Search results for "Human genetics"

showing 10 items of 203 documents

An autosomal dominant retinitis pigmentosa family with close linkage to D7S480 on 7q.

1995

Retinitis pigmentosa is the most prevalent inherited disorder of the retina. It can be autosomal dominant (adRP), autosomal recessive (arRP) or X-linked (XLRP). A form of adRP mapping to chromosome 7q was reported in a large Spanish pedigree. We have typed DNA from the members of another Spanish family for polymorphic markers from the known candidate genes. Positive lod scores were obtained only for the markers located on 7q31-35, giving a maximum lod score of 2.98 (3.01 by multipoint analysis) at theta = 0.00 for D7S480. A brief clinical evaluation is given.

Genetic MarkersMaleCandidate genecongenital hereditary and neonatal diseases and abnormalitiesgenetic structuresBiologyAutosomal dominant retinitis pigmentosaGene mappingRetinitis pigmentosaGeneticsmedicineHumansGeneGenetics (clinical)Genes DominantLinkage (software)GeneticsChromosome Mappingmedicine.diseaseHuman geneticseye diseasesPedigreeGenetic markerFemaleLod ScoreChromosomes Human Pair 7Retinitis PigmentosaHuman genetics
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Transgene detection by digital droplet PCR

2014

Somatic gene therapy is a promising tool for the treatment of severe diseases. Because of its abuse potential for performance enhancement in sports, the World Anti-Doping Agency (WADA) included the term 'gene doping' in the official list of banned substances and methods in 2004. Several nested PCR or qPCR-based strategies have been proposed that aim at detecting long-term presence of transgene in blood, but these strategies are hampered by technical limitations. We developed a digital droplet PCR (ddPCR) protocol for Insulin-Like Growth Factor 1 (IGF1) detection and demonstrated its applicability monitoring 6 mice injected into skeletal muscle with AAV9-IGF1 elements and 2 controls over a 3…

Genetics and Molecular Biology (all)Gene Identification and AnalysisGene TransferBiochemistryPolymerase Chain Reaction796 Athletic and outdoor sports and gamesMiceMedicine and Health SciencesTransgenesInsulin-Like Growth Factor IIntramuscularMedicine (all)QRDependovirusDependoviruMedicineGenetic VectorResearch ArticleBiotechnologyHumanScienceGenetic VectorsReproducibility of ResultIn Vitro TechniquesInjections IntramuscularInjectionsBiomaterialsMolecular GeneticsTransgeneAnimals; Dependovirus; Erythropoietin; Genetic Vectors; Humans; In Vitro Techniques; Injections Intramuscular; Insulin-Like Growth Factor I; Mice; Polymerase Chain Reaction; Reproducibility of Results; Transgenes; Agricultural and Biological Sciences (all); Biochemistry Genetics and Molecular Biology (all); Medicine (all)Genetic ElementsGeneticsAnimalsHumansSports and Exercise MedicineMolecular Biology TechniquesMolecular BiologyErythropoietinClinical GeneticsBiochemistry Genetics and Molecular Biology (all)796 SportAnimalIn Vitro TechniqueGene AmplificationBiology and Life SciencesReproducibility of ResultsHuman GeneticsDNAAgricultural and Biological Sciences (all)Mutation
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Phosphoglucomutase (EC 2.7.5.1.) and adenylate kinase (EC 2.7.4.3.) typings in Koreans and Irish.

1969

PGM1 and AK phenotypes were determined in samples from Korea and Ireland. the frequencies of PGM 1 1 genes amount to 0.916 in Koreans and 0.864 in Irish. AK1 frequencies come to 0.933 in Koreans and 0.873 in Irish.

GeneticsAdultMaleKoreaPolymorphism GeneticPhosphotransferasesAdenylate kinaseBiologyPhenotypeMolecular medicinelanguage.human_languageHuman geneticsIrishPhosphoglucomutasePGM1GeneticslanguageHumansPhosphoglucomutaseFemaleGeneIrelandGenetics (clinical)Humangenetik
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A high-throughput genome-wide siRNA screen for ciliogenesis identifies new ciliary functional components and ciliopathy genes.

2015

Defects in primary cilium biogenesis underlie the ciliopathies, a growing group of genetic disorders. We describe the first whole genome siRNA-based reverse genetics screen for defects in biogenesis and/or maintenance of the primary cilium, obtaining a global resource for investigation and interventions into the processes that are critical for the ciliary system. In total, we identified 83 candidate ciliogenesis and ciliopathy genes, including 15 components of the ubiquitin-proteasome system. The validated hits also include 12 encoding G-protein-coupled receptors, and three encoding pre-mRNA processing factors (PRPF6, PRPF8 and PRPF31) mutated in autosomal dominant retinitis pigmentosa. Com…

GeneticsCandidate genePRPF31CiliumCell BiologyBiologymedicine.diseaseCiliopathiesHuman geneticsCiliopathyCiliogenesismedicineOral PresentationExome sequencing
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Lack of linkage between gene(s) controlling the synthesis of the seventh component of complement and the HLA region on chromosome No. 6 in man.

