Search results for "INTERACTION"

showing 10 items of 5710 documents

Hepatitis B Virus Subverts the Autophagy Elongation Complex Atg5-12/16L1 and Does Not Require Atg8/LC3 Lipidation for Viral Maturation

2018

ABSTRACT Previous studies indicated that hepatitis B virus (HBV) stimulates autophagy to favor its production. To understand how HBV co-opts autophagy as a proviral machinery, we studied the roles of key autophagy proteins in HBV-replicating liver cell cultures. RNA interference-mediated silencing of Atg5, Atg12, and Atg16L1, which promote autophagophore expansion and LC3 membrane conjugation, interfered with viral core/nucleocapsid (NC) formation/stability and strongly diminished virus yields. Concomitantly, the core/NC membrane association and their sorting to envelope-positive compartments were perturbed. A close inspection of the HBV/autophagy cross talk revealed that the virus depended…

0301 basic medicineHepatitis B virusATG8Autophagosome maturationImmunologyATG5Autophagy-Related ProteinsBiologymedicine.disease_causeVirus ReplicationMicrobiologyVirusAutophagy-Related Protein 5ATG1203 medical and health sciencesVirologyCell Line TumormedicineAutophagyHumansHepatitis B virusAutophagyAutophagy-Related Protein 8 FamilyHepatitis BCell biologyVirus-Cell Interactions030104 developmental biologyViral replicationInsect ScienceGene Knockdown TechniquesMultiprotein ComplexesMicrotubule-Associated ProteinsAutophagy-Related Protein 12
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Rab33B Controls Hepatitis B Virus Assembly by Regulating Core Membrane Association and Nucleocapsid Processing

2017

Many viruses take advantage of cellular trafficking machineries to assemble and release new infectious particles. Using RNA interference (RNAi), we demonstrate that the Golgi/autophagosome-associated Rab33B is required for hepatitis B virus (HBV) propagation in hepatoma cell lines. While Rab33B is dispensable for the secretion of HBV subviral envelope particles, its knockdown reduced the virus yield to 20% and inhibited nucleocapsid (NC) formation and/or NC trafficking. The overexpression of a GDP-restricted Rab33B mutant phenocopied the effect of deficit Rab33B, indicating that Rab33B-specific effector proteins may be involved. Moreover, we found that HBV replication enhanced Rab33B expres…

0301 basic medicineHepatitis B virusBiologymedicine.disease_causeVirusArticleCell LineCell membraneRab33B03 medical and health sciencesnucleocapsid assemblyTranscription (biology)RNA interferenceVirologymedicineHumansSecretionNucleocapsidcore/capsid membrane associationHepatitis B virus030102 biochemistry & molecular biologyEffectorVirus AssemblyCell MembraneVirologyHepatitis B Core Antigenshepatitis B virus; Rab GTPase; Rab33B; core/capsid membrane association; nucleocapsid assembly; virus traffickingTransport proteinProtein Transport030104 developmental biologyInfectious Diseasesmedicine.anatomical_structurevirus traffickingrab GTP-Binding ProteinsHost-Pathogen InteractionsHepatocytesRab GTPaseViruses; Volume 9; Issue 6; Pages: 157
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The immunoglobulin γ marker 17 allotype and KIR/HLA genes prevent the development of chronic hepatitis B in humans

2020

Hepatitis B virus (HBV) infection causes a self-limiting disease in most individuals. However, < 10% of infected subjects develop a chronic disease. Genetic host variability of polymorphic genes at the interface of innate and acquired immunity, such as killer immunoglobulin-like receptors (KIR), their human leucocyte antigen (HLA) and IgG allotypes (GM), could explain this different clinical picture. We previously showed a protective role of the KIR2DL3 gene for the development of chronic hepatitis B (CHB), and a detrimental role of the KIR ligand groups, HLA-A-Bw4 and HLA-C2. We have expanded the previous analysis genotyping patients for GM23 and GM3/17 allotypes. The comparison of the …

0301 basic medicineHepatitis B virusKIR LigandImmunologyhepatitis B viruHuman leukocyte antigenHLA-C Antigensmedicine.disease_causeRisk Assessment03 medical and health sciences0302 clinical medicineHepatitis B ChronicGene FrequencyImmunoglobulin Gm AllotypesRisk Factorskiller immunoglobulin-like receptorImmunology and AllergyMedicineHumansGenetic Predisposition to DiseaseGenotypingHepatitis B virusSettore MED/04 - Patologia Generalebiologybusiness.industryOriginal ArticlesProtective FactorsAcquired immune systemAllotypeγ marker030104 developmental biologyPhenotypeHLA-B AntigensReceptors KIR2DL3Case-Control StudiesImmunologyHost-Pathogen Interactionsbiology.proteinGene polymorphismAntibodyhepatitis B virus; human leucocyte antigen; killer immunoglobulin-like receptor; ? markerbusiness030215 immunologyhuman leucocyte antigen
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Parasitic wasp-associated symbiont affects plant-mediated species interactions between herbivores

