Search results for "Homeostasis"

showing 10 items of 630 documents

Association of plasma markers of cholesterol homeostasis with metabolic syndrome components. A cross-sectional study.

2011

Abstract Background and aims Increased plasma phytosterols, which reflect enhanced cholesterol absorption, have been related to an increased risk of cardiovascular disease (CVD). However, high CVD risk conditions, such as obesity, diabetes and the metabolic syndrome (MetS) have been associated with reduced cholesterol absorption. We investigated associations between plasma noncholesterol sterols and MetS components. Methods and results With a cross-sectional design, we related MetS components to plasma noncholesterol sterol-to-cholesterol ratios measured by gas chromatography in 674 dyslipidemic patients and 361 healthy subjects participating in a prospective cohort study. Plasma phytostero…

Apolipoprotein EAdultMalemedicine.medical_specialtyGenotypeEndocrinology Diabetes and MetabolismMedicine (miscellaneous)LathosterolBiologychemistry.chemical_compoundHigh-density lipoproteinApolipoproteins ERisk FactorsInternal medicineDiabetes mellitusmedicineHomeostasisHumansProspective StudiesProspective cohort studyMetabolic SyndromeNutrition and DieteticsPhytosterolsOdds ratioMiddle Agedmedicine.diseaseLipid MetabolismSitosterolsEuropean Prospective Investigation into Cancer and NutritionEndocrinologyCholesterolCross-Sectional StudiesPhenotypechemistryCardiovascular Diseaseslipids (amino acids peptides and proteins)FemaleMetabolic syndromeCardiology and Cardiovascular MedicineBiomarkersNutrition, metabolism, and cardiovascular diseases : NMCD
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Haptoglobin interacts with apolipoprotein E and beta-amyloid and influences their crosstalk.

2014

Beta-amyloid accumulation in brain is a driving force for Alzheimer's disease pathogenesis. Apolipoprotein E (ApoE) represents a critical player in beta-amyloid homeostasis, but its role in disease progression is controversial. We previously reported that the acute-phase protein haptoglobin binds ApoE and impairs its function in cholesterol homeostasis. The major aims of this study were to characterize the binding of haptoglobin to beta-amyloid, and to evaluate whether haptoglobin affects ApoE binding to beta-amyloid. Haptoglobin is here reported to form a complex with beta-amyloid as shown by immunoblotting experiments with purified proteins, or by its immunoprecipitation in brain tissues …

Apolipoprotein EMalePhysiologyDiseaseBeta-amyloidBiochemistryAmyloid beta-Protein PrecursorAlzheimer' diseasepolycyclic compoundsskin and connective tissue diseasesapolipoprotein EbiologyChemistryMedicine (all)Haptoglobinfood and beveragesBrainApoE/A? complexGeneral MedicineMiddle AgedhaptoglobinCrosstalk (biology)ApoE/Aβ complexSettore MED/26 - Neurologialipids (amino acids peptides and proteins)FemaleAlzheimer's diseaseProtein BindingAdultmedicine.medical_specialtyImmunoprecipitationCognitive NeuroscienceEnzyme-Linked Immunosorbent AssayCHO CellsTransfectionAlzheimer' disease; ApoE/Aβ complex; Apolipoprotein E; Beta-amyloid; Haptoglobin; Human brain tissue; Adult; Aged; Alzheimer Disease; Amyloid beta-Peptides; Amyloid beta-Protein Precursor; Analysis of Variance; Animals; Apolipoproteins E; Brain; CHO Cells; Cricetulus; Enzyme-Linked Immunosorbent Assay; Female; Haptoglobins; Humans; Immunoprecipitation; Male; Middle Aged; Mutation; Protein Binding; Transfection; Biochemistry; Cell Biology; Physiology; Cognitive Neuroscience; Medicine (all)NOApolipoproteins ECricetulusAlzheimer DiseaseInternal medicinemental disordersmedicineAnimalsHumansImmunoprecipitationAgedAnalysis of VarianceAmyloid beta-PeptidesHaptoglobinsNeurotoxicityAlzheimer’diseaseCell Biologymedicine.diseasehuman brain tissueEndocrinologyMutationbiology.proteinAlzheimer'diseaseHomeostasisACS chemical neuroscience
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Alzheimer’s disease and genetics of inflammation: a pharmacogenomic vision

2007

Inflammation plays a key role in Alzheimer disease, and dissecting the genetics of inflammation may provide an answer to the possible treatment. The next-generation therapy is based on a pharmacogenomics that will reconure new approaches to a drug used on definite people with specific dosage. The translation of pharmacogenomics into clinical practice will allow bold steps to be taken toward personalized medicine. In response to tissue injury elicited by trauma or infection, the inflammatory response sets in as a complex network of molecular and cellular interactions, directed to facilitate a return to physiological homeostasis and tissue repair. The role of an individual’s genetic backgroun…

