Search results for "Human medicine"

showing 10 items of 256 documents

Mapping local patterns of childhood overweight and wasting in low- and middle-income countries between 2000 and 2017

2020

Artículo con numerosos autores, sólo se mencionan el primero, los de la UAM y grupo colectivo

MaleLocal patternsDouble burdenBörnResearch & Experimental MedicineSjúkdómseinkenniDOUBLE BURDENChildhood overweightLífefnafræðiLæknisfræði0302 clinical medicineSyndemicChild11 Medical and Health Sciencesunder 5 years of ageGeneral Medicine3. Good healthGeographyMedicine Research & ExperimentalChild PreschoolIncomeGROWTHAFRICAmedicine.medical_specialtyBiochemistry & Molecular BiologyRJMedicinaImmunologyeducationMODELSwa_395General Biochemistry Genetics and Molecular BiologyArticleG03 medical and health sciencesHumansAuthor CorrectionDeveloping CountriesPovertyBiologyLBD Double Burden of Malnutrition CollaboratorsDemographyScience & TechnologyWasting SyndromePublic healthMORTALITYInfantNæringarskorturmedicine.diseaseObesityTRENDSsigns and symptomsSocial ClassRisk factorsSameindalíffræðiITC-ISI-JOURNAL-ARTICLEUNDERNUTRITIONHuman medicineClinical Medicine030217 neurology & neurosurgeryPediatric ObesityobesityOffitaÁhættuþættirGeographic MappingOverweightRA0421Global healthrisk factors030212 general & internal medicineSigns and symptomsWastingMalnutrition Global Burden of Diseases Global Nutrition low- and middle-income countries2. Zero hungerPublic health1. No povertyPublic Health Global Health Social Medicine and EpidemiologyA900 Others in Medicine and DentistryChildhood wastingPREVALENCEChemistryMappingFemaleLýðheilsamedicine.symptomLife Sciences & BiomedicineGROWTH FAILURENutritional StatusmalnutritionITC-HYBRIDws_115childrenEnvironmental healthmedicineErfðafræðiObesitywd_200MalnutritionInfant NewbornKlinisk medicinCell BiologyOverweightMalnutritionFolkhälsovetenskap global hälsa socialmedicin och epidemiologi3121 General medicine internal medicine and other clinical medicineNA
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Fish and seafood consumption during pregnancy and the risk of asthma and allergic rhinitis in childhood

2017

Background: It has been suggested that prenatal exposure to n-3 long-chain fatty acids protects against asthma and other allergy-related diseases later in childhood. The extent to which fish intake in pregnancy protects against child asthma and rhinitis symptoms remains unclear. We aimed to assess whether fish and seafood consumption in pregnancy is associated with childhood wheeze, asthma and allergic rhinitis. Methods: We pooled individual data from 60 774 mother-child pairs participating in 18 European and US birth cohort studies. Information on wheeze, asthma and allergic rhinitis prevalence was collected using validated questionnaires. The time periods of interest were: infancy (0-2 ye…

MaleMediterranean dietEpidemiologyCHILDRENCohort Studies0302 clinical medicineFish Fruit and Other FoodSurveys and QuestionnairesSeafood consumptionPrevalence030212 general & internal medicineseafoodChildPrenatal Nutritional Physiological Phenomena2. Zero hungermedicine.diagnostic_testJAPANESE INFANTSGeneral Medicine3. Good healthEuropeMEDITERRANEAN DIETLUNG-FUNCTIONChild PreschoolRegression AnalysisFemaleGeneration RpregnancyFATTY-ACIDSmedicine.symptomallergic rhinitiCohort studyFish consumptionWHEEZEAsthma - ChildrenECZEMA03 medical and health scienceschildrenEnvironmental healthWheezeFatty Acids Omega-3medicineAnimalsHumansRespiratory soundsRespiratory SoundsAsthmaPRENATAL EXPOSUREfishPregnancyallergic rhinitisWheezingwheezingbusiness.industryWheezing; allergic rhinitis; asthma; children; fish; pregnancy; seafoodMATERNAL FOOD-CONSUMPTIONInfant NewbornInfantasthmamedicine.diseaseRhinitis AllergicUnited States030228 respiratory systemAllergic rhinitis - ChildrenRelative riskGENERATION RHuman medicinebusinessInternational Journal of Epidemiology
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Phenotypic and biochemical analysis of an international cohort of individuals with variants in NAA10 and NAA15.

