Search results for "Hormone"

showing 10 items of 2056 documents

Phenotypic variation in hyperandrogenic women influences the findings of abnormal metabolic and cardiovascular risk parameters.

2005

In hyperandrogenic women, several phenotypes may be observed. This includes women with classic polycystic ovary syndrome (C-PCOS), those with ovulatory (OV) PCOS, and women with idiopathic hyperandrogenism (IHA), which occurs in women with normal ovaries. Where other causes have been excluded, we categorized 290 hyperandrogenic women who were seen consecutively for this complaint between 1993 and 2004 into these three subgroups. The aim was to compare the prevalence of obesity, insulin resistance, and dyslipidemia as well as increases in C-reactive protein and homocysteine in these different phenotypes with age-matched ovulatory controls of normal weight (n = 85) and others matched for body…

medicine.medical_specialtyendocrine system diseasesHomocysteineEndocrinology Diabetes and Metabolismmedicine.medical_treatmentClinical BiochemistryBiochemistryBody Mass IndexNORMAL MENSESchemistry.chemical_compoundEndocrinologyInsulin resistanceRisk FactorsInternal medicineparasitic diseasesmedicineHumansInsulinANDROGEN EXCESSDEHYDROEPIANDROSTERONE SULFATEPLASMAbusiness.industryInsulinBiochemistry (medical)Hyperandrogenismnutritional and metabolic diseasesCholesterol LDLPOLYCYSTIC-OVARY-SYNDROMELuteinizing Hormonemedicine.diseaseObesityPolycystic ovaryfemale genital diseases and pregnancy complicationsEndocrinologyC-Reactive ProteinPhenotypechemistryCardiovascular DiseasesFemaleSENSITIVITYInsulin ResistancebusinessBody mass indexDyslipidemiaPolycystic Ovary SyndromeThe Journal of clinical endocrinology and metabolism
researchProduct

Transient hypothyroidism during lactation arrests myelination in the anterior commissure of rats. A magnetic resonance image and electron microscope …

2018

Thyroid hormone deficiency at early postnatal ages affects the cytoarchitecture and function of neocortical and telencephalic limbic areas, leading to impaired associative memory and in a wide spectrum of neurological and mental diseases. Neocortical areas project interhemispheric axons mostly through the corpus callosum and to a lesser extent through the anterior commissure (AC), while limbic areas mostly project through the AC and hippocampal commissures. Functional magnetic resonance data from children with late diagnosed congenital hypothyroidism and abnormal verbal memory processing, suggest altered ipsilateral and contralateral telencephalic connections. Gestational hypothyroidism aff…

medicine.medical_specialtyendocrine system diseasesNeuroscience (miscellaneous)030209 endocrinology & metabolismAnterior commissureCorpus callosumNerve conduction velocitylcsh:RC321-571lcsh:QM1-69503 medical and health sciencesCellular and Molecular Neuroscience0302 clinical medicineLimbic systemlimbic systemInternal medicinemedicineAxonlcsh:Neurosciences. Biological psychiatry. NeuropsychiatryOriginal Researchthyroid hormonesiodine dietmedicine.diagnostic_testbusiness.industryThyroidcongenital hypothyroidismpsychiatric diseasesMagnetic resonance imaginglcsh:Human anatomymedicine.diseaseCongenital hypothyroidismschizophreniaNeuroanatomyEndocrinologymedicine.anatomical_structurebipolar disordersrodent behaviorAnatomybusiness030217 neurology & neurosurgery
researchProduct

Resistin: An Inflammatory Cytokine. Role in Cardiovascular Diseases, Diabetes and the Metabolic Syndrome

