Search results for "IMMUNOSENESCENCE"

showing 10 items of 90 documents

Update of Immunosenescence in Cerebral Small Vessel Disease

2020

Aging of the central nervous system (CNS) is closely associated with chronic sterile low-grade inflammation in older organisms and related immune response. As an amplifier for neuro-inflammaging, immunosenescence remodels and deteriorates immune systems gradually with the passage of time, and finally contributes to severe outcomes like stroke, dementia and neurodegeneration in elderly adults. Cerebral small vessel disease (CSVD), one of the major causes of vascular dementia, has an intensive connection with the inflammatory response and immunosenescence plays a crucial role in the pathology of this disorder. In this review, we discuss the impact of immunosenescence on the development of CSV…

0301 basic medicinelcsh:Immunologic diseases. AllergyImmunologyInflammationDiseaseReview03 medical and health sciences0302 clinical medicineImmune systemmedicineDementiaAnimalsHumansImmunology and AllergyVascular dementiaStrokeimmunosenescenceMechanism (biology)business.industrycerebral small vessel diseasepathogenesisagingImmunosenescencemedicine.disease030104 developmental biologyCerebral Small Vessel DiseasesImmunologyinflammagingmedicine.symptombusinesslcsh:RC581-607030217 neurology & neurosurgeryFrontiers in Immunology
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Immune-inflammatory responses in the elderly: An update

2018

EDITORIAL NO ABSTRACT

0301 basic medicinelcsh:Immunologic diseases. Allergymedicine.medical_specialtyAgingAllergyImmunestimulationImmunosenescenceImmunologyMEDLINEClinical nutritionlcsh:Geriatrics03 medical and health sciencesImmune systemElderlymedicineCancerInflammationbiologybusiness.industryPublic healthVaccinationCancerImmunosenescencemedicine.diseaseVaccinationlcsh:RC952-954.6030104 developmental biologyEditorialImmunologybiology.proteinAntibodybusinesslcsh:RC581-607
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The role of immunogenetics in covid‐19

2021

Coronavirus disease 2019 (COVID-19) is induced by SARS-CoV-2 and may arise as a variety of clinical manifestations, ranging from an asymptomatic condition to a life-threatening disease associated with cytokine storm, multiorgan and respiratory failure. The molecular mechanism behind such variability is still under investigation. Several pieces of experimental evidence suggest that genetic variants influencing the onset, maintenance and resolution of the immune response may be fundamental in predicting the evolution of the disease. The identification of genetic variants behind immune system reactivity and function in COVID-19 may help in the elaboration of personalized therapeutic strategies…

0301 basic medicinemedicine.medical_specialtyCoronavirus disease 2019 (COVID-19)ReviewDiseaseImmunogeneticsHuman leukocyte antigenSeverity of Illness IndexCatalysisSARS‐CoV‐2ABO Blood-Group SystemInorganic Chemistrylcsh:Chemistry03 medical and health sciences0302 clinical medicineCOVID‐19HLA AntigensmedicineImmunogeneticsHumansGenetic Predisposition to DiseaseGenetic variabilityPhysical and Theoretical ChemistryIntensive care medicineMolecular Biologylcsh:QH301-705.5SpectroscopyAB0business.industrySARS-CoV-2Organic ChemistryImmunityCOVID-19General MedicineImmunosenescencemedicine.disease3. Good healthComputer Science ApplicationsKIRHLA030104 developmental biologylcsh:Biology (General)lcsh:QD1-999030220 oncology & carcinogenesisIdentification (biology)Disease SusceptibilityCytokine stormbusiness
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A double-negative (IgD−CD27−) B cell population is increased in the peripheral blood of elderly people

2009

The T cell branch of the immune system has been extensively studied in the elderly and it is known that the elderly have impaired immune function, mainly due to the chronic antigenic load that ultimately causes shrinkage of the T cell repertoire and filling of the immunologic space with memory T cells. In the present paper, we describe the IgD(-)CD27(-) double-negative B cell population which (as we have recently described) is higher in the elderly. Most of these cells were IgG(+). Evaluation of the telomere length and expression of the ABCB1 transporter and anti-apoptotic molecule, Bcl2, shows that they have the markers of memory B cells. We also show that these cells do not act as antigen…