2019

Abstract N-alpha-acetylation is one of the most common co-translational protein modifications in humans and is essential for normal cell function. NAA10 encodes for the enzyme NAA10, which is the catalytic subunit in the N-terminal acetyltransferase A (NatA) complex. The auxiliary and regulatory subunits of the NatA complex are NAA15 and Huntington-interacting protein (HYPK), respectively. Through a genotype-first approach with exome sequencing, we identified and phenotypically characterized 30 individuals from 30 unrelated families with 17 different de novo or inherited, dominantly acting missense variants in NAA10 or NAA15. Clinical features of affected individuals include variable levels…

MaleModels Molecular0301 basic medicineProtein ConformationMicrophthalmia0302 clinical medicineEnzyme StabilityMissense mutationN-Terminal Acetyltransferase EChildN-Terminal Acetyltransferase AExome sequencingGenetics (clinical)GeneticsbiologyGeneral MedicinePhenotypeRecombinant ProteinsChemistryPhenotypeChild PreschoolHMG-CoA reductaseCohortFemaleGeneral ArticleCorrigendumAdultNatA complexmedicine.medical_specialtyAdolescentGenotypeFrameshift mutationStructure-Activity RelationshipYoung Adult03 medical and health sciencesMolecular geneticsGeneticsmedicineHumansGenetic Predisposition to DiseaseGenetic TestingAlleleBiologyMolecular BiologyAllelesGenetic Association StudiesComputational BiologyFaciesGenetic VariationInfantmedicine.diseaseEnzyme ActivationLenz microphthalmia syndrome030104 developmental biologyGenetic LociMutationbiology.proteinHuman medicineBiomarkers030217 neurology & neurosurgeryNAA15Human molecular genetics
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Haploinsufficiency of the NOTCH1 receptor as a cause of Adams-Oliver syndrome with variable cardiac anomalies

2015

Background— Adams–Oliver syndrome (AOS) is a rare disorder characterized by congenital limb defects and scalp cutis aplasia. In a proportion of cases, notable cardiac involvement is also apparent. Despite recent advances in the understanding of the genetic basis of AOS, for the majority of affected subjects, the underlying molecular defect remains unresolved. This study aimed to identify novel genetic determinants of AOS. Methods and Results— Whole-exome sequencing was performed for 12 probands, each with a clinical diagnosis of AOS. Analyses led to the identification of novel heterozygous truncating NOTCH1 mutations (c.1649dupA and c.6049_6050delTC) in 2 kindreds in which AOS was segregat…

MaleModels MolecularProbandreceptorGene ExpressionHaploinsufficiencyNOTCH1Ectodermal DysplasiaMissense mutationExomeReceptor Notch1ChildExomeGenetics (clinical)GeneticsReverse Transcriptase Polymerase Chain ReactionAutosomal dominant traitMiddle AgedPedigreeembryonic structuresheart defectscardiovascular systemFemaleCardiology and Cardiovascular MedicineHaploinsufficiencySignal TransductionAdultHeart Defects CongenitalAdolescentLimb Deformities CongenitalNotch signaling pathwayBiologyArticleYoung AdultAdams-Oliver syndromeGeneticsmedicineHumansGenetic Predisposition to DiseaseGeneFamily HealthBase SequencecongenitalAdams-Oliver syndrome; genetics; haploinsufficiency; heart defects; congenital; receptor; NOTCH1; Cardiology and Cardiovascular Medicine; Genetics (clinical); GeneticsSequence Analysis DNAmedicine.diseaseProtein Structure TertiaryScalp DermatosesHuman medicineAdams–Oliver syndromeCirculation. Cardiovascular genetics
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Rare variants in the genetic background modulate cognitive and developmental phenotypes in individuals carrying disease-associated variants