2014

Resistin is an adipocyte- and monocyte-derived cytokine which has been implicated in the modulation of insulin action, energy, glucose and lipid homeostasis. Resistin has been associated with insulin resistance and many of its known complications. As a molecular link between metabolic signals, inflammation, and vascular dysfunction, resistin can be proposed as playing a significant role in the heightened inflammatory state induced by metabolic stress linked to excessive caloric intake, thus contributing to the risk for metabolic syndrome (MetS), type 2 diabetes (T2DM), and cardiovascular diseases (CVD). In this review, we highlighted the role of resistin, as an inflammatory cytokine, in the…

medicine.medical_specialtyendocrine system diseasesmedicine.medical_treatmentInflammationType 2 diabetesModels BiologicalType 2 diabetemetabolic syndrome03 medical and health scienceschemistry.chemical_compound0302 clinical medicineInsulin resistanceAdipocyteInternal medicineDiabetes mellitusDrug DiscoverymedicineHumansResistin030304 developmental biologyMetabolic SyndromePharmacology0303 health sciencesbusiness.industryDrug Discovery3003 Pharmaceutical ScienceMedicine (all)Insulinnutritional and metabolic diseasesCardiovascular diseasemedicine.disease3. Good healthcardiovascular diseasesEndocrinologyDiabetes Mellitus Type 2chemistryCardiovascular Diseases030220 oncology & carcinogenesisImmunologyResistintype 2 diabetesInflammation MediatorsMetabolic syndromemedicine.symptombusinesshormones hormone substitutes and hormone antagonists
researchProduct

Control of Gastric Acid Secretion in Somatostatin Receptor 2 Deficient Mice: Shift from Endocrine/Paracrine to Neurocrine Pathways

2007

The gastrin-enterochromaffin-like (ECL) cell-parietal cell axis is known to play an important role in the regulation of gastric acid secretion. Somatostatin, acting on somatostatin receptor type 2 (SSTR2), interferes with this axis by suppressing the activity of the gastrin cells, ECL cells, and parietal cells. Surprisingly, however, freely fed SSTR2 knockout mice seem to display normal circulating gastrin concentration and unchanged acid output. In the present study, we compared the control of acid secretion in these mutant mice with that in wild-type mice. In SSTR2 knockout mice, the number of gastrin cells was unchanged; whereas the numbers of somatostatin cells were reduced in the antru…

medicine.medical_specialtyendocrine systemCell CountGalaninBiologyHistidine DecarboxylaseArticleGastric AcidMiceEndocrinologyParietal Cells GastricInternal medicineParacrine CommunicationmedicineEnterochromaffin CellsSomatostatin receptor 2AnimalsReceptors SomatostatinEnterochromaffin-like cellGastrinMice KnockoutDelta cellSomatostatin receptorGastrin-Secreting Cellsdigestive oral and skin physiologyNeurosecretory SystemsMicroscopy ElectronEndocrinologySomatostatinGastric MucosaVesicular Monoamine Transport ProteinsG cellReceptors Galaninhormones hormone substitutes and hormone antagonists
researchProduct

Increased Connexin 43 Expression as a Potential Mediator of the Neuroprotective Activity of the Corticotropin-Releasing Hormone

2009

CRH is a major central stress mediator, but also a potent neuroprotective effector. The mechanisms by which CRH mediates its neuroprotective actions are largely unknown. Here, we describe that the gap junction molecule connexin43 (Cx43) mediates neuroprotective effects of CRH toward experimentally induced oxidative stress. An enhanced gap junction communication has been reported to contribute to neuroprotection after neurotoxic insults. We show that CRH treatment up-regulates Cx43 expression and gap junctional communication in a CRH receptor-dependent manner in IMR32 neuroblastoma cells, primary astrocytes, and organotypic hippocampal slice cultures. MAPKs and protein kinase A-cAMP response…

medicine.medical_specialtyendocrine systemCorticotropin-Releasing HormoneMAP Kinase Signaling SystemCarbenoxoloneConnexinBiologyNeuroprotectionModels BiologicalArticleRats Sprague-DawleyCorticotropin-releasing hormoneMiceEndocrinologyMediatorInternal medicineCell Line Tumormedicinepolycyclic compoundsAnimalsHumansProtein kinase AMolecular BiologyGap junctionBrainGap JunctionsGeneral MedicineCell biologyRatsEndocrinologyNeuroprotective Agentsnervous systemGene Expression RegulationConnexin 43cardiovascular systemSignal transductionhormones hormone substitutes and hormone antagonistsmedicine.drugSignal Transduction
researchProduct