AdultAgingATP Binding Cassette Transporter Subfamily BT cellAntigens CD19B-Lymphocyte Subsetschemical and pharmacologic phenomenaYoung AdultB lymphocyte Immunosenescence IgD CD27 Elderly Immunologic memorymedicineHumansCytotoxic T cellATP Binding Cassette Transporter Subfamily B Member 1IL-2 receptorCD40 AntigensCD154Antigen-presenting cellCells CulturedAgedAged 80 and overSettore MED/04 - Patologia Generalebusiness.industryAge FactorsHLA-DR AntigensImmunoglobulin DMiddle AgedTelomereFlow CytometryAcquired immune systemTumor Necrosis Factor Receptor Superfamily Member 7B-1 cellKi-67 Antigenmedicine.anatomical_structureProto-Oncogene Proteins c-bcl-2ImmunologyB7-1 AntigenbusinessImmunologic MemoryCD80Developmental BiologyMechanisms of Ageing and Development
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B cell immunosenescence: different features of naive and memory B cells in elderly.

2011

Elderly people show a reduced protection against new infections and a decreased response to vaccines as a consequence of impairment of both cellular and humoral immunity. In this paper we have studied memory/naive B cells in the elderly, evaluating surface immunoglobulin expression, production of the pro- and anti-inflammatory cytokines, tumor necrosis factor (TNF)-α and interleukin (IL)-10, and presence of somatic hypermutation, focusing on the IgG(+)IgD(-)CD27(-) double negative (DN) B cells that are expanded in the elderly. Our results show that naive B cells from young donors need a sufficiently strong stimulus to be activated "in vitro", while naive B cells from old subjects are able t…

AdultAgingNaive B cellSomatic hypermutationImmunoglobulinsInflammationBiologyLymphocyte ActivationElderlymedicineHumansCytokineB cellCellular SenescenceAgedSettore MED/04 - Patologia GeneraleAged 80 and overB-LymphocytesHypermutationIonomycinGerminal centerImmunosenescenceMiddle AgedMemory B cellsInterleukin-10B-1 cellInterleukin 10medicine.anatomical_structureImmunologyTetradecanoylphorbol AcetateGeriatrics and GerontologyGerontologyCell agingImmunologic MemoryBiogerontology
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Immune profiling of Alzheimer patients

2011

Abstract Alzheimer's disease (AD) is characterized by extracellular senile plaques in the brain, containing amyloid-β peptide (Aβ). We identify immunological differences between AD patients and age-matched controls greater than those related to age itself. The biggest differences were in the CD4 + rather than the CD8 + T cell compartment resulting in lower proportions of naive cells, more late-differentiated cells and higher percentages of activated CD4 + CD25 + T cells without a Treg phenotype in AD patients. Changes to CD4 + cells might be the result of chronic stimulation by Aβ present in the blood. These findings have implications for diagnosis and understanding the aetiology of the dis…

AdultCD4-Positive T-LymphocytesMaleImmunosenescenceT cellImmunologyStimulationDiseaseCD8-Positive T-LymphocytesBiologyYoung AdultAlzheimer DiseaseExtracellularmedicineHumansImmunology and AllergySenile plaquesAgedAged 80 and overSettore MED/04 - Patologia GeneraleGene Expression ProfilingAβ42Age FactorsT cellCell DifferentiationImmunosenescenceMiddle AgedAlzheimer's diseasePhenotypeCD4 Lymphocyte Countmedicine.anatomical_structureNeurologyImmunologyEtiologyFemaleNeurology (clinical)BiomarkersJournal of Neuroimmunology
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Age-related changes in the expression of CD95 (APO1/FAS) on blood lymphocytes☆

1999

Abstract Aging is associated with alterations of the immune system, thought to be related to an increased susceptibility to infectious diseases, and possibly to cancer and autoimmunity in the elderly. In the present paper we report data obtained on freshly collected blood from 148 healthy subjects of different ages (from cord blood to 102 years old). The subjects were divided into seven age classes (cord blood, 3–11 years, 15–39 years, 41–60 years, 61–74 years, 75–84 years, 85–102 years) and their lymphocyte subsets and the expression of the apoptosis-related molecule CD95 were evaluated. In respect of lymphocyte subsets, the major differences were found in the cord-blood samples compared w…

