Search results for "Hyla"

showing 10 items of 2227 documents

Dysregulation of DNA methylation induced by past arsenic treatment causes persistent genomic instability in mammalian cells

2015

The mechanisms by which arsenic-induced genomic instability is initiated and maintained are poorly understood. To investigate potential epigenetic mechanisms, in this study we evaluated global DNA methylation levels in V79 cells and human HaCaT keratinocytes at several time points during expanded growth of cell cultures following removal of arsenite exposures. We have found altered genomic methylation patterns that persisted up to 40 cell generations in HaCaT cells after the treatments were withdrawn. Moreover, mRNA expression levels were evaluated by RT-PCR for DNMT1, DNMT3A, DNMT3B, HMLH1, and HMSH2 genes, demonstrating that the down regulation of DNMT3A and DNMT3B genes, but not DNMT1, o…

0301 basic medicineGenome instabilityEpidemiologyHealth Toxicology and MutagenesisMethylationEpigenomeBiology03 medical and health sciences030104 developmental biologyDNA methylationCancer researchDNA mismatch repairEpigeneticsReprogrammingGenetics (clinical)DNA hypomethylationEnvironmental and Molecular Mutagenesis
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Basic Concepts in Molecular Biology Related to Genetics and Epigenetics.

2017

The observation that "one size does not fit all" for the prevention and treatment of cardiovascular disease, among other diseases, has driven the concept of precision medicine. The goal of precision medicine is to provide the best-targeted interventions tailored to an individual's genome. The human genome is composed of billions of sequence arrangements containing a code that controls how genes are expressed. This code depends on other nonstatic regulators that surround the DNA and constitute the epigenome. Moreover, environmental factors also play an important role in this complex regulation. This review provides a general perspective on the basic concepts of molecular biology related to g…

0301 basic medicineGenome-wide association study030204 cardiovascular system & hematologyGenomeEpigenesis Genetic03 medical and health sciences0302 clinical medicineComputational epigeneticsMedicineHumansEpigeneticsExerciseGeneticsbiologybusiness.industrySmokingGeneral MedicineEpigenomeDNA MethylationPrecision medicineMolecular biologyDietDNA-Binding ProteinsHistone Code030104 developmental biologyHistoneCardiovascular Diseasesbiology.proteinHuman genomeGene-Environment InteractionbusinessRevista espanola de cardiologia (English ed.)
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Free energy profiles for two ubiquitous damaging agents: methylation and hydroxylation of guanine in B-DNA

2017

International audience; DNA methylation and hydroxylation are two ubiquitous reactions in DNA damage induction, yet insights are scarce concerning the free energy of activation within B-DNA. We resort to multiscale simulations to investigate the attack of a hydroxyl radical and of the primary diazonium onto a guanine embedded in a solvated dodecamer. Reaction free energy profiles characterize two strongly exergonic processes, yet allow unprecedented quantification of the barrier towards this damage reaction, not higher than 6 kcal mol−1 and sometimes inexistent, and of the exergonicities. In the case of the [G(C8)-OH]˙ intermediate, we challenge the functional dependence of such simulations…

0301 basic medicineGuanineGuanineDNA damageStereochemistryEntropyGeneral Physics and Astronomy010402 general chemistryHydroxylation01 natural sciencesHydroxylation03 medical and health scienceschemistry.chemical_compoundComputational chemistry[CHIM.ANAL]Chemical Sciences/Analytical chemistryPhysical and Theoretical ChemistryExergonic reactionchemistry.chemical_classificationHydroxyl RadicalBiomoleculeDNA Methylation0104 chemical sciences030104 developmental biologychemistryEnergy TransferDNA methylationHydroxyl radicalDNA B-Form[CHIM.RADIO]Chemical Sciences/RadiochemistryDNA
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Novel deletion in 11p15.5 imprinting center region 1 in a patient with Beckwith-Wiedemann syndrome provides insight into distal enhancer regulation a…

2016

Background Beckwith–Wiedemann syndrome (BWS) is an early-onset overgrowth disorder with a high risk for embryonal tumors. It is mainly caused by dysregulation of imprinted genes on chromosome 11p15.5; however, the driving forces in the development of tumors are not fully understood. Procedure We report on a female patient presenting with macrosomia, macroglossia, organomegaly and extensive bilateral nephroblastomatosis. Adjuvant chemotherapy was initiated; however, the patient developed hepatoblastoma and Wilms tumor at 5 and 12 months of age, respectively. Subsequent radiofrequency ablation of the liver tumor and partial nephrectomy followed by consolidation therapy achieved complete remis…

