadnaviridaeDNA ViralImmunologyFemaleViral diseasebusinessLiver
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Correlation of hepatitis B virus, hepatitis D virus and human immunodeficiency virus type I infection markers in hepatitis B surface antigen positive…

1992

The hepatitis D virus (HDV) infection plays a major role in severe liver damage caused by hepatitis. To establish the prevalence of HDV infection in haemophilic patients and patients without haemophilia, 87 patients with chronic hepatitis B virus (HBV) infection were examined for serological evidence of delta hepatitis. In addition HBV, HDV and human immunodeficiency virus type 1 (HIV) infection markers were compared to clinical and histopathological outcome of hepatitis. Out of 46 haemophiliacs 30 (65%) were anti-HD-seropositive; 10 out of 30 anti-HD-positive patients (33%) had pathological liver function tests compared to 2 out of 16 anti-HD-negative haemophiliacs (13%). The rate of HIV i…

AdultMaleHepatitis B virusCirrhosisAdolescentvirusesBiopsyRadioimmunoassaymedicine.disease_causeHaemophiliaHemophilia AmedicineHumansHepatitis AntibodiesHepatitis B e AntigensChildAntigens ViralHepatitis ChronicHepatitis B virusHepatitisAcquired Immunodeficiency SyndromeHepatitis B Surface Antigensbiologymedicine.diagnostic_testbusiness.industryvirus diseasesMiddle Agedmedicine.diseasebiology.organism_classificationHepatitis BVirologyHepatitis DHepatitis DHepadnaviridaeLiverLiver biopsyChild PreschoolPediatrics Perinatology and Child HealthDNA ViralHepatitis D virusHepatitis Delta VirusbusinessEuropean journal of pediatrics
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Hepatitis B and C virus variants in long-term immunosuppressed renal transplant patients in Latvia.

2004

The incidence of genome variants of hepatitis B and hepatitis C viruses among 38 long-term (2–15 years) immunosuppressed patients after renal transplantation and 10 patients undergoing dialysis was investigated. Twelve patients had only HBV infection, 9 had only HCV infection and 14 were co-infected. Regions corresponding to the HBV X/EnII/BCP, preC/C, preS/S and to the HCV core were sequenced for molecular characterization of the HBV and HCV genomes. Fifty-seven percent of HBV DNA isolates belonged to genotype D and 42% to genotype A, whereas 77% of HCV RNA isolates belonged to genotype 1b and only 17% to genotype 3a. One sample (6%) was of genotype 2c. Detailed analysis of the above-menti…

AdultMaleHepatitis B virusGenes ViralGenotypeHepatitis B virus DNA polymerasemedicine.medical_treatmentSequence HomologyHepacivirusVirusHepatitis B virus PRE betaVirologymedicineImmune ToleranceHumansPoint MutationDialysisPhylogenyAgedbusiness.industryIncidence (epidemiology)Hepatitis CHepatitis BMiddle Agedmedicine.diseaseHepatitis BVirologyHepatitis CKidney TransplantationLatviaTransplantationInfectious DiseasesAmino Acid SubstitutionFemalebusinessSequence AnalysisIntervirology
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Virological profiles in patients with chronic hepatitis C and overt or occult HBV infection

2002

Abstract OBJECTIVES: The virological profiles of hepatitis B and C viruses (HBV and HCV) and their interplay in cases of coinfection are undefined. A suppressed and occult HBV infection may occur in hepatitis B surface antigen (HBsAg) negative patients with chronic hepatitis C. The HCV core protein is able to inhibit HBV “in vitro,” and serines at positions 99 and 116 are essential for such inhibition. We aimed to assess the HBV and HCV virological profiles in cases of coinfection and to evaluate the relationship between HCV core gene variability and HBV activity. METHODS: Eighty-two anti-HCV positive patients were examined: 35 cases were HBsAg positive, 24 were HBsAg negative with “occult”…

AdultMaleHepatitis B virusHBsAgHCV RNAHepacivirusHepatitis C virusDUAL INFECTION; INTERFERON THERAPY; HEPATOCELLULAR-CARCINOMA; CHRONIC LIVER-DISEASE; HCV core protein; Hepatitis B Surface Antigens; HCV RNAGenome ViralHepacivirusDUAL INFECTIONVirus Replicationmedicine.disease_causeCHRONIC LIVER-DISEASEHepatitis B ChronicINTERFERON THERAPYOrthohepadnavirusHEPATOCELLULAR-CARCINOMAmedicineHumansAgedHepatitis B virusHepatitis B Surface AntigensHepatologybiologybusiness.industryHCV core proteinGastroenterologyvirus diseasesHepatitis C ChronicMiddle AgedViral LoadHepatitis Bbiology.organism_classificationmedicine.diseaseVirologydigestive system diseasesHepadnaviridaeDNA ViralImmunologyCoinfectionRNA ViralFemalebusinessThe American Journal of Gastroenterology
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Occult hepatitis B virus in liver tissue of individuals without hepatic disease

2008

Abstract BACKGROUND/AIMS: While many data are available concerning occult hepatitis B virus (HBV) infection in patients with hepatic disorders, there is little information about this cryptic infection in individuals without liver disease. The aim of this study was to investigate the prevalence of occult HBV in the general population by examining liver specimens from a large series of HBV-surface-antigen negative individuals with no clinical and biochemical evidence of liver disease. METHODS: The presence of HBV DNA was evaluated by testing, through polymerase chain reaction techniques, DNA extracts from 98 liver-disease-free individuals who underwent liver resection or needle biopsy during …

AdultMaleHepatitis B virusHBsAgHepatitis C virusPopulationhepatitis B virus liver tissuemedicine.disease_causeLiver diseaseNormal liverOrthohepadnavirusOccult HBVOccult HBV; HBV DNA; Normal liver; Anti-HBc; HBV-seronegativemedicineHumansHBV-seronegativeHepatitis B AntibodieseducationAgedHepatitis B viruseducation.field_of_studyHepatologybiologybusiness.industryvirus diseasesMiddle AgedHepatitis Bmedicine.diseasebiology.organism_classificationHepatitis B Core AntigensOccultdigestive system diseasesLiverHepadnaviridaeHBV DNACarrier StateDNA ViralImmunologyFemalebusinessAnti-HBcJournal of Hepatology
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