2019

Purpose: To assess the contribution of rare variants in the genetic background toward variability of neurodevelopmental phenotypes in individuals with rare copy-number variants (CNVs) and gene-disruptive variants. Methods: We analyzed quantitative clinical information, exome sequencing, and microarray data from 757 probands and 233 parents and siblings who carry disease-associated variants. Results: The number of rare likely deleterious variants in functionally intolerant genes (“other hits”) correlated with expression of neurodevelopmental phenotypes in probands with 16p12.1 deletion (n=23, p=0.004) and in autism probands carrying gene-disruptive variants (n=184, p=0.03) compared with thei…

MaleParents0301 basic medicineProbandNeuronalGenetic Carrier Screening16p11.2 deletion030105 genetics & heredityCognitionFamily historyNeural Cell Adhesion MoleculesGenetics (clinical)Exome sequencingSequence DeletionGeneticsGenetic Carrier ScreeningPhenotypePenetrancePedigreePhenotypeAutistic Disorder/genetics; Autistic Disorder/physiopathology; Cell Adhesion Molecules Neuronal/genetics; Chromosomes Human Pair 16/genetics; Cognition/physiology; DNA Copy Number Variations/genetics; Female; Gene Expression Regulation/genetics; Genetic Background; Genetic Carrier Screening; Humans; Male; Methyltransferases/genetics; Nerve Tissue Proteins/genetics; Parents; Pedigree; Phenotype; Proteins/genetics; Sequence Deletion/genetics; Siblings; 16p11.2 deletion; CNV; autism; modifier; phenotypic variabilityFemaleGenetic BackgroundHumanDNA Copy Number VariationsCell Adhesion Molecules NeuronalCNVautismNerve Tissue ProteinsBiologyChromosomesArticle03 medical and health sciencesmental disordersmedicineHumansAutistic DisorderBiologyGenemodifierPair 16SiblingsCalcium-Binding ProteinsProteinsMethyltransferasesmedicine.disease16p11.2 deletion; autism; CNV; modifier; phenotypic variability; Genetics (clinical)Cytoskeletal Proteins030104 developmental biologyGene Expression Regulation[SDV.GEN.GH]Life Sciences [q-bio]/Genetics/Human geneticsAutismphenotypic variabilityHuman medicine16p11.2 deletion; autism; CNV; modifier; phenotypic variability; Autistic Disorder; Cell Adhesion Molecules Neuronal; Chromosomes Human Pair 16; Cognition; DNA Copy Number Variations; Female; Gene Expression Regulation; Genetic Background; Humans; Male; Methyltransferases; Nerve Tissue Proteins; Parents; Pedigree; Phenotype; Proteins; Sequence Deletion; Siblings; Genetic Carrier ScreeningCell Adhesion MoleculesChromosomes Human Pair 16Transcription FactorsGenetics in Medicine
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A multi-centre clinico-genetic analysis of the VPS35 gene in Parkinson disease indicates reduced penetrance for disease-associated variants

2012

Abstract: Background Two recent studies identified a mutation (p.Asp620Asn) in the vacuolar protein sorting 35 gene as a cause for an autosomal dominant form of Parkinson disease. Although additional missense variants were described, their pathogenic role yet remains inconclusive. Methods and results We performed the largest multi-center study to ascertain the frequency and pathogenicity of the reported vacuolar protein sorting 35 gene variants in more than 15,000 individuals worldwide. p.Asp620Asn was detected in 5 familial and 2 sporadic PD cases and not in healthy controls, p.Leu774Met in 6 cases and 1 control, p.Gly51Ser in 3 cases and 2 controls. Overall analyses did not reveal any sig…