2018

Abstract Microbial mutualistic symbiosis is increasingly recognised as a hidden driving force in the ecology of plant–insect interactions. Although plant-associated and herbivore-associated symbionts clearly affect interactions between plants and herbivores, the effects of symbionts associated with higher trophic levels has been largely overlooked. At the third-trophic level, parasitic wasps are a common group of insects that can inject symbiotic viruses (polydnaviruses) and venom into their herbivorous hosts to support parasitoid offspring development. Here, we show that such third-trophic level symbionts act in combination with venom to affect plant-mediated interactions by reducing colon…

0301 basic medicineHerbivore colonisationInsecta[SDV]Life Sciences [q-bio]tritrophic interactionWaspsVenomParasitoidchenilleLaboratory of EntomologyTrophic levelbiologyEcologyPolydnavirusHost-Parasite Interactionfood and beveragesplant–insect interactionsHerbivore colonisation parasitoid plant–insect interactions polydnaviruses tritrophic interactionsPE&RCsymbiosisParasiteinternationalpolydnaviruse[SDE]Environmental Sciencespolydnavirussymbioseécologie des populationsinteraction plante insecteherbivoreplant–insect interactionHost-Parasite Interactions03 medical and health sciencesSymbiosisAnimalsParasitesHerbivoryguêpeCaterpillarparasitoidEcology Evolution Behavior and SystematicsHerbivoreAnimaltritrophic interactionsfungi15. Life on landplant-insect interactionsLaboratorium voor Entomologiebiology.organism_classificationplant insect interactionherbivore colonisationpolydnavirusesColonisationSettore AGR/11 - Entomologia Generale E Applicata030104 developmental biologycaterpillarEPS
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HIPPIE v2.0: Enhancing meaningfulness and reliability of protein-protein interaction networks

2016

The increasing number of experimentally detected interactions between proteins makes it difficult for researchers to extract the interactions relevant for specific biological processes or diseases. This makes it necessary to accompany the large-scale detection of protein-protein interactions (PPIs) with strategies and tools to generate meaningful PPI subnetworks. To this end, we generated the Human Integrated Protein-Protein Interaction rEference or HIPPIE (http://cbdm.uni-mainz.de/hippie/). HIPPIE is a one-stop resource for the generation and interpretation of PPI networks relevant to a specific research question. We provide means to generate highly reliable, context-specific PPI networks …

0301 basic medicineHippieReliability (computer networking)BiologyWeb BrowserBioinformaticsProtein protein interaction networkComputational biology03 medical and health sciences0302 clinical medicineResource (project management)GeneticsHumansDatabase IssueGraph algorithmsProtein Interaction MapsDatabases ProteinResearch questionGraphical user interfacebusiness.industryReproducibility of ResultsData science030104 developmental biologyComputingMethodologies_PATTERNRECOGNITIONProtein interaction mappingbusiness030217 neurology & neurosurgeryProtein Interaction MapSoftware
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An android architecture for bio-inspired honest signalling in Human-Humanoid Interaction

2017

Abstract This paper outlines an augmented robotic architecture to study the conditions of successful Human-Humanoid Interaction (HHI). The architecture is designed as a testable model generator for interaction centred on the ability to emit, display and detect honest signals. First we overview the biological theory in which the concept of honest signals has been put forward in order to assess its explanatory power. We reconstruct the application of the concept of honest signalling in accounting for interaction in strategic contexts and in laying bare the foundation for an automated social metrics. We describe the modules of the architecture, which is intended to implement the concept of hon…

0301 basic medicineHonest signals; Geminoid robot; Social robotics; Human-Humanoid InteractionHonest signalsShared environmentComputer scienceCognitive NeuroscienceExperimental and Cognitive Psychology02 engineering and technology03 medical and health sciencesArtificial IntelligenceHuman–computer interactionSocial robotic0202 electrical engineering electronic engineering information engineeringHuman-Humanoid InteractionArchitectureGeminoid robotHonest signalSettore ING-INF/05 - Sistemi Di Elaborazione Delle InformazioniSocial robotSocial metricsSocial robotics020207 software engineering030104 developmental biologySignallingSettore M-FIL/04 - EsteticaBiological theoryAndroid (robot)Settore M-PSI/05 - Psicologia Sociale
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Overview of the interaction of helminth extracellular vesicles with the host and their potential functions and biological applications.

2021

Helminth Extracellular Vesicles (EVs) have emerged as important mediators in host-parasite communications, participating in the parasite survival and its pathogenic effects. In the last decade, a growing amount of information reporting the isolation and characterization of EVs from different helminth species has appeared, but unfortunately, few reports have focused on functional studies of helminth EVs in different cell lines, organoids or animal models. We here review these in vitro and in vivo studies, which clearly demonstrate that helminths secrete EVs, which affect their environment. Helminth EVs are actively internalized by different cell lines, modulating cellular functions important…