Apolipoprotein E2alzheimerInflammationDiseaseAlzheimer DiseaseGeneticsHumansMedicineSettore MED/05 - Patologia ClinicaClinical significancePhysiological HomeostasisInflammationPharmacologyGeneticsSettore MED/04 - Patologia Generalebusiness.industryAnti-Inflammatory Agents Non-Steroidalmedicine.diseaseToll-Like Receptor 4PharmacogeneticsPharmacogenomicsTLR4CytokinesMolecular MedicinePersonalized medicinemedicine.symptomAlzheimer's diseasebusiness
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Temporal aspects of copper homeostasis and its crosstalk with hormones

2015

To cope with the dual nature of copper as being essential and toxic for cells, plants temporarily adapt the expression of copper homeostasis components to assure its delivery to cuproproteins while avoiding the interference of potential oxidative damage derived from both copper uptake and photosynthetic reactions during light hours. The circadian clock participates in the temporal organization of coordination of plant nutrition adapting metabolic responses to the daily oscillations. This timely control improves plant fitness and reproduction and holds biotechnological potential to drive increased crop yields. Hormonal pathways, including those of abscisic acid, gibberellins, ethylene, auxin…

Arabidopsis thalianaEstrès oxidatiuCircadian clockFisiologiahormone signallinghormone signalingMetal toxicityOryza sativaReviewPlant ScienceBiologyCircadian clocklcsh:Plant culturechemistry.chemical_compoundAuxinhormone biosynthesisoxidative stresslcsh:SB1-1110Abscisic acidchemistry.chemical_classificationGeneticsfood and beveragescopper homeostasiscopper transportersCell biologyOxidative stress.Crosstalk (biology)chemistryGibberellinHomeostasisHormoneFrontiers in Plant Science
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Comparison of global responses to mild deficiency and excess copper levels in Arabidopsis seedlings

2013

[EN] Copper is an essential micronutrient in higher plants, but it is toxic in excess. The fine adjustments required to fit copper nutritional demands for optimal growth are illustrated by the diverse, severe symptoms resulting from copper deficiency and excess. Here, a differential transcriptomic analysis was done between Arabidopsis thaliana plants suffering from mild copper deficiency and those with a slight copper excess. The effects on the genes encoding cuproproteins or copper homeostasis factors were included in a CuAt database, which was organised to collect additional information and connections to other databases. The categories overrepresented under copper deficiency and copper e…

ArabidopsisBiophysicsFunctional homologchemistry.chemical_elementCircadian clockTransporterBiochemistryBiomaterialsTranscriptomeSuperoxide dismutaseStomatal closureGene Expression Regulation PlantIron homeostasisArabidopsisThalianamedicineHomeostasisArabidopsis thalianaGeneOligonucleotide Array Sequence AnalysisGeneticsDose-Response Relationship DrugbiologyArabidopsis ProteinsReverse Transcriptase Polymerase Chain ReactionSuperoxide DismutaseProteinMetals and AlloysBindingMicronutrientbiology.organism_classificationmedicine.diseaseCopperDNA-Binding ProteinschemistryBiochemistrySeedlingsChemistry (miscellaneous)biology.proteinFeedback loopTranscription factorTranscriptomeCopper deficiencyCopperTranscription FactorsMetallomics
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Effect of ADH II Deficiency on the Intracellular Redox Homeostasis in Zymomonas mobilis

2011

Mutant strain of the facultatively anaerobic, ethanol-producing bacteriumZymomonas mobilis, deficient in the Fe-containing alcohol dehydrogenase isoenzyme (ADH II), showed impaired homeostasis of the intracellular NAD(P)H during transition from anaerobic to aerobic conditions, and also in steady-state continuous cultures at various oxygen supplies. At the same time, ADH II deficiency in aerobically grown cells was accompanied by a threefold increase of catalase activity and by about 50% increase of hydrogen peroxide excretion. It is concluded that ADH II under aerobic conditions functions to maintain intracellular redox homeostasis and to protect the cells from endogenous hydrogen peroxide.

Article Subjectlcsh:MedicineBiologyZymomonas mobilislcsh:TechnologyGeneral Biochemistry Genetics and Molecular Biologychemistry.chemical_compoundOxygen ConsumptionSpecies SpecificityHomeostasisHydrogen peroxidelcsh:ScienceGeneral Environmental ScienceAlcohol dehydrogenaseZymomonaslcsh:Tlcsh:RAlcohol DehydrogenaseHydrogen PeroxideGeneral Medicinebiology.organism_classificationOxygenBiochemistrychemistryCatalasebiology.proteinlcsh:QNAD+ kinaseOxidation-ReductionAnaerobic exerciseNADPHomeostasisIntracellularResearch ArticleThe Scientific World Journal
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Isolation of T Cells from the Skin

2014

T cells can be found in skin under steady-state conditions as well as in inflammatory processes. T cells in skin play an important role in immune homeostasis as well as control of infectious, inflammatory diseases or tumors. In addition, several important and frequent skin diseases such as psoriasis, atopic dermatitis, autoimmune disease, and contact allergy are initiated by T cells. In skin diseases, the majority of antigen-specific T cells can be found in the tissue, not the peripheral blood. Here, we present a protocol suitable for isolation of skin-resident (inflammatory) T cells that can be used for an in-depth characterization of their frequency, function, and role for the respective …

Autoimmune diseaseintegumentary systemIsolation (health care)business.industryfood and beveragesAtopic dermatitismedicine.diseasePeripheral bloodContact allergyPsoriasisImmunologymedicineImmune homeostasisbusiness
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TGF-β signalling is required for CD4⁺ T cell homeostasis but dispensable for regulatory T cell function.