Controlled Ovarian Stimulation Induces a Functional Genomic Delay of the Endometrium with Potential Clinical Implications

2008

Context: Controlled ovarian stimulation induces morphological, biochemical, and functional genomic modifications of the human endometrium during the window of implantation. Objective: Our objective was to compare the gene expression profile of the human endometrium in natural vs. controlled ovarian stimulation cycles throughout the early-mid secretory transition using microarray technology. Method: Microarray data from 49 endometrial biopsies obtained from LH+1 to LH+9 (n = 25) in natural cycles and from human chorionic gonadotropin (hCG) +1 to hCG+9 in controlled ovarian stimulation cycles (n = 24) were analyzed using different methods, such as clustering, profiling of biological processes…

medicine.medical_specialtyendocrine systemEndocrinology Diabetes and Metabolismmedia_common.quotation_subjectClinical BiochemistryStimulationLuteal PhaseBiologyEndometriumChorionic GonadotropinBiochemistryHuman chorionic gonadotropinEndometriumEndocrinologyOvulation InductionReference ValuesInternal medicinemedicineHumansMenstrual CycleMenstrual cycleOligonucleotide Array Sequence Analysismedia_commonRegulation of gene expressionGlutathione PeroxidaseGenome HumanReverse Transcriptase Polymerase Chain ReactionMicroarray analysis techniquesurogenital systemBiochemistry (medical)Luteinizing HormoneInsulin-Like Growth Factor Binding ProteinsGene expression profilingInsulin-Like Growth Factor Binding Protein 3Endocrinologymedicine.anatomical_structureGene Expression RegulationGene chip analysisRNAFemaleAlgorithms
researchProduct

Cortisol suppression and hearing thresholds in tinnitus after low-dose dexamethasone challenge

2012

Abstract Background Tinnitus is a frequent, debilitating hearing disorder associated with severe emotional and psychological suffering. Although a link between stress and tinnitus has been widely recognized, the empirical evidence is scant. Our aims were to test for dysregulation of the stress-related hypothalamus-pituitary adrenal (HPA) axis in tinnitus and to examine ear sensitivity variations with cortisol manipulation. Methods Twenty-one tinnitus participants and 21 controls comparable in age, education, and overall health status but without tinnitus underwent basal cortisol assessments on three non-consecutive days and took 0.5 mg of dexamethasone (DEX) at 23:00 on the first day. Corti…

medicine.medical_specialtyendocrine systemHearing lossAudiologyStressCortisol03 medical and health sciencesTinnitus0302 clinical medicineMineralocorticoid receptorGlucocorticoid receptorotorhinolaryngologic diseasesMedicineDexamethasone030304 developmental biology0303 health sciencesAbsolute threshold of hearingbusiness.industryHPA axisHearing thresholdlcsh:OtorhinolaryngologyHearing sensitivityLow-dose dexamethasone suppression testlcsh:RF1-547Hearing disorderOtorhinolaryngologyDexamethasone suppression testmedicine.symptombusiness030217 neurology & neurosurgeryTinnitushormones hormone substitutes and hormone antagonistsResearch Articlemedicine.drugBMC Ear, Nose and Throat Disorders
researchProduct

Atrial natriuretic peptide secretion during development of the rat supraoptic nucleus

2009

Since a relationship between atrial natriuretic peptide and oxytocin was recently demonstrated in the heart (Gutkowska et al., 1997), the aim of this study was to determine whether a relationship between the two peptides is present also in the rat hypothalamus. For this purpose, we measured ANP-ontogeny in the rat hypothalamus immunohistochemically and compared it with oxytocin-ontogeny which we previously studied. The results showed that the ANP-peptide and mRNA-ANP start at the 18th day of the fetal life. Our earlier data for oxytocin in the rat hypothalamus showed that only mRNA-oxytocin appeared the 18th day of foetal life (Farina Lipari et al., 2001); thus, at the 18th day of foetal li…

