Search results for "Immune system"

showing 10 items of 2885 documents

Nucleic acid recognizing Toll-like receptors and autoimmunity

2007

The understanding of autoimmune diseases experienced an impressive boost since the Toll-like receptors (TLRs) have been identified as possible key players in autoimmune pathophysiology. Although these receptors recognize a variety of structures derived from viruses, bacteria, and fungi leading to subsequent initiation of the relevant immune responses, recent data support the idea that TLRs are crucial in the induction and perpetuation of certain autoimmune diseases, especially the systemic lupus erythematosus (SLE). In this review, we will summarize recent data on involvement of TLRs in the development of autoimmune diseases. We will focus on TLRs 7, 8, and 9 that were originally identified…

ImmunologyGene ExpressionReceptors Antigen B-CellAutoimmunityContext (language use)Biologymedicine.disease_causeAutoimmune DiseasesAutoimmunityImmune systemAntigenGene expressionmedicineAnimalsHumansImmunology and AllergyReceptorToll-Like ReceptorsRNADNADendritic CellsToll-Like Receptor 7Toll-Like Receptor 8Toll-Like Receptor 9ImmunologyRNASignal transductionSignal TransductionCurrent Opinion in Immunology
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Modulation of C1q mRNA Expression and Secretion by Interleukin-1,Interleukin-6, and Interferon-g in Resident and Stimulated Murine Peritoneal Macroph…

2002

The complement system plays an important role in the humoral immune response. Activation of the classical complement pathway is mediated by its subcomponent, C1q. Among the main C1q-synthesising tissues, macrophages have been attributed as a source of particular importance. We investigated the effects of cytokines (IL-1, IL-6 and Interferon-gamma) on local C1q mRNA expression and C1q secretion in resident and in thioglycollate-stimulated murine peritoneal macrophages in vitro. The macrophages were isolated from murine peritoneal lavage fluid, maintained in culture and incubated with the cytokines. Among the cytokines, only IL-6 had a stimulatory effect on C1q production (25% increase vs. co…

ImmunologyGene Expressionchemical and pharmacologic phenomenaIn Vitro TechniquesProinflammatory cytokineInterferon-gammaMiceClassical complement pathwayImmune systemmedicineAnimalsImmunology and AllergyMacrophageInterferon gammaRNA MessengerInterleukin 6Macrophage inflammatory proteinMice Inbred BALB CbiologyInterleukin-6ChemistryComplement C1qInterleukinHematologyMacrophage ActivationRecombinant ProteinsCell biologyThioglycolatesMacrophages Peritonealbiology.proteinInterleukin-1medicine.drugImmunobiology
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Altered metabolism of gut microbiota contributes to chronic immune activation in HIV-infected individuals.

2015

Altered interplay between gut mucosa and microbiota during treated HIV infection may possibly contribute to increased bacterial translocation and chronic immune activation, both of which are predictors of morbidity and mortality. Although a dysbiotic gut microbiota has recently been reported in HIV + individuals, the metagenome gene pool associated with HIV infection remains unknown. The aim of this study is to characterize the functional gene content of gut microbiota in HIV + patients and to define the metabolic pathways of this bacterial community, which is potentially associated with immune dysfunction. We determined systemic markers of innate and adaptive immunity in a cohort of HIV-in…

ImmunologyHIV InfectionsBiologyGut floraAdaptive ImmunityMicrobiologyMetabolomicsImmunityAntiretroviral Therapy Highly ActiveRNA Ribosomal 16SMetabolomeImmunology and AllergyCluster AnalysisHumansMetabolomicsGeneCase-control studyBayes TheoremBiodiversityAcquired immune systembiology.organism_classificationImmunity InnateMarkov ChainsGastrointestinal MicrobiomeMetabolic pathwayCase-Control StudiesImmunologyDisease ProgressionHIV-1MetabolomeMetagenomeMucosal immunology
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Predominant role of NF-kappa B p65 in the pathogenesis of chronic intestinal inflammation.