AdultMaleAgingAdolescentT-LymphocytesPopulationchemical and pharmacologic phenomenaBiologymedicine.disease_causeBiochemistryCD19AutoimmunityLeukocyte CountEndocrinologyImmune systemAntigens CDGeneticsmedicineHumansLymphocyte CountLymphocytesfas ReceptorChildeducationMolecular BiologyAgedAged 80 and overeducation.field_of_studyAge FactorsInfant NewbornGene Expression Regulation Developmentalhemic and immune systemsCell BiologyImmunosenescenceMiddle AgedFetal BloodFas receptorLymphocyte SubsetsChild PreschoolCord bloodImmunologybiology.proteinFemaleCD8Experimental Gerontology
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Impairment of gamma/delta T lymphocytes in elderly: implications for immunosenescence

2004

Gamma/delta T lymphocytes cells recognize the antigen in a non-classical way and are considered the third branch of the immune system devoted to defend the integrity of the body. Ageing is characterized by an impairment of the main way of protection (the adaptive branch) but, successfully aged people show compensatory mechanisms of defense such as proneness to inflammation. Moreover, very old subjects show an increased number of NK cells. We have previously demonstrated that gamma delta T lymphocytes are reduced in elderly. In the present paper we have studied some characteristics of these cells to evaluate the possibility that these cells might balance the decreased action of the adaptive …

AdultMaleAgingApoptosisInflammationBiologyLymphocyte ActivationBiochemistryPeripheral blood mononuclear cellEndocrinologyImmune systemAntigenT-Lymphocyte SubsetsGeneticsmedicineHumansLymphocyte CountMolecular BiologyCells CulturedAgedAged 80 and overGamma/Delta T-LymphocyteReceptors Antigen T-Cell gamma-deltaCell BiologyImmunosenescenceMiddle AgedApoptosisAgeingImmunologyFemalemedicine.symptomCell Division
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Evidence for Less Marked Potential Signs of T-Cell Immunosenescence in Centenarian Offspring Than in the General Age-Matched Population

2014

People may reach the upper limits of the human life span at least partly because they have maintained more appropriate immune function, avoiding changes to immunity termed "immunosenescence." Exceptionally long-lived people may be enriched for genes that contribute to their longevity, some of which may bear on immune function. Centenarian offspring would be expected to inherit some of these, which might be reflected in their resistance to immunosenescence, and contribute to their potential longevity. We have tested this hypothesis by comparing centenarian offspring with age-matched controls. We report differences in the numbers and proportions of both CD4(+) and CD8(+) early- and late-diffe…

AdultMaleAgingImmunosenescenceOffspringHealth StatusT-LymphocytesT cellmedia_common.quotation_subjectLongevityPopulationCD4-CD8 RatioT cellsBiologyLymphocyte Activation03 medical and health sciences0302 clinical medicineImmune systemAntigenmedicineHumanseducationAged030304 developmental biologymedia_commonAged 80 and overSettore MED/04 - Patologia Generale0303 health scienceseducation.field_of_studyAge FactorsLongevityImmunosenescencemedicine.anatomical_structureCase-Control StudiesCentenarian offspring.ImmunologyAdult ChildrenFemaleGeriatrics and GerontologyCentenarian030215 immunologyThe Journals of Gerontology Series A: Biological Sciences and Medical Sciences
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Granulocyte and natural killer activity in the elderly

1999

The deterioration of the immune system in ageing, 'immunosenescence', is thought to contribute to increased morbidity and mortality from infections and possibly autoimmune diseases and cancer. The most profound changes involve effector and immunoregulatory T-cell functions. Immunosenescence appears also to be related to changes in non specific immunity as well. In the present study we have assessed superoxide production, chemotaxis and the expression of the apoptosis-related molecule APO1/Fas (CD95) on neutrophils (PMN) from young and old subjects. Furthermore, we have measured the basal natural killer (NK) activity of young and elderly subjects and we have compared the number of CD16+ cell…

AdultMaleAgingmedicine.medical_specialtyNeutrophilschemical and pharmacologic phenomenaBiologyCD16Natural killer cellImmune systemInternal medicinemedicineHumansAgedAged 80 and overInnate immune systemEffectorChemotaxisImmunosenescenceMiddle AgedFas receptorKiller Cells Naturalmedicine.anatomical_structureEndocrinologyImmunologyFemaleDevelopmental BiologyMechanisms of Ageing and Development
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