MaleParkinson's diseasePopulationVesicular Transport ProteinsLocus (genetics)DiseaseBiologyVPS35 protein humanBioinformaticsgenetics [Vesicular Transport Proteins]genetics [Parkinson Disease]Risk Factorsmedicinemetabolism [Vesicular Transport Proteins]GeneticsMissense mutationVPS35 GeneHumansGenetic epidemiologyGenetic Predisposition to Diseaseddc:6101506Genome-wideeducationGenetics (clinical)Genetic Association StudiesGeneticsVacuolar protein sortingeducation.field_of_studyGenotype-Phenotype CorrelationsParkinson DiseaseComplex traitsmedicine.diseasePenetranceddc:MutationFemaleHuman medicineParkinson-s diseaseJournal of Medical Genetics
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Pulmonary vascular endothelialitis, thrombosis, and angiogenesis in Covid-19

2020

Abstract: BackgroundProgressive respiratory failure is the primary cause of death in the coronavirus disease 2019 (Covid-19) pandemic. Despite widespread interest in the pathophysiology of the disease, relatively little is known about the associated morphologic and molecular changes in the peripheral lung of patients who die from Covid-19. MethodsWe examined 7 lungs obtained during autopsy from patients who died from Covid-19 and compared them with 7 lungs obtained during autopsy from patients who died from acute respiratory distress syndrome (ARDS) secondary to influenza A(H1N1) infection and 10 age-matched, uninfected control lungs. The lungs were studied with the use of seven-color immun…

MalePathologymedicine.medical_specialtyAngiogenesisPneumonia ViralAutopsy030204 cardiovascular system & hematologyNeovascularizationBetacoronavirus03 medical and health sciencesInfluenza A Virus H1N1 Subtype0302 clinical medicineInfluenza HumanmedicineHumans030212 general & internal medicineLungPandemicsAgedCause of deathAged 80 and overRespiratory Distress SyndromeNeovascularization PathologicSARS-CoV-2business.industryCOVID-19ThrombosisGeneral MedicineMiddle Agedmedicine.diseaseThrombosisPathophysiology3. Good healthPneumoniaFemaleAutopsyEndothelium VascularHuman medicinemedicine.symptomCoronavirus InfectionsRespiratory InsufficiencybusinessHuman PathologyThe New England journal of medicine
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Fish Intake in Pregnancy and Child Growth: A Pooled Analysis of 15 European and US Birth Cohorts

2016

Importance Maternal fish intake in pregnancy has been shown to influence fetal growth. The extent to which fish intake affects childhood growth and obesity remains unclear. Objective To examine whether fish intake in pregnancy is associated with offspring growth and the risk of childhood overweight and obesity. Design, Setting, and Participants Multicenter, population-based birth cohort study of singleton deliveries from 1996 to 2011 in Belgium, France, Greece, Ireland, Italy, the Netherlands, Norway, Poland, Portugal, Spain, and Massachusetts. A total of 26 184 pregnant women and their children were followed up at 2-year intervals until the age of 6 years. Exposures Consumption of fish dur…

MalePediatric ObesityPediatricsChild growthFetal Development/physiologyOverweightBody Mass IndexCohort StudiesFetal DevelopmentChild Development0302 clinical medicinePregnancyRisk Factors030212 general & internal medicineChild2. Zero hungereducation.field_of_studyFish intakeFishes3. Good healthEuropePediatric Obesity/epidemiologyChild PreschoolFemalemedicine.symptomCohort studymedicine.medical_specialtyOffspringPopulation030209 endocrinology & metabolismOverweight/epidemiologyArticleChildhood obesity03 medical and health sciencesmedicineAnimalsHumanseducationNutritionPregnancyPrenatal nutritionbusiness.industryInfant NewbornInfantOverweightmedicine.diseaseUnited StatesDietPregnancy ComplicationsSeafoodPediatrics Perinatology and Child HealthHuman medicinebusinessBody mass indexFollow-Up StudiesDemography
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Incidence of severe critical events in paediatric anaesthesia (APRICOT): a prospective multicentre observational study in 261 hospitals in Europe