0301 basic medicineHepatoblastomaPathologymedicine.medical_specialtyBeckwith-Wiedemann SyndromeBeckwith–Wiedemann syndrome030105 genetics & hereditymedicine.disease_cause03 medical and health sciencesGenomic ImprintingInsulin-Like Growth Factor IIMacroglossiaMedicineHumansImprinting (psychology)NephroblastomatosisSequence Deletionbusiness.industryChromosomes Human Pair 11Infant NewbornWilms' tumorHematologyDNA Methylationmedicine.diseasePrognosis030104 developmental biologyCell Transformation NeoplasticPhenotypeOncologyPediatrics Perinatology and Child HealthCancer researchFemalemedicine.symptombusinessGenomic imprintingCarcinogenesisPediatric bloodcancer
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Integrative Genome-Scale DNA Methylation Analysis of a Large and Unselected Cohort Reveals 5 Distinct Subtypes of Colorectal Adenocarcinomas

2019

BACKGROUND & AIMS: Colorectal cancer is an epigenetically heterogeneous disease, however, the extent and spectrum of the CpG island methylator phenotype (CIMP) is not clear. METHODS: Genome-scale methylation and transcript expression were measured by DNA Methylation and RNA expression microarray in 216 unselected colorectal cancers, and findings were validated using The Cancer Genome Atlas 450K and RNA sequencing data. Mutations in epigenetic regulators were assessed using CIMP-subtyped Cancer Genome Atlas exomes. RESULTS: CIMP-high cancers dichotomized into CIMP-H1 and CIMP-H2 based on methylation profile. KRAS mutation was associated significantly with CIMP-H2 cancers, but not CIMP-H1 can…

0301 basic medicineHepatologyCpG Island Methylator PhenotypeColorectal cancerGastroenterologyMethylationBiologymedicine.disease_causemedicine.diseasedigestive system diseases3. Good health03 medical and health sciences030104 developmental biology0302 clinical medicineDNA methylationCancer researchmedicinelcsh:Diseases of the digestive system. Gastroenterology030211 gastroenterology & hepatologyKRASEpigeneticslcsh:RC799-869neoplasmsGeneExome sequencingCellular and Molecular Gastroenterology and Hepatology
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The DNA methylation profile of human spermatogonia at single-cell- and single-allele-resolution refutes its role in spermatogonial stem cell function…

2019

Human spermatogonial stem cells (hSSCs) have potential in fertility preservation of prepubertal boys or in treatment of male adults suffering from meiotic arrest. Prior to therapeutic application, in vitro propagation of rare hSSCs is mandatory. As the published data points to epigenetic alterations in long-term cell culture of spermatogonia (SPG), an initial characterisation of their DNA methylation state is important. Testicular biopsies from five adult normogonadotropic patients were converted into aggregate-free cell suspensions. FGFR3-positive (FGFR3+) SPG, resembling a very early stem cell state, were labelled with magnetic beads and isolated in addition to unlabelled SPG (FGFR3-). DN…

0301 basic medicineHomeobox protein NANOGMaleEmbryologyBiologyEpigenesis Genetic03 medical and health sciences0302 clinical medicineGeneticsmedicineHumansReceptor Fibroblast Growth Factor Type 3EpigeneticsSpermatogenesisMolecular BiologyAllelesMEG3030219 obstetrics & reproductive medicineKCNQ1OT1Stem CellsObstetrics and GynecologyCell DifferentiationCell BiologyMethylationDNA MethylationMolecular biologySpermatozoaSpermatogonia030104 developmental biologymedicine.anatomical_structureReproductive MedicineDNA methylationGenomic imprintingGerm cellDevelopmental BiologyMolecular human reproduction
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Wiedemann-Steiner syndrome as a major cause of syndromic intellectual disability: A study of 33 French cases.

2018

International audience; Wiedemann-Steiner syndrome (WSS) is a rare syndromic condition in which intellectual disability (ID) is associated with hypertrichosis cubiti, short stature, and characteristic facies. Following the identification of the causative gene (KMT2A) in 2012, only 31 cases of WSS have been described precisely in the literature. We report on 33 French individuals with a KMT2A mutation confirmed by targeted gene sequencing, high-throughput sequencing or exome sequencing. Patients' molecular and clinical features were recorded and compared with the literature data. On the molecular level, we found 29 novel mutations. We observed autosomal dominant transmission of WSS in 3 fami…

0301 basic medicineHypertrichosisMalePediatrics[SDV]Life Sciences [q-bio]MESH: Magnetic Resonance ImagingPathognomonicMESH: ChildIntellectual disabilityMESH: SyndromeChildMESH: High-Throughput Nucleotide SequencingGenetics (clinical)Exome sequencingComputingMilieux_MISCELLANEOUSbiologyWiedemann-Steiner syndromeHigh-Throughput Nucleotide SequencingSyndromeKMT2AMESH: Amino Acid SubstitutionMagnetic Resonance Imaginghypertrichosis3. Good healthhairinessKMT2APhenotypeWiedemann-Steiner syndromeChild Preschoolcardiovascular systemFemaleDisease SusceptibilityFrancemedicine.symptomMESH: Tomography X-Ray ComputedMyeloid-Lymphoid Leukemia Proteinmedicine.medical_specialtyMESH: MutationAdolescentMESH: Disease SusceptibilityMESH: PhenotypeShort statureMESH: Intellectual Disability03 medical and health sciencesHypertrichosis cubitiIntellectual DisabilityGeneticsmedicineHumanshistone methylationMESH: Adolescent[SDV.GEN]Life Sciences [q-bio]/GeneticsMESH: Humansbusiness.industryMESH: Child PreschoolMESH: Histone-Lysine N-MethyltransferaseHistone-Lysine N-Methyltransferasemedicine.diseaseMESH: MaleMESH: France030104 developmental biology[SDV.GEN.GH]Life Sciences [q-bio]/Genetics/Human geneticsAmino Acid SubstitutionMESH: Myeloid-Lymphoid Leukemia ProteinMutationbiology.proteinbusinessTomography X-Ray ComputedMESH: FemaleClinical genetics
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Influence of gut microbiota on neuropsychiatric disorders