1998

NF-kappa B is a key transcription factor of lymphocytes and macrophages with important regulatory functions in the immune system and inflammatory processes. These functions are at least partially based on its ability to regulate the promoters of a variety of genes whose products, such as cytokines, adhesion molecules and acute phase proteins, are critical for inflammatory processes. In the present study, we describe a method to treat established intestinal inflammation by local or systemic application of antisense phosphorothioate oligonucleotides targeting the translation start site of the p65 subunit of NF-kappa B. Specific downregulation of p65 by administration of antisense phosphorothi…

ImmunologyInflammationBiologyPathogenesisMiceImmune systemDownregulation and upregulationCrohn DiseasemedicineImmunology and AllergyAnimalsHumansCells CulturedInflammationPhosphorothioate OligonucleotidesOligonucleotideInterleukin-6Tumor Necrosis Factor-alphaMacrophagesAcute-phase proteinNF-kappa BTranscription Factor RelAHematologyOligonucleotides AntisenseNFKB1ColitisIntestinesDisease Models AnimalImmunologyChronic DiseaseCancer researchFemalemedicine.symptomInterleukin-1Immunobiology
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Mast cells are crucial for early inflammation, migration of Langerhans cells, and CTL responses following topical application of TLR7 ligand in mice.

2007

Abstract Until recently, IgE-activated mast cells have been regarded merely as effector cells of adaptive immune responses, involved in allergic reactions and mucosal immunity to parasites. Herein, we report that murine dermal mast cells, activated by local administration of a cream containing the synthetic TLR7 ligand imiquimod, are essential to initiate an early inflammatory reaction. The mast-cell–derived cytokines TNF-α and IL-1β play an important role in this process. Furthermore, TLR7-activated mast cells are also able to promote the emigration of Langerhans cells, which partly depends on the expression of mast-cell–derived IL-1β. We have previously shown that TLR7 ligation enhances t…

ImmunologyInterleukin-1betaInflammationImmunoglobulin ELigandsBiochemistryMiceImmune systemAdjuvants ImmunologicCell MovementmedicineCytotoxic T cellAnimalsMast CellsAntigensSkinInflammationImmunity CellularMice Inbred BALB CVaccinesImiquimodMembrane GlycoproteinsbiologyTumor Necrosis Factor-alphaDegranulationCell BiologyHematologyTLR7Immunoglobulin EAcquired immune systemImmunity InnateInterleukin 33Toll-Like Receptor 7Langerhans CellsImmunologybiology.proteinAminoquinolinesImmunizationmedicine.symptomAgranulocytosisT-Lymphocytes CytotoxicBlood
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CD4-mediated functional activation of human CD4+CD25+ regulatory T cells

2007

Naturally occurring CD4(+)CD25(+)FoxP3(+) regulatory T cells (CD25(+) Tregs) constitute a specialized population of T cells that is essential for the maintenance of peripheral self-tolerance. The immune regulatory function of CD25(+) Tregs depends upon their activation. We found that anti-CD4 antibodies activate the suppressive function of human CD25(+) Tregs in a dose-dependent manner. We demonstrate that CD4-activated CD25(+) Tregs suppress the proliferation of CD4(+) and CD8(+) T cells, their IL-2 and IFN-gamma production as well as the capacity of CD8(+) T cells to re-express CD25. By contrast, anti-CD4 stimulation did not induce suppressive activity in conventional CD4(+) T cells. Thes…

ImmunologyInterleukin-2 Receptor alpha SubunitAntibodies MonoclonalFOXP3hemic and immune systemschemical and pharmacologic phenomenaCD8-Positive T-LymphocytesBiologyFlow CytometryLymphocyte ActivationT-Lymphocytes RegulatoryCoculture TechniquesImmune toleranceCell biologyInterleukin 21Immune systemCD4 AntigensImmunologyImmune ToleranceHumansImmunology and AllergyCytotoxic T cellIL-2 receptorCell activationCD8European Journal of Immunology
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Mast Cells Are Key Promoters of Contact Allergy that Mediate the Adjuvant Effects of Haptens

2011

SummaryA prominent feature of sensitizing environmental compounds that cause allergic contact dermatitis is the rapid induction of an innate inflammatory response that seems to provide danger signals for efficient T cell priming. We generated mouse models of mast cell deficiency, mast cell-specific gene inactivation, and mast cell reporter mice for intravital imaging and showed that these adjuvant effects of contact allergens are mediated by mast cells and histamine. Mast cell deficiency resulted in impaired emigration of skin DCs to the lymph node and contact hypersensitivity was dramatically reduced in the absence of mast cells. In addition, mast cell-specific inactivation of the Il10 gen…