2017

Background Little is known about the incidence of severe critical events in children undergoing general anaesthesia in Europe. We aimed to identify the incidence, nature, and outcome of severe critical events in children undergoing anaesthesia, and the associated potential risk factors. Methods The APRICOT study was a prospective observational multicentre cohort study of children from birth to 15 years of age undergoing elective or urgent anaesthesia for diagnostic or surgical procedures. Children were eligible for inclusion during a 2-week period determined prospectively by each centre. There were 261 participating centres across 33 European countries. The primary endpoint was the occurenc…

MalePediatricsHealth StatusOPERATING-ROOMRespiratory Tract DiseasesCHILDREN0302 clinical medicineREGIONAL ANESTHESIAPostoperative Complications030202 anesthesiologyCARDIAC-ARRESTMedicineGeneral anaesthesiaProspective StudiesProspective cohort studyChildIntraoperative ComplicationsCOMPLICATIONSddc:617Incidence (epidemiology)Mortality ratemusculoskeletal neural and ocular physiologyIncidenceAge FactorsHospitalsEuropeCardiovascular DiseasesChild PreschoolRESPIRATORY ADVERSE EVENTSFemaleClinical CompetenceCohort studyPulmonary and Respiratory Medicinemedicine.medical_specialtyAdolescentmacromolecular substancesAnesthesia GeneralDrug Hypersensitivity03 medical and health sciencesMORBIDITYJournal ArticleHumansbusiness.industryInfant NewbornInfant030208 emergency & critical care medicinePerioperativenervous systemPROSPECTIVE COHORTRelative riskRISK-FACTORSObservational studyHuman medicineNervous System DiseasesbusinessSYSTEM
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NEXMIF encephalopathy: an X-linked disorder with male and female phenotypic patterns

2021

Contains fulltext : 231688.pdf (Publisher’s version ) (Closed access) PURPOSE: Pathogenic variants in the X-linked gene NEXMIF (previously KIAA2022) are associated with intellectual disability (ID), autism spectrum disorder, and epilepsy. We aimed to delineate the female and male phenotypic spectrum of NEXMIF encephalopathy. METHODS: Through an international collaboration, we analyzed the phenotypes and genotypes of 87 patients with NEXMIF encephalopathy. RESULTS: Sixty-three females and 24 males (46 new patients) with NEXMIF encephalopathy were studied, with 30 novel variants. Phenotypic features included developmental delay/ID in 86/87 (99%), seizures in 71/86 (83%) and multiple comorbidi…

MalePediatricsmedicine.medical_specialtyINTELLECTUAL DISABILITYAutism Spectrum DisorderEncephalopathyNerve Tissue ProteinsILAE COMMISSIONMOSAICISMEpilepsy/geneticsCLASSIFICATIONEpilepsyBrain Diseases/geneticsGenes X-LinkedSeizuresIntellectual disabilityGenotypemedicineHumansdevelopmental and epileptic encephalopathyMYOCLONIAAtonic seizureGenetics (clinical)Brain Diseasesddc:618Neurodevelopmental disorders Donders Center for Medical Neuroscience [Radboudumc 7]KIAA2022business.industryMUTATIONSmedicine.diseasePhenotypeAutism Spectrum Disorder/geneticsGenes X-Linked/geneticsAutism spectrum disorderintellectual disabilityNEXMIFAutismepilepsyFemaleINACTIVATIONHuman medicineSeizures/geneticsbusinessPOSITION PAPERGenetics in Medicine
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