medicine.medical_specialtyendocrine systemHistologyBiophysicsOxytocinSupraoptic nucleusAtrial natriuretic peptidePregnancyInternal medicinesupraopticMedicineAnimalsSecretionRNA MessengerRats Wistarlcsh:QH301-705.5FetusMessenger RNAbusiness.industryReverse Transcriptase Polymerase Chain ReactionnucleushypothalamicCell BiologyImmunohistochemistryRatsEndocrinologyOxytocinAnimals NewbornGene Expression Regulationlcsh:Biology (General)Hypothalamuscardiovascular systemFemalebusinessSupraoptic NucleusAtrial Natriuretic FactorCell Nucleolushormones hormone substitutes and hormone antagonistscirculatory and respiratory physiologymedicine.drugEuropean Journal of Histochemistry
researchProduct

Bisphenol A Effects on the Growing Mouse Oocyte Are Influenced by Diet1

2009

Growing evidence suggests that exposure to bisphenol A (BPA) has the ability to disrupt several different stages of oocyte development. To date, most attention has focused on the effects of BPA on the periovulatory oocyte, and considerable variation is evident in the results of these studies. In our own laboratory, variation in the results of BPA studies conducted at different times appeared to correlate with changes in mill dates of animal feed. This observation, coupled with reports by others that dietary estrogens in feed are a confounding variable in studies of endocrine-disrupting chemicals, prompted us to evaluate the effect of diet on the results of BPA studies of the periovulatory o…

medicine.medical_specialtyendocrine systemOffspringmedia_common.quotation_subjectPhytoestrogensBiologyEndocrine DisruptorsAndrologychemistry.chemical_compoundMiceMeiosisPhenolsInternal medicinemedicineAnimalsBenzhydryl compoundsEstrogens Non-SteroidalMatingBenzhydryl Compoundsmedia_commonurogenital systemCell BiologyGeneral MedicineOocyteAneuploidyAnimal FeedIsoflavonesDietMice Inbred C57BLMeiosisEndocrinologymedicine.anatomical_structureReproductive MedicinechemistryOocytesGametePhytoestrogensFemaleReproductionhormones hormone substitutes and hormone antagonistsResearch Article
researchProduct

Thyrotropin Receptor Blocking Antibodies.

2018

AbstractAutoantibodies (Ab) against the thyroid-stimulating hormone receptor (TSHR) are frequently found in autoimmune thyroid disease (AITD). Autoantibodies to the TSHR (anti-TSHR-Ab) may mimic or block the action of TSH or be functionally neutral. Measurement of anti-TSHR-Ab can be done either via competitive-binding immunoassays or with functional cell-based bioassays. Antibody-binding assays do not assess anti-TSHR-Ab functionality, but rather measure the concentration of total anti-TSHR binding activity. In contrast, functional cell-based bioassays indicate whether anti-TSHR-Ab have stimulatory or blocking activity. Historically bioassays for anti-TSHR-Ab were research tools and were u…

medicine.medical_specialtyendocrine systemendocrine system diseasesEndocrinology Diabetes and MetabolismGraves' diseaseClinical Biochemistry030209 endocrinology & metabolismHashimoto DiseaseReviewBiochemistryThyroiditisThyrotropin receptor03 medical and health sciences0302 clinical medicineEndocrinologyInternal medicineBlocking antibodymedicineAnimalsHumansReceptorAntibodies BlockingAutoantibodiesbinding assaycell-based bioassaybiologybusiness.industryBiochemistry (medical)AutoantibodyReceptors ThyrotropinGeneral MedicineHashimoto’s thyroiditismedicine.diseaseTSH receptor blocking autoantibodieseye diseasesEndocrinologyHormone receptor030220 oncology & carcinogenesisImmunologybiology.proteinBiological AssayAntibodybusinessGraves’ diseasehormones hormone substitutes and hormone antagonistsHormone and metabolic research = Hormon- und Stoffwechselforschung = Hormones et metabolisme
researchProduct