ImmunologyMedizinPriming (immunology)BiologyMicechemistry.chemical_compoundImmune systemAdjuvants ImmunologicCell MovementmedicineAnimalsImmunology and AllergyMast CellsInterleukin 5Allergic contact dermatitisNeovascularization PathologicDendritic CellsHypertrophymedicine.diseaseMast cellImmunity InnateMice Inbred C57BLInterleukin 33Interleukin 10medicine.anatomical_structureInfectious DiseaseschemistryDermatitis Allergic ContactMutationImmunologyLymph NodesHaptensHistamineHistamineImmunity
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Major histocompatibility complex regulation of the class of the immune response: the H-2d haplotype determines poor interferon-γ response to several …

1990

The lymph node cells of CBA (H-2k), but not BALB/c (H-2d) mice, release interferon (IFN)-gamma into the supernatant when immunized with picryl chloride epicutaneously and then exposed to antigen (haptenized cells) in vitro 4 days later. The failure in IFN-gamma production maps to the major histocompatibility complex (MHC; H-2d) in the congenic BALB/c, BALB/k and BALB/b mice. The evidence that this is an MHC regulation of the class of response to a range of antigens and not a classical Ir gene effect is (a) the difference is seen with several antigens including picryl chloride, "oxazolone" and purified protein derivative of tuberculin and (b) BALB/c mice, which fail to produce IFN-gamma, sho…

ImmunologyMice Inbred StrainsDermatitis ContactMajor histocompatibility complexMajor Histocompatibility ComplexPicryl chlorideOxazoloneInterferon-gammaMicechemistry.chemical_compoundImmune systemH-2 AntigensAntigenInterferonmedicineAnimalsImmunology and AllergyInterferon gammaHistocompatibility Antigen H-2DbiologyH-2 AntigensImmunityHaplotypeschemistryImmunologybiology.proteinmedicine.drugEuropean Journal of Immunology
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The collagen-like component of the complement system, C1q, is recognized by 7 S autoantibodies and is functionally impaired in synovial fluids of pat…

1996

Cross-reactivity between type II collagen (CII) and C1q, the collagen-like subunit of the first component of complement, has been demonstrated in synovial fluid (SF) from rheumatoid arthritis (RA) patients. Many authors have studied autoimmunity to CII in RA, but little work has been done on autoimmunity to C1q in RA. In the data presented here, we have been able to show that in addition to native C1q, an altered form of C1q is present in SF from RA patients. Furthermore, a low molecular weight form of C1q is present in RA SF, although its role, if any, in the pathogenesis of RA is unclear. The presence in these RA SF of C1q-specific antibodies (IgG and IgM) has been studied and we have par…

ImmunologyMolecular Sequence DataType II collagenArthritischemical and pharmacologic phenomenamedicine.disease_causeurologic and male genital diseasesAutoimmunityArthritis Rheumatoidfluids and secretionsimmune system diseasesSynovial FluidmedicineImmunology and AllergySynovial fluidHumansAmino Acid Sequenceskin and connective tissue diseasesAutoantibodiesbiologybusiness.industryComplement C1qAutoantibodyAntibodies Monoclonalmedicine.diseaseComplement systemMolecular WeightRheumatoid arthritisImmunoglobulin GImmunologybiology.proteinCollagenAntibodybusinessResearch ArticleImmunology
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Mechanisms underlying lineage commitment and plasticity of human γδ T cells.

2012

Phenotypic and functional heterogeneity are the hallmarks of effector and memory T cells. Upon antigen stimulation, γδ T cells differentiate into two major types of memory T cells: central memory cells, which patrol the blood and secondary lymphoid organs, and effector memory cells, which migrate to peripheral tissues. γδ T cells display in vitro a certain degree of plasticity in their function that is reminiscent of that which is observed in conventional CD4 T cells. Similar to CD4 T cells, in which a plethora of specialized subsets affect the host response, γδ T cells may readily and rapidly assume distinct Th1-, Th2-, Th17-, T(FH) and T regulatory-like effector functions, suggesting that…

ImmunologyReviewT-Lymphocytes RegulatoryInterleukin 21Cell MovementImmunology and AllergyCytotoxic T cellHumansIL-2 receptorAntigen-presenting cellhuman gamma delta T cells lineage subsets.Interleukin 3Settore MED/04 - Patologia GeneraleImmunity CellularCD40biologycytokines; effector and memory cells; γδ T cells; lineage-specifying factors; T-cell subsetsCell DifferentiationReceptors Antigen T-Cell gamma-deltaT-Lymphocytes Helper-InducerNatural killer T cellAcquired immune systemImmunity HumoralInfectious DiseasesImmunologybiology.proteinImmunologic Memory
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