0301 basic medicineHost (biology)ImmunologyCellular functionsHelminthiasisComputational biologyBiologyExtracellular vesiclesHost-Parasite Interactions03 medical and health sciencesExtracellular Vesicles030104 developmental biology0302 clinical medicineImmune systemHelminthsparasitic diseasesHelminthsAnimalsHumansSecretionFunctional studiesInformation reportingMolecular Biology030215 immunologyMolecular immunology
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Identifying Host Molecular Features Strongly Linked With Responses to Huanglongbing Disease in Citrus Leaves

2018

© 2018 Balan, Ibáñez, Dandekar, Caruso and Martinelli. A bioinformatic analysis of previously published RNA-Seq studies on Huanglongbing (HLB) response and tolerance in leaf tissues was performed. The aim was to identify genes commonly modulated between studies and genes, pathways and gene set categories strongly associated with this devastating Citrus disease. Bioinformatic analysis of expression data of four datasets present in NCBI provided 46–68 million reads with an alignment percentage of 72.95–86.76%. Only 16 HLB-regulated genes were commonly identified between the three leaf datasets. Among them were key genes encoding proteins involved in cell wall modification such as CESA8, pecti…

0301 basic medicineHuanglongbing HLB citrus protein–protein interaction network transcriptomics RNA-SeqPlant BiologyHuanglongbingRNA-SeqPlant Sciencelcsh:Plant cultureBiologycitrusTranscriptometranscriptomics03 medical and health sciencesExpansinSettore AGR/07 - Genetica AgrariaHeat shock proteinGenetics2.1 Biological and endogenous factorslcsh:SB1-1110RNA-SeqAetiologyGeneTranscription factorOriginal Research2. Zero hungerGeneticsHuanglongbing; HLB; citrus; protein–protein interaction network; transcriptomics; RNA-SeqPectinesteraseSettore AGR/03 - Arboricoltura Generale E Coltivazioni ArboreeHLB030104 developmental biologyPectate lyaseprotein–protein interaction networkprotein-protein interaction networkBiotechnologyFrontiers in Plant Science
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Role of Immunogenetics in the Outcome of HCMV Infection: Implications for Ageing

2019

The outcome of host-virus interactions is determined by a number of factors, some related to the virus, others to the host, such as environmental factors and genetic factors. Therefore, different individuals vary in their relative susceptibility to infections. Human cytomegalovirus (HCMV) is an important pathogen from a clinical point of view, as it causes significant morbidity and mortality in immunosuppressed or immunosenescent individuals, such as the transplanted patients and the elderly, respectively. It is, therefore, important to understand the mechanisms of virus infection control. In this review, we discuss recent advances in the immunobiology of HCMV-host interactions, with partic…

0301 basic medicineHuman cytomegalovirusAgingCellular immunityvirusesCytomegalovirusReviewlcsh:Chemistry0302 clinical medicineHLA AntigensGenotypeMedicineantibodieslcsh:QH301-705.5SpectroscopyimmunosenescenceImmunity CellularbiologyGeneral MedicineImmunosenescenceGMComputer Science ApplicationsKIRHLAantibodieCytomegalovirus InfectionsHost-Pathogen InteractionsAntibodyGenotypeNKCongenital cytomegalovirus infectionHuman leukocyte antigenelderlyCatalysisVirusInorganic Chemistry03 medical and health sciencesImmunogeneticsAnimalsHumansGenetic Predisposition to DiseasePhysical and Theoretical ChemistryMolecular BiologyHCMVSettore MED/04 - Patologia Generalebusiness.industryOrganic Chemistrymedicine.diseaseImmunity Humoral030104 developmental biologylcsh:Biology (General)lcsh:QD1-999Immunologybiology.proteinbusiness030215 immunology
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Regulatory Interaction between the Cellular Restriction Factor IFI16 and Viral pp65 (pUL83) Modulates Viral Gene Expression and IFI16 Protein Stabili…

2016

ABSTRACT A key player in the intrinsic resistance against human cytomegalovirus (HCMV) is the interferon-γ-inducible protein 16 (IFI16), which behaves as a viral DNA sensor in the first hours postinfection and as a repressor of viral gene transcription in the later stages. Previous studies on HCMV replication demonstrated that IFI16 binds to the viral protein kinase pUL97, undergoes phosphorylation, and relocalizes to the cytoplasm of infected cells. In this study, we demonstrate that the tegument protein pp65 (pUL83) recruits IFI16 to the promoter of the UL54 gene and downregulates viral replication, as shown by use of the HCMV mutant v65Stop, which lacks pp65 expression. Interestingly, at…

0301 basic medicineHuman cytomegalovirusViral proteinviruses030106 microbiologyImmunologyCytomegalovirusDNA-Directed DNA PolymeraseBiologymedicine.disease_causeVirus ReplicationMicrobiologyViral Matrix Proteins03 medical and health sciencesViral ProteinsVirologymedicineHumansNuclear proteinPromoter Regions GeneticGeneCells CulturedViral matrix proteinIFI16Protein Stabilityvirus diseasesNuclear ProteinsViral tegumentmedicine.diseasePhosphoproteinsMolecular biologyVirus-Cell Interactions030104 developmental biologyViral replicationInsect ScienceDNA ViralHost-Pathogen InteractionsProtein BindingJournal of virology
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