1976

The family of an individual was studied who lacks the seventh component of complement in his serum (C7 homozygous deficiency). Both parents are C7 heterozygousdeficient. In this investigation, the following parameters were determined: complement components in functional and immunochemical tests; HLA-A,B antigens, HLA-D (MLC) determinants; the Bf system; glyoxalase I and B cell antigens. No evidence for linkage between the immunogenetic linkage group on chromosome 6 and gene(s) controlling the synthesis of the seventh component of complement was obtained. This is in accordance with the assumption that only genes controlling components of the initiating rather than the membrane attack unit of…

GeneticsChromosomes Human 6-12 and XMaleGenetic LinkageChromosomeHuman leukocyte antigenComplement System ProteinsBiologyComplement factor BHuman geneticsComplement C7Complement (complexity)medicine.anatomical_structureAntigenHLA AntigensHistocompatibility AntigensGeneticsmedicineHumansLymphocyte Culture Test MixedChildGeneGenetics (clinical)B cellHuman genetics
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Molecular and clinical characterization of a small duplication Xp in a human female with psychiatric disorders

2011

CGH techniques allow us to detect small duplications thatoccur in humans with phenotypic manifestations and demon-strate the importance of these duplications in the etiologyof neurodevelopmental impairment. As in the case of otherX-linked disorders, X-inactivation plays a major role in theclinical expression of such X chromosomal imbalances withusually milder symptoms in females than in males. Mostmale patients carrying Xp duplication have mental retarda-tion (X-linked mental retardation) and variable facial dys-morphic features (Gimelli

GeneticsChromosomes Human XComparative Genomic HybridizationMental Disordershuman geneticsBiologyPhenotypeHuman geneticspsychiatric disorderfunctional Xp disomySettore MED/38 - Pediatria Generale E SpecialisticaSettore MED/03 - Genetica MedicaX Chromosome InactivationChild PreschoolGene duplicationChromosome DuplicationGeneticsMental Retardation X-LinkedHumansarray CGHFemaleChildfunctional Xp disomy; array CGH; psychiatric disorders; human geneticsGenetic Association StudiesSex Chromosome Aberrations
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Human complement C81 (C8 A) polymorphism: detection and segregation of new variants

1993

In addition to the earlier detected C81(A) rare variants A1, A2 (now A3) and B1 (now B2), six new rare variants (C81 A2 new, A4, A5, A6, M1 and B1new) are described within the polymorphism of the eighth component of human complement (alpha-gamma chain subunit). Except for A3, all rare C81 A variants are only detected by isoelectric focusing, and not by SDS polyacrylamide gel electrophoresis (PAGE), in the alpha-gamma subunit. In one individual out of approximately 700 individuals studied, a reversed position of the common allele (B vs A) was observed by SDS PAGE and the isofocusing technique. The segregation of A1, A3 and A4 could be followed in putative father/child combinations.

GeneticsComplement (group theory)Polymorphism GeneticIsoelectric focusingProtein subunitBiologyComplement C8Molecular biologyAllotypeHuman geneticsPolymorphism (computer science)GeneticsHumansElectrophoresis Polyacrylamide GelIsoelectric FocusingAllelePolyacrylamide gel electrophoresisAllelesGenetics (clinical)Human Genetics
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Investigations on the genetics and population genetics of the ?2 polymorphism

1970

The results of studies on 49 families with 107 children and various populations of Caucasoid, Negroid and Mongoloid origin concerning the genetics and population genetics of the β2-glycoprotein I polymorphism are reported. In general the genetical model proposed by Cleve (1968) is confirmed: two autosomal alleles BgN and BgD controlling the phenotypes Bg N-N, Bg N-D and Bg D-D. However, divergences from this model were found in two families. They indicate the assumption of non-genetic factors influencing the phenotype expression rather than more complicated genetical control mechanisms. Within Caucasoid populations phenotype and gene frequencies show almost a homogeneous distribution. This …

GeneticsEvolutionary biologyGeneticsBeta 2-Glycoprotein IPopulation geneticsMongoloidAlleleBiologyGeneAllele frequencyPhenotypeGenetics (clinical)Human geneticsHuman Genetics
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Reciprocal translocation t(1;18)(p32;q21) in a patient with some phenotypical anomalies

1987

The authors report on a case of 1;18 translocation and request contact with any colleagues who have observed similar cases.

GeneticsEyelashesChromosomal translocationBiologyPhenotypeMolecular medicineTranslocation GeneticHuman geneticsPhenotypeChromosomes Human Pair 1GeneticsHumansFemaleEyebrowsMetabolic diseaseChildChromosomes Human Pair 16Genetics (clinical)Human Genetics
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Inherited pericentric inversion of human chromosome 5.

1983

Une inversion pericentrique heterozygote du chromosome 5 a ete decouverte dans les cellules de la moelle osseuse et dans les lymphocytes peripheriques stimules par le phytohemagglutinine d'une femme de 75 ans atteinte de myelofibrose et de metaplasie myeloide. L'inversion etait presente dans 100% des metaphases etudiees. Les points de cessure du chromosome 5 etaient localises en k15 et q10. Une etude de la famille est presentee

GeneticsHeterozygoteChromosomeHeterozygote advantageBiologyHuman geneticsPrimary MyelofibrosisChromosome InversionGeneticsHumansFemaleChromosomes Human 4-5Genetics (clinical)Chromosomal inversionAgedHuman genetics
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