2017

The last decade has witnessed a growing appreciation of the fundamental role played by an early assembly of a diverse and balanced gut microbiota and its subsequent maintenance for future health of the host. Gut microbiota is currently viewed as a key regulator of a fluent bidirectional dialogue between the gut and the brain (gut-brain axis). A number of preclinical studies have suggested that the microbiota and its genome (microbiome) may play a key role in neurodevelopmental and neurodegenerative disorders. Furthermore, alterations in the gut microbiota composition in humans have also been linked to a variety of neuropsychiatric conditions, including depression, autism and Parkinson’s dis…

0301 basic medicineHypothalamo-Hypophyseal SystemGut–brain axisPituitary-Adrenal SystemDiseaseGut floraBioinformaticsdigestive systemEpigenesis Genetic03 medical and health sciences0302 clinical medicinemedicineAnimalsHumansBrain-gut axisMicrobiomePsychiatric conditionsbiologyMicrobiotaGastrointestinal MicrobiomeGastroenterologyBrainNeurodegenerative DiseasesMinireviewsGeneral MedicineDNA MethylationFecal Microbiota TransplantationMental illnessmedicine.diseasebiology.organism_classificationGastrointestinal MicrobiomeTransplantationDisease Models Animal030104 developmental biologyNeurodevelopmental DisordersDysbiosisMental healthMicrobiomeDysbiosisStress Psychological030217 neurology & neurosurgeryWorld Journal of Gastroenterology
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Epigenetic IVD Tests for Personalized Precision Medicine in Cancer

2019

Epigenetic alterations play a key role in the initiation and progression of cancer. Therefore, it is possible to use epigenetic marks as biomarkers for predictive and precision medicine in cancer. Precision medicine is poised to impact clinical practice, patients, and healthcare systems. The objective of this review is to provide an overview of the epigenetic testing landscape in cancer by examining commercially available epigenetic-based in vitro diagnostic tests for colon, breast, cervical, glioblastoma, lung cancers, and for cancers of unknown origin. We compile current commercial epigenetic tests based on epigenetic biomarkers (i.e., DNA methylation, miRNAs, and histones) that can actua…

0301 basic medicineIn Vitro Diagnostic (IVD)lcsh:QH426-470precision medicineReviewBioinformatics03 medical and health sciences0302 clinical medicinemicroRNAGeneticsMedicineEpigeneticscfDNAGenetics (clinical)miRNAEpigenetic biomarkersDNA methylationbiologybusiness.industryCancerepigenetic biomarkerPrecision medicinemedicine.diseaselcsh:Genetics030104 developmental biologyHistone030220 oncology & carcinogenesisDNA methylationcirculating nucleosomesbiology.proteinMolecular MedicinebusinessGlioblastomaFrontiers in Genetics
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Loss-of-function and missense variants in NSD2 cause decreased methylation activity and are associated with a distinct developmental phenotype

2021

Purpose Despite a few recent reports of patients harboring truncating variants in NSD2, a gene considered critical for the Wolf–Hirschhorn syndrome (WHS) phenotype, the clinical spectrum associated with NSD2 pathogenic variants remains poorly understood. Methods We collected a comprehensive series of 18 unpublished patients carrying heterozygous missense, elongating, or truncating NSD2 variants; compared their clinical data to the typical WHS phenotype after pooling them with ten previously described patients; and assessed the underlying molecular mechanism by structural modeling and measuring methylation activity in vitro. Results The core NSD2-associated phenotype includes mostly mild dev…

0301 basic medicineIn silicoBiologyArticleREGION03 medical and health sciencesROGERS-DANKS-SYNDROME0302 clinical medicineMissense mutationHISTONE H3GeneGenetics (clinical)Loss functionGeneticsNeurodevelopmental disorders Donders Center for Medical Neuroscience [Radboudumc 7]DELETIONDEFECTSMethylationPhenotypeLYSINE 36030104 developmental biologyMolecular mechanismWOLF-HIRSCHHORN-SYNDROME030217 neurology & neurosurgeryFunction (biology)Rare cancers Radboud Institute for Health Sciences [Radboudumc 9]Genetics in Medicine
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