2013

Signalling by the cytokine TGF-β regulates mature CD4+ T cell populations but is not involved in the survival and function of regulatory T cells.

Autoimmunity10263 Institute of Experimental ImmunologyT-Lymphocytes RegulatoryMiceInterleukin 210302 clinical medicineTransforming Growth Factor beta2400 General Immunology and MicrobiologyHomeostasisCytotoxic T cellIL-2 receptorBiology (General)0303 health sciencesGeneral Neuroscience2800 General NeurosciencePeripheral toleranceFOXP3ColitisNatural killer T cell3. Good healthCell biologymedicine.anatomical_structureGeneral Agricultural and Biological SciencesResearch ArticleSignal TransductionRegulatory T cellQH301-705.5Receptors Antigen T-Cell610 Medicine & health1100 General Agricultural and Biological SciencesThymus GlandBiologyGeneral Biochemistry Genetics and Molecular Biology03 medical and health sciences1300 General Biochemistry Genetics and Molecular BiologyLymphopeniamedicineAnimalsAntigen-presenting cellCell Proliferation030304 developmental biologyInflammationIntegrasesGeneral Immunology and MicrobiologyReproducibility of ResultsMice Inbred C57BLTamoxifenImmunologyNIH 3T3 Cells570 Life sciences; biologyGene Deletion030215 immunologyPLoS Biology
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Assessing autophagy in archived tissue or how to capture autophagic flux from a tissue snapshot

2020

This article belongs to the Special Issue Autophagy in Cancer.

Bioquímicaautophagy:Ciências da Saúde [Ciências Médicas]Ciências Médicas::Ciências da SaúdeCellular homeostasisAutofagia610 Medicine & healthBiologyGeneral Biochemistry Genetics and Molecular Biology:Organisms::Eukaryota::Animals::Chordata::Vertebrates::Mammals::Primates::Haplorhini::Catarrhini::Hominidae::Humans [Medical Subject Headings]03 medical and health sciences0302 clinical medicineHuman disease:Chemicals and Drugs::Biological Factors::Biological Markers [Medical Subject Headings]:Phenomena and Processes::Cell Physiological Phenomena::Cell Physiological Processes::Cell Death::Autophagy [Medical Subject Headings]lcsh:QH301-705.5030304 developmental biology0303 health sciencesdiseaseBiología molecularScience & TechnologyGeneral Immunology and MicrobiologyMechanism (biology)CommunicationAutophagyautophagy; biomarkers; pathology; diseasebiomarkersPatología3. Good healthCell biology:Health Care::Environment and Public Health::Public Health::Epidemiologic Methods::Epidemiologic Research Design::Reproducibility of Results [Medical Subject Headings]BiomarcadoresTejidoslcsh:Biology (General)030220 oncology & carcinogenesis570 Life sciences; biologypathologyGeneral Agricultural and Biological SciencesFlux (metabolism)Enfermedad:Phenomena and Processes::Physiological Phenomena::Physiological Processes::Homeostasis [Medical Subject Headings]
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GLP2: An underestimated signal for improving glycaemic control and insulin sensitivity

2016

Glucagon-like peptide 2 (GLP2) is a proglucagon-derived peptide produced by intestinal enteroendocrine L-cells and by a discrete population of neurons in the brainstem, which projects mainly to the hypothalamus. The main biological actions of GLP2 are related to the regulation of energy absorption and maintenance of mucosal morphology, function and integrity of the intestine; however, recent experimental data suggest that GLP2 exerts beneficial effects on glucose metabolism, especially in conditions related to increased uptake of energy, such as obesity, at least in the animal model. Indeed, mice lacking GLP2 receptor selectively in hypothalamic neurons that express proopiomelanocortin show…

Blood Glucose0301 basic medicinemedicine.medical_specialtyEndocrinology Diabetes and MetabolismPopulation030209 endocrinology & metabolismEnteroendocrine cellType 2 diabetesBiologyCarbohydrate metabolismDiet High-FatModels BiologicalType 2 diabeteMice03 medical and health sciences0302 clinical medicineInsulin resistanceEndocrinologyInternal medicineGlucagon-Like Peptide 2medicineAnimalsHomeostasisHumansGlucose homeostasisObesityeducationeducation.field_of_studyGLP2Insulin resistanceGlucagon-like peptide-2medicine.disease030104 developmental biologyEndocrinologyDiabetes Mellitus Type 2GLP2; Insulin resistance; Obesity; Type 2 diabetes; Endocrinology; Endocrinology Diabetes and MetabolismHomeostasisSignal Transduction
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